L28 malaria Flashcards
(30 cards)
algid malaria
skin is cold but internal temp is high
from falciparum
enlarged organs
chronic phase of chagas disease
identifiable features of vivax
enlarged infected RBCs with schuffners dots
doesn’t infect many RBCs
vivax is most prevalent in
caribbean and latin america
RARE in west africa
this type of malaria has lots of cellular debris causing:
cerebral malaria
algid malaria
gastric malaria
falciparum
people with sickle cell are resistant to
falciparum
parasitic protozoan transmitted through feces
trypanosoma
poops while it bites your face
(chagas dz)
Blackwater fever
seen in falciparum:
black urine from lots of Hgb in the urine
+ fever, chills, rigor
due to high parasite numbers
cerebral malaria causes
necrosis, hemorrhages, mania, convulsons
due to
falciparum
chagas protozoa life cycle
bug poops
trypamastigotes enter
become amastigotes
amastigotes replicate
become trypamastigotes
trypanosoma vector
triatomine bugs
aka kissing bugs
(causes chagas dz)
cross like formation in RBCs
babesia microti
frequent vomiting similar to whooping cough
gastric FALCIPARUM malaria
romanas sign
extreme swelling of eyelid near where the parasite entered
acute phase of chagas
receptor for vivax
duffy antigen
many people from West African don’t have this
paroxysms every 4th day
plasmodium malariae
identifiable features of ovale
common relapses
most common in West Africa
malaria’s main means of resistance
(there are many)
efflux pumps
identifiable features of plasmodium knowlesi
zoonotic
life threating w/ enormous parasite load
SE Asia
schuffners dots
vivax
this malaria species tends to infect young erythrocytes
vivax
relapses occur frequently with this common malaria…
why?
vivax
due to activation of liver hyponzoites
New England
warm months
malaria like sxs
small pinpoint lesions
babesia microti
malaria sxs
anemia, hypotension
N/V/D, HA, back pain, myalgia, cough
