case pres Flashcards

(87 cards)

1
Q

measured bpe
what is this & discuss results

A

BPE is a screening tool NOT a diagnostic tool; helps give a guide for tx
pt had bpe of 3 in all sextants measured
‘walk’ the probe around all of the teeth using enough pressure that will blanch a finger nail ~ 15-20g
WHO 0.5mm diameter ball ended probe

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2
Q

bpe of 3

A

black band partially visible
probing depths of at least 4mm present (3.5-5.5)
calculus / overhangs / plaque / BoP

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3
Q

how do you carry out mpbs

A

ramfjords teeth
16 21 24
36 41 44
using a WHO probe (ball end 0.5mm diameter)
teeth used in this case was
18 21 24
36 41 44
used to monitor pt engagement

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4
Q

why OPT & PAs

A

pt initially came in to emergency clinic where PA of 35 36 37 was taken to assess 36
pt refused xla on emergency clinic of 36 due to dental anxiety
struggled with gag reflex when taking PA so decided at r/v that an opt supplemented with PAs of 11 and 21 (due superimposition of cervical spine) would be better for this patient

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5
Q

OPT v PA

A

would need at least 7 or more PAs to get a full mouth of PAs which is 4 microsv per radiograph whereas one OPT is 20 microsv
radiation doses according to international atomic energy agency

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6
Q

what regulations do we follow regarding radiation dosage

A

IRMER17 & IRR17 in UK
IRR17 - deals with occupational exposures & exposures of general public. annual dose limits are specified
IRMER17 - deals with medical exposure of patients. roles inc referrer / practitioner / operator / employer (dentist can be all of these)

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7
Q

what is ICRP and what do they say regarding radiographs

A

international commission on radiological protection
3 basic principles
1. justified - must do more good than harm
2. optimised - should use as little radiation as is practicable (ALARP)
3. limited - individual dose limits are used to ensure no one has unacceptable dose

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8
Q

periodontal diagnosis and why

A

generalised periodontitis stage 3 grade c currently unstable with risk factor of ex smoker
generalised - >30% bone loss
stage 3 - interproximal bone loss mid 1/3 of root
grade c - % bone loss divided by pt age which in this case is 60/54 which is >1 so rapid progression
currently unstable - pockets greater than or equal to 4mm & BoP
(NOTE - typo in poster; remember to say this)
risk factors - ex smoker (smoked 10 a day for 15 years so 7.5 pack years) & currently socially smokes cannabis

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9
Q

what is periodontal disease

A

Periodontal diseases (gingivitis and periodontitis) are a group of inflammatory conditions that affect the
hard and soft supporting tissues of the teeth and can lead to poor aesthetics, tooth loss, loss of function and reduced quality of life. A plaque biofilm is essential for development.
Damage from periodontal disease causes irreversible bone loss which manifests as clinical loss of attachment

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10
Q

why is smoking a risk factor for periodontal disease

A

smoking alters the balance present in the biofilm of the mouth triggering an immune attack
increased production of inflammatory mediating cytokines causes tissue breakdown
there is reduced gingival blood flow - signs & symptoms are suppressed
impaired white cell function causes impaired wound healing

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11
Q

what defines an engaging patient

A

improvement in OH; greater than or equal to 50% improvement in plaque & bleeding scores OR
plaque levels less than or equal to 20% & bleeding levels less than or equal to 30%
OR
pt has met targets outlined in their personal self care plan as determined by their health care practitioner

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12
Q

why re evaluate 3mths after BSP step 2

A

3 mths gives enough time for healing & decreased pocket depths for:
- oedema to reduce (causing gingival recession)
- increased clinical attachment due to formation of junctional epithelium
this increases tissue tone & causes resistance to probing

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13
Q

what constitutes periodontal health

A

<10% BoP
pocket depth less than or equal to 4mm
no BoP at 4mm sites
note - this can be adjusted to the patient i.e. pt may have PPD of 5-6mm in absence of BoP which may not represent active disease

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14
Q

what is stability and how do we measure it

A

stability is a reflection of the current level of disease activity
assessing stability relies on measuring extent of BoP and level of PPD across dentition

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15
Q

what are the different results we can see at the 3mth re evaluation mark

A
  1. stable - BoP <10% / PDD <4mm / no BoP at 4mm sites
  2. in remission - BoP >10% / PPD <4mm / no BoP at 4mm sites
  3. unstable - PPD >5mm / PPD >4mm & BoP
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16
Q

according to BSP what does a code 3 mean

A

initial perio therapy and r/v in 3mths with localised 6PPC in involved sextants
- if no pockets greater than or equal to 4mm & no radiographic evidence of bone loss due to periodontitis then continue with code 0/1/2 pathway
- if pockets > than or equal to 4mm remain and / or radiographic evidence of bone loss due to periodontitis then continue with code 4 pathway

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17
Q

what is bsp step 1

A

building foundations for optimal treatment outcomes
- explain disease, risk factors & tx alternatives, risks & benefits inc no tx
- explain importance of OHI, encourage & support behaviour change for OH improvement
- reduce risk factors inc removal of plaque retentive features, smoking cessation, diabetes control interventions
- provide tailored OH inc ID cleaning, tp & MW, PMPR inc supra and sub gingival scaling of clinical crown

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18
Q

what is bsp step 2

A

if pt engaging with step 1 then can progress to step 2
engagement assessed via MPBs
- reinforce OH / risk factor control / behaviour change
- subgingival instrumentation (hand or powered) either alone or in combination

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19
Q

what happens after 3mth evaluation according to bsp

A

if unstable go to step 3 which is re performing subgingival PMPR for moderate pockets of 4-5mm & considering alternative causes / referral for deep residual pocketing of >6mm

if stable go to step 4 which is maintenance; supportive perio care encouraged as well as regular targeted PMPR (time frame decided with what pt needs dictate)

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20
Q

what is sciatica

A

sciatic nerve which runs from lower back to feet is irritated or compressed
nerve starts in lower back and runs down back of each leg
usually clears up in 12wks but can recur; influenced by poor sleep, stress & emotional wellbeing
issues with dental tx with regards to time in chair & getting into chair

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21
Q

pt manages sciatica flare ups with tramadol what is this

A

opioid painkiller
used to tx moderate to severe pain (often prescribed if weaker painkillers no longer work and you have long term pain)
pt taking standard tablets that contain 50mg
taking as and when required
can have withdrawal effects (dose should be gradually reduced) - agitation / anxious / shaking / sweating

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22
Q

tramadol mechanism of action

A

centrally acting synthetic opioid analgesic & SNRI (serotonin / norepinephrine reuptake inhibitor) it:
1. is an agonist of the opioid receptor (release of nociceptive neurotransmitters i.e. GABA / substance P / noradrenaline / acetylcholine is inhibited as decreases intracellular cAMP which modulates the release of nociceptive neurotransmitters)
2. inhibits serotonin reuptake
these pathways are complementary & synergistic; improving ability to modulate the perception of & response to pain
+ enantiomer inhibits serotonin reuptake & - enantiomer inhibits norepinephrine reuptake enhancing inhibitory effects on pain transmission in spinal cord
-> closes voltage gated calcium channels & opens calcium dependent potassium channels resulting in hyperpolarisation & a reduction in neuronal excitability

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23
Q

side effects of tramadol

A

dry mouth !!!!
arrythmia
respiratory depression
confusion
withdrawal

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24
Q

caution when prescribing tramadol

A

opioids co prescribed with benzodiazepines & benzodiazepine drugs can produce additive CNS depressant effects thereby increasing risk of sedation, respiratory depression, come & death

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25
risk factors for candida
long term corticosteroid use immunosuppressive drugs broad spectrum ABs diabetes nutrition dialysis smoking denture wearing anaemia steroid inhaler use
26
pseudomembranous candidiasis
white pronounced plaques wiped away easily to leave bleeding erythematous surface
27
other types of candida
erythematous - red & angry; either atrophic from HIV or denture stomatitis hyperplastic - white patches, less pronounced & plaque like than pseudomembranous, can't be wiped off, associated with candidal leukoplakia which can be pre malignant angular cheilitis - seen at commissures & angles of mouth
28
systemic candida tx
azoles - i.e. fluconazole / miconazole gel. inhibit ergosterol synthesis (fungistatic). c.glabrata resistant. avoid in warfarin / statins. polyenes - i.e. nystatin. binds directly to ergosterol on cell membranes causing pores to open & cell contents to be lost (fungicidal).
29
nystatin prescription sdcep
nystatin oral suspension 100,000 units / ml send 30ml 1ml after food 4x daily for 7 days
30
why was pt given nystatin
pt request no tablets as doesn't like taking tablets
31
issue with chronic candida
yeasts in candida can metabolise simple sugars to alcohol & CO2 gas acetaldehyde produced from subsequent alcoholic breakdown is a carcinogen (cancer causing chemical)
32
virulence factors of candida
phospholipase - degrades lipids, aids host cell penetration haemolysin - degrades RBCs, facilitates hyphal invasion proteinase - degrades protein, aids adhesion to epithelial cells alters target site to prevent azoles from binding changing cell membrane composition to prevent insertion of polyene
33
pt doesn't have any of usual risk factors for candida what has caused this
pt not seen dentist in 10yrs & only quit smoking 1yr prior could have had candida for a long time and not realised
34
perio guidelines followed
SDCEP BSP S3 guidelines - S3 format is the highest level of guideline production considering both a systematic appraisal of published evidence & clinical experience
35
to ensure you have covered everything in medical history
a systems enquiry HEALABLES H - heart E - endocrine A - a musculoskeletal L - lungs A - a joint issue B - bleeding L - liver / kidney E - brain e.g. FFS / epilepsy S - stomach / GI
36
benefits of keeping retained roots
no need to undergo surgical xla to remove can maintain alveolar bone for prosthodontic purposes
37
how to consent pt prior to perio disease
- signed perio consent form is proof pt knows what is going on & what they need to do - pt must know; they have perio, it is not curable, it is in their control to make it better - need to provide them with tx options; do nothing / perio tx / xla / perio surgery - need to discuss what will happen if perio not controlled i.e. inflammation / tooth mobility / tooth loss - need to discuss what will happen if they do control it i.e. less bleeding / less swelling so formation of black triangles / gums going from red to pink / gum recession & lengthening of teeth / sensitivity (recession exposes more root so more sensitive)
38
symptomatic irreversible pulpitis
subjective & objective findings that vital inflamed pulp is incapable of healing & RCT indicated sharp pain upon thermal stimulus lingering pain (often 30secs or longer following stimulus removal) spontaneity (unprovoked pain) referred pain pain may be accentuated by postural changes such as lying down or bending over OTC analgesics typically ineffective common aetiologies inc deep caries, extensive restorations, fractures exposing pulp tissues can be difficult to diagnose as inflammation has not yet reached the PA tissues thus resulting in no pain or discomfort to percussion
39
symptomatic apical periodontitis
represents inflammation usually of apical periodontium producing clinical symptoms involving a painful response to biting and / or percussion or palpation may or may not be accompanied by radiographic changes i.e. depending on stage of disease there may be normal width of the pdl or there may be PA radiolucency severe pain to percussion and / or palpation is highly indicative of a degenerating pulp & RCT is needed
40
why was 36 xla * come back to this question*
severe caries unrestorable unable to gain adequate moisture control to place suitable restoration caries nearly subcrestal ? unable to place adequate dam so cannot carry out rct
41
what is grade I mobility
increased mobility of the crown of the tooth to at the most 1mm in a horizontal direction
42
attritive wear
physiological wear of teeth due to tooth to tooth contact
43
signs of attritive wear
polished facet flattening of cusps shortened clinical crown restorations show same wear as tooth substance can see present in pt on lower anterior 3-3; note this could also be due to MCC opposing these teeth pt identified parafunctional habits - grinds teeth at night
44
smith & knight
tooth wear index pt scored 2 surface - incisal criteria - loss of enamel exposing dentine for less than 1/3 of surface loss of enamel just exposing dentine defect <1mm deep
45
why remove crowns
unaesthetic poor margins
46
how to remove crowns
drill off using tungsten carbide / diamond bur
47
options for 11 & discussion with pt
1. no tx, not causing any pain, PA radiolucency present but no symptoms; might require treatment in the future 2. replace 21 and not 11 but this will leave uneven looking crowns 3. can replace but may make it worse, depends on quality of tooth structure remaining underneath crowns, may need RCT but no clear canal visible may not be possible would require referral to specialist with microscope equipment. Possible sensitivity following this & will not last forever.
48
why MCC crowns
- cheaper - functional - less tooth tissue removal required - not as good aesthetics - can see metal collar - metal shine through - less destructive than all ceramic
49
why not all ceramic crowns
definitely more aesthetic but more expensive and require increased removal of healthy tissue so increased chance of pulp exposure upon prep emax less prep but all ceramic more prep
50
dimensions for emax / mcc
emax 2mm all the way round ? mcc - 1.3mm labial should and 0.5mm palatal chamfer ?
51
why not place bridge for missing gaps
18 and 48 are in occlusion replacing 47 with nothing above it is pointless; would be for aesthetic rather than functional purposes same with the 37 as nothing in upper arch opposing this mesially tilted so difficult to get parallel prep for conventional cantilever
52
why soft splint
protect remaining tooth structure identify where parafunction is worst cheap & easily constructed prior to more definitive hard splint following restorative tx
53
tmd
symptom complex of multifactorial aetiology which almost certainly represents a psychological response to stress that becomes chronic through increasing muscle tension affecting MoM namely masseter, lateral pterygoid, temporalis. muscle hyperactivity can release lactic acid & other components that can cause pain
54
effect of cannabis on dental tx / teeth
1. xerostomia - decreased saliva production 2. increased risk of perio - contains toxins & carcinogens that can irritate gum tissue leading to increased inflammation 3. tooth discolouration - adheres to enamel leading to yellow / brown staining 4. oral cancer risk - not as high as tobacco. long term exposure increases risk of OC 5. healing - may impair ability to heal due to reducing blood flow to surgical site & interfering with body's natural healing process
55
discussion with pt re prosthetic tx
Long term stability of crowns due to lack of posterior support, lot of weight on the front so contributing to attrition of lower anteriors due to parafunction denture would help relieve this pressure provide more function
56
calculus
57
why restore in composite over amalgam
more aesthetic bonds to tooth
58
constituents of composite
59
what is addiction
chronic, primary, neurobiological condition influenced by genetic, psychosocial & environmental factors
60
effects of cannabis
euphoria slowed thinking & reaction time confusion impaired balance & coordination cough respiratory infection increased HR anxiety addiction
61
risk factors for substance abuse
social deprivation poor housing adverse childhood experiences poor role models low educational aspiration lack of opportunities high unemployment
62
cannabis & LA
1. potency alteration - cannabis can alter perception of pain & alter sensitivity to pain so could influence effectiveness of LA 2. metabolic interactions - can impact metabolism & clearance of LA from body altering duration of action or potency 3. cardiovascular effects - with adrenaline can prolong acute tachycardia (HR >100bpm) 4. CNS depression - both cannabis & LA depress CNS leading to sedation / drowsiness so combination can increase effect risking excessive sedation / respiratory depression if high doses are used 5. individual variability - some people may experience heightened sensitivity to pain or altered anaesthesia
63
how does LA work
reversibly binds to sodium channels preventing the entry of sodium into the cells thereby inhibiting the propagation of nerve impulses consequently nociceptive impulses associated with painful stimuli do not reach the brain and pt does not perceive pain note - lidocaine fully metabolised in liver
64
mx of attrition in this pt
prevention & monitoring this is tx pts usually in this phase for 6mths advice re TMD & provision of splint
65
cannabis facts
most abused drug in UK with 18.7% of adults aged 16-59yrs reporting having used it in previous yr THC is 1 of 66 cannabinoids within cannabis & is most associated with hallucinogenic / psychoactive effects THC reacts with 2 main receptors: CB1 (mainly found in brain) and CB2 (mainly found in immune cells and GI tract) most common route of admin is smoking through which 50% of THC is inhaled high often felt within mins and lasts 2-3hrs THC accumulates in adipose tissue & slowly released back into the body total elimination can take up to 30 days
66
further dental health impact following cannabis use
- poor OH & perio - reduced salivary flow - appetite enhancing nature of drug - LA containing adrenaline may prolong tachycardia following acute dose of cannabis - chronic use results in increased risk of oral leukoplakia / OSCC / candidiasis / oral infections - cannabis may compound effects of anaesthetic agents thus increasing arterial pressure & HR to possibly life threatening levels - pts should be advised not to use cannabis for at least 72hrs before tx under conscious sedation in order to reduce the likelihood of drug interactions & unpredictable sedation quality
67
options for LA without adrenaline
prilocaine & mepivicaine have weak vasodilation properties. both are short acting anaesthetics & good options for children, elderly & pts with contraindications to adrenaline
68
why is adrenaline added to LA
to act as a vasoconstrictor and delay drug absorption which prolongs duration of LA and reduces toxicity risk
69
LA preparation
LA base reducing agent (sodium metabisulfide) preservatives fungicide +/- vasoconstrictor
70
adrenaline v felypressin
felypressin = less effective vasoconstrictor but is an alternative for hypertensive pts or those wanting adrenaline free LA f - acts on ADH receptors a - acts on adrenoreceptors
71
prilocaine & mepivicaine
prilocaine (citanest) 3% HCl w felypressin (pulp infil = 30-45mins, pulp block = 60mins, soft tissue = 3-6hrs) max dose = 8 cartridges (8mg/kg) mepivicaine 3% HCl plain (pulp infil = 20mins, pulpal block = 40mins, soft tissue = 2hrs) , max dose = 6 cartridges (3mg/kg)
72
aims of TMD tx
1. control immediate pain 2. lower psychological stress 3. eliminate TMJ damage
73
possible tx for TMD pts
rest heat analgesics injections CBT hypnosis psychiatric tx rest masticate bilaterally avoid trauma splint
74
use of splint
increase vertical dimension, may reduce joint loading, eliminate faulty occlusal interferences, provide cognitive awareness of damaging oral habits
75
reasons for parafunction in this patient
STRESS - due to caffeine intake, cannabis intake etc
76
why is this patient at high risk for candida
dry mouth can use gel / spray / pastilles to help salivary flow
77
benefits of this pt receiving a denture
78
what kind of denture would you start with and why
acrylic - cheaper, better transitional denture, would aim for upper and then discuss provision of lower ask at each appointment highlight positives of getting denture but ensure it is something that pt wants; let pt come to you and ask you for denture would then transition to a chrome denture and then discuss provision of lower RPD if that was something pt was interested in
79
why leave RR
preserve bone height for future implant placement near vital structures i.e. IAN present for no of yrs with absence of PA pathology can be left & monitored to ensure caries free / no PA pathology
80
reasons why these RR were extracted
infection risk sitting in soft tissue so straightforward low risk procedure
81
pros / cons acrylic denture
cheap non toxic / non irritant bulkier less heat transfer quicker to make easier to add to (can add stainless steel clasps to acrylic denture)
82
pros / cons CoCr denture
stronger thinner more expensive more difficult to add to takes longer to make
83
3 methods of testing for candida
1. swab / oral rinse for culture 2. biopsy for hp using PAS stain 3. smear for microscopy
84
why does dry mouth impact tx
less saliva saliva is antibacterial, acts as a buffer, aids digestion
85
sclerosed canal
calcified deposits within canal 2ndary dentine gets laid down an calcified blocking canal system can be idiopathic, age related, trauma related
86
healthy gingiva
pink knife edge, no erythema, stippled, no BoP
87
how many units in 4 glasses of wine
9.6