Week 11: Digestive Flashcards

(34 cards)

1
Q

retching

A

glottis closes and intrathoracic P v
esophagus becomes distended
abd muscles contract, ^ P
duodenum and antrum spasm
reverse peristalsis and P gradient force chyme up
chyme does not enter mouth and falls back down

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2
Q

constipation patho

A

neurogenic disorders
functional/mech disorders, affected by weakness/pain
low residue diet can ^ chance
sedentary lifestyle ^ chance
depression and anticholinergics contribute

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3
Q

systemic effects of diarrhea

A

dehydration
electrolyte imbalance
weight loss
metabolic acidosis

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4
Q

steatorrhea

A

fat in the feces, more common in malabsorption syndromes

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5
Q

types of abdominal pain

A

parietal, visceral, referred

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6
Q

hematochezia

A

blood in stool

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7
Q

occult bleeding

A

Blood in the feces or vomit that is not visible upon gross inspection but is detected in tests used to screen for colon cancer.

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8
Q

duodenal ulcers

A

Blood in the feces or vomit that is not visible upon gross inspection but is detected in tests used to screen for colon cancer.

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9
Q

gastric ulcers

A

ulcers of the stomach
dt ^ in mucosal permeability to HCl
normal/v acid prod
tend to be chronic, more vomiting, anorexia

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10
Q

pyloric obstruction

A

the blocking or narrowing of the opening between the stomach and the duodenum
cause distress, anorexia, copious vomiting

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11
Q

intestinal obstruction

A

partial or complete blockage of the small or large intestine caused by a physical obstruction

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12
Q

simple obstruction

A

mechanical blockage of the lumen

functional blockage dt lack of motility

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13
Q

gi obstruction patho

A

consequences are variable
gas can cause distension
distension v ability to absorb H2O and electrolytes
if distension severe, may cause necrosis, ischemia, perforation, peritonitis

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14
Q

ulcerative colitis

A

chronic condition of colonic mucosa, usually in rectum and sigmoid
oft 20-40, may be familial
causes incl: dietary, infections, immunologic

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15
Q

uc patho

A

begins w inflammation in colon w neutrophil infiltration
inflammatory cytokines cause tissue damage
abscesses form in crypts, causing blding, pain, cramps
urge to defecate (diarrhea w bld and purulent mucus)

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16
Q

uc cm

A

begins w inflammation in colon w neutrophil infiltration
inflammatory cytokines cause tissue damage
abscesses form in crypts, causing blding, pain, cramps
urge to defecate (diarrhea w bld and purulent mucus)

17
Q

appendicitis

A

inflammation of the appendix
most common abd sx emerg
may arise from obstruction/bacterial infection

18
Q

appendicitis cm

A

epigastric/periumbilical pain, getting worse
RLQ pain associated w inflammation
rebound tenderness
^ WBC and C-reactive proteins

19
Q

appendicitis complications

A

perforation, peritonitis, abscess

20
Q

obesity

A

BMI> 120% ideal body wt
associated w CV disease, CA, DM
imbalance between energy intake and expense

21
Q

anorexia nervosa

A

an eating disorder in which an irrational fear of weight gain leads people to starve themselves
oft associated w nausea, abd pain, diarrhea, CA
less WBC = ^ infection
loss of 25-30% of body wt leads to starvation induced CF

22
Q

anorexia nrevosa cm

A
ED denial
skeletal look
postural hypoTN
hypoK, hypoT
sleep disturbances
23
Q

bulimia nervosa

A

an eating disorder characterized by binge eating followed by purging
teeth damaged dt acid, tracheoesophageal fistulas
rectal bleeding dt laxatives

24
Q

hav

A

spread by fecal-oral, bld route (rapidly in unsanitary)

Incubation 4-6 wks, most contagious 4 days pre symp to 3 mo post, IgG elevated for yrs

25
hbv
Spread through fluids, bld, needles | incubation 4-6 wks IgG are elevated for yrs
26
hcv
Most cases of post-transfusion hep (screened in bld) incurable RNA virus Risk fx in chronic liver disease, cirrhosis, carcinoma
27
hep patho
similar lesions to other viruses Hepatic necrosis, scarring, Kupffer cell hyperplasia and phagocyte infiltration occur, regen w/in 48 hrs damage worst in HBV/HCV
28
hep cm
prodromal: Begins 2 wks post-exposure, ends w jaundice icteric: 1-2 wks after prodromal, lasts 2-6 wks posticteric: Begins w resolution of jaundice, liver function normal after 2-12 wks
29
chronic hep
Persistence of CM and liver inflammation after hep (most common in HBV)
30
hep dx
Use HBsAg: marker for HBV, or anti HAV for HAV/HCV | Liver function tests
31
hep tx
Less fat, ^ carb diet if bile flow is obstructed precautions Prophylactic Ig can prevent HBV
32
cholithiasis patho
Enzyme defect: ^ cholesterol synth Less secretion of bile acids to emulsify fats Less resorption of bile salts from ileum Gallbladder hypomobility/stasis ^ secretion of gallbladder mucin and biliary Ca genetics
33
acute pancreatitis
Usually dt damage to biliary duct by ETOH, drugs, infection, trauma Characterized by severe epigastric pain radiating to back, fever, anorexia, N+V, jaundice
34
chronic pancreatitis tx
oral lipase, insulin corticosteroids if autoimmune cessation of ETOH/smoking sx