Equine Flashcards

(51 cards)

1
Q

population

A

US (10.2m), china, mexico, brazil, argentina, colombia, mongolia
150,000 in MT

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

earliest horse

A

sifrhippus (genus)
over 55 million years ago
about the size of a house cat

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

modern horse

A

equus
reduction in the number of digits- variation from four toes on the ground
adaption and change to different food- advanced molars for grasses and hays
increase in size
increase in size and complexity of the brain

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

reasons for evolution

A

changes in environment
wet, woody habitats–> grassy prairies

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

stay apparatus

A

a passive, inherent, force resisting system that supports the weight of the body, enables energy efficient locomotion and allows a horse to stand and rest while little energy exertion
tendons and ligmaments

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

history

A

Columbus brought horses to the West Indies in 1493, reintroduced by Cortez in 1519 and de Soto in 1539 (mexico and US), native americans spread the horse across the western plains through 1750, “wild horse” adapted and flourished through 1850, horse used for mostly draft 1890-1920, 1920-1960 horse numbers declined due to automotives and locomotives
increasing from 1980s - recreational horse

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

leading states

A

texas, california, florida, oklahoma

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

horses in the world

A

60 million

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

gene G

A

causes horses to be gray

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

gene E

A

ee: red
E_: black

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

gene A (agouti)

A

determines distribution of black

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

gene W

A

makes horse unable to form pigment in skin and hair, albino
WW- fatal

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

gene C

A

cream dilution gene
CC- color remains same
Cc- incompletely dominant- red pigment diluted to yellow–palomino

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

gene D

A

produces dun coloring

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

gene CH

A

champagne gene, metalic sheen

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

gene Z

A

silver gene

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

gene RN

A

controls roan

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

gene LP

A

controls whether or not a horse has the complex of spotting
spotting- white superimposed on another color

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

gene F

A

F_: chestnut
ff: flaxen mane on chestnut

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
19
Q

draft horse

A

1400lbs or more
14.2-17.2
“cold blooded”
strength, patient, docile
Shire, Belgian, Percheron, Clydesdale

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
20
Q

clydesdale

A

draft
originated from Scotland
founded 1877
bay, brown, black, roan

21
Q

belgian

A

most popular draft breed
16.2-17hh, 2000lbs
brabant or belgian heavy draft
many colors

22
Q

arabian

A

light breed
oldest purebreed of horse
originated on arabian peninsula
no palomino, dun, or buckskin
characteristically dished face
speed, courage, endurance

23
Q

thoroughbred

A

light breed
originated in england
greatest single influence on the world horse population
high spirited and brave
original sires: Byerly Turk, Godolphin Arabian, and

24
morgan
light breed one common foundation sire -Justin Morgan developed in northeast, 1894
25
american saddlebred
light breed exaggerated gaits
26
tennessee walking horse
light breed flat walk and running walk
27
appaloosa
light breed developed from spanish horses by Nez Perce tribe mid 1700
28
american quarter horse
light most versatile breed largest breed registry 3.1m worldwide AQHA 1940 Wimpy P1 was first AQHA registered quarter horse
29
ponies
less than 14.2hh shetland, welsh, dartmoor can be draft, heavy harness, or saddle type
30
birthdates
northern hemisphere: january 1 southern hemisphere: august 1
31
why mules and hinny are sterile
horses and donkeys have different chromosomes
32
mule
jack x mare
33
hinny
stallion x jenny
34
mare
sexual maturity 18 months average gestation length 334 days (11 months) can become pregnant 1-2 weeks after foaling seasonally polyestrous, stop cycling in winter
35
stallion
colts reach sexual maturity at 12-18 months fertile year round but decreased sperm production september-february
36
seasonal polyestrous
long day breeders melatonin on pineal gland (affected by light) roughly april/may until september/october signs of estrous: winking of the vulva, urination, squatting, seeking the stallion
37
horse digestion
complex hindgut (cecum), require high-quality forage, unable to use microbial cell protein, good fat digestion, methane production is minimal
38
incisors
selection and prehension
39
molars
chewing and grinding; particle size reduction
40
hipsadonic teeth
continuously growing teeth
41
equine jaw
upper jaw is wider than the lower jaw elliptical chewing pattern -molars wear irregularly and can become sharp -teeth must be periodically floated
42
colic
characterized by severe abdominal pains symptoms: loss of appetite, grinding teeth, stretching posture, flank watching, uneasiness and rolling, sweating, increased heart rate (>60bpm), increased respiration rate physiology: intestinal distention (gas or ingeseta) stretching of mesenteric support inflammation of intestine causes: intestinal spasm- rapid diet change, endotoxins, obesity constipation- inadequate forage, dehydration, ingestion of sand or soil
43
laminitis
inflammation of hood laminae resulting in tissue damage symptoms: lameness, abnormal hoof growth, irregular gait physiology: chronic laminitis leads to founder, sensitive and insensitive laminae of the hoof wall separate, the bond between the sensitive and insensitive laminae fails, the attachment of the coffin bone to the hoof breaks down causes: over-consumption of grain or lush pasture, excessive intake of cold water by over-heated horses, obesity
44
recurrent airway obstruction (heaves)
similar to asthma in humans, lung aveoli become fibrous and eventually rupture symptoms: shortness of breath, increased respiration rate, chronic coughing, presence of a heave line on the flank due to exertion of abdominal muscles, nasal discharge physiology: irritants in the trachea and lungs causes hyper-secretion of mucous and thickening of the airways, lung alveoli become scarred and brittle, increasing the chance of rupture causes: allergic reactions to fungal spores in feed, bedding, and the environment, dusty or moldy hay, dusty environments
45
forages
daily forage consumption should be between 1-2% of body weight (DM basis)
46
grain
must be stored properly to prevent mold, do not feed in excess of 0.75% of body weight at a single feeding, feed at the same time each day, avoid abrupt changes in the grain ration
47
basic feeding requirements
NRC maximize forage- pasture and hay feed concentrate to make-up difference between nutrients needed and nutrients in roughage
48
weight and body condition
maintenance requires an intake of 2-3% BW body condition refers to the amount of fat on a horse's body fat is energy storage and is produced when the horse is digesting more energy than is needed for maintenance and production
49
body weight measurements
simplest and most accurate method: weight scale, weight can fluctuate-time of day weighed, feeding interval, defecation weight tapes: variables-conformation and proportion
50
body condition
1. poor 2. very thin 3. thin 4. moderately thin 5. moderate 6. moderate to fleshy 7. fleshy 8. fat 9. extremely fat