root resorption Flashcards

(29 cards)

1
Q

root resorption definition

A

non bacterial destruction of dental hard & soft tissue due to interaction of clastic cells

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2
Q

key features of osteoclast cell (3)

A
  1. very mobile
  2. ruffled border (well adapted to resorb hard tissue)
  3. in contact with dentine
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3
Q

what induces differentiation to osteoclasts

A

RANKL - receptor activator NF kappa b ligand
note - OPG (osteoprotegrin) downregulates therefore inhibiting RANKL & so development of osteoclasts

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4
Q

what causes stimulation of RANKL (4)

A
  1. parathyroid hormone B3 & interleukin B1
  2. bacterial lipopolysaccharides
  3. trauma (physical / chemical)
  4. chronic inflammation
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5
Q

surfaces involved (3)

A
  1. pdl
  2. cementum
  3. pre dentine
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6
Q

main categories of resorption & sub categories

A
  1. INTERNAL - inflammatory / replacement
  2. EXTERNAL - inflammatory / replacement / cervical / surface
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7
Q

o/e what probe should be used

A

PCP12 - as BPE will inhibit examination of pdl

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8
Q

radiographic examination

A

require 2 angles with 30 degree mesial / distal beam shift ( if shift in what you are looking at strong chance it is external) or CBCT

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9
Q

how does internal resorption appear on radiograph

A

comes out from middle of root canal so loses parallel lines

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10
Q

how does external resorption appear on radiograph

A

crucially can see parallel lines of root canal system

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11
Q

presentation of internal inflammatory resorption

A

positive to sensibility tests, no obvious signs, no pockets unless perforated root surface, no sinus unless peri-radicular disease
radiographically centred in canal, doesn’t move with bean shift & see ballooning of RC i.e. loss of parallel lines

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12
Q

pathogenesis of internal inflammatory resorption

A

coronal pulp necrotic
apical pulp vital
lesion inc inflammatory & vascular tissue (if perforated will communicate with pd)
lesion will continue to progress until apical pulp goes completely necrotic too

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13
Q

tx of internal inflammatory resorption

A

orthograde endodontics ONLY
- possible haemorrhage
- active irrigation
- inter visit medicament
- thermal obturation

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14
Q

presentation of internal replacement resorption

A

no clinical signs
very rare
usually incidental finding
radiographically -> radiopaque expansion of the pulp i.e. replacement element via bone / pdl / cementum (something mineralised) so RCT unpredictable & unlikely to benefit pt

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15
Q

presentation of external surface resorption

A

clinically normal, only sign may be slightly increased mobility
radiographically -> key thing to note is that PDL IS INTACT

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16
Q

aetiology of external surface resorption (4)

A
  1. orthodontics - 90% of teeth have some form of ESR; teeth used for anchorage usually worse 2-5% severe
  2. ectopic teeth - in lateral incisor which guides in canine
  3. pathological lesions - pressure from adjacent pathological lesion
  4. idiopathic
17
Q

tx of external surface resorption

A

pulp is healthy so endo tx will not have any effect so must remove source to stop the resorption i.e. remove ortho bracket & splint if mobile

18
Q

presentation of external inflammatory resorption

A

find that tooth is usually restored
may have increased mobility depending on extent
sensibility testing is NEGATIVE as pulp necrotic
radiographically -> usually has a PA radiolucency

19
Q

aetiology of external inflammatory resorption

A

pulp is necrotic from bacterial or dental trauma in origin
periapical inflammatory lesion precipitates the resorption process

20
Q

tx of external inflammatory resorption

A

remove the cause of the inflammation
this is usually via:
orthograde endo tx / re tx
possiibly surgical endo
XLA
note - apical stop can be challenging so consider hydraulic cement rather than GP

21
Q

presentation of external replacement resorption

A

historically trauma related
can be unrestored but infra occluded
not TTP but high pitched note
no physiological mobility
radiographically -> pulp appears normal, no PA radiolucency but crucially there is PDL degeneration

22
Q

aetiology of external replacement resorption

A

TRAUMA - significant injuries to periodontium such that osteoclasts then in contact with external root dentine to begin resorption e.g. avulsion or lateral luxation

23
Q

tx of external replacement resorption

A

DECORONATION
if infraocclusion >1mm in growing pt
remove crown to alveolar lever & allow root to resorb
this preserves bone volume
adjacent teeth & periodontium develop normally
tooth replacement via denture / RBB

if pt fully grown can monitor & add comp incisally if infraoccluded
endo intervention will not stop resorption

24
Q

consequence of delayed decoronation in external replacement resorption

A

no pdl so tooth fused to bone so ankylotic percussion note

25
presentation of external cervical resorption
can be unrestored, perio pocketing if extensive and profuse BoP, notable pink spot, POSITIVE to sensibility testing pink spot - middle of crown can have +/- bleeding / erythematous gingivae radiographically -> radiolucency but still maintains parallel canals
26
classification of external cervical resorption
apico coronal direction: 1. crestal 2. coronal 1/3 2. middle 1/3 4. apical 1/3 circumferential: 1/4 1/2 3/4 > 3/4
27
risk factors for developing external cervical resorption (6)
1. ortho 2. trauma - avulsion & luxation 3. historical non vital whitening when heat was applied 4. wind instruments 5. viral infection 6. systemic disturbance i.e. thyroid
28
tx for external cervical resorption
1. monitor - resorption likely to continue 2. XLA - + prosthetic replacement 3. endo - internal repair & orthograde endo
29
summary of tx options
orthograde endo = external & internal inflammatory surgical endo = external cervical no endo = external replacement & surface