Clin Med: Pulmonary Vascular Disorders Flashcards

(34 cards)

1
Q

Pulmonary Circulation route

A

RV
Pulmonary trunk
L and R Pulmonary arteries
re-enter via pulmonary veins into the LA

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2
Q

Pulmonary trunk is how big

A

5 cm

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3
Q

main branches of the pulm arteries are about ______ of the aortas thickness

A

1/3

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4
Q

Hypoxia will trigger

A

vasoconstriction

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5
Q

Vasoconstriction in the lungs will lead to

A

shunting of blood to the more aerated areas within the lung —> so other areas get more blood

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6
Q

If the entire lung is vasoconstricted this can lead to

A

pulm HTN

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7
Q

Pulmonary lymphatics remove

A

both infectious material and excess fluid

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8
Q

Pulmonary lymphatics drain into

A

the right thoracic lymphatic duct

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9
Q

Pulmonary embolism is

A

a blood clot within the pulmonary vasculature

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10
Q

Causes of PE’s

A

DVT
fat embolism
air embolism
amniotic embolism

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11
Q

Treatment for PE

A

provoked - 3 month anticoag
unprovoked - forever anticoag
tPA (severe sx)

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12
Q

Fat embolism occurs with injury to

A

long bones - femur fracture

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13
Q

Septic emboli most commonly associated with

A

IVDU and s. aureus - will affect right side of the heart

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14
Q

Air embolism is most commonly due to

A

scuba diving
iatrogenic

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15
Q

In an air embolism bubbles enter

A

the pulmonary circulation and lodge in the same fashion a clot would

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16
Q

On physical exam what would you find with an air embolism

17
Q

Amniotic embolism is when

A

amniotic fluid is able to get into Mom’s circulation
m/c occurs during lab or immediate postpartum

18
Q

Amniotic embolism tx

A

treat like it is a clot - anticoag, tPA if not stable
watch for bleeding to death since she just gave birth and will be on anticoags - have OB/GYN with you to counteract these

19
Q

PE presentation

A

pleuritic chest pain (breathing pain)
SOB, hypoxia
syncope
shock (right sided HF)
findings of DVT
cough
hemoptysis
tachycardia

20
Q

Classifications of PEs

A

Massive - hemodynamically unstable
Submassive - without right sided heart strain

21
Q

Complications of a PE

A

right sided heart failure
decreased gas exchange - lack of profusion leading to lung ischemia

22
Q

Pulmonary HTN is

A

elevation in the normal pulmonary vasculature leading to increased RV pressure

23
Q

Treatment of Pulm HTN

A

inhaled agents - nitro

24
Q

EKG findings in a PE

25
PE can result in
Pulmonary HTN
26
Classifications of Pulm HTN
group 1 group 2 group 3 group 4 group 5
27
PULM HTN - Group 1
idiopathic, familial, drug/toxin, connective tissue disease, congenital (Primary)
28
PULM HTN - Group 2
secondary cause of LV dysfunction - pulmonary venous hypertension
29
PULM HTN - Group 3
chronic lung diseases
30
PULM HTN - Group 4
PE
31
PULM HTN - Group 5
IDK
32
Cor Pulmonale is
downstream cardiac effect of PE and Pulm HTN that leads to hypertrophy and dilation of the RV
33
Causes of pulmonary vasoconstriction
chronic hypoxemia acidosis
34
Cor pulmonale presentation mirrors
right sided heart failure