headache Flashcards

(47 cards)

1
Q

are most headaches primary or secondary?

A

primary

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2
Q

what would be types of secondary headaches?

A
Tumour
Meningitis
Vascular disorders
Systemic infection
Head injury
Drug-induced
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3
Q

what would be types of primary headaches?

A

Tension Type Headache
Migraine
Cluster Headache

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4
Q

what is tension type headache?

A

Mild, bilateral headache which is often pressing or tightening in quality, has no significant associated features and is not aggravated by routine physical activity

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5
Q

what are tension type headache treatments?

A

Aspirin or paracetamol
NSAIDs
Limit to 10 days per month (~2 days per week) to avoid the development of medication overuse headache

Preventative treatment
Rarely required
Tricyclic antidepressants
amitriptyline, dothiepin, nortriptyline

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6
Q

what is the most frequent dsiabling primary headache?

A

migraine

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7
Q

what is a migraine?

A

A chronic disorder with episodic attacks

Complex changes in the brain

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8
Q

what happens during migraine attacks?

A

Headache
Nausea, photophobia, phonophobia
Functional disability

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9
Q

what happens in between migraine attacks?

A

Enduring predisposition to future attacks

Anticipatory anxiety

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10
Q

what are migraine triggers?

A
dehydration
diet
environmental stimuli
changes in oestrogen level in women
sleep disturbance
hunger
stress
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11
Q

what percentage of people does aura affect?

A

33%

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12
Q

what is aura?

A

Transient neurological symptoms resulting from cortical or brainstem dysfunction

May involve visual, sensory, motor or speech systems

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13
Q

how long does aura occur for?

A

15-60 minutes

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14
Q

how would you define a chronic migraine?

A

Headache on ≥ 15 days per month, of which ≥ 8 days have to be migraine, for more than 3 months

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15
Q

how does medication overuse in headaches occur?

A

Headache present on ≥15 days / month which has developed or worsened whilst taking regular symptomatic medication

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16
Q

what is the treatment for migraines?

A

Abortive treatment
Aspirin or NSAIDs
Triptans
Limit to 10 days per month (~2 days per week) to avoid the development of medication overuse headache

Prophylactic treatment
Propranolol, Candesartan
Anti-epileptics
Topiramate, Valproate, Gabapentin
Tricyclic antidepressants
amitriptyline, dothiepin, nortriptyline 
Venlafaxine
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17
Q

when are you likely to get your first migraine as a woman?

A

during pregnancy

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18
Q

what treatment would you use for someone that is haveing an acute attack of migraines but is pregnant?

A

paraceamol

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19
Q

what preventatives would you give a pregnant person with migraines

A

Propranolol or Amitriptyline

20
Q

what are Trigeminal Autonomic Cephalalgias

A

group of headache disorders characterised by attacks of moderate to severe unilateral pain in the head or face, with associated ipsilateral cranial autonomic features such as lacrimation, conjunctival injection, rhinorrhoea, nasal congestion, eyelid oedema and ptosis.

21
Q

where would the pain most likely be due to a cluster headache?

A

mainly orbital and temporal

22
Q

how long would a cluster headache be?

A

15 mins to 3 hours

23
Q

what are the different types of Trigeminal Autonomic Cephalalgias

A

Paroxysmal Hemicrania
SUNCT
cluster headache

24
Q

which of the TAC’s attack the most frequent

A

Short-lasting unilateral neuralgiform headache with conjunctival injection and tearing
(SUNCT)

25
which os the TAC's last the longest?
cluster headache
26
which of the TAC's have a burning sensation?
SUNCT
27
where would the pain be due to paroxysmal hemicrania?
mainly orbital and temporal
28
how long would a paroxysmal hemicrania attack last?
2-30 mins
29
what are cutaneous triggers for SUNCT?
Wind , cold Touch Chewing
30
what type of a pain is trigeminal neuralgia?
stabbing
31
how long does trigeminal neuralgia last?
5-10 seconds
32
what are the treatments for cluster headache?
Abortive (Headache) Subcutaneous sumatriptan 6mg or nasal zolmatriptan 5mg 100% oxygen 7-12 l/min via a tight fitting non-rebreathing max is effective and safe Abortive (Headache bout) Occipital depomedrone injection (same side as the headache) Or tapering course of oral prednisone ``` Preventative Verapamil (high doses may be required) Lithium Methysergide (risk of retroperitoneal fibrosis) Topiramate ```
33
what are the treatments for paroxysmal hemicrania?
No abortive treatment Prophylaxis with indometacin Alternatives – COX-II inhibitors, Topiramate
34
what are the treatments for SUNCT?
No abortive treatment ``` Prophylaxis: Lamotrigine Topiramate Gabapentin Carbamazepine / Oxcarbazepine ```
35
what are the treatments for trigeminal neuralgia?
No abortive treatment Prophylaxis: Carbamazepine Oxcarbazepine Surgical intervention: Glycerol ganglion injection Steriotactic radiosurgery Decompressive surgery
36
what are presentations of a secondary headache?
``` Associated head trauma First or worst Sudden (thunderclap) onset New daily persistent headache Change in headache pattern or type Returning patient ```
37
what are red flags for a secondary headache?
new onset headache new or change in headache aged over 50 Immunosupression or cancer change in headache frequency, characteristics or associated symptoms focal neurological symptoms non-focal neurological symptoms abnormal neurological examination
38
what is a thunderclap headache?
A high intensity headache reaching maximum intensity in less than 1 minute Majority peak instantaneously
39
what are differential diagnosis for thunderclap headaches?
``` Primary (migraine, primary thunderclap headache, primary exertional headache, primary headache associated with sexual activity) Subarachnoid haemorrhage Intracerebral haemorrhage TIA / stroke Carotid / vertebral dissection Cerebral venous sinus thrombosis Meningitis / encephalitis Pituitary apoplexy Spontaneous intracranial hypotension ```
40
who is likely to get a subarachnoid haemorrhage?
``` All patients presenting with a sudden severe headache that peaks within a few minutes and lasts for at least 1 hour Examination is often normal! Never consider a patient ‘too well’ for SAH ```
41
what are features suggestive of a space occupying lesion or raised intracranial pressure?
Progressive headache with associated symptoms and signs Headache worse in morning or wakes patient from sleep Headache worse lying flat or brought on by valsalva (cough, stooping, straining) Focal symptoms or signs Non-focal symptoms e.g. cognitive or personality change, drowsiness Seizures Visual obscurations and pulsatile tinnitus
42
what is intracranial hypotension
a condition in which there is negative pressure within the brain cavity
43
causes of intracranial hypotension?
Dural CSF leak
44
investigations for intracranial hypotension
MRI brain and spine
45
treatment of intracranial hypotension
Bed rest, fluids, analgesia, caffeine (e.g. 1 can red bull qds) i.v. caffeine Epidural blood patch
46
when would you consider someone with arteritis of large arteries?
in any patient over the age of 50 years presenting with new headache
47
what are specific features of giant cell arteritis?
include scalp tenderness, jaw claudication and visual disturbance Prominent, beaded or enlarged temporal arteries may be present An elevated ESR supports the diagnosis (usually >50, often much higher, rarely normal) Raised CRP and platelet count are other useful markers