Clin Med: Pulmonary Vascular Disorders Flashcards

1
Q

Pulmonary Circulation route

A

RV
Pulmonary trunk
L and R Pulmonary arteries
re-enter via pulmonary veins into the LA

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2
Q

Pulmonary trunk is how big

A

5 cm

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3
Q

main branches of the pulm arteries are about ______ of the aortas thickness

A

1/3

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4
Q

Hypoxia will trigger

A

vasoconstriction

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5
Q

Vasoconstriction in the lungs will lead to

A

shunting of blood to the more aerated areas within the lung —> so other areas get more blood

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6
Q

If the entire lung is vasoconstricted this can lead to

A

pulm HTN

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7
Q

Pulmonary lymphatics remove

A

both infectious material and excess fluid

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8
Q

Pulmonary lymphatics drain into

A

the right thoracic lymphatic duct

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9
Q

Pulmonary embolism is

A

a blood clot within the pulmonary vasculature

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10
Q

Causes of PE’s

A

DVT
fat embolism
air embolism
amniotic embolism

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11
Q

Treatment for PE

A

provoked - 3 month anticoag
unprovoked - forever anticoag
tPA (severe sx)

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12
Q

Fat embolism occurs with injury to

A

long bones - femur fracture

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13
Q

Septic emboli most commonly associated with

A

IVDU and s. aureus - will affect right side of the heart

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14
Q

Air embolism is most commonly due to

A

scuba diving
iatrogenic

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15
Q

In an air embolism bubbles enter

A

the pulmonary circulation and lodge in the same fashion a clot would

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16
Q

On physical exam what would you find with an air embolism

A

crepitus

17
Q

Amniotic embolism is when

A

amniotic fluid is able to get into Mom’s circulation
m/c occurs during lab or immediate postpartum

18
Q

Amniotic embolism tx

A

treat like it is a clot - anticoag, tPA if not stable
watch for bleeding to death since she just gave birth and will be on anticoags - have OB/GYN with you to counteract these

19
Q

PE presentation

A

pleuritic chest pain (breathing pain)
SOB, hypoxia
syncope
shock (right sided HF)
findings of DVT
cough
hemoptysis
tachycardia

20
Q

Classifications of PEs

A

Massive - hemodynamically unstable
Submassive - without right sided heart strain

21
Q

Complications of a PE

A

right sided heart failure
decreased gas exchange - lack of profusion leading to lung ischemia

22
Q

Pulmonary HTN is

A

elevation in the normal pulmonary vasculature leading to increased RV pressure

23
Q

Treatment of Pulm HTN

A

inhaled agents - nitro

24
Q

EKG findings in a PE

A

S1 Q3 T3

25
Q

PE can result in

A

Pulmonary HTN

26
Q

Classifications of Pulm HTN

A

group 1
group 2
group 3
group 4
group 5

27
Q

PULM HTN - Group 1

A

idiopathic, familial, drug/toxin, connective tissue disease, congenital (Primary)

28
Q

PULM HTN - Group 2

A

secondary cause of LV dysfunction - pulmonary venous hypertension

29
Q

PULM HTN - Group 3

A

chronic lung diseases

30
Q

PULM HTN - Group 4

A

PE

31
Q

PULM HTN - Group 5

A

IDK

32
Q

Cor Pulmonale is

A

downstream cardiac effect of PE and Pulm HTN that leads to hypertrophy and dilation of the RV

33
Q

Causes of pulmonary vasoconstriction

A

chronic hypoxemia
acidosis

34
Q

Cor pulmonale presentation mirrors

A

right sided heart failure