flashcards from articles

1
Q
A
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2
Q

What is the neuromatrix theory of pain?

A

A theory that pain is generated by a ‘neurosignature’ within a brain network (body-self neuromatrix), not just from injury.

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3
Q

What is a neurosignature?

A

A pattern of neural activity that produces the pain experience, influenced but not caused by sensory input.

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4
Q

What are the main brain structures involved in the neuromatrix?

A

Thalamus, cortex, and limbic system.

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5
Q

How does the neuromatrix explain phantom limb pain?

A

It shows that pain can be felt without any physical input, as it is generated by the brain.

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6
Q

What role does stress play in chronic pain according to Melzack?

A

Stress activates hormonal responses (e.g., cortisol) that can damage tissues and contribute to pain.

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7
Q

What are the four key conclusions from the neuromatrix model?

A

1) Body is felt due to brain processes; 2) Pain can exist without input; 3) The self is unified; 4) Brain patterns are genetically programmed.

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8
Q

What are the three stages of tendinopathy in the continuum model?

A

Reactive tendinopathy, tendon dysrepair, and degenerative tendinopathy.

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9
Q

What happens in reactive tendinopathy?

A

Swelling due to acute overload, with increased proteoglycans and water—reversible with load management.

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10
Q

What defines the degenerative stage of tendinopathy?

A

Irreversible changes like matrix disorganization and cell death, often without pain.

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11
Q

Why might tendon structure and pain not align?

A

Many people show structural changes without pain; pain is influenced by load-related signaling.

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12
Q

What is reactive-on-degenerative tendinopathy?

A

A new reactive flare-up on an existing degenerative tendon.

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13
Q

What does the quote ‘treat the doughnut, not the hole’ mean in tendinopathy?

A

Focus on strengthening the healthy part of the tendon, not the degenerated portion.

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14
Q

What is the key idea of Bialosky’s updated MT model?

A

MT relieves pain through neurophysiological responses, not just mechanical correction.

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15
Q

What are the three zones in the MT mechanism model?

A

Zone 1: Provider-patient interaction; Zone 2: Nervous system response; Zone 3: Clinical outcomes.

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16
Q

How can MT alter pain processing in the nervous system?

A

Via peripheral, spinal, and supraspinal effects, including fMRI evidence of reduced brain activation in pain regions.

17
Q

What are two psychophysical tools used to assess pain modulation?

A

Conditioned Pain Modulation (CPM) and Temporal Summation.

18
Q

What makes MT effects patient-specific?

A

Patient beliefs, expectations, and provider interaction all influence outcomes.

19
Q

Why might people resist identifying as ‘patients’?

A

It can feel disempowering and reduce their perceived value in care settings.

20
Q

What is the significance of the ‘8760 hours’?

A

Most people manage their condition themselves outside the few hours spent with a provider.

21
Q

How can providers better engage patients?

A

By asking about their life, validating their efforts, acknowledging loss of control, and practicing empathy.

22
Q

What is a key critique of patient-centered care in this article?

A

Systems often focus more on procedures than truly seeing and respecting the person.

23
Q

How should exercise for OA be tailored?

A

To the patient’s needs and preferences.

24
Q

When is aquatic exercise recommended for OA?

A

If land-based exercises are too painful.

25
How long should supervised therapy for OA last initially?
At least 6 weeks; some may need 12 weeks.
26
What is the role of 'booster' sessions for OA?
To support long-term OA pain and health management after initial therapy.
27
Why are home exercises important for OA?
They optimize long-term outcomes.
28
What should patients understand about exercise and pain flare-ups?
How to manage and modify exercises during flare-ups.