14. Myostatic Reflexes & Golgi Tendon Organ Flashcards

1
Q

what reflexes are controled in the cortex

A

placing reaction (brush babies feet)

hopping reaction

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2
Q

what reflexes are controlled in the brainstem/midbrain

A

vestibular

right reflex

suckle

yawn

eye/head movement

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3
Q

what are the spinal reflexes

A

stretch (myotatic)

golgi tendon reflex

cross extensor

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4
Q

what is the purpose of a reflex

A

protect

create movement thats required w/o voluntary control

= fast

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5
Q

what is the key to being an effective reflex

A

must be very PRECISE

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6
Q

how do you tell the different btn volitional movement & normal reflexes

A

normal reflexes = faster than fastest volitional movement

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7
Q

what are the characteristics of a myotatic reflex

A

-initated by M spindles to contract (shorten) a stretched M

= protection from tearing due to over stretching

= monosynaptic & segmental (stay in same seg of sp cord)

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8
Q

what is a muscle spindle

A

w/i sk. muscle - embedded in fusiform capsule

= parallel to M fibers

-contain afferent & efferent components

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9
Q

compare intrafusal vs extrafusal fibers

A

intrafusal: fibers w/i capsule
extrafusal: bulk of the M (the M you see)

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10
Q

what are characteristics of intrafusal fibers

A

contain BOTH motor/sensory components

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11
Q

what are characteristics of the sensory portion of a M spindle

A

= sensitive to length

  • 2 sensors : nuclear bag afferent & nuclear chain afferent

not contractile

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12
Q

what does the 1a fibers do

A

innervate nuclear bag & nuclear chain structures

Large & myelinated

sensitive to BOTH: length & how faxt its changing

increase firing with stretch

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13
Q

what do Group II fibers do

A

innervate ONLY nuclear chain

small & myelinated (still pretty fast)

sensitive to ONLY length of M

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14
Q

what does the motor portion of the m. spindle contain

A

similar histo as sk. M

inneraved by GAMMA MNs

control the length of the sensory portion

=contractile

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15
Q

what is the role of gamma MNs

A

control length of sensory part of M spindle

control sensitivity / cause contraction

does NOT directly lead to motion

–> contract intrafusal fibers stretch the sensory part –> increase the sensitivity of 1a & II fibers

(slighty smaller/slower than alpha MNs)

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16
Q

describe the stretch reflex (myotatic)

A

M stretched –> 1a activated –> synapse DIRECTLY w/ alpha MN –> activate alpha MN by releasing EAA –> alpha MN contract the M to shorten (decrease the stretch)

17
Q

how does the antagonist M come into play in the stretch reflex

A

when 1a stimulated it synapse with alpha MN

AND an interneuron (stimulates interneuron with EAA –> interneuron release GABA and inhibit alpha MN of antagoinst M –> fewer APs to anagonist –> relax)

==> the originally stretched M is no longer opposed

18
Q

what is the Golgi Tendon reflex

aka inverse myotatic

aka autogenic inhibition

A

initiated by golgi tendon organ

= polysynaptic, segmental reflex

= sudden relaxation of contracted M –> protects M from damage from excess force

19
Q

what is the golgi tendon organ

A

tendon with bare N endings w lots of branches

APs increase w/ tension

–> 1b fibers

must have LARGE contracting force to activate GTO (passive stretch initiates M. spindles not GTO)

20
Q

what are the steps to the golgi tendon reflex

A

strong contraction of golgi tendon –> 1b afferent activated –> release EAA & activate interneuron –> interneuron inhibits (hyperpolarizes) alpha MN of contracting M –> decrease APs = sudden relaxation

21
Q

do higher regions of the brain stimulate or inhibit reflexes?

A

often = inhibitory

by some regions provide excitation

22
Q

what happens to a reflex below a transection

A

fail to occur, even if they’re intact

23
Q

how doe recovery of reflexes occur

A

axons sprouting below levels on transection

expression of self activating receptor phenotypes (5HTC receptor)

24
Q

what occurs in decerebrate posturing

A

loss of all structures above red nucleus

–> rigidity (motion restricted in all directions); maintained M contrction & continual activation of alpha MNs (activated by Br.st)

—> spasticity: resist motion in a direction; hyperactive myotatic reflex & continual activation of gamma MNs (make m spindles more sensitive –> spontanously active)

25
Q

explain spasticity in decerebate posture

A

resist motion in a direction; hyperactive myotatic reflex & continual activation of gamma MNs (make m spindles more sensitive –> spontanously active)

-loss of cortex –> br.st not activated –> leaves the brainstem faciliatory region in charge

==> uncontrolled brainstem faciliatroy region –> stretch reflexes that fight any passive motion ==> spasiticity

26
Q

what is decorticate postureing

A

-rigidity; flexion of Upper limbs (disinhibition of red nucleus)

;extension of lower limbs (disinhibition of reticulospinal and vestibulospinal paths

=dep on head position

=strokes in vicinity of IC