Peri-parturient mare (Yr4) Flashcards

1
Q

what can cause pre-parturient colic?

A

foal movement
normal GI colic
infarctions/necrosis
uterine torsion

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2
Q

what can cause post-parturient colic?

A

uterine cramps
normal GI colic
uterine haemorrhage
colonic torsions
uterine inversion
infarctions/necrosis

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3
Q

what would be the appropriate treatment for colic caused by foal-movement?

A

buscopan and phenylbutazone (mild analgesia)

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4
Q

how severe is the pain caused by foal movement?

A

mild/moderate

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5
Q

why is treatment and diagnosis of ischaemia, necrosis and rupture of the caecum and colon of pre-parturient mares difficult?

A

weight of foal causing stretching and pressure on vessels, the signs are vague so have look for signs of peritonitis/toxaemia
the lesions however are often inaccessible

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6
Q

why is a vaginal exam not useful for diagnosing uterine torsion in mares?

A

they twist cranial to the cervix

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7
Q

how are uterine torsions treated?

A

laparotomy (coupled with caesarian)

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8
Q

when do some mares develop ventral oedema?

A

foal compresses lymphatic drainage system towards the end of gestation

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9
Q

what mares is pre-pubic tendon rupture most commonly seen in?

A

older mares

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10
Q

what causes pre-pubic tendon rupture?

A

the weight of the foal

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11
Q

how does pre-pubic tendon rupture present?

A

large painful oedema with a dropped udder appearance
bloody discharge can be seen in milk
(mare often spend more time recumbent)

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12
Q

how is pre-pubic tendon rupture treated?

A

phenylbutazone and assistance when foaling (or caesarian)
can resolve after foaling but can become progressively more painful and result in euthanasia
(shouldn’t be bred from again)

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13
Q

how is hydrops amnion/allantois treated?

A

induce foaling/abortion
dilate cervix and slowly drain fluid whilst monitoring blood pressure
manual removal of foal

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14
Q

what is a common clinical sign of placentitis?

A

premature udder development and lactation (alongside vaginal discharge and eventual abortion)

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15
Q

how can placentitis be treated?

A

potentiated sulphonamides and phenylbutazone

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16
Q

what is the most common cause of vaginal bleeding in mares?

A

varicose veins

17
Q

how problem are varicose veins for the pregnant mare?

A

typically no treatment required and of little clinical significance

18
Q

what is controlled vaginal delivery?

A

mare GA and hindquarters hoisted to allow easier manipulation of foal

19
Q

should perineal lacerations due to foaling be repaired?

A

if the anatomy has been substantially disrupted then repair is indicated, if not allow healing without intervention

20
Q

what is a third degree perineal laceration?

A

full thickness tear (by foal foot) into the rectum and through the anus

21
Q

how should third degree perineal lacerations be treated?

A

don’t repair immediately as they will breakdown, instead give antibiotics, NSAIDs and tetanus and delay for 4-6 weeks once its granulated

22
Q

how should recto-vaginal fistulas be treated?

A

don’t repair immediately as they will breakdown, instead give antibiotics, NSAIDs and tetanus and delay for 4-6 weeks once its granulated (same as third degree perineal lacerations)

23
Q

when should cervical tears be treated?

A

once involution as occurred (most heal on their own)

24
Q

how are uterine cramps causing colic treated?

A

phenylbutazone and buscopan (mild/moderate pain)

25
Q

why could inversion of the uterine horns occur after foaling?

A

forceful foaling or too forceful removal or retained membranes

26
Q

how should inversions of the uterine horns be treated?

A

analgesia, buscopan/clenbuterol, manual replacement and lavage

27
Q

why are postpartum mares prone to colonic torsions?

A

sudden increase in space within the abdomen post foaling

28
Q

what is the prognosis for rupture of the uterine artery?

A

if contained within the broad ligament if often responds well to treatment but haemorrhage into the abdomen can be rapidly fatal

29
Q

how severe is the pain caused by uterine artery rupture?

A

mild/moderate but can progress to signs of haemorrhagic shock

30
Q

how can rupture of the uterine artery be treated?

A

sedate (keep quiet)
analgesia
IV fluids an blood transfusion
clotting agents

31
Q

how is a uterine prolapse treated?

A

clean and replace under an epidural
give oxytocin when replaced
NSAIDs and antibiotics

32
Q

when are foetal membranes considered retained?

A

if not passed within 4 hours

33
Q

why are retained foetal membranes an issue?

A

they rapidly decompose to produce a metritis which will induce an endometritis and eventual death

34
Q

how should retained foetal membranes be treated?

A

oxytocin, antibiotics, flunixin, tetanus
can attempt manual removal by gentle traction

35
Q

what is the most likely part of the placenta to be retained?

A

tip of the non-pregnant horn

36
Q

if the foetal membranes don’t look compete when removed, what should be done?

A

lavage (initially with tap water) until it runs clear then give oxytocin

37
Q
A