Biom9 Flashcards

(48 cards)

1
Q

Which term describes the body’s process of anticipating and preparing for changes in internal and external conditions, such as a temperature increase?

A) Homeostasis
B) Allostasis
C) Positive feedback
D) Negative feedback

A

Answer: B) Allostasis.
Explanation: Allostasis refers to the body’s ability to anticipate needs and make adjustments based on environmental and internal cues, such as increasing thirst and reducing urine production when anticipating dehydration due to heat.

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2
Q

Which of the following is a key feature of homeostasis?

A) It only applies to maintaining body temperature.
B) It involves behavior modifications like moving to a cooler or warmer area.
C) It keeps physiological variables within a set range through negative feedback.
D) It anticipates the body’s future needs rather than simply responding to disturbances.

A

Answer: C) It keeps physiological variables within a set range through negative feedback.
Explanation: Homeostasis maintains body variables (e.g., temperature, blood pressure) within a narrow range using negative feedback mechanisms. For instance, the body shivers to increase heat when it’s cold.

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3
Q

What is the primary disadvantage of endothermy (the ability to regulate body temperature internally)?

A) Increased energy expenditure for temperature regulation
B) Limited adaptability to environmental changes
C) Inability to engage in vigorous activity in cold temperatures
D) Inability to enter hibernation during food scarcity

A

Answer: A) Increased energy expenditure for temperature regulation.
Explanation: Endothermic animals, like mammals and birds, need to continuously expend energy to maintain a constant body temperature. This comes at a metabolic cost, requiring more food and energy compared to ectotherms that rely on external sources for heat.

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4
Q

Which of the following mechanisms is NOT typically involved in regulating body temperature in humans?

A) Shivering to generate heat in cold conditions
B) Sweating to dissipate heat in hot conditions
C) Changing metabolic set points based on external temperature
D) Decreasing blood flow to the skin to preserve core temperature

A

Answer: C) Changing metabolic set points based on external temperature.
Explanation: Humans do not change their metabolic set points like ectotherms. Instead, they regulate body temperature through mechanisms such as sweating, shivering, and adjusting blood flow to the skin. Metabolic set points are relatively constant and only adjust slightly for physiological needs like fever.

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5
Q

What is the function of brown adipose tissue in mammals?

A) To store excess nutrients
B) To produce heat through metabolism
C) To store fat for long-term energy use
D) To regulate body temperature by constricting blood vessels

A

Answer: B) To produce heat through metabolism.
Explanation: Brown adipose tissue, found in some mammals, burns fuel to produce heat (thermogenesis) instead of storing it as fat. This helps maintain body temperature, especially in colder environments.

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6
Q

Which of the following brain areas primarily controls physiological mechanisms of body temperature regulation such as sweating and shivering?

a) Hippocampus
b) Preoptic area/anterior hypothalamus (POA/AH)
c) Raphe nucleus
d) Medulla oblongata

A

Answer: b) Preoptic area/anterior hypothalamus (POA/AH)
Explanation: The POA/AH is the primary area involved in regulating physiological temperature mechanisms, including sweating and shivering, based on input from temperature receptors in the body.

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7
Q

What happens to body temperature regulation in mammals after damage to the POA/AH?

a) Mammals cannot regulate body temperature at all.
b) They rely on behavioral mechanisms similar to those of reptiles and amphibians.
c) They become unable to sense temperature changes.
d) They exhibit enhanced sweating and shivering.

A

Answer: b) They rely on behavioral mechanisms similar to those of reptiles and amphibians.
Explanation: After damage to the POA/AH, mammals can still regulate body temperature, but they do so through behavioral means like seeking a warmer or colder environment, similar to how reptiles and amphibians adjust to temperature changes.

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8
Q

Which of the following best explains the mechanism behind a fever?

a) The body temperature rises due to an infection’s direct effect on the hypothalamus.
b) The hypothalamus sets a higher temperature threshold, causing the body to generate heat.
c) The body fails to regulate temperature, causing a direct rise in body heat.
d) Increased shivering and sweating are triggered by infection-induced changes in the immune system.

A

Answer: b) The hypothalamus sets a higher temperature threshold, causing the body to generate heat.
Explanation: Fever results from the hypothalamus setting a higher body temperature set point in response to infection, prompting the body to generate heat (e.g., through shivering) to maintain that higher temperature.

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9
Q

How does vasopressin help the body conserve water?

a) By increasing blood volume during dehydration
b) By promoting the excretion of diluted urine
c) By constricting blood vessels and promoting reabsorption of water in the kidneys
d) By stimulating the hypothalamus to induce thirst

A

Answer: c) By constricting blood vessels and promoting reabsorption of water in the kidneys
Explanation: Vasopressin, also known as antidiuretic hormone (ADH), constricts blood vessels to raise blood pressure and promotes the kidneys’ ability to reabsorb water, leading to more concentrated urine and helping the body conserve water during dehydration.

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10
Q

What causes osmotic thirst?

A) Eating salty foods
B) Losing fluid through bleeding or sweating
C) A decrease in blood volume
D) A drop in blood pressure

A

Answer: A) Eating salty foods
Explanation: Osmotic thirst is triggered by the consumption of salty foods, which increases the concentration of solutes outside cells, causing water to move out of cells and creating a need for water.

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11
Q

What is osmotic pressure?

A) The force that moves solutes from low to high concentration
B) The force that drives water across a semipermeable membrane from low to high solute concentration
C) The pressure that blood exerts on blood vessel walls
D) The pressure created by hormones like aldosterone

A

Answer: B) The force that drives water across a semipermeable membrane from low to high solute concentration
Explanation: Osmotic pressure refers to the movement of water across a membrane to balance the concentration of solutes, moving from an area of low solute concentration to one of higher concentration.

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12
Q

Which brain structure detects osmotic pressure and sodium content of the blood?

A) Hippocampus
B) Subfornical organ (SFO)
C) Organum vasculosum laminae terminalis (OVLT)
D) Medulla oblongata

A

Answer: C) Organum vasculosum laminae terminalis (OVLT)
Explanation: The OVLT, located around the third ventricle, detects osmotic pressure and sodium content of the blood, triggering mechanisms to restore balance.

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13
Q

Which hormone is involved in both osmotic thirst and the regulation of blood pressure?

A) Cortisol
B) Insulin
C) Vasopressin
D) Aldosterone

A

Answer: C) Vasopressin
Explanation: Vasopressin helps regulate osmotic thirst by promoting water retention in the kidneys, and it also constricts blood vessels to manage blood pressure.

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14
Q

What is hypovolemic thirst triggered by?

A) An increase in solute concentration in the blood
B) A decrease in blood volume
C) Increased intake of salty food
D) Dehydration caused by sweating

A

Answer: B) A decrease in blood volume
Explanation: Hypovolemic thirst occurs when there is a loss of body fluid (e.g., through bleeding or sweating), and the body needs to restore blood volume rather than just solute concentration.

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15
Q

What causes sodium-specific hunger?

A) An increase in blood sugar levels
B) A decrease in sodium reserves
C) An increase in osmotic pressure
D) A drop in water levels

A

Answer: B) A decrease in sodium reserves
Explanation: Sodium-specific hunger arises when sodium levels are low, causing the body to crave salty foods to replenish its sodium reserves.

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16
Q

Which brain region is responsible for controlling drinking behavior during osmotic thirst?

A) Hypothalamus
B) Thalamus
C) Medulla
D) Cerebellum

A

Answer: A) Hypothalamus
Explanation: The hypothalamus is involved in regulating thirst and controlling drinking behavior, responding to signals of osmotic and hypovolemic thirst.

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17
Q

What is the role of aldosterone in sodium-specific hunger?

A) It increases salt excretion in urine
B) It stimulates the release of vasopressin
C) It causes the kidneys and glands to retain sodium
D) It dilutes the concentration of solutes in the blood

A

Answer: C) It causes the kidneys and glands to retain sodium
Explanation: Aldosterone increases the retention of sodium by the kidneys, salivary glands, and sweat glands, playing a role in sodium-specific hunger when sodium levels are low.

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18
Q

Which of the following is true about the relationship between osmotic and hypovolemic thirst?

A) Osmotic thirst is triggered by a loss of blood volume, while hypovolemic thirst is triggered by a high concentration of solutes in the blood
B) Osmotic thirst involves water balance, while hypovolemic thirst involves salt and water balance
C) Osmotic thirst requires a higher intake of salt, while hypovolemic thirst requires pure water
D) Osmotic thirst is caused by dehydration, while hypovolemic thirst is not related to fluid loss

A

Answer: B) Osmotic thirst involves water balance, while hypovolemic thirst involves salt and water balance
Explanation: Osmotic thirst is related to the need for water due to an imbalance of solutes, while hypovolemic thirst involves the need for both salt and water due to a decrease in blood volume.

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19
Q

How does the body anticipate osmotic thirst before it occurs?

A) By reducing the production of vasopressin
B) By activating neurons in the digestive tract that detect salt intake
C) By decreasing the sensitivity of taste receptors to salty foods
D) By stimulating the release of aldosterone

A

Answer: B) By activating neurons in the digestive tract that detect salt intake
Explanation: The OVLT receives input from the digestive tract, allowing the body to anticipate the need for water after eating salty foods, before osmotic pressure changes occur.

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20
Q

Which of the following best describes the function of the small intestine in digestion? A) It absorbs water and minerals
B) It digests carbohydrates, proteins, and fats and absorbs nutrients into the blood
C) It produces bile to digest fats
D) It stores food for later digestion

A

Answer: B) It digests carbohydrates, proteins, and fats and absorbs nutrients into the blood
Explanation: The small intestine is primarily responsible for digesting food with enzymes and absorbing the resulting nutrients into the bloodstream.

21
Q

Why do some adults in certain regions have the ability to metabolize lactose, while others do not?
A) The genetic ability to digest lactose evolved only in Europe
B) Lactase persistence is a universal trait in mammals
C) The prevalence of the necessary genes varies due to historical domestication of cattle
D) Lactase production is linked to childhood diet

A

Answer: C) The prevalence of the necessary genes varies due to historical domestication of cattle
Explanation: The ability to digest lactose in adulthood is primarily found in societies with a long history of cattle domestication, showing a genetic adaptation to dairy consumption.

22
Q

Which statement is true regarding the process of satiety in eating?
A) Sham feeding satisfies hunger completely
B) Stomach distension is a critical factor for ending a meal
C) Tasting food is not necessary for satiety
D) The duodenum does not send signals about food intake

A

Answer: B) Stomach distension is a critical factor for ending a meal
Explanation: Stomach distension is one of the main signals for satiety. When the stomach stretches, it triggers mechanisms to end a meal.

23
Q

What role does the hormone cholecystokinin (CCK) play in meal regulation?
A) It enhances digestion by increasing gastric acid production
B) It prevents hunger signals from reaching the brain
C) It slows down digestion by constricting the stomach sphincter and signaling satiety
D) It helps in the digestion of lactose

A

Answer: C) It slows down digestion by constricting the stomach sphincter and signaling satiety
Explanation: CCK reduces meal size by constricting the stomach sphincter and signaling the brain about fullness, thus contributing to meal termination.

24
Q

Which factor is most likely responsible for the regulation of food intake through feedback from the intestines?
A) The amount of calories consumed
B) The taste receptors in the digestive tract
C) The secretion of insulin from the pancreas
D) The absorption of vitamins in the stomach

A

Answer: B) The taste receptors in the digestive tract
Explanation: The digestive tract contains taste receptors that help regulate food intake by altering brain activity, contributing to the sensation of satiety.

25
What triggers the release of insulin before and after a meal? a) High blood glucose levels b) The sight and smell of food c) Low blood glucose levels d) The release of leptin
Answer: b) The sight and smell of food Explanation: Insulin is released in response to the anticipation of food (triggered by the sight and smell), as well as after a meal, to help regulate glucose levels and enable cells to absorb glucose.
26
What happens when insulin levels remain high after a meal? a) Glucose enters cells more slowly, leading to increased hunger. b) Glucose continues to enter cells, and blood glucose levels drop, leading to increased hunger. c) The body becomes resistant to glucose. d) Blood glucose levels increase rapidly.
Answer: b) Glucose continues to enter cells, and blood glucose levels drop, leading to increased hunger. Explanation: High insulin levels promote the movement of glucose into cells, which lowers blood glucose levels, leading to hunger as the body seeks to replenish its energy store
27
What is the primary function of glucagon in regulating blood glucose levels? a) It increases insulin secretion. b) It stimulates the liver to convert glycogen to glucose. c) It reduces glucose absorption by cells. d) It promotes fat storage in adipose tissue.
Answer: b) It stimulates the liver to convert glycogen to glucose. Explanation: Glucagon is released when blood glucose levels drop, stimulating the liver to break down glycogen into glucose and release it into the blood to maintain blood sugar levels.
28
What is leptin primarily responsible for regulating in the body? a) Appetite and energy balance based on fat reserves. b) The release of insulin from the pancreas. c) Blood pressure and sympathetic nervous system activation. d) The digestion of glucose in the intestines.
Answer: a) Appetite and energy balance based on fat reserves. Explanation: Leptin helps regulate appetite and energy balance by signaling to the brain about the amount of fat in the body. Lower leptin levels signal a need for more food, while higher levels signal satiety.
29
Which of the following describes the role of leptin in puberty? a) Leptin inhibits puberty in individuals with low fat stores. b) Leptin triggers the onset of puberty when fat reserves reach a certain level. c) Leptin delays puberty in individuals with high fat stores. d) Leptin has no effect on puberty.
Answer: b) Leptin triggers the onset of puberty when fat reserves reach a certain level. Explanation: Leptin levels must reach a certain threshold to trigger puberty, indicating that the body has sufficient energy reserves to support reproduction.
30
What happens to the body when insulin levels are low, as in people with type 1 diabetes? a) Blood glucose levels remain normal, and energy is efficiently stored. b) Blood glucose levels are elevated, but cells cannot absorb glucose, leading to hunger. c) Blood glucose levels drop significantly, leading to hypoglycemia. d) The pancreas stops producing insulin entirely.
Answer: b) Blood glucose levels are elevated, but cells cannot absorb glucose, leading to hunger. Explanation: In type 1 diabetes, insulin is insufficient or absent, so glucose cannot enter cells, causing elevated blood glucose levels, while the body experiences hunger because it is unable to use the glucose.
31
Which of the following neurotransmitters is primarily associated with hunger signals in the arcuate nucleus of the hypothalamus? A) Serotonin B) Ghrelin C) Leptin D) Melanocortins
Answer: B) Ghrelin Explanation: Ghrelin, a hunger hormone released by the stomach, stimulates hunger signals by acting on the arcuate nucleus of the hypothalamus. It promotes appetite and food-seeking behavior, especially during periods of food deprivation.
32
Which of the following brain regions is crucial for inhibiting hunger and controlling satiety? A) Lateral hypothalamus B) Paraventricular nucleus (PVN) C) Amygdala D) Hippocampus
Answer: B) Paraventricular nucleus (PVN) Explanation: The paraventricular nucleus plays a significant role in inhibiting the lateral hypothalamus, which is important for controlling eating behavior. The PVN integrates signals from satiety-sensitive neurons to regulate appetite.
33
What role does orexin play in feeding behavior? A) It reduces food-seeking behavior. B) It increases persistence in seeking food and motivates activity. C) It inhibits hunger signals in the arcuate nucleus. D) It acts as a satiety signal in the brain.
Answer: B) It increases persistence in seeking food and motivates activity Explanation: Orexin, released from neurons in the lateral hypothalamus, increases persistence in seeking food and plays a role in motivation and activity, thus promoting feeding behavior.
34
What happens if there is damage to the melanocortin receptors in the paraventricular nucleus? A) Increased food intake B) Decreased food intake C) Increased satiety signals D) Decreased ghrelin secretion
Answer: A) Increased food intake Explanation: Damage to melanocortin receptors in the paraventricular nucleus leads to overeating. These receptors play a role in limiting food intake, and their dysfunction results in excessive eating.
35
Which of the following is true regarding the arcuate nucleus and its role in hunger and satiety? A) It has two sets of neurons, one sensitive to hunger signals and the other to satiety signals. B) It only processes satiety signals. C) It is located in the brainstem and is unaffected by hormonal input. D) It releases leptin to regulate appetite.
Answer: A) It has two sets of neurons, one sensitive to hunger signals and the other to satiety signals. Explanation: The arcuate nucleus contains two types of neurons: hunger-sensitive neurons that respond to signals like ghrelin, and satiety-sensitive neurons that respond to signals like leptin and insulin.
36
How does ghrelin contribute to the regulation of appetite? A) It inhibits hunger signals in the hypothalamus. B) It is released by fat cells to signal satiety. C) It is released by the stomach during food deprivation and stimulates appetite. D) It directly inhibits the paraventricular nucleus.
Answer: C) It is released by the stomach during food deprivation and stimulates appetite. Explanation: Ghrelin is produced by the stomach during food deprivation. It stimulates the arcuate nucleus in the hypothalamus, promoting hunger and motivating food intake.
37
Which of the following is the main long-term satiety signal in the body? A) Ghrelin B) Insulin C) Leptin D) Orexin
Answer: C) Leptin Explanation: Leptin is a long-term satiety signal produced by fat cells. It helps regulate energy balance by signaling the brain about the body’s fat stores, reducing appetite when fat stores are adequate.
38
The sight of highly appealing food increases appetite. Which brain area is involved in this response? A) Lateral hypothalamus B) Arcuate nucleus C) Amygdala D) Paraventricular nucleus
Answer: C) Amygdala Explanation: The amygdala is involved in processing emotions and sensory information, including responses to appealing food stimuli. It plays a role in increasing appetite when presented with highly appealing food.
39
Why might developing drugs that target melanocortin receptors for appetite control be challenging? A) The receptors are located in the brainstem, making them difficult to access. B) Stimulation of these receptors causes severe side effects. C) These receptors have no known effect on appetite. D) Damage to these receptors causes no change in eating behavior.
Answer: B) Stimulation of these receptors causes severe side effects. Explanation: While melanocortin receptors are involved in controlling appetite, drugs that stimulate these receptors have not been successful due to unacceptable side effects, making it difficult to use them as a reliable treatment for appetite control.
40
Which of the following best describes the role of the lateral hypothalamus in feeding behavior? A) It inhibits feeding by increasing the release of insulin. B) It enhances food intake by altering taste sensation and increasing salivation. C) It suppresses hunger by releasing leptin. D) It inhibits food intake through activation of the paraventricular nucleus.
Answer: B) It enhances food intake by altering taste sensation and increasing salivation. Explanation: The lateral hypothalamus promotes feeding by altering taste sensation, increasing salivation, and facilitating ingestion and swallowing. It also influences the response to the sight, smell, or taste of food.
41
Damage to the lateral hypothalamus has which of the following consequences? A) Increased food intake without loss of arousal or activity. B) A failure to recognize the taste of food. C) A complete loss of the ability to eat and drink unless force-fed. D) Increased responsiveness to food stimuli without any significant behavioral changes.
Answer: C) A complete loss of the ability to eat and drink unless force-fed. Explanation: Damage to the lateral hypothalamus results in an inability to eat and drink, causing the animal to avoid food as if it were distasteful. Force-feeding is necessary for survival, but the ability to eat gradually recovers.
42
What is a key function of the axons originating from the lateral hypothalamus? A) They activate the release of leptin from fat cells. B) They suppress food intake through the paraventricular nucleus. C) They send signals to the pituitary gland to increase insulin secretion. D) They control hunger signals directly from the arcuate nucleus.
Answer: C) They send signals to the pituitary gland to increase insulin secretion. Explanation: The lateral hypothalamus plays a role in increasing insulin secretion by sending axonal signals to the pituitary gland. It also regulates other aspects of feeding, including taste and food intake.
43
Damage to the ventromedial hypothalamus (VMH) leads to which of the following outcomes? A) Overeating due to a loss of satiety signals. B) Reduced hunger due to a lack of hunger signals. C) Increased food intake without weight gain. D) Reduced insulin production leading to weight loss.
Answer: A) Overeating due to a loss of satiety signals. Explanation: Damage to the VMH leads to overeating because this region normally inhibits feeding. Without the VMH's inhibitory control, animals consume larger meals and gain excessive weight.
44
Which neurotransmitter is associated with the promotion of food intake in the lateral hypothalamus? A) Serotonin B) Dopamine C) Neuropeptide Y (NPY) D) Glutamate
Answer: B) Dopamine Explanation: The lateral hypothalamus contains dopamine-rich axons, which play a role in promoting feeding, arousal, and motivation to seek food. Damage to these fibers leads to a loss of these behaviors.
45
Which of the following genetic factors is most associated with an increased likelihood of obesity in individuals? A) A mutation in the gene for melanocortin receptor B) An increase in the ghrelin hormone C) A decrease in FTO gene expression D) A mutation in the gene for leptin receptors
Answer: A) A mutation in the gene for melanocortin receptor Explanation: A mutation in the melanocortin receptor gene is associated with monogenic obesity, where individuals overeat and become obese from childhood.
46
Which type of obesity results from a mutation in a single gene, like the melanocortin receptor, without other physical abnormalities? A) Syndromal obesity B) Polygenic obesity C) Monogenic obesity D) Epigenetic obesity
Answer: C) Monogenic obesity Explanation: Monogenic obesity occurs when a single gene mutation leads to obesity without other physical abnormalities, such as the mutation in the melanocortin receptor.
47
Which hormone is found to be elevated in people with bulimia nervosa, and may play a role in the disorder's cycle? A) Insulin B) Leptin C) Ghrelin D) CCK
Answer: C) Ghrelin Explanation: People with bulimia nervosa typically have elevated ghrelin levels, a hormone associated with increased appetite, which is thought to be a consequence of the binge-and-purge cycle.
48
9. Which brain chemical is abnormal in individuals with anorexia nervosa, potentially due to prolonged weight loss? A) Dopamine B) Serotonin C) Ghrelin D) Insulin
Answer: A) Dopamine Explanation: People with anorexia nervosa often exhibit abnormalities in dopamine release, which may be a result of prolonged starvation and weight loss rather than the cause of the disorder.