Review of posters 23/04/2016 Flashcards

1
Q

Causes of myocarditis

A
Cocksackie viridans
Rheumatic fever- streptococcus infection where the immune system  makes antibodies that attack the myocardium)
Idiopathic
Lyme disease
Cardiotoxic drugs e.g. cocaine, alcohol
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2
Q

Lab tests for myocarditis

A
CRP
CPK- elevated in damage to brain, heart or muscle
Troponin- specific to heart
Erythrocyte sedimentation rate increased
White cell count increased
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3
Q

Treatment of myocarditis

A

ACE inhibitor
Beta blocker
Diuretic (for any fluid retention)
Antibiotics- not in viral cases

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4
Q

Causes of pericarditis

A
Viral- herpes simplex, HIV
Bacterial- TB
Trauma
Cancer
Post MI- Dresslers syndrome- 2-3 weeks after an MI, the immune system starts attacking the hearts pericardium.
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5
Q

Signs of pericarditis

A

Elevated JVP
Possible murmurs
Distant heart sounds

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6
Q

Dilated cardiomyopathy symptoms

A

Heart failure esc symptoms- oedema, SOB, fatigue, chest pain, syncope (possibly in extreme cases)

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7
Q

Hypertrophic cardiomopathy symptoms

A

Intermittent- patient may be asymptomatic or have symptoms. When symptoms show they include:
SOB
Fainting
Can cause sudden death

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8
Q

Signs of hypertrophic cardiomyopathy

A

Systolic ejection murmur- valsalva manouvre will accentuate it because it briefly decreases flow back to the heart and therefore the heart chambers constrict and the obstruction worsens, therefore accentuating the murmur.

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9
Q

Restrictive cardiomyopathy symptoms

A

Decreased filling may cause syncope
odema
SOB
weakness and fatigue

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10
Q

Treatment of hypertrophic cardiomyopathy

A

Septal reduction

Beta blockers or calcium channel blockers

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11
Q

Treatment of restrictive cardiomyopathy and dilated cardiomyopathy

A

Diuretics, ACE inhibitors/ARB and lifestyle changes to decrease fluid.
Possible pacemaker insertion
Beta blockers/ calcium channel blockers in dilated cardiomyopathy.
Digoxin

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12
Q

Symptoms in severe cases of acute pancreatitis

A

Tachycardia
Hypotension
Grey Turners sign
Cullens sign

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13
Q

Blood tests for acute pancreatitis

A

SERUM AMYLASE #1

Urinary amylase

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14
Q

Pathogenesis of chronic pancreatitis

A

Excess alcohol. Causes premature activation of trypsinogen to trypsin. This leads to protein depositation in the pancreatic duct forming plugs. These may obstruct the duct- lead to pancreatic hypertension and increased intracellular conc of calcium.

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15
Q

Tests for chronic pancreatitis

A

Serum amylase- may be ok in chronic

Faecal elastase! raised.

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16
Q

Treatment for chronic pancreatitis

A

NSAIDs and opiates to manage abdominal pain

17
Q

Causes of chronic pancreatitis

A

Alcohol or autoimmune chronic pancreatitis. Defect in the IgG4, causing increase in serum. Causes dense lymphoplasthytic infiltrates. Responds to steroids.

18
Q

Cholelithasis

A

Gallstones

Two types- cholesterol or pigment stones

19
Q

How are cholesterol stones formed?

A

Difference in ratio of cholesterol to bile salts.

20
Q

How are pigment gall stones formed?

A

Too much bile can’t all be formed into bile salts.

21
Q

Risk factors for developing gall stones

A
Obesity
Too much cholesterol
Diabetes
Female
Genetic
22
Q

Cholecystitis

A

Inflammation of the bile duct due to impaction of gall stones.
Acute- gall stone obstructing ampulla of Vater.
Chronic- Gall bladder wall becomes thickened (associated with gall stones)

23
Q

Symptoms of cholecystitis

A

Acute
Epigastric pain
Jaundice
May develop peritonitis if it becomes infected