Lab Exam 2- Lab 8 Flashcards

(47 cards)

1
Q

what is the best overall indicator of cardiorespiratory function

A

VO2 max

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2
Q

what provides a more sensitive measurement of changes in cardiorespiratory function

A

cardiac and hemodynamic assessments (BP)

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3
Q

common measurements for all GXT procedures include

A

cardiac electrical conduction through 12 lead ECGs
RPE
O2 consumption
BP

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4
Q

what is one of the most valuable and convenient indicators of CV health

A

BP

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5
Q

how many americans have high blood pressure

A

one third of Americans have hypertension which can be present without symptoms

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6
Q

blood pressure is recorded as

A

a ratio of systolic pressure/diastolic pressure
BP= sBP/dBP

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7
Q

systolic blood pressure

A

the pressure in the arteries when the heart contracts

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8
Q

diastolic pressure

A

the pressure in the arteries when the heart is filling in between beats

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9
Q

pulse pressure (PP)

A

the difference between diastolic and systolic pressures
PP= sBP-dBP

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10
Q

healthy blood pressure is defined as

A

less than 120/80 mmHg

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11
Q

as we age what happens to blood pressure

A

BP rises steadily with age due to arterial stiffness and plaque buildup

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12
Q

mean arterial pressure (MAP)

A

the average pressure in the arteries during a cardiac cycle

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13
Q

importance of MAP

A

determines the rate of blood flow through vasculature

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14
Q

how to calculate MAP at rest

A

1/3 PP + dBP

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15
Q

why is MAP calculated the way it is

A

because the heart is in diastole (relaxation) for 2/3 of the cardiac cycle AT REST** meaning it is not accurate during exercise because the heart is in SYSTOLE the majority of the time compared to rest

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16
Q

how is MAP determined centrally

A

by cardiac output (Q)

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17
Q

how is MAP calculated peripherally

A

via TPR or total peripheral resistance

MAP = Q*TPR

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18
Q

TPR

A

the sum of the resistance of blood flow in the vasculature

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19
Q

blood pressure is influenced by

A

anything that influences Q and TPR and increasing ANY of these variables will increase BP

20
Q

why does BP increase during exercise

A

due to a large increase in Q

21
Q

how does Q increase during exercise

A

elevated HR and LVR contractility as a result of SNS + increased venous return (preload)

22
Q

MAP refers to

A

afterload and is a barrier to the ejection of blood from the ventricles

23
Q

if you have a higher afterload what happens to SV

A

increases because SV must increase more than BP to pump blood to the body

24
Q

what helps reduce the effects of increased afterload after exercise

A

vasodilation of the arterioles supplying the blood to the active muscles

25
net effect of exercise on BP
increase in systolic BP due to increased Q and no change in dBP due to vasodilation
26
aerobic exercise training effects on BP
decreases resting and submaximal exercise BP and decreases TPR (vascular tone)
27
what happens to BP at maximal exercise
dBP changes sBP and MAP do not change
28
does weight loss have favorable or unfavorable influences on BP
favorable
29
benefits of performing GXT
powerful predictive and diagnostic capabilities useful for testing different things in different individuals extending medical history and physical evaluating exertional discomfort evaluating hidden CHD identifying risk in patients with known CVD Rx exercise
30
prior to GXT subjects are classified into 3 risk categories based on
age known disease risk factors categories are low, medium, and high
31
when can the GXT be stopped
at a predetermined point (%HRmax) or point of volitional exhaustion chest pain/breathlessness
32
relative criteria for stopping GXT
subjects discretion
33
absolute criteria for stopping GXT
always stopped
34
ECG and BP abnormalities during an exercise test may be indicative of or predictive of
CAD and suggest need for further testing
35
changes in cardiac function during GXT
linear increase in HR to max linear increase in SV until a certain intensity
36
when does SV plateau in untrained individuals
approx 50%
37
when does SV plateau in trained individuals
at higher than 50% intensities and may not ever plateau in elite athletes
38
what happens to Q during GXT
Q increases greatly at first and less as SV plateaus
39
ECG
electrocardiogram an indicator of the hearts ability to function normally under increased myocardial O2 demand
40
what is normal and can be seen in reduced RR intervals and minor QRS complex and P wave changes
altered AP duration, conduction velocity and contractile velocity associated with an increase in HR during exercise
41
arrhythmias not present at rest can be present during
execise as conduction disturbances and myocardial ischemia (reduced blood flow)
42
most common sign of myocardial ischemia during GXT is
ST segment depression
43
central feature of GXT is
functional exercise capacity since VO2 is the best predictor of CV function
44
prognostic capabilities of GXT are defined by normal and abnormal cardiac and hemodynamic changes . what is considered normal
achieve >80% predicted HRR sBP increases 5 mmHg per MET HRrecovery decreases 12 bpm in 1 minute 3 min post exercise sBP <90%max sBP normal cardiac rythyms
45
prognostic capabilities of GXT are defined by normal and abnormal cardiac and hemodynamic changes . what is considered abnormal
achieve <80% predicted HRR slow increase or sudden decrease in sBP sBP >250 mmHg or increases >140 mmHg from rest dBP increases more than 10mmHg dysrhythmias, ST segment depression or elevation
46
BP measurement positions
one standing and one sitting
47
how to measure BP
seated position wrap cuff over brachial artery inflate cuff to no more than 200 mmHg slowly release until you hear Kortokoff sound (sBP) continue releasing until sound disappears (dBP)