chapter 2 Flashcards

1
Q

what are the two main thoughts on mental disorders

A

biological and environmental

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2
Q

Biological aspects

A

downplay the influence of experience

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3
Q

Environmental influences

A

emphasize external factors (poverty and parenting styles)

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4
Q

what are the three main approaches to viewing mental disorder

A

Biological & psychodynamic, Humanistic & existential, Behavioural & cognitive

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5
Q

Biological & psychodynamic

A

view dysfunctional behaviour as the product of forces beyond the individual’s control

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6
Q

Bio encourages a physical basis for disorders – leads to

A

formulation of a diagnostic system that classifies people as disorder & implies that physical interventions should be the treatment

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7
Q

Humanistic & existential

A

lay responsibility for action & choices on the individual

Personal experience provides the basis for the development of self-directed behaviour

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8
Q

Behavioural & cognitive

A

a mix of external and internal factors produce dysfunctions

The way that people are conditioned to learn and the way they think or perceive the world causes the
development

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9
Q

what does the behavioural cognitive approach emphasize

A

classification of behaviours not people

Seek environmental events that shape dysfunctional responses

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10
Q

behavioural and cogntive approach treatment

A

manipulating environment or modifying perception / schema / beliefs of people
regarding experience and self

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11
Q

Adopting one of these theories is influenced by

A

the prevailing social belief system & the individual’s disposition to see human behaviour as being determined by factors beyond or within their control

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12
Q

what are the leves of theories

A

single factor explanation and interactionist explanation

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13
Q

sing-factor explanation

A

: attempts to trace the origins of a disorder to one factor

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14
Q

Interactionist explanation

A

behaviour is the product of interactions between a variety of factors

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15
Q

Can be classified according to their level of explanation

A

Ex: Maslow’s theory of self-actualization tries to explain all human behaviour while Freud’s try to explain abnormal behaviour

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16
Q

Theories embody 3 features

A
  1. Integrate most of what is currently known about the phenomena in the simplest way (parsimony)
  2. Make testable predictions about aspects of the phenomena that weren’t previously thought of
  3. Make possible to specify what evidence would deny the theory
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17
Q

Why do theories gain strength?

A

Evidence supports their predictions

Alternative explanations are rejected

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18
Q

Null Hypothesis

A

proposes that the prediction made from the theory is false

When you reject it, it provides support for a theory BUT theories are NOT factors and can NEVER be PROVEN to be true

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19
Q

Etiology

A

the causes or origins of a disorder

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20
Q

General aims of theories

A

Explain the etiology of behaviour

Identify factors that maintain the behaviour

Predict the course of the disorder

Design effective treatments

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21
Q

Factors involved in the etiology may not be involved in

A

its maintenance

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22
Q

In disorders where there is a clear biological cause

A

environmental manipulations may alleviate or prevent
the development of the most serious symptoms

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23
Q

Phenylketonuria (PKU) found in the diet

A

When detected in newborns, it’s possible to prevent development of severe symptoms like
retardation by administering a diet low in PKU-containing foods

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24
Q

using cognitive therapy methods in depressed & anxious people can change their neurobiology

A

Decreases activation of the amygdala / hippocampal regions associated w/ negative affect

Increased activation of areas involved in cognitive control of negative emotion

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25
Theories of the etiology of mental disorders
Biological Psychodynamic Behavioural & Cognitive Humanistic & Existentialist Socio-cultural Integrative
26
Biological Models
Adopt the language of medicine = patients, symptoms, treatments CNS damage is the focus + PNS dysfunction (somatic + autonomic) + endocrine system dysfunction
27
CNS
Brain has ~100 billion neurons & thousands of billions of glia cells
28
what three brain regions are in the CNS
forebrain, midbrain, hindbrain
29
forebrain
speech, perception, memory, learning, planning
30
midbrain
reticular activating system = control arousal and attention
31
hindbrain
directs the function og the autonomic NS
32
Current theories about the brain bases of abnormal behaviour
They focus on the role of neurotransmitters and not neuronal damage Most research is done on GABA, norepinephrine, serotonin, dopamine
33
Abnormal behaviour can result from disturbances in ligands in many ways
Too little / much of the ligand produced or released into the synapse Too few / many receptors on the dendrites Too few / many ligand-deactivating subs in the synapse The reuptake process is too rapid / slow
34
problems with any of the ligand =
alterations in the brain circuits
35
current research shows that disturbance sin the ligan systems have
general effects interactions of ligands and subtypes are related to behaviour
36
Inferring a causal relationship btw disturbances in ligands & abnormal behaviour
Dopamine antagonists are used to treat schizophrenia neurochemistry and behaviour are bidirectional
37
Brain Plasticity
capacity of the brain to reorganize its circuitry – influenced by experienced that occur pre- and postnatally
37
Ligand function affects behaviour but
behaviour affects neurochemistry
38
PNS
Somatic and autonomic NS
39
Autonomic (ANS)
sympathetic + parasympathetic
40
These systems work cooperatively but in terms of stress
they act antagonistically Its response is exaggeratedly strong or remarkably week
41
Sympathetic
readies body for action
42
Parasympathetic
shuts down digestive process
43
Overactive ANS
increase readiness to acquire phobias or other anxiety disorders
44
Deficits in regulation of ANS functions can influence
disordered behaviour
45
patient with GAD tend to show
decreased parasympathetic regulation of heart rate & respiration – also show chronic muscle tension (somatic system)
46
there’s an inflexibility of
the autonomic and somatic systems in GAD
47
ES and CNS interact in a
feedback loop It maintains homeostatic levels of hormones circulating in the bloodstream
48
order of ES2
ES hypothalamus pituitary (“master gland”) adrenal cortex cortisol (anti-inflammatory)
49
HPA: hypothalamic pituitary-adrenal cortex axis
Has been studied in regard to anxiety and depression It involves the release of cortisol into the bloodstream by the adrenal cortex It increases the # of intracellular glucocorticoid receptors, leading to anti-inflammatory effects & other survival benefits
50
Sensitivity to stress is implicated in
the etiology of depression and anxiety
51
Cretinism
dwarf-life appearance and mental disability – result of defective thyroid gland
52
Hypoglycemia
pancreas fails to produce insulin
53
Genetics and behaviour
Idea that human behaviour is inherited Inherited features interact with the environment to produce behaviour
54
Genetic Determinism
who a person is, is determined largely by inherited characteristics
55
Behavioural Genetics
offers insight into the biological bases of abnormal functioning BUT in psychopathology, genes confer a liability – not a certainty.
56
Genotype-environment interaction
genes may influence behaviour that contribute to environmental stressors which increases the risk of psychopathology
57
what does the G-E interaction suggest
a reciprocal relationship between genetic predisposition and environmental risk factors Neither one can explain the onset of the disorder- A complex interaction of both is required
58
Study: investigated the link between
A gene involved in serotonin transmission Stressful life events Depression
59
what were the results of the study
People with two LL alleles of the gene coped better than people with two SS alleles of the gene No direct link between the gene and depression
60
what can be determined from the results of the study
people with two SS alleles developed depression only if they also experienced stressful life events
61
Behavioural research into the genetic bases of psychiatric disorders
Family studies – pedigree  Twin studies  Adoption studies  Genetic linkage studies  Molecular biology studies
62
molecular biology studies
When comparing one with another, if the problem arises in both, they are said to be concordant for the problem. BUT this isn’t always true because environmental influences can be involved
63
(2) Psychodynamic Theories
Suggest that behaviour is motivated by unconscious processes acquired during the formative years of life See the person as having little control over their action (Similar to the biological theories)
64
freud on psychodynamic theories
: traumatic experiences early on become repressed because they’re too distressing
65
Features of Freud that determine current behaviour and thinking
1. Levels of consciousness 2. Structures of personality 3. Psychosexual stages of development
66
levels of consciousness
3 levels conscious preconscious unconscious
67
conscious
info that we are aware of
68
preconscious
info which we can bring into awareness
69
unconcscious
info that can only be brought to awareness with difficulty and techniques i. Most of our memories, motivations and drives are unconscious ii. Kept unconscious by Defense Mechanisms
70
Defense Mechanisms
1. The use of these mechanism depleted psychic energy  psych dysfunction
71
structures of personality
3 that are in constant conflict ID Ego Superego
72
ID
present at birth & contains/represents our biologically instinctual drives i. Drives that demand instant gratification w/o regard for consequences ii. Acts according to the pleasure principle
73
Ego
first year of life, curbs the desires of the ID so that the person doesn’t suffer i. It has no concern of right vs. wrong ii. Concern for avoidance of pain and discomfort + maximization of unpunished pleasure iii. Acts by the Reality principle
74
superego
internalization of the moral standards of society & parents i. Acts on the Moral Principle ii. Serves as the person’s conscience by monitoring the ego
75
psychosexual stages of development
oral, anal, phallic, latency, genital
76
oral
birth – 18months, focus on oral activities
77
anal
18months – 3yrs, toilet training cooperation or resistance
78
phallic
3-6yrs, Oedipal or Electra complex
79
latency
6-12yrs, consolidation of behavioural skills and attitudes
80
genital
adolescence, achievement of personal and sexual maturity
81
defense mechanisms
repression regression projhection intellectualization denial displacement reaction formation sublimation
82
repression
inability to recall something
83
regression
acting childlike
84
projection
attributing your own desires to others
85
intellectualization
hiding the real issues behind abstract analyses
86
denial
refusal to aknowledge
87
displacement
angry at your spouse because you got fired ex
88
reaction formation
expressing the opposite viewpoint
89
behavioural theories
watson
90
what did watson say
classicla conditioning is the basis upon which human behaviour is learned
91
stimulus-stimulus learning
transfer of a conditioned or unconditioned response from one stimulus to another
92
acquisition of phobias
a neutral stimulus, over time and experience, elicits a conditioned fear response
93
what is the problem with watsons view on acquisition of phobias
over-generalization it cannot explain the many facets of phobias
94
two factor theory of conditioning
solution by Mowrer 2 types of learning take place in the acquisition and maintenance of phobias
95
classical conditioning
establishes the aversive response to a previously neutral stimulus
96
what happens after classical conditioning
human avoids the ocnditioned stimulus to prevent fear and is why we prevent extinction
97
operant conditioning skinner
all behaviours are guided by consequence
98
positive reinforcement
behaviour is increase bc it leads to pleasant consequences
99
neg reinforcement
behaviour is increased bc it leads to a reduction of stress
100
pos punishment
behaviour is reduced bc it leads to unpleasant experiences
101
neg punishment
behviour is reduced due to the removal of something desirable
102
social learning theory
learn by observing others rather than by direct personal experience
103
cognitive behavioural theory
thinking and behaviour aare learned therefore both can be changed
104
cognitive theories
aim to help people shift from unhealthy appraisals to more realistic adaptive ones
105
what is the treatment used in cogntiive theories
change schemas and underlying beliefs/ develop mroe realistic and adaptive cognition
106
rational-emotive behavioural therapy
consequences of life events are not contigent upon the activating event but are mediated by ones beliefs about these experiences we can learn to change how we think
107
cognitive theory and therapy
emotions and behaviours are influence by perception or cogntiive appraisals of events
108
what are the levels of cognition
schemas information-processing biases and intermediate beliefs automatic thoughts
109
shcemas
early maladaptive schemas can originate from repetitious, aversive experiences in childhood
110
what are the three things schemas do
a. Can influence how someone processes life experiences b. Develop early but don’t become active until triggered by negative life events c. content specific
111
Content-specific
different types of beliefs are considered to be related to different kinds of abnormal behaviour
112
infromation-processing biases and intermediate beliefs
a. Selective attention and enhanced memory for info that’s schema-consistent b. Persistent “if-then” statements c. All or nothing thinking d. Inaccurate causal attributions
113
automatic thoughts
people who experience psychopathology have a greater # of neg and threat related automatic thougths
114
socio-cultural influences
role that society and close others play in the etiology and maintenance of psychiatric disorders
115
stigma
 Plays a role in the maintenance  One of the largest barriers to people seeking treatment 1/5 Canadians have mental health issues – 1/3 seek help
116
lebeling theory
a person being identified as having a mental illness results in other people perceiving them as dysfunctional and different.
117
what does the labelling theory perceive
results in the person being treated disadvantageously and disrespectfully
118
public vs self stigma
Being aware that there’s a negative perception about mental illness doesn’t alter health-seeking behaviours The internalization of these perceptions (self-stigma) hinders help-seeking
119
social support
Helps prevent and reduce intensity of psychological problems Associated with decreased psychiatric symptoms quality over quantity
120
absence of social support is
the causal chain leading to dysfunction
121
gender
Many disorders are more prevalent in women and vice versa Personality disorders – dependent personality or antisocial personality – may be an exaggeration of gender roles
122
prejudice and lack of opportunity
stress in minority and underprivileged populations
123
resentment in race and poverty
generation fo behaviours that are viewed by others as antisocial or dysfunctional
124
professionals from priviledged classes are more inclined to
apply denigrating diagnoses to patients from lower classes
125
integrative theories
integrate biological and environmental factors
126
systems theory
the whole is more than the sum of its parts
127
causation is the result of
multiple factors interacting its bidirectional process
128
diathesis-stress perspective
predisposition to developing a disorder (diathesis) interacts with the experience of stress to cause mental disorders
129
The diathesis can be
biological or psychological or social
130
a diathesis will not produce a disorder without
the trigger of some stress
131
a diathesis allows the possibility that people may develop disorders through
the influence of different diatheses and different stressors
132
biosychosocial methodel
biological, psychological and social factors
133
Brain functions have been found to influence and to be influenced by
psychological and social processes; This relationship is reciprocal
134
early maladaptive schemas
1. Disconnection and rejection 2. Impaired autonomy and performance 3. Impaired limits 4. Other directedness 5. Over-vigilance and inhibition
135