L22- BW (bacterial skin)

Question 1

bronze skin with tense edema, tenderness, and crepitant

Answer 1

Gas gangrene

Question 2

what do the borders look like in cellulitis

Answer 2

the blend in elevation and color to surrounding tissues

Question 3

what differentiates cellulitis and necrotizing fasciitis

Answer 3

failure to respond to abx w/in 24-48 hours

Question 4

superficial cellulitis with focal dermal lymphatic invovlement

Answer 4

erysipelas caused by GAS

Question 5

person to person staph transmission

Answer 5

difficult to stop!

Question 6

impetigo has 2 forms:

Answer 6

nonbullous and bullous

Question 7

destruction of muscle fascia and overlying structures

Answer 7

streptococcal gangrene

Question 8

streptococcal gangrene tx

Answer 8

AGGRESSIVE SURGERY + abx + IV fluids

Question 9

usual cause of folliculitis

Answer 9

staph aureus

Question 10

staphylcocci inoculum are what size? and how can you prevent the disease

Answer 10

usually not large, disease is preventable with cleansing (soap)

Question 11

what bacteria are normal skin and mucous membrane inhabitants

Answer 11

staphylococci

Question 12

how do you tx cellulitis

Answer 12

empirically b/c cx rarely ID agent and abx usually lead to quick resolution

Question 13

clusters of furuncles that extend into the dermis and SQ often on the back, neck, thighs

Answer 13

carbuncles

Question 14

how are bullous impetigo created

Answer 14

action of exfoliative toxin that disrupts epidermal cell connections

Question 15

what makes an infection complicated

Answer 15

(1) pre-existing wound involved (2) deeper tissues (3) requires sx (4) unresponsive to tx/ recurrent (5) associated with underlying dz (ie DM)

Question 16

streptococcal gangrene type 2 risk factors

Answer 16

anyone can get it, it effects ANY AGE GROUP

Question 17

fascia is swollen and dull gray with NO TRUE PUS ANYWHERE only thin brownish exudate

Answer 17

necrotizing fasciitis

Question 18

SQ infection with pain out of proportion to clinical signs

Answer 18

necrotizing SQ infection

Question 19

2 primary pathogens for superficial folliculitis

Answer 19

staph aureus, pseudomonas aeruginosa

Question 20

bullous impetigo toxin spread

Answer 20

does NOT disseminate beyond the local sites of infection

Question 21

mortality rate with ritters disease

Answer 21

low, often caused by secondary infections

Question 22

contains no organisms or leukocytes; a toxin mediated response

Answer 22

ritters disease

Question 23

streptococcal gangrene type 1 risk factors

Answer 23

DM

Question 24

nikolskys sign, and desquamated areas look scalded

Answer 24

ritters disease

Question 25

HEAT- Heat Erythema Edema Tenderness

Answer 25

cellulitis hallmarks (also seen in necrotizing fasciitis though!)

Question 26

clusters of vesicles that rupture and crust over

Answer 26

nonbullous impetigo

Question 27

acute infection of skin and deeper SQ tissues

Answer 27

cellulitis

Question 28

what makes an infection uncomplicated

Answer 28

responds to abx and wound care \*\*\* still has potential to become serious\*\*\*

Question 29

slight pressure disrupts the skin, causing it to peel easily

Answer 29

nikolskys sign (seen in ritters)

Question 30

what drastically drops the infectious dose of staphylococci

Answer 30

foreign body like stiches or splinter

Question 31

with acne vulgaris, what is disease NOT related to

Answer 31

skin cleansing

Question 32

gas gangrene dx

Answer 32

tissue bx showing muscle necrosis, gram variable rods, and tissue destruction

Question 33

H2 gas

Answer 33

crepitant

Question 34

how do you treat extensive ritters skin wounds

Answer 34

as burns

Question 35

abscesses involving a hair follicle and surrounding tissue often on neck, thighs, butt, face... what causative agent

Answer 35

furuncle aka boil... staph aureus

Question 36

\_\_\_ and ___ account for \_\_\_% of cellulitis

Answer 36

S. aureus and S. pyrogenes (GAS) account for 90% of cellulitis

Question 37

superficial skin infection with crusting or bullae

Answer 37

impetigo (pyoderma)

Question 38

enlarged vesicles that form staph aureus colonized fluid filled bullae

Answer 38

bullous impetigo

Question 39

nonbullous impetigo pathogens

Answer 39

mostly S. aureus, can have co-infection with Strep pyogenes

Question 40

what is important for diagnosis of ritters disease

Answer 40

clinical presentation