Epi Mix D 601-800 Flashcards

1
Q

Maedi/visna virus is shed only in the tracheal discharge

A

F

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2
Q

Clinical signs of maedi are generally seen above 3-4 years of age

A

T

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3
Q

Caprine arthritis-encephalitis virus can cause persistent infection

A

T

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4
Q

Sheep are resistant against caprine arthritis encephalitis virus

A

F

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5
Q

Arthritis caused by caprine arthritis encephalitis virus is mainly seen in lambs

A

Arthritis caused by caprine arthritis encephalitis virus is mainly seen in lambs

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6
Q

Encephalitis caused by caprine arthritis encephalitis virus is generally seen in 2-4 month old kids

A

T

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7
Q

Caprine arthritis encephalitis virus can be transmitted by milk

A

T

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8
Q

There is intensive vaccination against Caprine arthritis encephalitis in endemic countries

A

F

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9
Q

Caprine arthritis encephalitis virus is more frequent in dairy goats than in rural breeds

A

T

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10
Q

Caprine arthritis encephalitis virus is shed in the milk

A

T

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11
Q

In the case of caprine arthritis encephalitis the signs of encephalitis can be seen in 2-4 months old kids

A

T

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12
Q

In the case of caprine arthritis encephalitis, arthritis is less frequent than encephalitis

A

F

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13
Q

Kids are recommended to be isolated in a herd where caprine arthritis encephalitis is present

A

T

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14
Q

Kids can be infected with caprine arthritis encephalitis virus through the milk

A

T

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15
Q

Arthritis caused by caprine arthritis-encephalitis virus is typically seen in kids below half a year of age

A

F

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16
Q

Caprine arthritis encephalitis is characterized by CNS signs in young goats

A

T

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17
Q

Caprine arthritis encephalitis virus replicates in the intestinal tract

A

F

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18
Q

Equine infectious anaemia virus is transmitted with blood of the infected animals.

A

T

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19
Q

Blood sucking arthropods can transmit Equine infectious anaemia virus

A

T

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20
Q

Mosquitoes are the main vectors of equine infectious anaemia virus; the virus can replicate in them

A

F

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21
Q

Agar gel diffusion test is used to detect antibodies against equine infectious anaemia

A

T

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22
Q

Equine infectious anaemia is an acute disease; it does not have a chronic form

A

F

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23
Q

Iatrogenic transmission of Equine infectious anaemia can happen

A

T

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24
Q

Equine infectious anaemia virus can damage the bone marrow

A

T

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25
Q

There are no vaccines for the prevention of equine infectious anaemia

A

T

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26
Q

Equine infectious anaemia virus is more resistant than other retroviruses

A

T

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27
Q

Equine infectious anaemia virus disappears from animals after the viraemia

A

F

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28
Q

Fever is a major clinical sign of equine infectious anaemia

A

T

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29
Q

Equine infectious anaemia virus is resistant, it can survive several months in the environment

A

T

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30
Q

Only Equidae are susceptible to equine infectious anaemia virus

A

T

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31
Q

Ticks are vectors of equine infectious anaemia virus

A

F

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32
Q

Febrile waves can be seen in equine infectious anaemias

A

T

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33
Q

Equine infectious anaemia is a vector borne disease

A

T

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34
Q

Equine infectious anaemia virus causes persistent infection

A

T

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35
Q

There is no immune reaction in the case of equine infectious anaemia

A

F

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36
Q

Equine infectious anaemia is zoonotic

A

F

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37
Q

Equine infectious anaemia is caused by a lentivirus

A

T

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38
Q

Blood sucking arthropods are mechanical vectors of equine infectious anaemia virus.

A

T

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39
Q

Agar gel precipitation test can be used to the detection of antibodies against equine infectious anaemia virus.

A

T

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40
Q

Equine infectious anaemia can damage the medulla of the bone.

A

T

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41
Q

Equine infectious anaemia has a weak resistance

A

F

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42
Q

You cannot diagnose Equine infectious anaemia with serology

A

F

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43
Q

The resistance of equine infectious anaemia is very low

A

F

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44
Q

Horses and cattle are susceptible to equine infectious anaemia virus

A

F

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45
Q

In the case of equine infectious anaemia, haemorrhages cannot be seen

A

F

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46
Q

Animals infected with equine infectious anaemia virus are lifelong carriers.

A

T

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47
Q

Equine infectious anaemia virus is mainly transmitted with tracheal discharge

A

F

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48
Q

Attenuated vaccines are used for the prevention of equine infectious anaemia

A

F

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49
Q

Equine infectious anaemia can be asymptomatic

A

T

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50
Q

Equine infectious anaemia may cause recurrent fever in horses

A

T

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51
Q

Horseflies are mechanical vectors equine anaemia virus

A

T

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52
Q

Equine anaemia is a notifiable disease

A

T

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53
Q

Animals showing clinical signs of anthrax have to be killed, treatment is not allowed

A

F

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54
Q

The agent of anthrax is spreading in the herd very fast from animal to animal

A

F

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55
Q

Anthrax is frequently a peracute disease in cattle

A

T

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56
Q

Anthrax is caused by Clostridium anthracis

A

F

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57
Q

Enlargement of the spleen is a frequent postmortem lesion of anthrax

A

T

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58
Q

Anthrax can occur only in ruminants

A

F

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59
Q

Anthrax can be diagnosed by staining blood smear

A

T

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60
Q

Anthrax is zoonosis

A

T

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61
Q

Incomplete blood clotting is a typical postmortem finding in the case of anthrax

A

T

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62
Q

Animals are infected with the agent of anthrax mainly per os

A

T

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63
Q

There are no vaccines for the prevention of Anthrax

A

F

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64
Q

Anthrax can cause clinical signs in pigs

A

T

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65
Q

Horses are resistant to Anthrax

A

F

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66
Q

Generally live vaccines are used for the prevention of anthrax

A

T

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67
Q

There is a metachromatic staining in the case of Bacillus Anthracis

A

T

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68
Q

Capsule and oedema factor are virulence factors of Bacillus anthracis

A

T

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69
Q

Carbon dioxide is needed to the spore production of Bacillus Anthracis

A

F

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70
Q

Capsule is a virulence factor of the agent of anthrax

A

T

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71
Q

Pigs are more susceptible to anthrax than sheep

A

F

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72
Q

Oedema factor is a virulence factor of the agent of anthrax

A

T

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73
Q

Human anthrax cannot be treated with antibiotics

A

F

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74
Q

Inactivated vaccines are used for the prevention of Anthrax

A

F

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75
Q

Only herbivorous animals can show clinical signs of Anthrax

A

F

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76
Q

The spore of Bacillus anthracis can survive several decades in the soil

A

T

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77
Q

Bacillus Anthracis cannot produce spores in the infected animals

A

T

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78
Q

Dogs are more susceptible to Bacillus Anthracis than sheep

A

F

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79
Q

Europe is already free from anthrax

A

F

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80
Q

Cattle are infected with B. anthracis mainly from the soil

A

T

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81
Q

Anthrax is a per-acute or acute diseases in cattle

A

T

82
Q

Colic is a typical clinical sign of anthrax in horses

A

T

83
Q

Anthrax can be diagnosed with microscopic examination of blood

A

T

84
Q

Bacillus anthracis main virulence factor is in the capsule

A

T

85
Q

Bacillus anthracis spores: after 1 hour of boiling they are still alive

A

T

86
Q

Bacillus anthracis makes spores only without oxygen

A

F

87
Q

Anthrax important symptom is high fever

A

T

88
Q

If the animals have Anthrax and they have a fever, you have to vaccinate them immediately

A

F

89
Q

For anthrax we use inactive vaccine

A

F

90
Q

Humans infected with Anthrax, primarily per os

A

F

91
Q

Bacillus anthracis, herbivores are especially susceptible

A

T

92
Q

Bacillus anthracis is not in pig

A

F

93
Q

Anthrax spreads rapidly in a herd

A

F

94
Q

Bacillus anthracis is in the soil

A

T

95
Q

In anthrax, tracheitis common in carnivores

A

F

96
Q

Anthrax causes necrotic foci in liver

A

F

97
Q

Anthrax diagnosis with blood/staining

A

T

98
Q

Anthrax cannot occur in dogs and cats

A

F

99
Q

The agent of anthrax can infect only herbivorous animals

A

F

100
Q

The agent of anthrax is not spreading from animal to animal

A

T

101
Q

There is a septicaemia in cattle in the case of anthrax

A

T

102
Q

Anthrax is caused by Bacillus bovin

A

F

103
Q

The capsule of the agent of anthrax is polypeptide

A

T

104
Q

Anaerobic conditions are needed to the spore formation of the agent of anthrax

A

F

105
Q

Pigs are the most susceptible animals to the agent of anthrax.

A

F

106
Q

Animals showing clinical signs of anthrax are not allowed to be treated with antibiotics

A

F

107
Q

Only capsulated strains of Bacillus anthracis can cause anthrax

A

T

108
Q

Oedema factor and lethal factor are important virulence factors of Bacillus anthracis

A

T

109
Q

The clinical signs of anthrax in pigs are more severe than in cattle

A

F

110
Q

Dogs and cats are resistant against the agent of anthrax

A

F

111
Q

Only capsulated strain of B. anthracis is virulent

A

T

112
Q

Toxin is a virulence factor of B. anthracis

A

T

113
Q

Lethal factor is a virulence factor of B. anthracis

A

T

114
Q

Cell wall antigen is a virulence factor of B. anthracis

A

F

115
Q

Oxygen is needed to the spore production of B. anthracis

A

T

116
Q

Spore is a virulence factor of B. anthracis

A

F

117
Q

B. anthracis can cause blackleg

A

F

118
Q

Anthrax is generally seen as a chronic disease in cattle

A

F

119
Q

In case of anthrax, febrile animals have to be separated and vaccinated

A

F

120
Q

Animals with anthrax can be treated with penicillin

A

T

121
Q

B. anthracis can only be diagnosed by bacterial culture

A

F

122
Q

B. anthracis can only be diagnosed by Ascoli test.

A

F

123
Q

Animals suspected of being infected with anthrax should be vaccinated

A

T

124
Q

Animals infected with anthrax should be treated with antibiotics

A

T

125
Q

Virulence factors of anthrax: capsule, toxin, protective antigen

A

T

126
Q

Virulence factors of anthrax: capsule, toxin, cilia

A

F

127
Q

Virulence factors of anthrax: capsule, toxin, oedema factor

A

T

128
Q

Virulence factors of anthrax: capsule, toxin, cell wall antigen

A

F

129
Q

Anthrax is an epidemic disease that rapidly develops

A

F

130
Q

Anthrax is a quickly spreading, contagious infectious disease

A

F

131
Q

For lab examination of Anthrax you always have to send a spleen sample

A

F

132
Q

Animals can only be infected by anthrax on the pasture

A

F

133
Q

Sheep, cattle, and goats are the most sensitive animals to anthrax infection.

A

T

134
Q

Flagella is a virulence factor of B. anthracis

A

F

135
Q

The source of anthrax infection on animals is generally the soil

A

T

136
Q

Anthrax appears generally in the form of a local infection in pigs

A

T

137
Q

Fever is a typical sign of acute anthrax

A

T

138
Q

Anthrax can be prevented by using a live vaccine.

A

T

139
Q

Anthrax is caused by Clostridium chauvoei

A

F

140
Q

Anthrax is not spreading from animal to animal

A

T

141
Q

Ruminants are the most sensitive to anthrax

A

T

142
Q

Animals suffering from anthrax should be treated with antibiotics and hyperimmune sera, they should not be slaughtered

A

T

143
Q

Causative agent of anthrax is spore-forming bacterium in air

A

F

144
Q

Anthrax spreads in a herd by direct contact

A

F

145
Q

In order to diagnose anthrax all carcasses have to be dissected

A

F

146
Q

Anthrax is an acute disease in cattle with high fever

A

T

147
Q

Swine is highly susceptible in anthrax

A

F

148
Q

Splenic fever causes suffocation

A

T

149
Q

Splenic fever in cattle is a per-acute/acute disease

A

T

150
Q

Splenic fever is similar in every species

A

F

151
Q

Carnivorous animals are resistant to Bacillus anthracis

A

F

152
Q

Incomplete clotting of the blood is a post mortem lesion of anthrax

A

T

153
Q

Fibrinous pneumonia is a common post mortem lesion of anthrax

A

F

154
Q

Animals showing clinical signs of anthrax have to be treated with antibiotics immediately

A

T

155
Q

Horses are more susceptible to Bacillus anthracis than pigs

A

T

156
Q

Only vaccinated animals are allowed to graze on pastures infected with Bacillus anthracis

A

T

157
Q

Gastric juice can kill Bacillus anthracis in the meat, so per os infection does not occur in humans

A

F

158
Q

Most clostridia have low invasive capacity

A

T

159
Q

Spores of clostridia are generally very resistant against heat

A

T

160
Q

The habitat of clostridia is the gut and the soil

A

T

161
Q

Clostridia are obligate aerobic bacteria

A

F

162
Q

Clostridium perfringens is an obligate pathogenic bacterium

A

F

163
Q

Clostridium perfringens can produce main and auxillary toxins

A

T

164
Q

Extracellular enzymes and toxins are virulence factors of clostridia

A

T

165
Q

There are no vaccines for the prevention of diseases caused by clostridia

A

F

166
Q

Clostridium is anaerobe spore forming bacteria

A

T

167
Q

Clostridium bacteria is not in the environment, because it cannot tolerate oxygen

A

F

168
Q

Clostridium spreads usually rapid in a herd

A

F

169
Q

Clostridium spread mostly with insecticides

A

F

170
Q

Clostridium difficile can be treated with metronidazole

A

T

171
Q

Clostridium difficile is seen in foal and piglets

A

T

172
Q

Many Clostridium species have flagella

A

F

173
Q

Clostridium species are only found in the subtropics

A

F

174
Q

Clostridium can cause severe contagious diseases

A

F

175
Q

Clostridium are obligate pathogens

A

F

176
Q

Anaculture or anatoxin vaccines are used for the prevention of malignant oedema

A

T

177
Q

Cl. chauvoei is the agent of malignant oedema

A

F

178
Q

Lesions of malignant oedema are mainly seen in the large muscles

A

T

179
Q

Malignant oedema is generally endogenous in cattle

A

F

180
Q

Malignant oedema is generally a consequence of wound infection

A

T

181
Q

Movement difficulties are frequently seen in the case of malignant oedema

A

T

182
Q

Clostridium novyi can cause malignant oedema

A

T

183
Q

Malignant oedema can be diagnosed based on clinical signs

A

F

184
Q

Malignant oedema is caused due to wound infection

A

T

185
Q

Malignant oedema is only in ruminants

A

F

186
Q

Malignant oedema, one of the clinical signs is lameness/movement problems

A

T

187
Q

Malignant oedema, attenuated vaccine for prevention

A

F

188
Q

Clostridium channel is the agent of malignant oedema

A

F

189
Q

Clostridium septicum is an agent of malignant oedema

A

T

190
Q

Clostridium histolyticum can cause malignant oedema

A

T

191
Q

Agents of malignant oedema can be detected by bacterium culture

A

T

192
Q

There are no vaccines for the prevention of malignant oedema

A

F

193
Q

Malignant oedema occurs in ruminants and pigs

A

T

194
Q

Malignant oedema is an acute fatal disease

A

T

195
Q

Malignant oedema can be treated with antibiotics

A

T

196
Q

Malignant oedema can occur in any warm-blooded animal

A

T

197
Q

Once an area is infected with gas gangrene re-occurrence is common

A

T

198
Q

Malignant oedema cannot occur in swine

A

F

199
Q

Malignant oedema usually develop following an endogenous infection

A

F

200
Q

Malignant oedema is well treated with long-term antibiotics therapy

A

F