Week 13 Handout Flipped Classrooms: OBSTRUCTIVE DISEASES & RLD Flashcards

1
Q

What is the screening tool for Obstructive Sleep Apnea (OSA)?

A

Use the STOP-BANG assessment: Snoring, Tiredness during the day, Observed apnea, Pressure (HTN), BMI > 35 kg/m², Age > 50, Neck circumference > 40 cm, Gender (male)

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2
Q

What are the key risk factors for OSA?

A

Obesity, male sex, large neck circumference, untreated or poorly controlled OSA

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3
Q

What is the plan for airway management in OSA?

A

High suspicion for difficult mask ventilation and intubation. Confirm whether patient uses CPAP/BiPAP at home.

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4
Q

What are intraoperative airway risks for OSA?

A

Redundant pharyngeal tissue and upper airway collapse risk. Difficult mask ventilation and laryngoscopy may occur.

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5
Q

What is the CNS depressant sensitivity in OSA patients?

A

Increased sensitivity to opioids, benzodiazepines, and inhaled agents. Prefer short-acting drugs and opioid-sparing techniques.

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6
Q

What are the extubation criteria for OSA patients?

A

Only extubate when patient is fully awake and responsive. Ensure full return of airway tone and protective reflexes.

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7
Q

What are the postoperative respiratory monitoring considerations for OSA?

A

Increased risk of post-extubation apnea or desaturation. Requires prolonged PACU monitoring or admission to a monitored bed.

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8
Q

What are the disposition concerns for OSA patients postoperatively?

A

May not qualify for same-day discharge. Must have access to their CPAP/BiPAP (encourage them to bring it to surgery).

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9
Q

What should be avoided in postoperative medications for OSA?

A

Avoid sedating meds post-op: If needed, use lowest effective dose and monitor closely.

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10
Q

What should be assessed for COPD preoperatively?

A

History of recent exacerbations, cough, sputum production, dyspnea. Determine baseline function (e.g., exercise tolerance).

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11
Q

What tools are used to classify COPD severity?

A

GOLD Classification (based on FEV₁ % predicted) and BODE Index (BMI, Obstruction, Dyspnea, Exercise capacity).

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12
Q

What is the goal for optimizing lung function in COPD?

A

Pre-op bronchodilator therapy (inhalers or nebulizers). Delay elective surgery during acute exacerbations.

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13
Q

What should be avoided regarding anesthesia in COPD?

A

Avoid neuraxial anesthesia at high levels, nitrous oxide, and use volatile agents cautiously.

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14
Q

What are the ventilation management goals for COPD?

A

Prevent air trapping, low tidal volumes, avoid high peak inspiratory pressures, use PEEP cautiously.

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15
Q

What are the postoperative considerations for COPD?

A

May require continued intubation and mechanical ventilation, especially after thoracic or upper abdominal surgery.

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16
Q

What should be evaluated for asthma preoperatively?

A

Evaluate asthma control before surgery/red flags: Frequent inhaler use, recent ER visits, nocturnal awakenings, recent increases in medication requirements, signs of upper respiratory tract infection.

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17
Q

What is the goal for intraoperative management of asthma?

A

Prevent airway reactivity and bronchospasm.

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18
Q

What are the preferred induction agents for asthma?

A

Propofol, Ketamine, Sevoflurane.

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19
Q

What should be avoided in asthma management?

A

Desflurane, isoflurane, morphine, atracurium, mivacurium, non-selective beta blockers, prostaglandins, and ergonovine.

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20
Q

What are the ventilation risks in asthma?

A

Air trapping, hyperinflation leading to decreased venous return and cardiac output.

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21
Q

What are the primary preoperative considerations for pulmonary hypertension (PH)?

A

Continue all PAH medications, assess ECG, Echo, ABG, and Chest X-ray.

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22
Q

What are the primary goals during intraoperative management of PH?

A

Avoid increased pulmonary vascular resistance (PVR) and decreased systemic vascular resistance (SVR).

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23
Q

What should be avoided in the management of pulmonary hypertension?

A

Hypoxemia, hypercarbia, acidosis, hypothermia, pain, ketamine, nitrous oxide, and desflurane.

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24
Q

What is the preferred induction agent for pulmonary hypertension?

A

Etomidate for induction – minimal cardiac depression.

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25
What monitoring is recommended for pulmonary hypertension?
Arterial line recommended, consider CVP or PA catheter if severe PH.
26
What should be done in case of hypotension in PH patients?
Use vasopressors if decreased SVR, inhaled nitric oxide, iloprost, and fluid cautiously if RV failure.
27
What are early signs of acute pulmonary embolism (PE)?
Tachycardia, sudden decrease in ETCO₂ waveform and value, moderate hypoxemia without CO₂ retention.
28
What are advanced signs of acute pulmonary embolism (PE)?
Hypotension, rapid rise in PAP and CVP (if monitored), ECG changes (sinus tachycardia, RBBB, S1Q3T3 pattern).
29
What indicators suggest a massive pulmonary embolism (PE)?
Sudden hypotension, severe tachycardia, loss of ETCO₂, may progress to cardiac arrest.
30
What is the first line treatment for hypotension in acute PE?
Norepinephrine.
31
What are the intraoperative management steps for acute PE?
Secure airway with 100% FiO₂, support circulatory volume with IVF or blood, manage ventricular dysrhythmias with lidocaine or amiodarone, and provide adequate PEEP.
32
What are common causes of bronchospasm?
Airway manipulation, surgical stress (especially in asthma patients), allergic reactions, histamine-releasing agents (e.g., morphine, atracurium, mivacurium).
33
What are signs and symptoms of bronchospasm?
Diffuse wheezing, increased peak inspiratory pressure (PIP), decreased tidal volumes, shark-fin ETCO₂ waveform, hypoxemia, increased mucous production.
34
What is the first step in the treatment approach for bronchospasm?
Deepen anesthesia using agents like sevoflurane, propofol, ketamine, or IV lidocaine.
35
What should be done to maximize alveolar oxygenation in bronchospasm?
Increase FiO₂ by delivering 100% oxygen immediately.
36
What is the role of beta-2 agonists in bronchospasm treatment?
Administer short-acting inhaled beta-agonists (e.g., albuterol).
37
When should epinephrine be administered for bronchospasm?
For moderate to severe bronchospasm, especially if progressing toward anaphylaxis.
38
What corticosteroids can be used to reduce inflammation in bronchospasm?
Methylprednisolone and dexamethasone.
39
What post-bronchospasm considerations should be taken?
Delay extubation until airway reactivity has subsided, ensure complete resolution of wheezing, monitor for recurrence in PACU, and consider post-op bronchodilator therapy or ICU monitoring.
40
What is Restrictive Lung Disease (RLD)?
RLD refers to conditions that impair the ability of the lungs to fully expand during inspiration.
41
How is Restrictive Lung Disease characterized?
It is characterized by a reduced Total Lung Capacity (TLC < 5th percentile).
42
What causes Restrictive Lung Disease?
It is caused by increased stiffness or tightness of the lungs, pleura, chest wall, or neuromuscular apparatus.
43
What are the results of Restrictive Lung Disease?
It results in decreased lung volume & capacity, reduced compliance, and impaired gas exchange.
44
What are the types of Restrictive Lung Disease?
The types are Intrinsic RLD, Extrinsic RLD, and Neuromuscular RLD.
45
What is Intrinsic RLD?
Intrinsic RLD refers to diseases directly affecting the lung interstitium.
46
What are examples of Intrinsic RLD?
Examples include Pneumoconiosis, Sarcoidosis, and Pneumonitis.
47
What is Extrinsic RLD?
Extrinsic RLD refers to impaired lung expansion due to external restriction.
48
What are examples of Extrinsic RLD?
Examples include Obesity, Flail chest, Scoliosis, Pneumothorax, and Ankylosing spondylitis.
49
What is Neuromuscular RLD?
Neuromuscular RLD is inadequate chest wall movement due to muscle or nerve disorders.
50
What are examples of Neuromuscular RLD?
Examples include Myasthenia gravis, Guillain-Barré syndrome, and Poliomyelitis.
51
What is Pneumoconiosis?
Pneumoconiosis is a lung disease caused by inhalation of airborne dust/fibers.
52
What are the subtypes of Pneumoconiosis?
Subtypes include Asbestosis, Silicosis, and Coal worker’s pneumoconiosis.
53
What is Sarcoidosis?
Sarcoidosis is a multisystem granulomatous disease formed by activated macrophages & lymphocytes.
54
What ethnic variation is noted in Sarcoidosis?
It is more common and severe in Black populations.
55
What is Pneumonitis?
Pneumonitis is inflammation of lung tissue of noninfectious origin.
56
What triggers Pneumonitis?
It is triggered by irritants, allergens, radiation, or medications.
57
What is Acute Intrinsic RLD: ARDS?
ARDS is caused by severe inflammation of the alveolar-capillary membrane.
58
What triggers ARDS?
It is triggered by cytokine storm, phospholipids, and immune activation.
59
What is Extrinsic RLD: Flail Chest?
Flail chest is chest wall trauma from multiple rib fractures causing paradoxical breathing.
60
What is Extrinsic RLD: Pneumothorax?
Pneumothorax is air in the pleural space causing partial or complete lung collapse.
61
What are the severity classifications of Pneumothorax?
Small: ≤15% collapse, Moderate: 15–60% collapse, Large: >60% collapse.
62
What are preoperative anesthetic considerations for RLD?
Perform thorough pulmonary evaluation, optimize comorbid conditions, and encourage pre-op pulmonary rehab.
63
What are intraoperative anesthetic considerations for RLD?
Prefer regional anesthesia, minimize sedation, and use lung-protective ventilation.
64
What should be monitored intraoperatively for RLD?
Monitor for hypoxia, CO₂ retention, and increased airway pressures.
65
What are postoperative anesthetic considerations for RLD?
Implement aggressive pulmonary hygiene, encourage early mobilization, and monitor for atelectasis.