4 - Aphasia Syndromes Flashcards

(66 cards)

1
Q

In the connectionist model, what are the three layers of units?

A

Semantic features

Words

Phonemes

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2
Q

In the connectionist model, what does each word corresponds to?

A

A single unit in the word layer

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3
Q

In the connectionist model, what links words to their semantic features and phonemes?

A

Bidirectional Excitatory Connections

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4
Q

In the connectionist model, what is each word connected to?

(2)

A

Semantic features

Phonemes

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5
Q

In the connectionist models, what is lexical access achieved by?

A

Interactive Spreading Activation

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6
Q

In the connectionist model, when Semantic Units are ___________, this activation spreads throughout the ___________, and ultimately the sounds of the ___________ are retrieved.

A

Activated

Neural network

Intended words

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7
Q

What is the FIRST step in the connectionist model?

(4)

A

[Semantic Step/Lemma Selection]

Activation begins and spreads for a fixed amount of time

Bilateral Excitatory Connections activate all three network levels (phonemic: /k, a, t/; words: “cat”, semantic: “pet with claws, etc.”)

Semantic neighbors (“dog”) become activated due to shared semantic features

Phonemically similar words are activated due to shared phonemes (“mat,” “rat”)

(The word with the highest level of activation is from the most appropriate grammatical category)

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8
Q

What is the SECOND step in the connectionist model?

(4)

A

[Phonological Step/Phonological Encoding]

Chosen word from first step is activated

Activation Process becomes nonlinear allowing network to handle the arbitrary mapping between semantic features and phonemes

The phonemes with the highest activation are linked into slots in a phonological frame (number and kind of syllables, stress patterns)

This linking concludes phonological encoding.

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9
Q

When do errors in phonological encoding tend to occur?

What to these errors tend to result in?

A

Due to noise, one or more wrong phonemes are more active than those of selected word.

Typically results in nonword (e.g., “lat” for CAT) but can be form‐related words (e.g. “mat” or “sat” for CAT).

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10
Q

What are the five error patterns that can occur in the connectionist model?

A

Semantic (“dog”)

Formal (“mat,” “cap)

Mixed (“rat”)

Unrelated (“log”)

Nonword (“lat”)

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11
Q

What aphasia syndromes could appear from cortical damage?

(3)

A

Broca’s

Wernicke’s

Global

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12
Q

What aphasia syndromes could result from damage of an unknown origin?

A

Anomic

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13
Q

What aphasia syndromes could appear when there is damage to the fiber tracts?

(4)

A

Conduction

Transcortical motor

Transcortical sensory

Mixed Transcortical

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14
Q

What is the most frequently occurring aphasia?

A

Anomic

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15
Q

Besides Anomia, what other aphasia types are common?

A

Broca’s

Wernicke’s

Global

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16
Q

What type of aphasia account for 50% of cases admitted to acute stroke units?

(2)

A

Global

Nonclassified aphasias

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17
Q

When are Anomia, Brocas, Conduction, Wernickes, and Transcortical more frequent in patients?

A

When patients have single lesions

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18
Q

What are different names for Broca’s Aphasia?

(4)

A

Expressive Aphasia

Motor Aphasia

Anterior Aphasia

Non-fluent Aphasia

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19
Q

What are the characteristics of Broca’s Aphasia?

(5)

A

Non-fluent (Slow, laboriously and halting speech + frequent pausing between syllables, words)

Agrammatic output (telegraphic)

Comprehension relatively preserved (as opposed to expression)

Poor, nonfluent repetition

Preserved self monitoring

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20
Q

Why do patients with Broca’s Aphasia struggle with repetition?

A

Poor language and motor planning

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21
Q

How does Broca’s Aphasia affect writing?

(4)

A

Writing similar to verbal output

Agrammatic

Large letters

Function words are absent

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22
Q

In Broca’s Aphasia, what produces Chronic Agrammatic Aphasia?

(2)

A

Lesions in Broca’s areas that extend into anterior insula cortex + temporal and parietal regions

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23
Q

What is needed to produce Broca’s Aphasia?

A

Damage to deep regions

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24
Q

What is considered Little Broca’s?

A

Smaller lesions limited to Brodman Area 44 cause aphasia that resolves quickly

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25
What is considered Big Broca's?
Large lesions producing full-blown, complex Broca's aphasia
26
What are different names for Wernicke's Aphasia? (4)
Sensory Aphasia Receptive Aphasia Posterior Aphasia Fluent Aphasia
27
What are characteristics of Wernicke's Aphasia? (8)
Fluent (Smooth + effortress output, long + syntactical sentences, normal intonation and stress patterns) Frequent paraphasia (Semantic, occasional phonemic) Paragrammatism Jargon (strings of neologism with connecting words) Impaired repetition Poor comprehension Poor self-monitoring Anosognosia (non-awareness of deficits)
28
Why do patients with Wernicke's Aphasia struggle with repetition?
Comprehension deficits
29
How does Wernicke's Aphasia affect writing? (3)
Handwriting mirrors verbal output Small Letters Significant word errors
30
What are characteristics of Global Aphasia? (4)
Severe impairments in all language functions Difficulty with basic comprehension deficits Some respond in conversation suggesting basic comprehension skills Limited verbal output + some stereotypical output.
31
What causes Conduction Aphasia? (2)
Lesions in upper temporal lobe, lower parietal lobe or insula This damages the arcuate fasiculus but spares Broca's and Wernicke's.
32
What are characteristics of Conduction Aphasia? (4)
Fluent verbal output with occasional verbal paraphasias Grossly Impaired Repetition Relatively preserved language comprehension Poor oral reading
33
How does Conduction Aphasia affect writing?
Handwriting typically functional - can be variable
34
What causes Transcortical Aphasias? (2)
Dominant hemisphere is damage but Broca's and Wernicke's areas are spared Damage (hypoperfusion) to watershed region surrounding perisylvian region (isolates this region, usually due to severe narrowing of MCA)
35
What is the defining characteristic of transcortical aphasias?
Preserved repetition
36
What are the characteristics of transcortical motor aphasia? (3)
[Damage to anterior superior frontal lobe of language dominant hemisphere] Reduced speech output Good Repetition (imp) Good Auditory comprehension
37
What are the characteristics of Transcortical Sensory Aphasia? (3)
[Posterior isolation syndrome - damage to high parietal lobe in dominant language hemisphere] Poor comprehension (listening and reading) Good repetition (imp) Fluent, empty speech with paraphasia (Like Wernicke's but with good repetition)
38
What are the characteristics of Mixed Transcortical Aphasia? (3)
[Isolation of Broca's, Wernicke's and arcuate fasciculus from rest of the brain] Good repetition (imp) Profound impairment in all communicative areas (Global???) Typically nonfluent
39
What are the characteristics of Anomic Aphasia? (4)
Word retrieval in verbal output and writing are the only obvious symptoms Fluent and grammatically correct spontaneous output Unusual pausing and circumlocutions Residual state of many aphasic syndrome AFTER time of improvement (imp)
40
What is fluency like for Broca Aphasia? Comprehension? Repetition?
Poor Intact words and simple sentences Poor, Nonfluent
41
What is fluency like for Wernicke Aphasia? Comprehension? Repetition?
Good Poor Poor, fluent jargon
42
What is fluency like for Conduction Aphasia? Comprehension? Repetition?
Fair-good Intact words and simple sentences Poor
43
What is fluency like for TCM Aphasia? Comprehension? Repetition?
Poor Intact words and simple sentences Good
44
What is fluency like for TCS Aphasia? Comprehension? Repetition?
Good Poor Good
45
What is fluency like for Anomic Aphasia? Comprehension? Repetition?
Good Good Good
46
What are the Aphasia taxonomies? (4)
Traditional Syndrome Classification Fluent Vs. Non-fluent Expressive Vs. Receptive Intentional (non-fluent) Vs. Attentional (fluent
47
Most of the tasks performed poorly by patients with Non-Thalamic Subcortical (NS) Lesions appear to involve some degree of \_\_\_\_\_\_\_\_\_\_\_\_-\_\_\_\_\_\_\_\_\_\_\_ flexibility in dealing with _______ \_\_\_\_\_\_\_\_\_\_, devising __________ \_\_\_\_\_\_\_\_\_\_\_\_\_\_\_, and organizing and monitoring responses.
Cogntive-linguistic Novel situations Linguistic strategies
48
When there is a stroke in the Basal Ganglia, language issues probably stem from what?
A combination of damage resulting from the stroke and hyperfusion affecting language issues.
49
Language deficits in Non-Thalamic Subcortical (NS) Lesions resemble what?
A frontal-type language disorder
50
Thalamic Aphasia most frequently found in lesions to dominant what?
Tuberothalamic and paramedian arteries
51
In Thalamic Aphasia, why is there a low probability of associated cortical dysfunction?
They are small arteries
52
What are disorders are associated with Thalamic Aphasia? (4)
Nuclei specific Dysfluency and conditions similiar to transcortical aphasias Arousal disturbances Comprehension deficits
53
What do we call Alexia with Agraphia?
Pareto-Temporal Alexia
54
What are the characteristics of Pareto-Temporal Alexia? (4)
Oral reading grossly impaired Cannot identify words spelled out by the examiner Copying printing material is better than writing to dictation Common component of aphasia syndromes and rare in isolation
55
What kind of profile are you likely to have when there is damage to the basal ganglia?
Atypical profile
56
What is alexia?
Decline in reading ability
57
What is agraphia?
Decline in writing ability
58
What do we call Alexia without Agraphia?
Occipital Alexia
59
What is an another name for Occipital Alexia?
Pure Word Blindness
60
What are characteristics of Occipital Alexia/Pure Word Blindness? (3)
Ability to write complex sentences or narratives without ability to read what they have written May retain the ability to identify familiar words Difficulty copying material
61
What type of lesions frequently cause Apraxia to accompany Aphasia? (2)
Frontal lobe lesions Anterior parietal lobe lesions
62
What are the two types of Apraxia which may accompany Aphasia?
Ideational Apraxia Ideomotor Apraxia
63
What is Ideational Apraxia?
Inability to Conceptualize, Plan, and Execute the complex sequence of motor actions involving the use of tools or objects in everyday life.
64
What is Ideomotor Apraxia?
Inability to correctly imitate hand gestures and voluntarily mime tool/object use.
65
What must you consider before diagnosing Ideomotor Apraxia? (4)
Paralysis Weakness Incoordination Poor comprehension
66
What is the Pseudobulbar Affect? (2)
Neurologic disorder characterized by involuntary, severe emotional displays Usually uncontrollable crying and/or laughing