CV TEST ONE: HYPERTENSION Flashcards

(46 cards)

1
Q

Hydrochlorothiazide

A

Diuretic HTN Med, potassium wasting (no potassium) potassium rich diet suggested

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2
Q

Peripheral resistance

A

Arteries’ resistance to blood flow, arteries constrict=increased resistance , arteries dilate= decreased resistance

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3
Q

Modifiable/non modifiable HTN factors

A

•Modifiable—diet, lifestyle, exercise
•non modifiable—race, gender, heredity, age, pre existing conditions

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4
Q

BP categories

A

*normal–less than 120/80 mmHg
*elevated–120-129/<80 mmHg
*stage 1–130-139/80-89 mmHg, poss. Med. intervention, lifestyle change
*stage 2–BP < 140/< or = 90 mmHg, meds (ANTIHYPERTENSIVES) lifestyle change
*urgent–180/120 or <, immediate reduction of BO to prevent organ damage, PT resting while 911 contacted

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5
Q

HTN diagnosis includes:

A

Confirmed >2 occasions of hypertensive measurements, risk factors, presence/absence of symptoms, history of heart/kidney disease, current meds used

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6
Q

HTN S/S

A

Headache, bloody nose, anxiety, SOB

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7
Q

Cardiac catheterization

A

•Studies heart’s anatomy/physiology, assesses pressure in coronary arteries/chambers/great vessels, assesses C.O./O2 saturation
•dye injected into catheter>inserted into femoral vein

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8
Q

Pre & post cardiac catheter

A

•pre– consent, assess for iodine/dye allergies, NPO/conscious sedation
•post–monitor for bleeding, pressure device at site, bedrest, no flexion of extremity for hrs, V.S. Taken, peripheral pulses taken

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9
Q

PTs radial pulse is different than their apical. What should nurse proceed to do and why

A

Report pulse deficit to doctor, could indicate arrhythmia/cardiac dysfunction

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10
Q

Trans esophageal Echocardiogram (TEE Test)

A

•Clearer picture, transducer probe in esophagus (doesn’t cross lung/rib tissue), sedation/local anesthetic in oropharynx, NPO 6 hrs pre-test
•complications—esophageal perforation, throat bleeding, hypoxia, dysthymias

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11
Q

Pre/Post TEE

A

•Pre TEE—assess allergy hx/sedation reactions/local & general anesthesia
•post TEE–asses VS, monitor bleeding, MONITOR GAG REFLEX!, keep suction & resuscitation equipment available

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12
Q

Pericardial rub

A

Heard in inflammatory processes (pericarditis), sounds like rubbing tissue papers, can be heard closely if PT sitting up & leaning forward

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13
Q

S3 & S4 sounds

A

•S3—ventricular gallop or LUBB DUBB DUBB
•S4— atrial gallop or LUBB LUBB DUBB

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14
Q

3 membranes of heart

A

•fibrous pericardium– outermost layer, forms sac around heart
•parietal pericardium–middle layer
•visceral pericardium aka epicardium– innermost layer

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15
Q

Where does blood start at in the heart ?

A

Superior and inferior vena cava

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16
Q

If a PT is struggling financially to pay for care costs they should be redirected to:

A

The financial dept within the clinical setting

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17
Q

The left ventricle pumps __x times more force because _____

A

5, accommodate systemic circulation

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18
Q

Cardiac pathway

A

AV node>SA node>Bundle of His>purkinje

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19
Q

SA node

A

Pacemaker of the heart, where cardiac impulse is first carried out, heartbeat initiation, located in R.A.’s wall, gives normal rhythm

20
Q

Cardiac cycle

A

One heartbeat, 0.8 seconds

21
Q

During cardiac cycle, both atrias and ventricles contract and relax, known as

A

•systole–contraction(emptying occurs)
•diastole–relaxation phase

22
Q

Cardiac output

A

•amount of blood ejected from left ventricle in a minute
•calculation –stoke volume X heart rate

23
Q

Too much or too little potassium can cause

24
Q

ANP (Atrial natriuetic) is secreted in response to

A

Vessels stretching from increased volume

25
Uretic
Sodium and water loss, BP increased
26
If there’s a different reading on the PTs arms go with
The higher reading for ongoing use
27
Complications of HTN
CAD, atherosclerosis, M.I., H.F., stroke, kidney damage, eye damage, L.V. hypertrophy
28
Meds to lower Bp (diuretics)
•K-sparing: Spironolactone (Aldactone) •k-wasting: thiazide (hydrochlorothiazide) (chlorothiazide) •Loop: furosemide (lasix), torsemide (demadex) •oldest most studied antiHTN med •PTs should swing legs on bed’s edge for some minute before standing to avoid syncope
29
most common heart disease manifestation
chest pain (angina)
30
Dyspnea may be a sign of
LV failure or transient CHF
31
Heart sounds produced by closing valves start at
the aortic
32
murmur
turbulent blood flow through heart/vessels, prolonged sound>narrowed valve closing
33
cardiac enzyme elevated values
CPK and troponin
34
C.O., blood viscosity, blood volume, PVR
factors determining BP
35
What is HTN? what could it cause?
Increase in blood pressure against vessel walls... coronary heart disease, CHF, stroke, M.I., eye damage, LV hypertrophy
36
can a clot be caused by HTN
Yes due to blood being forced thru narrow vessels
37
Increased C.O., PVR, blood viscosity, blood volume, hormone imbalances, kidney dysfunction
pathophysiologic changes of HTN
38
Lifestyle mods. should be done ______ drug therapy unless PT is at high risk
before....
39
PTs at risk for urgent HTN
untreated HTN, non compliant with med, using nitroprusside as its a vasodilation
40
beta 1
beta 1 antagonist: iontropic, chronotropic effects beta 1 blocker: decreased contractility, HR, decreased C.O.
41
BETA 2
agonist: bronchodilation blocker: bronchoconstriction
42
alpha 1
agonist: vasoconstriction blocker: vasodilation
43
secreted by adrenal medulla, sympathomimetic response
epinephrine
44
produced by adrenal cortex, regulates Na & K levels
aldosterone
45
ANP
secreted by atria, increases Na excretion
46