Management Diseases Flashcards
(26 cards)
The outdoor sow
- cold
- sunburn
- salt poisoning
- bird strike
- stavation/bullying
Thin sow syndrome
- causes (5)
- bodyweight
- inadequate bodily condition to function properly
- feeding too little in pregnancy
- feeding too little in lactation
- bullying
- excess energy loss e.g. cold and no increase in winter feed
- parasitism
12.5kg increase in body weight from conception to conception
Thin sow syndrome
- results
- commonly
- thin layer of SC fat do not cushion the bony prominences
- animals hosed on concrete may develop sores
commonly:
- during farrowing indoors
- over shoulder
- over hips and back
Parasites
- hyostrongylus rubidus
- oesophagostomum
- ascaris suum
- trichuris suis
- metastrongylus apri
- sarcoptes scabeii var suis
Hyostrongylus rubidus
- Facts (2)
- pathogenesis
- CS (3)
- Dx
- Tx
- 1cm red stomach worm
- infects gastric mucosal glands and gives a morroccan leather appearance
- goes into gastric mucosa in larval stages and lie there inhibited until half way through gestation when they recommence development until lactation –> damage causes protein leakage
CS:
- weightloss in lactation which continues post weaning
- no diarrhoea
- reduced fertility (increased W-S interval, increased irregular returns and decreased fecundity
Dx:
- FEC
- plamsa pesinogen
larval stages do not respond to anthelmintics
Oesophagostomum
- Facts (2)
- CS (4)
- Pathology
- Dx
- 1.5 cm, found in colon and and caecum
- periparturinet rise mid gestation and peaks during lactation
CS:
- poor weight gain
- reduced productivity
- ± diarrhoea
- contributes to thin cow syndrome and reduced milk yields
Pathology:
- diffuse enteritis ± nodules of larvae in mucosal surface
Dx: FEC
Ascaris Suum
- facts (2)
- CS (3)
- larvae migrate through the liver (milk spot) with mature stages in the SI
- mainly transmitted through environmental contamination but can be passed (rarely) dam –> offspring
CS:
- decreased growth
- decreased fecundity
- soft cough
Trichuris Suis (4)
- mature worms are 3-8cm long
- LI worm
- larval stages invade the crypts of the LI, causing mucosal damage –> may be invaded by secondaries e.g. brachyspira
- mild diarrhoea and reduced weight gain
- immunity develops with age
Metastrongylus Apri
- facts (2)
- CS (3)
- PM (3)
- lungworm
- IMG = earthworm (common in outdoor pigs)
CS:
- coughing
- dyspnoea
- reduced growth rate
PM:
- pale patches of emphysema in diaphragmatic lobes
- thin, white worms (4-5cm) can be squeezed out of this lung tissue
- may see embryonated eggs in faeces
Sarcoptes scabeii var suis (5)
- burrowing mite
- causes intense pruritis (hypersensitivity reaction) and contributes to thin cow syndrome
- mites commonly found around ears –> will flap ears/ have crusting around edges/ wax in ears
- spread pig-pig and from environment
- mite survives up to 2 weeks in environment
Anthelmintic protocols
- types
- programmes
- in feed, water and parenterally (no drenches)
- may include ascaracidal activity by using avermectin where herd has sarcoptes or haematopinus suis (sucking louse)
Premixes;
- Ivermectin
- benzimadazoles
Water soluble:
- fenbendazole
Parenternal wormers
- Ivermectins
Worming programmes:
- breeding stock: done 2/year: spring and autumn
- fattening stock: every 2 months form weaning
Infertility Problems (4)
- gilt management prior to first service
- supervision of service
- AI timing
- Pregnancy checks and returns
Farrowing Problems (5)
- farrowing crate adjustment
- use of prostaglandin on wrong day of gestation
- positioning of creep lights
- poor colostral intake (supervised farrowing)
- poor hygiene at interferance
Use of prostaglandin on wrong day of gestation (4)
- used to induce farrowing
- piglets are viable from day 111 (for success, service dates must be precise)
- undersized piglets are more vulnerable and more likely to have splay leg
- colostral quantity and quality may be compromised if induced early
Positioning of creep lights (2)
- should be placed to the rear of sow and in creep area
- rear light should be switched off after the sow has finished farrowing –> piglets will then be safe in the creep, away from the sow
Poor colostral intake (supervised farrowing) (4)
- colostrum protects against general diseases, stimulates metabolism to warm the pig and acts as a laxative
- intakes is essential to protect the offspring of vaccinated sows
- split sucking may be required
- colostral substitutes may be given by stomach tube, but cross fostering should be avoided in the first 24hours of life
Poor Hygiene at interferance
- dirty hands, damage or faecal contamination can lead to intrauterine infection, infected postpartum discharge and reduced fertility at subsequent service
Trauma in the Farrowing crate (4)
- piglets may be overlain (most common in first 48 hours): raised edge may prevent
- piglets may be crushed against filiments
- piglets may be savaged (most commonly gilts)
- all crushings/overlying problems should be confirmed by PM
Water supply (5)
- unpalatable water/ furred up pipes and nipples depress water intake
- need min. of 2L/minute water delivery to avoid frustration
- if inadequate water will result n overheating, reduced milk production and salt poisoning
- water sprays may be required in hot weather so sows and piglets
- piglet drinkers will reduce mortality fomr diarrhoea dna improve growth rates
Salt Poisoning
- faces (3)
- CS of total water deprivation (4)
- CS of excessive salt intake/ incomplete deprivation (6)
- can affect any age
- due to excess dietary salt or water deprivation (freezing/drought/burst pipes) or if individuals are too immobile and unable to reach water
- CS may be exacerbated by re-introduction of water
CS of total water deprivation
- seizures
- prostration with paddling
- coma
- death
CS of excessive salt intake/ incomplete deprivation:
- blindness
- unresponsiveness to external stimuli
- inappetence
- head pressing
- circling
- convulsions
Salt poisoning
- Dx (4)
- Tx (5)
Dx:
- no evidence of fever or arthritis = not strep suis
- no evidence of oedema = not oedema disease
- no grows PM change apart from congestion of meninges
- meningoencephalitis with oedema ad eosinophil accumulations around vessels of cerebral cortex and meninges
Tx:
- low sodium diet
- re introduce water slowly: water trough so all can access but with a limited supply
- betamethasone
- keep in subdued light
- euthanase if no improvement after 36 hours
Iron deficiency/anaemia
- pathogenesis
- requirements
- CS (5)
- hypochromic, microcytic anaemia of rapidly growing pigs housed on concrete/plastic (iron is found in soil)
- piglets require 15mg/day but sow only supply 1mg/day
- by 3-4 weeks, haemoglobin will have dropped form 120g/l to 35g/l if no access to iron
- iron is contained in creep feed
- clinically apparent by 10-14 days
CS:
- reduced growth
- falling behind rest of litter
- appear pale/jaundiced
- lethargy
- dyspnoea
Tooth Clipping (5)
- not permitted as routine in UK
- advantage of 0.5kg/piglet
- injury can occure (particularly to face) resulting in staph. hyicus –> exudative epidermitis (greasy pig)
- injury to sows udder can occur
- take sharp point off only by clipping and grinding
Tail bitin
- causes (2)
- triggers (6)
- control (3)
- considerations
- natural tendency to chew
- attracted to blood (metallic taste) once initial damage has occurred
Triggers:
- temperature variation
- draughts
- high stocking rates
- competition for food/water
- mixed genetic lines
- mixed tail lengths
control:
- docking (not routinely permitted in uk)
- increase salt content of diet if house on slats to 1%
- remove affected piglets ASAP
- if no tail remaining, open wound or selling/abscessation at the tail head, euthanise pig on farm
- if the wound has healed without evidence of abscessation: send for slaughter highlighting tail bite on food information form