knee lig sprains CPG 2017 Flashcards

(60 cards)

1
Q

Q: What is the primary classification label for patients with knee ligament sprains in these guidelines?

A

A: Knee stability and movement coordination impairments.

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2
Q

Q: What key clinical findings support the classification of an acute ACL sprain?

A

A: Hearing or feeling a “pop,” immediate swelling, giving way, and a positive Lachman test.

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3
Q

Q: How is a grade III MCL sprain commonly identified?

A

A: Excessive gapping with valgus stress at 30° knee flexion and localized medial pain/swelling.

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4
Q

Q: What combination of tests is most accurate for diagnosing an ACL injury?

A

A: Lachman, anterior drawer, and pivot shift tests.

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5
Q

Q: What clinical presentation is typical for posterior cruciate ligament (PCL) sprains?

A

A: Posterior knee pain, a positive posterior drawer test, and a “sag sign” on inspection.

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6
Q

Q: What mechanism of injury often leads to ACL tears?

A

A: Non-contact deceleration, cutting, or pivoting maneuvers.

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7
Q

Q: What timeframe defines the acute phase of a ligament injury?

A

A: Within 6 weeks of the initial trauma.

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8
Q

Q: How are multi-ligament injuries typically identified?

A

A: Signs of instability in more than one direction, often following high-energy trauma.

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9
Q

Q: What key finding suggests a chronic ACL injury with movement coordination deficits?

A

A: Complaints of knee instability or “giving way” during sport or activity.

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10
Q

Q: How is knee hypermobility incorporated into the classification?

A

A: Considered when laxity exists without pain or trauma, distinguishing from ligament sprains.

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11
Q

Q: How can a meniscal tear be differentiated from an MCL sprain?

A

A: Meniscal tears often present with joint line tenderness, locking, or clicking, while MCL sprains show medial pain with valgus stress and no mechanical symptoms.

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12
Q

Q: What finding helps distinguish a PCL injury from an ACL tear?

A

A: A positive posterior drawer test and posterior sag sign suggest PCL injury, while a Lachman or pivot shift test indicates ACL involvement.

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13
Q

Q: How does patellar subluxation differ from ACL injury?

A

A: Patellar subluxation involves lateral displacement and apprehension with lateral glide, not the instability and “pop” typical of ACL tears.

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14
Q

Q: What symptom pattern suggests a tibial plateau fracture rather than a ligament sprain?

A

A: Acute swelling with inability to bear weight and bony tenderness may indicate fracture, confirmed by imaging.

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15
Q

Q: How can clinicians differentiate pes anserine bursitis from MCL injury?

A

A: Pes anserine bursitis presents with tenderness distal to the joint line, not directly over the MCL.

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16
Q

Q: What distinguishes a quadriceps tendon rupture from a ligament sprain?

A

A: Inability to extend the knee actively and a palpable defect above the patella suggest quadriceps rupture.

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17
Q

Q: How does septic arthritis differ from ligamentous injury?

A

A: Septic arthritis presents with fever, joint warmth, and severe pain with passive motion—urgent referral is needed.

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18
Q

Q: What clinical sign helps rule out patellar tendon rupture?

A

A: A high-riding patella (patella alta) and inability to perform a straight leg raise suggest tendon rupture.

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19
Q

Q: How can referred hip pathology mimic knee ligament injury?

A

A: Hip conditions may cause anterior knee pain without local knee findings—hip ROM testing helps differentiate.

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20
Q

Q: What distinguishes a chronic ACL-deficient knee from other causes of instability?

A

A: Recurrent giving way during cutting or pivoting activities without acute trauma suggests chronic ACL insufficiency.

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21
Q

Q: How does shoe-surface interaction influence knee ligament injury risk?

A

A: High friction between footwear and playing surface can increase torsional forces, raising injury risk.

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22
Q

Q: What anatomical feature is associated with increased ACL injury risk?

A

A: A narrow femoral notch width may predispose individuals to ACL tears.

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23
Q

Q: How does increased joint laxity affect ligament injury risk?

A

A: Greater laxity can reduce passive stability, making ligaments more vulnerable to sprain.

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24
Q

Q: What hormonal factor may influence ligament injury risk in females?

A

A: The pre-ovulatory (follicular) phase of the menstrual cycle may increase ligament laxity.

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25
Q: How does BMI relate to knee ligament injury risk?
A: Higher BMI increases joint loading and may elevate injury risk.
26
Q: What neuromuscular pattern is linked to ACL injury risk?
A: Strong quadriceps activation during eccentric loading with limited knee flexion can increase strain on the ACL.
27
Q: How does poor neuromuscular control contribute to injury risk?
A: Faulty movement patterns like valgus collapse during landing or cutting increase ligament stress.
28
Q: What role does previous injury play in ligament sprain risk?
A: A history of knee ligament injury significantly increases the risk of future sprains.
29
Q: How does sport type influence ligament injury risk?
A: Cutting and pivoting sports (e.g., soccer, basketball) carry higher risk due to rapid directional changes.
30
Q: Why is fatigue considered a risk factor for ligament injury?
A: Fatigue impairs neuromuscular control, increasing the likelihood of poor landing mechanics and instability.
31
Q: What subgroup includes patients with recent trauma and high tissue irritability?
A: Acute phase subgroup—characterized by pain, swelling, and limited ROM requiring protection and gradual loading.
32
Q: What clinical signs define the movement coordination impairment subgroup?
A: Reports of instability, poor neuromuscular control, and positive special tests like pivot shift or Lachman.
33
Q: Which subgroup benefits most from neuromuscular re-education and proprioceptive training?
A: Movement coordination impairments subgroup.
34
Q: What impairment profile is typical in patients post-ACL reconstruction?
A: Quadriceps weakness, altered gait, and impaired dynamic stability.
35
Q: How are patients with knee hypermobility classified?
A: As having generalized ligamentous laxity with potential for recurrent sprains and instability.
36
Q: What subgroup includes individuals with chronic instability but minimal pain or swelling?
A: Chronic phase subgroup—focused on restoring function and preventing reinjury.
37
Q: What impairments are prioritized in the acute protection phase?
A: Pain control, inflammation reduction, and gentle ROM restoration.
38
Q: What subgroup is appropriate for patients with isolated MCL sprains and localized tenderness?
A: Local tissue injury subgroup—managed with bracing and progressive loading.
39
Q: What is the focus of treatment in the return-to-sport subgroup?
A: High-level neuromuscular control, sport-specific drills, and psychological readiness.
40
Q: How does effusion grading help guide subgroup classification?
A: Higher effusion grades suggest acute irritability, guiding clinicians toward protection and unloading strategies.
41
Q: What is the primary special test used to assess ACL integrity?
A: The Lachman test—highly sensitive for detecting ACL tears, especially in acute settings.
42
Q: Which test is most specific for diagnosing ACL rupture?
A: The pivot shift test, though it may be difficult to perform in acute or guarded patients.
43
Q: What test is used to assess PCL integrity?
A: The posterior drawer test, often accompanied by the posterior sag sign.
44
Q: How is MCL integrity evaluated during examination?
A: Valgus stress test at 30° knee flexion to isolate the MCL.
45
Q: What examination technique helps assess joint effusion?
A: The stroke test or bulge sign to detect intra-articular swelling.
46
Q: What functional test is recommended to assess dynamic stability post-ACL injury?
A: Single-leg hop tests (e.g., hop for distance, triple hop, crossover hop).
47
Q: How is quadriceps strength commonly assessed in ligament injury rehab?
A: Isometric or isokinetic testing, or functional measures like single-leg squat.
48
Q: What tool is recommended to assess psychological readiness for return to sport?
A: The ACL-Return to Sport after Injury (ACL-RSI) scale.
49
Q: Why is range of motion assessment important in early rehab?
A: To monitor for extension lag or flexion deficits that may impair recovery.
50
Q: What movement pattern should clinicians observe during gait analysis?
A: Antalgic gait, quadriceps avoidance, or knee hyperextension during stance phase.
51
Q: What is the recommended approach during the acute phase of a knee ligament sprain?
A: Use of cryotherapy, protected weight bearing, and gentle ROM to reduce pain and swelling.
52
Q: How soon should clinicians initiate early weight bearing after ACL reconstruction?
A: As tolerated, typically within 1 week, to promote joint health and function.
53
Q: What is the role of neuromuscular electrical stimulation (NMES) post-ACL surgery?
A: NMES is recommended to improve quadriceps strength, especially in the early rehab phase.
54
Q: What type of exercise is emphasized during the subacute and return-to-activity phases?
A: Progressive strengthening and neuromuscular control exercises targeting the hip and knee.
55
Q: When is immediate postoperative mobilization recommended?
A: To increase ROM, reduce pain, and minimize adverse responses like arthrofibrosis.
56
Q: What is the guideline stance on supervised rehabilitation vs. home-based programs?
A: Supervised rehab is preferred for better outcomes, especially in the early stages.
57
Q: How should clinicians address movement coordination deficits in chronic ACL-deficient knees?
A: Through perturbation training and dynamic stability exercises.
58
Q: What is the evidence for bracing after ACL reconstruction?
A: Routine use of functional bracing is not strongly supported for improving long-term outcomes.
59
Q: What psychological factor should be addressed during rehab?
A: Fear of reinjury—clinicians should incorporate education and graded exposure to activity.
60
Q: What criteria should guide return-to-sport decisions?
A: Objective strength and hop test symmetry, psychological readiness, and sport-specific performance.