week 12 (vision + recognition) Flashcards
(43 cards)
explain: retinotopic organization in LGN
- topographically mapped
- each layer = info from 1 eye
⤷ monocular - 1, 4, 6 get info from contralateral eye (nasal retina)
- 2, 3, 5 get info from ipsilateral (lateral retina)
**each layer = monocular but overall, each LGN = binocular
explain: functional segregation in LGN
- M ganglions project to M layers
⤷ good for large fast moving objects - P projects to P layers
⤷ good for details and stationary
question: where else do the RGC project to? (aside from LGN) (3)
- superior colliculus
- pretectum and edinger-westphal nucleus
- suprachiasmatic nucleus (in hypothalamus
explain: other structures tha LGN projects to (aside from LGN) (3)
-
superior colliculus
- integrates vis and somatosensory
⤷ coordinates eye and head mvt. w/ stim.
⤷ moves focal point if needed (saccades) -
pretectum and edinger-westphal nucleus)
- controls lens and iris
- iris = in too bright/dim -> sends motor output to eye from cranial nerve II to adjust pupil
- lens = out of focus -> sneds motor output to ciliary musc. to change accommodation -
suprachiasmatic nucleus (SCN)
- in hypothalamus
- photosensitive (resp. to ambient light)
- circadian clock
⤷ in less light -> projects to pinneal gland -> prod. melatoning
explain: striate cortex
- V1
- in occipital lobe
- 6 layers
⤷ 2 - 3 = coordinate w/ other visual areas
⤷ 4 = primary receiving area
⤷ 5 - 6 = comms w/ subcortical struc. (LGN, SCN)
explain: layer 4 of the striate cortex
- sublayers A, B, C
- primary receiving area
⤷ thickest layer - most LGN neurons terminate on 4C leurons
⤷ M axons (layers 1 - 2 of LGN) on upper 4C
⤷ P axons (layers 3 - 6 of LGN) on lower 4C
question: does V1 have topographical mapping or cortical magnification?
- both
- topo. map
⤷ R of V1 has map of L vis. field - cortical mag
⤷ fovea = magnified compared to peripheral vision
explain: cortical magnification in V1
- center of vis field = closer to fovea -> more representation
- peripheral = further from fovea -> less cortical space
- 20 degrees of vis field has same cortical space as fovea
⤷ fovea in reality is only 2.5 degrees of vis field
explain: consequences of cortical magnification (2)
-
acuity declines w/ eccentricity
- eccentricity = how far stim. is from center of gaze
- stim. further away from center -> less acuity bc most cortical space is for the fovea
- solution: move head to foveate -
visual crowding in periphery
- objects that can be easily identified in isolation get jumbled when crowded
- deleterious effect of clutter impairs ability to see objects in the clutter
explain: binocularity and ocular dominance of V1
- LGN = first place where input from both eyes intermingles
**above and below 4C = binocular but 4C = monocular
**layers 1, 4, 6 = contralat (nasal), 2, 3, 5 = ipsilat (lat)
**LGN layers/cells are each monocular but each LGN is binocular - ocular dominance caused by diff. neurons having preference for L or R eye
⤷ layers 2 - 6 are binocular
⤷ 1 = contralateral
⤷ 7 = ipsilateral
question: role of binocular neurons
- perceive depth
- increases sensitivity of vis. system
explain: orientation selectivity in V1
-preference of a V1 neuron for a vis. stim of a specific orientation
⤷ along a certain axis
⤷ ex. bars of light
- firing more = more like preferred orientation
⤷ more light falling on the excitatory region of that neuron
explain: directional motion sensitivity in V1
- neuron shows greater firing to stim. mvt. in a particular direction
- represented by circle drawn on a cartesian plane
- if all distances same = non-directional neuron bc all same sensitivity
- closer to center = fires less = does not prefer (vv)
explain: colour contrast detection of V1
- parvo path = red green light
⤷ 3 - 6 - konio path = blue yellow light
⤷ koniocellular = in between each layer of LGN - causes blobs of colour sensitive cells in V1
question: simple vs complex cells?
- simple = clearly defined excitatory and inhibitory regions
- complex = receptive fields don’t have defined regions
question: ocular dominance columns vs orientation columns?
- ocular dominance = vertical columns in V1 where neurons arranged in alternating L-preferring or R-preferring manner
- orientation columns = vertical columns where orientation preference changes in a regular manner unal all oritations are represnted
explain: columns in V1 (ice cube model)
- have ocular dominance vs orientation columns
- hypercolumn = one pair (both L and R eye) of ocular dominance columns including all orientation columns
⤷ not totally correct bc Hubel and Wiesel only measured one neuron at a time
explain: new model exp. instead of ice cube columns
- shine light onto brain and see a pop. of neurons
⤷ not just a single one - shows active areas
⤷ absorb more light -> appear darker - use detector to see where light is absorbed
⤷ more absorbed = less light to camera = darker is more active
question: what is the updated model of colouring in V1?
- lines marking borders of orientation columns are in radial pinwheel fashion
question: how is colour sensitivity represented in V1?
- blobs and interblobs
⤷ magno path = interblob
⤷ parvo path = blob - parvo gets fovea -> cones -> colour
⤷ so blobs mean colour - stained cortex w/ cytochrome oxidase to see blobs
explain: new model for hypercolumn
- 1mm block of V1 that has all the machinery needed to see everything the V1 is responsible for
question: how does orientation preference change after adaptation?
ORIGINAL = prefer 0 degrees
- adapt to 20 degree stripes
NEW = prefer -10 degrees
- 20 degrees cells = most tired
⤷ followed by 10 degrees and 0 degrees - so the -10 degrees cells fire the best -> bars look like they bend to the left
define: interocular transfer
- transfer of adaptation from an adapted eye to a nonadapted one
**input doesn’t converge until V1 so adaptation must happen in cortical neurons (V1 or beyond)
explain: spatial freq. channel
- pattern analyzer by cortical neurons
- each set of neurons is tuned to limited range of special freq.
⤷ high freq. = detail
⤷ low freq. = broad outlines (large changes in contrast)