Cerebral Palsy Flashcards

1
Q

Description

A

Disorder of movement, muscle tone, and posture

Caused by lesion that affects the immature brain

Non-progressive
underlying neurological lesion is static, process that led to brain dysfunction is no longer active

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2
Q

Incidence

A

2-3/1000 live births

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3
Q

When does the damage occur

A

CNS damage can occur prenatally, perinatally (period around childbirth), postnatally

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4
Q

Risk Factors

A

Maternal/Intraueterine infections

Prematurity&raquo_space; peri or intra ventricular hemorrhage

Low birth weight, SGA, IUGR

Multiple births, congenital malformations

Neonatal infection, hyperbilrubinemia

CVA, encephalopathy, anoxia

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5
Q

Clinical Exam

A

Early hypotonia

After 3-4 months, increased tone

Persistent and pervasive primitive reflexes

Delayed motor development

Asymmetrical upper limb use or hand preference in 1st year

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6
Q

Cerebral Palsy Clinical Types

A

Spastic Hemiparesis

Spastic Diplegia

Spastic Quadriparesis

Dyskinetic

Hypotonic

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7
Q

Spastic Hemiparesis

A

Gross motor delay

Most walk by age 3

Hemiparetic gait, upper > lower limb

Cortical sensory deficit

Hemiparetic limbs remain underdeveloped

May develop mild scoliosis

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8
Q

Spastic Diplegia

A

Predominant extensor posture with scissoring in lower limbs

Crouched gait secondary to hip flexor weakness

Later, internal rotation of 1 or both legs, pes planovagus, pelvic obliquity, hip subluxation/dislocation common

Scoliosis

May have mild upper limb dysfunction

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9
Q

Spastic Quadriparesis

A

More extensive cortical damage, severe disability

More frequent musculoskeletal complications and pseudobulbar palsy signs

1/3 never ambulate

1/3 ambulate with assistive devices

1/3 ambulate independently

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10
Q

Dyskinetic

A

Athetoid

Chreiform

Ballistic

Ataxic

Athetosis may become dystonia

Drooling, dysarthric speech

Upper limbs may be more involved than lower

Equinovarus, scoliosis

75% ambulate, 50% by age 3

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11
Q

Hypotonic

A

Persistent hypotonia/atonia with hip adduction, external rotation, and hip flexion contractures

Severe neuromuscular dysfunction

Intellectual deficit

Poorest overall prognosis for ambulation and independence

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12
Q

Associated Disabilities

A

Mental retardation 50%

Communication disorder

Hearing Impairment

Language delay

Perceptual problems

Apraxia

ADHD, PDD

Seizure disorders

Visual and Extraocular motor deficits

Feeding difficulties

Pulmonary disease

Usually Maximal level of motor function by age 7

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13
Q

CP Treatment

A

PT, OT, SLP

Training gross and fine motor skill, ADL

Orthoses and adaptive equipment

Orthopedic surgery
Tendon Lengthening or transfer, osteotomy, arthrodesis

Neurosurgical procedures: posterior rhizotomy

Medical management: medications, monitoring

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