Dominance Related Issues Flashcards Preview

Adult Language Disorders > Dominance Related Issues > Flashcards

Flashcards in Dominance Related Issues Deck (44):

Cerebral Asymmetry

Biologic landmark of the human cerebral evolution
asymmetrical control for language


Equivalent lesion in right hemisphere

No aphasia


Visual-spatial deficits

RH medial functions


Hypotheses of cerebral dominance

1. Equipotentiality: both sides of the brain are equally as capable of serving language
2. LH dominance --> diluting effects of RH dominance (left brain becomes more dominant for language around age 5)
3. RH for nonlinguistic functions: right hemisphere takes the lead on nonlinguistic functions previously served by LH
4. LH damage = interruption of nonlinguistic behaviors
5. Intact RH = possible compensatory source in recovery


Implicating Theories: The Coarse Coding Hypothesis

Differences: ways of access to semantic representations & ways semantic representations are suppressed


Implicating Theories: The LH semantic coding

activating small fields, inhibiting all but central features (brings precision)


Implicating Theories: RH Coding

Activation of large semantic fields (brings redundancy -- too much info)


Implicating Theories: Suppression Deficit Hypothesis

Reconciliation of multiple meaning hampered due to impaired suppressive strategy (activation of multiple representations for words)


Implicating Theories: Cognitive Resource Hypothesis

Attentional allocation and task demand
RD patients falter on high concentration tasks


Methods to study dominance

lesion-to-stroke data, dichotic listening, tachistoscopic data, commissurotomy


Visual-Tactile association

right hemisphere can match and identify objects through touch


RH recognition, but no naming

patient can read the word "nut" and he will pick up a nut, but cannot say the word


Left Brain

right visual field, left olfaction, speech, writing, main language center, calculation


Right Brain

Right olfaction, spatial abilities, simple language comprehension, nonverbal ideation, left visual field


RH Cog-Communication Profile
(characteristics if someone has a lesion in the right brain)

Disorders of contextually-based communication from disturbed underlying cognitive, perceptual, and pragmatic processes
1. copious output with poor communication
2. superficial treatment of information/question
3. breakdown on communicative demands
4. inappropriate verbal output on careful scrutiny
5. often confabulatory speech
6. literal interpretation of question
7. irrelevant/excessive details


RH communication profile (if lesioned)

1. Reduced emotions, melodies, pitch contours, creative language functions
2. No organized information, isolation of salient points, or integration of crucial information
3. Addresses to but no answer
4. Elusive nature of the deficit


Nonlanguage functions of the right brain

1. Visual functions: recognition of visual stimuli, image generation, mental rotation, visual-spatial attributes
2. Block designs
3. Tactile-Visual orientation
4. Reproduction (copying, memorization of graphic stimuli, Figure of Rey)
5. Visual-Spatial Orientation (spatial object/shape placement, location relationship, map reading, reading/writing deficits --> no space, spelling errors, additional strokes, extra capitalization)
6. Orientation to time
7. Musical Processing (Naive -RH-vs. Experienced -LH- subjects)
8. Attention
9. Visual Inattention/neglect
10. Affection/Emotion
11. Pragmatics


Non-language functions of RH: Musical Processing

Naive subjects vs. Experienced Subjects
1. Naive: RH for processing & identification, pitch & melody, dichotic listening, familiar melodies
2. Experienced: analytic approach, LH for expression and information processing


Non-language functions of RH: Attention

RH: Neural circuitry for both extra-personal spaces (why there is only left neglect with RH lesion, but never right neglect)
LH: Circuitry for contralateral space (still can access other side because right hemisphere will cover it)


Divided attention

Executive control


Selective attention



Non-language functions of RH: Visual attention/neglect

unilateral spatial neglect, not interested in events on one side, ignorance of people on affected side, fail to carry out basic self-care tasks, dramatic recover - 85% within 3 days


Affection/Emotion representation in both hemispheres

RH association with experience and expression of negative emotions. Lesion diminishes negative affection (inappropriate jocularity and lack of concerns)

LH association with positive emotions. Lesion = display of catastrophic reactions (anxiety, tears) to failures


Right hemisphere lesion --> prosody, stress, intonation

flat in tone, lack of facial expression/gestures, apathy indifferent reaction, impaired sensitivity to moods in pictures


RH lesion Pragmatic Functions

Insensitive to turn taking, eye contact, context sensitive language, gestures-coverbal properties


Paralinguistic Tasks

Jokes, Metaphors, Idioms


Eliott Ross' Work: looked at prosody of a variety of patients

Inadequate consideration of paralinguistic functions
Prosody: intonation, melody, pauses, stress, accents, attitudes

Dyprosody: Parkinson's
Hyperprosody: Manic patients
Hypoprosody: Broca's aphasics



study of limb, body and facial movements with nonverbal communication



referential purposes (LH property)
motions with meaning



non referential - enhance communicative content (don't carry, but add meaning)


Goodglass and Kaplan ideas regarding Kinesics

1. pantomime decoding disorders in aphasics with incomprehension is due to general inability to comprehend symbols
2. difficulty in the execution of pantomimes in aphasics with no comprehension disorders
3. due to idea-motor apraxia


Diagnosis of RH lesion/damage

No RH sensitive task
•No literal language deficit
•True deficit -on complex tasks
•Testing for: Meaningful organization, Impulsive responses, Trivial details, Differentiation between imp/trivia inf., Assimilation of contextual cues, Depersonalization of events, Literal interpretations to figurative language, Sensitivity pragmatic/context aspects


Components of Evaluations

1. Spontaneous use of affection
2. Prosodic Content: expression of affective prosody, comprehension of affective prosody
3. Visual comprehension of gestural components
4. Nonverbal reasoning

Ask: What makes a joke funny, what example similes/metaphors mean, to read sentences while looking for prosody, ask for the meaning of sentences based on the stress patterns, ask to read sentences with suitable emotion


Aphasia in Deaf Signers

sign language & verbal language are two DIFFERENT modes of communication

Interplay between: linguistic and spatial/visual functions (different hemispheric specialization for each process)


Linguistic Nature of ASL

1. Hand configurations, facial expressions, and shifting of body positions
2. Phonological Code (obeys the physical constraints on motor gestures related with word formation)
3. Internal morphology (Packs many morphologically salient units into a single lexical item tied to language modality)
4. Complex Syntax (spatially organized)
5. Additional attributes (Classifier, verbs, motion, and distinctive location of signs)


Cerebral Dominance relating to static and dynamic stimuli

RH dominance for static signs (LH advantage in hearing subjects)

Changing pattern of cerebral dominance on computer generated moving signs (shift from the RH)


RH lesions in deaf signers

Signers with RH lesion
1. no aphasia
2. fluent, grammatical, error free signing
3. impaired processing of non-language spatial tasks (drawing, lock designs, attention to visual space, line orientation, facial recognition, and visual closures)


RH in signers

Dominance for non-language visual-spatial functions


Aphasia in signers with LH lesion

3 case studies
PD (Wernicke's aphasia)
1. fluent-type sign aphasia
2. long strings of
motorically facile signs
3. exhibited impaired morphology and syntax
4. preserved phonology and lexical sign properties

KL (Wenicke's aphasia)
1. long strings of signs effortlessly
2. wide range of syntactic morphological signs
3. phonological impairments making frequent errors in selection of correct hand configuration, place and movement

1. grossly impaired sign output
2. nonfluent signing reduced to single sign utterances
3. largely referential signs without any of the required grammatical and syntactic markings



accompanying and/or replacing speech



deliberate use of body/manual movements to convey a message


Coverbal or Paraverbal

Behaviors accompanying speech: facial expressions, gestures


Aphasia (pantomime)

impairment of pantomimic expressions and recognition


Paraverbal behavior in aphasia

gestures/facial expressions: generally spared in aphasia
*usually with RH lesion