effects of aerobic and anaerobic training - week 9 Flashcards

1
Q

what is training overload ?

A

when a physiological system is exercised at a level beyond its normally accustomed

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2
Q

what is specificity ? (4)

A

training being specific to …

  • muscle fibres recruited
  • energy system involved
  • velocity of contraction
  • type of contraction
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3
Q

what is reversibility ?

A

gains are lost when training ceases

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4
Q

what’s is involved in training to increase VO2 max ? (4)

A

large muscle groups, dynamic activity

20-60 mins
≥3 times a week
≥50% VO2 max

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5
Q

how much does VO2 max change with endurance training ? (2)

A

15-20% increase

if high VO2 already, increase is smaller

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6
Q

how does genetics impact VO2 max ?

A

heritability determines 50% in sedentary adults

also key role in determining training response

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7
Q

why does VO2 max change with training ?

A

due to changes in Cardiac output and stroke volume
- changes O2 consumption and VO2 max

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8
Q

how does training increase stroke volume ? (4)

A

increased preload
increased plasma volume
increased venous return

increased O2 uptake, improves systems, improves functions of heart

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9
Q

what is eccentric hypertrophy ? (2)

A

chamber size and wall thickness increase of heart

helps deal w increased volumes of heart so can pump more blood around - stronger heart

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10
Q

how does endurance training increase stroke volume ? (2)

A

increased blood, reduced resistance

increased contractility
- greater force produced

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11
Q

what factors influence stroke volume ? (3)

A

end diastolic volume (preload)

contractility

totally peripheral resistance (after load)

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12
Q

what factors influence end diastolic volume ? (3)

A

plasma volume

filling time + venous return

ventricular volume

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13
Q

what’s the equation for CO?

A

stroke volume x HR

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14
Q

what happens to max CO with training ?

A

increases !

mostly linear

increases efficiency of using O2

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15
Q

at sub-man workload HR is __ following trainmen due to increase in __ so __ can be achieved with fewer bpm

A

lower

stroke volume

CO

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16
Q

what happens due to increased muscle blood flow due to increased arteriovenous O2 difference from training?

A

decreased SNS vasoconstriction

increased diameter and compliance of arteries

17
Q

what happens to improved ability of muscle fibres to utilise O2 due to increased arteriovenous O2 difference from training?

A

increased capillary density - increased diffusion due slower blood flow through muscle

increased mitochondria

18
Q

transit time of RBCs _ due to bigger __ so they take longer to pass through

A

increase

capillary network

19
Q

during sub-max exercise blood flow in trained muscles is lower _ the A-V difference is greater

A

because

20
Q

during maximal exercise blood flow in trained muscles is higher _ the A-V difference is greater

A

and

21
Q

what are the consequences of endurance training shifting fibre types ? (2)

A

reduction of fast fibres and increase in slow

magnitude of fibre type dependent on training, duration . genetics

22
Q

what are the consequences of endurance training increasing capillaries surrounding muscle fibres ?

A

enhanced diffusion of oxygen

improved removal of waste

23
Q

how does endurance training increase mitochondrial volume + turnover in muscles?

A

increased volume subsarcolemmal + intermyofibrillar mitochondria

greater capacity for oxidative phosphorylation

increased mitochondrial volume
- reduced systolic ADP so less lactate + H+ formation

24
Q

how does endurance training change fuel utilisation ? (3)

A

increased plasma FFA transport and oxidation to muscle

transport of FFA from cytoplasm to mitochondria

mitochondria oxidation of FFA
- increased acetyl CoA formed
- slowed glycolysis

25
Q

how does endurance training improve antioxidant capacity of muscle ? (3)

A

(when muscle contract they produce free radicals)

increases in endogenous antioxidant enzymes
- improved ability to remove radicals
- protects against oxidative damage + muscle fatigue

26
Q

how does endurance training improve acid-base balance during exercise ? (3)

A

(lactate production creates H+ ions which can increase fatigue)

increased mitochondria - less carbs used- less pyruvate

increased NADH shuttles
- less NADh for lactate production

change in LDH isoform
- less lactate formation

27
Q

how does training promote protein synthesis in fibres? (3)

A

‘stress’ activates gene transcription

contraction activates primary and secondary messengers

results in expression of genes and synthesis of new proteins

28
Q

what are some of the primary signals for post exercise adaptations ?

A

mechanical stretch
calcium
AMP/ATP
free radicals

these drive proteins and enzymes that can shift/drive the adaptation

29
Q

what is the secondary messenger AMP kinase ?

A

promotes glucose uptake and linked to gene expression by activation of transcriptional activating factors

30
Q

what is the secondary messenger PGC-1a?

A

regulator of mitochondrial biogenesis, promotes angiogenesis and synthesis of antioxidant enzymes
- activated by AMPK

31
Q

how does detraining effect body and vo2 max? (3)

A

rapid decrease in VO2 max

decrease in SV max

decrease in maximal a-v O2 difference
- less mitochondria
- less oxidative capacity of muscle
- type 2a fibres and increase type 2 x fibres