Class 3 Flashcards

(49 cards)

1
Q

Etiology of inflammation

A

What caused it?

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2
Q

Duration of inflammation

A

how long is it?

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3
Q

Morphology of inflammation

A

What does it look like?

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4
Q

Acute inflammation

A

Sudden onset, short duration

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5
Q

Chronic inflammation

A

Longer duration (months/years)

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6
Q

Acute inflammation stops when?

A

Injurious stimulus removed

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7
Q

Acute inflammation initiated by

A

Local vessels near injury

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8
Q

Vessels initiate inflammation by

A

Alter permeability, allow leukocytes in

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9
Q

Inflammatory process stops by

A

Removal of stimuli

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10
Q

Mildest form of inflammation

A

Serous inflammation

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11
Q

Serous inflammation occurs?

A

Early stages and resolves easily

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12
Q

Inflammation rich in fibrin?

A

Fibrinous inflammation

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13
Q

Causes of Fibrinous inflammation

A

Bacterial infections

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14
Q

Inflammation caused by pus-forming bacteria

A

Purulent inflammation

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15
Q

Abscess

A

Local collection of pus

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16
Q

Inflammation of body surfaces or mucosa

A

Ulcerative inflammation

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17
Q

What ulcerative inflammation leads to

A

Ulceration or necrosis of epithelial lining

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18
Q

Ulcer is a ?

A

Defect in epithelium

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19
Q

Combination of ulcerative, fibrinous and purulent inflammation

A

Pseudomembranous inflammation

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20
Q

What is a pseudomembrane?

A

Exudate of fibrin, pus, cellular debris

21
Q

Diphteria

A

Pseudomembrane on throat

22
Q

Exudate containing monocytes, lymphocytes, macrophages, plasma cells is indication of?

A

Chronic inflammation

23
Q

Why does chronic inflammation perpetuate

A

Stimulate proliferation of fibroblasts &
Constantly recruit new inflammatory cells

24
Q

Chronic inflammation may lead to

A

Loss of function

25
Chronic inflammation type not preceded by acute
Granulomatous Inflammation
26
What accumulates at site of injury during Granulomatous inflammation
T-lymphocytes and macrophages
27
t-lymphocyte and macrophage function in Granulomatous inflammation
Lymphocytes release cytokines Cytokines turn macrophage into epithelioid cells
28
Epithelioid functions during Granulomatous inflammation
Cells fuse together forming multinucleated giant cells
29
Granulomas consist of
Lymphocytes, macrophages, multinucleated giant cells
30
Leukocytosis
increase circulating WBC's
31
Continually dividing or mitotic cells (Tissue healing)
Stem cells divide and differentiate, replace lost cells
32
Result of continually dividing cells (tissue healing)
Minimal tissue damage
33
Quiescent Cells (tissue healing)
Don't divide regularly, divide if necessary
34
Outcome of quiescent cells (tissue healing)
Regeneration - replace w identical tissue
35
Non-dividing cells (tissue healing)
Not able to divide and regenerate Eg. neuron
36
Outcome of non-dividing cells? (tissue healing)
Loss of functional capacity
37
by first intention healing
would clean, necrotic tissue/edges close together
38
by second intention healing
Large break, more inflammation, longer healing
39
Leukocytes (WBC's) **PMNS**
Scavenge initial injury site
40
Connective tissue cells
Prod. scar tissue
41
Epithelial cells
Divide and extend across wound
42
Macrophages
Stay at healing site, produce cytokines/growth factors/meadiators
43
Myofibroblasts
Smooth muscle cells and fibroblasts
44
Angioblasts
blood vessel precursor
45
Fibroblast produces?
prod. extracellular matrix
46
Loss of function, deficient scar formation, excessive scar formation, infection are signs of?
Complications of healing
47
Keloids
scar tissue overgrowth - excessive collagen
48
Contracture
fixation/deformity of joint
49
Adhesions
scar tissue bands joining 2 normally separated surfaces