Chapter 5 Shit Flashcards

1
Q

What are adaptations of Anaerobic training?

A

-Increased agonist recruitment
-Improved neural firing rates
-Greater synchronization of timing and neural discharge
-Reduction of inhibitory mechanisms such as GTO activation

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2
Q

What is cross-education?

A

When you do a unilateral resistance exercise, the non working side receives increases in strength and neural activity from the movement. Sully taught you this in music

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3
Q

What is the bilateral deficit?

A

Bilateral deficit is when the sum of unilateral force production is greater than the sum of bilateral force production

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4
Q

What is bilateral facilitation?

A

Occurs in trained individuals, force produced with both limbs is greater than the sum of unilateral force production

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5
Q

How do motor units adapt to anaerobic training?

A

-Increased frequency of firing increases force production
-Increased synchronization of agonist, synergist, and antagonist muscles
-Increased activation and firing rate are primary motor unit adaptations to anaerobic training
-Heavy lifting results in hypertrophy of all muscle fibers due to the size principle
-Advanced lifters can selectively recruit type 2 fibers - allows “skipping over” the type 1 fiber recruitment associated with the size principle to rapidly generate force by immediately recruiting type 2 fibers
I.E. olympic weightlifters

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6
Q

What is the size principle?

A

Motor units are recruited based on recruitment threshold and firing rate (units are recruited from smallest to largest depending on the intensity)

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7
Q

How do smaller and larger muscles increase force production?

A

-Smaller increase contraction frequency
-Larger increase contraction frequency and increasing motor unit recruitment

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8
Q

How does the structure of a muscle change with anaerobic training?

A

-Pennation angle can increase which allows a greater protein deposition and CSA growth (resistance training can also do this)
-Fascicle length can also increase (sprint and jump training can increase vastus lateralis fascicle length)
-Increase in myofibrillar volume
-Increased cytoplasmic density
-Increased sarcoplasmic reticulum and T-tubule density
-Increased sodium-potassium ATPase activity
-Reduced mitochondrial density due to muscle CSA increase being greater than increase in mitochondria (not caused by loss of mitochondria)
-Reduced capillary density caused by increase in muscle CSA
-Increased H+ buffering capacity
-Increased resting CP and ATP concentration
-Increased muscle glycogen content

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9
Q

What are CNS adaptations to anaerobic training?

A

-Neural changes in spinal cord elevate fast-twitch recruitment
-Untrained populations have limited ability for maximal recruitment
-Less than 71% of muscle tissue activated during max effort in untrained people

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10
Q

How does the neuromuscular junction and reflex potentiation adapt to anaerobic training?

A

Adaptations in the NMJ include an increased area of NMJ and greater length of terminal nerve branches
Anaerobic training can increase the intensity of myotatic reflex - enhances the involuntary elastic properties of muscle and connective tissue to increase force

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11
Q

How does bone tissue adapt?

A

-Mechanical loading helps remodel bones
-Osteoblasts migrate to the bone surface to begin remodeling
-Secrete proteins (mostly collagen) into spaces between bone cells to increase strength
-Collagens forms the bone matrix and eventually mineralizes into calcium phosphate crystals (hydroxyapatite)
-New bone formation occurs primarily on the outer bone surface (periosteum)
-Increase diameter and strength

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12
Q

What effects the rate of bone formation?

A

-Rate of bone formation varies in axial and appendicular skeleton
-Rate difference caused by different amounts of trabecular (spongy bone) and cortical (compact bone)
-Cortical bone - dense compact outer shell surrounding trabecular board
-Trabecular bone more capable of growth
-New bone formation requires minimal essential strain - the threshold stimulus for new bone formation
-MES level increases as bones become stronger, require progression to continue growth
-Bone remodeling requires six months or longer to occur
-Bone mineral density increases as strength and hypertrophy increase the demands on the bone

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13
Q

Talk about bone remodeling in response to mechanical loading

A

-Application of longitudinal weight-bearing bends bone
-Osteoblasts lay down additional collagen fibers at bending site
-Dormant osteoblasts migrate to bend site
-Collagen mineralizes - increasing the bone diameter

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14
Q

How do you get a specific area of bone to grow?

A

-Load the region of the skeleton where bone growth is desired
-Novel forces stimulate bone growth
-Important to load areas commonly affected by osteoporosis
-Osteoporosis is a disease where bone mineral density and bone mass are reduced to critically low levels
-High-impact cyclical loading increases BMD more than low-impact activity

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15
Q

How would you select exercises for osteogenic stimuli?

A

-Multiple joints
-Direct force vectors through the spine and hip (structural exercises)
-Squat, power clean, deadlift for the lower body
-Shoulder press for upper body
-Progressive overload
-Must place greater than normal demands on musculature to increase bone mass
-Bones respond to higher forces - i.e. 1rm-10rm loads
-Adaptive response reduces stress fractures
-Peak bone mass elevated by bone loading during adolescence and adulthood
-Changing distribution and direction of force vectors in resistance training presents unique stimulus for bone growth within a given region
-If the magnitude and rate of force application is sufficient, bone growth stimulus can be maximized with as few as 30 reps per workout

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16
Q

What is included under the connective tissue umbrella?

A

Connective tissue includes tendons, ligaments, fascia, and cartilage and forms the critical link between muscle and bone

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17
Q

What is collagen?

A

-Primary structural component of all connective tissue
-Includes type 1 collagen (bone, tendon, ligaments) and type 2 collagen (cartilage)
-Secreted by fibroblasts - the most common cells in connective tissue
-Enzymes cleave the protective extensions on procollagen molecule following secretion
-Collagen filaments are organized in parallel - pairs of collagen filaments are known as a microfibril
-Microfibrils are arranged into fibers - rely on cross-linking - strong chemical bonds between collagen molecules

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18
Q

What is procollagen?

A

Parent protein to collagen

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19
Q

What are properties of tendons and ligaments in regards to collagen and bones?

A

-Primarily consist of tightly packed parallel arrangements of collagen bundles
-Mature tendons and ligaments contain few cells
-Significant strength in bone attachment
-Ligaments contain elastin - allows some stretch to occur during normal joint motion
-Low blood flow means tendons and ligaments are slow to regenerate and slow to recover from injury

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20
Q

What is fascia?

A

-Fibrous connective tissue surrounding the muscles
-Bundles of collagen arranged in different planes
-Provides resistance from force in multiple directions
-Converges to form tendons at the end of the muscle

21
Q

What is cartilage?

A

-Dense connective tissue with high ability to withstand force without damage
-Provides a smooth joint articulating surface - -Hyaline cartilage
-Acts as a shock absorber
-Aids in connective tissue attachments to skeleton
-Fibrous cartilage - tough cartilage found in intervertebral disks and at the tendon-bone junctions
-Receives nutrient supply via diffusion from synovial fluid - Joint immobilization prevents proper diffusion of nutrients - results in the death of healthy cartilage cells

22
Q

How does connective tissue adapt to resistance training?

A

Intense anaerobic training can increase the following
-Collagen fibril diameter
-Number of covalent cross-links in the fiber
-Increase in the number of collagen fibrils
-Increase in packing density of collagen fibers
-Collectively increase tendon’s ability to withstand force as well as tendon stiffness - the amount of force required per unit of tendon elongation

23
Q

How does cartilage adapt to anaerobic training?

A

-Not fully understood
-Resistance training may prevent thinning or atrophy of cartilage
-Cartilage morphology likely determined by genetic factors
-Moderate-intensity exercise may be adequate for increasing cartilage thickness
-Strenuous exercise does not cause degenerative disease when overloaded appropriately
-Tissue viability can be maintained using a variety of exercise modalities and using a full range of motion

24
Q

How do tendons and ligaments adapt to anaerobic stimulus?

A

-Long-term adaptations stimulated through progressive high intensity loading patterns using external resistance
-Low-moderate loads do not change the collagen content of connective tissue
-Forces should be exerted through a full range of motion around the joint

25
Q

What are acute endocrine responses to anaerobic training?

A

-Elevated testosterone, GH variants, and cortisol in men
-Rapidly stabilize after 30 minutes
-Magnitude of elevation greatest when large muscle mass used, or workout intensity is moderate to high
-High correlation between blood lactate, H+ concentration, and cortisol and GH level
-IGF levels have a delayed response to training and depend on GH response
-Catecholamine levels reflect acute anaerobic demands

26
Q

What are chronic changes in acute hormonal response to anaerobic training?

A

-Acute hormonal responses likely mirror the ability to tolerate progressively heavier loads from consistent anaerobic training
-Long-term acute responses likely augment the ability to tolerate and sustain higher intensities

27
Q

What are chronic changes in resting hormonal concentrations to anaerobic training?

A

-Inconclusive research
-Resting hormone levels likely unaffected by long-term training
-Chronic elevation of hormones may be detrimental
-May reduce hormone receptor binding sensitivity

28
Q

What hormonal receptor changes occur as a response to anaerobic training?

A

-Receptor content mediates responses to hormones
-Resistance training upregulates androgen receptors within 48-72 hours post-workout
-Resistance training stimulus mediates the magnitude of androgen receptor (AR) changes

29
Q

What is the difference in response immediately an 1 hour post 1 set vs 6 sets of 10 squats?

A

-No difference in AR following single set
-Higher volume showed downregulation of AR content 1 hour post workout
-Protein-carbohydrate consumption post-workout attenuates the AR downregulation

30
Q

What are the acute cardiovascular responses to anaerobic training?

A

Elevation of the following
-Heart rate
HR highest 5 seconds following the work set
-Stroke volume
Highest during eccentric phase especially with Valsalva maneuver
-Cardiac output
Highest during eccentric phase especially with Valsalva maneuver
-Blood pressure
Peak BP of 320/250 and HR of 170 bpm reported during 95% 1RM leg press
Blood pressure elevation is nonlinear
Highest during concentric phase - especially at the “sticking point”
No data to suggest resistance training has a negative effect on resting BP
-Degree of blood flow increase depends on
Intensity of resistance
Length of time of the effort
Size of muscle mass used
-Blood flow to working muscles decreased during set at intensities above 20% 1rm because of the tissue occlusion of capillaries
Blood flow increases following the set - reactive hyperemia

31
Q

What are chronic cardiovascular responses to Anaerobic training?

A

-Heart rate
Some long term reduction in resting HR
-Blood pressure
Decrease of 2-4% BP following long term resistance training
-Rate-pressure product = heart rate x systolic blood pressure
Constant or decreases following resistance training
-Possible slight decrease in LDL, increase in HDL
-Increased left ventricular wall thickness
Does not increase relative to lean body mass - but larger overall compared to untrained population
-Chronic training reduces the acute cardiovascular response to resistance exercise at an absolute intensity

32
Q

What are ventilation responses to anaerobic exercise?

A

-Resistance exercise generally not limited by ventilation rate
-Ventilation rate unaffected or moderately improved by resistance training
-Ventilation elevated slightly during resistance training - most elevated in first minute of recovery
-Training adaptations include
-Increased tidal volume and breathing frequency during maximal exercise
-Breathing frequency reduced but tidal volume increased during submaximal exercise
-Improved ventilation efficiency is shown in trained individuals
Measured by a reduced ventilatory equivalent
Ratio of air ventilated to oxygen used by tissues

33
Q

How does aerobic training effect strength and power?

A

-aerobic training hinders strength and power gains.
-most detrimental to power.
-high volume aerobic has greatest negative effect

34
Q

How would you order anaerobic and aerobic training?

A

Aerobic is best done after resistance training as it will have a negative effect on the strength training

35
Q

How does anaerobic performance improve muscular strength?

A

Avg strength can increase from 20-40% depending on current training age

36
Q

How does anaerobic performance improve power?

A

Peak power output increases from resistance training

37
Q

How does anaerobic performance improve local muscular endurance?

A

-Enhanced local endurance associated with improved oxidative and buffering capacity
-Adaptations include improved mitochondrial and capillary numbers, type 2x-type 2a fiber transitions, and improved fatigue resistance

38
Q

How does anaerobic performance improve body composition?

A

Increases fat-free mass and lean body mass

39
Q

How does anaerobic performance improve flexibility?

A

Resistance training can improve flexibility - most noticeable when paired with flexibility training

40
Q

How does anaerobic performance improve aerobic capacity?

A

-Untrained individuals improve aerobic capacity via resistance training
-Trained individuals do not generally see improvements in aerobic capacity
-Circuit training can improve VO2 max

41
Q

How does anaerobic performance improve motor performance?

A

You can increase running economy, vertical jump, sprint speed, swing and throwing velocity, and kicking performance

42
Q

What are the phases of overtraining?

A

-Functional overreaching
-non-functional overreaching
-Overtraining syndrome

43
Q

What are the factors of functional overreaching?

A

-Excessive training leading to short term detriments in performance
-Recovery normally achieved within a few days or weeks
-Altered motor recruitment and sympathetic activity

44
Q

What are the factors of non-functional overreaching?

A

-Stagnation or decrease in performance, increased fatigue, decreased vigor, and hormonal disturbance
-Decreased circulation
-Altered excitation-contraction coupling
-Decreased glycogen
-Increased resting HR and BP
-Altered immune function and hormone concentration
-Recovery takes weeks to months

45
Q

What are the factors of overtraining syndrome and what are the 2 types of ots?

A

-sympathetic and parasympathetic overtraining syndrome are the 2 types
-Prolonged maladaptation of biological, neurochemical, and hormonal regulation mechanisms
-Can last six months or more
-Can ruin athletic career

46
Q

What is sympathetic OTS?

A

Increased sympathetic activity at rest

47
Q

What is parasympathetic OTS?

A

-Increased parasympathetic activity at rest and during exercise
-Decreased force production
-Decreased glycolytic activity
-Increased sickness, infection, and emotional disturbance
-Final phase of overtraining
-Chronically suppresses many physiological systems in the body

48
Q

What is detraining?

A

Loss of physiological adaptations once training stops or substantially reduces