MEDSURG 2 EXAM #3 Flashcards

Weeks 5, 6, & 7 (173 cards)

1
Q

What to study for EXAM #3

A

— Diarrhea, constipation, and incontinence
— What is the purpose and what does the RN need to do to help with issue
— Distinguish the process of food breakdown
— What to include in teaching for a procedure (pre/intra/post)
— Objective 1 for sureeee
— How to assess (order and direction)
— Difference b/w ostomies and kind of stools to expect; tx
— Types of hernias, location, which type is an considered an emergent event
— B12, Niacin for vitamin therapy with Cirrhosis patients
— Short gut syndrome
— Metabolic syndrome: detect DM
— Management for UGI if patient comes in = color of stool/vomitus vs. lower GI
— Guaiac test: detect if blood in stool + EGD = NPO + clear liquids + bowel prep (Go-Lytely), have gag reflux to make sure they can eat again
— Peptic Ulcer Disease
— Projectile vomiting may mean what? = ICP
— Protein formula = 0.8 * kg

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2
Q
  1. Describe the structures and functions of the organs of the gastrointestinal tract.
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3
Q

Hepatitis or Cirrhosis lab findings

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— Elevated AST, ALT levels

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4
Q
  1. Describe the structures and functions of the liver, gallbladder, biliary tract, and pancreas.
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5
Q

Elevated ALP levels indicates ________ damage

A

liver

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6
Q
  1. Distinguish the processes of ingestion, digestion, absorption, and elimination.
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7
Q
  1. Explain the processes of biliary metabolism, bile production, and bile excretion.
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8
Q
  1. Link the age-related changes of the gastrointestinal system to the differences in assessment findings.
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9
Q
  1. Obtain significant subjective and objective assessment data related to the gastrointestinal system from a patient.
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10
Q
  1. Perform a physical assessment of the gastrointestinal system using appropriate techniques.
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11
Q
  1. Distinguish normal from abnormal findings of a physical assessment of the gastrointestinal system.
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12
Q
  1. Describe the purpose, significance of results, and nursing responsibilities related to diagnostic studies of the gastrointestinal system.
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13
Q
  1. Describe the etiology, clinical manifestations, and interprofessional and nursing management of malnutrition.
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14
Q
  1. Describe the components of a nutrition assessment.
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15
Q

What is malabsorption syndrome + results from?

A

Malabsorption Syndrome: impaired absorption of nutrients from the GI tract

May result from:
— ↓ enzymes
— Drug side effects
— ↓ bowel surface area
— Fever increases BMR

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16
Q
  1. Explain the indications, complications, and nursing management related to the use of enteral nutrition.
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17
Q
  1. Explain the indications, complications, and nursing management related to the use of parenteral nutrition.
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18
Q
  1. Compare the etiology, clinical manifestations, and nursing management of eating disorders.
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19
Q
  1. Discuss the epidemiology and etiology of obesity.
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20
Q
  1. Explain the health risks associated with obesity.
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21
Q
  1. Use classification systems to determine body size.
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22
Q
  1. Discuss comprehensive therapy for the patient with obesity.
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23
Q
  1. Distinguish among the bariatric surgical procedures used to treat obesity.
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24
Q
  1. Describe the nursing and interprofessional management related to surgical therapies for obesity.
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25
8. Describe the etiology, complications, and interprofessional and nursing management of nausea and vomiting.
26
7. Describe the etiology, clinical manifestations, and nursing and interprofessional management of metabolic syndrome.
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9. Discuss the etiology, clinical manifestations, and interprofessional and nursing management of common oral inflammations and infections.
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10. Describe the etiology, clinical manifestations, complications, and interprofessional and nursing management of oral cancer.
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11. Explain the types, pathophysiology, clinical manifestations, complications, and interprofessional and nursing management of gastroesophageal reflux disease and hiatal hernia.
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12. Relate the pathophysiology, clinical manifestations, complications, and interprofessional management of esophageal cancer, diverticula, achalasia, and esophageal strictures.
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13. Compare acute and chronic gastritis, including etiology, pathophysiology, and interprofessional and nursing management.
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14. Distinguish gastric and duodenal ulcers, including etiology, pathophysiology, clinical manifestations, complications, and interprofessional and nursing management.
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15. Outline the clinical manifestations and interprofessional and nursing management of stomach cancer.
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16. Explain the common etiologies, clinical manifestations, and interprofessional and nursing management of upper gastrointestinal bleeding.
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17. Identify common types of foodborne illnesses and nursing responsibilities related to food poisoning.
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1. Explain common causes and interprofessional and nursing management of diarrhea, fecal incontinence, and constipation.
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2. Describe common causes of acute abdominal pain and nursing management of the patient after a laparotomy.
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3. Describe the interprofessional and nursing management of acute appendicitis, peritonitis, and gastroenteritis.
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4. Compare and contrast the inflammatory bowel diseases of ulcerative colitis and Crohn’s disease, including pathophysiology, clinical manifestations, complications, and interprofessional and nursing management.
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5. Distinguish among small and large bowel obstructions, including causes, clinical manifestations, and interprofessional and nursing management.
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Ulcerative Colitis
Onset: teens to mid-30s. >60YO Abdominal pain = common, severe constant Diarrhea = common Fever = during acute attacks Malabsorption/nutritional deficiencies = minimal Rectal bleeding = common Tenesmus [gotta go, gotta poooo!] = common Weight loss = rare Location/distribution: usually starts in RECTUM and spreads in continuous pattern UP the colon; continuous areas of inflammation Cobblestoning of mucosa? NOPE, rare Depth of involvement = just mucosa Peudopolyps = common, minimal small bowel movements *Increased risk of C.diff* Perinatal abscess/fistulas = rare Strictures = occasional Toxic mega colon = more common = common perforations
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6. Describe the clinical manifestations and interprofessional and nursing management of colorectal cancer.
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Crohn’s Disease
Onset: teens to mid-30s. >60yo *Abd pain/diarrhea/fever: common w/ cramping; common* Malabsorption: common Rectal bleeding + tenesmus: sometimes/rare Weight loss = common, may be severe Location = occurs anywhere along the GI tract; most common site = distal ileum **Cobblestoning of mucosa = common** Pseudopolyps = rare Small bowel involvement = common *Increased risk of C.diff* Perforation = common (b/c inflammation involves entire bowel wall) Abscess/fistulas/strictures = common
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7. Select nursing interventions to manage the care of the patient after bowel resection and ostomy surgery.
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8. Distinguish between diverticulosis and diverticulitis, including clinical manifestations and interprofessional and nursing management.
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9. Compare and contrast the types of hernias, including etiology and surgical and nursing management.
— Inguinal = most common — Femoral — Umbilical — Ventral — Incisional
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10. Describe the types of malabsorption syndromes and interprofessional care of celiac disease, lactase deficiency, and short bowel syndrome.
Celiac disease — AID that causes damage to small intesting Lactase deficiency — condition in which the lactase enzyme is deficient or absent Short gut syndrome — small intestestine doesn’t have enough S.A. to absorb nutrients, —> unable to meet E, F/E, nutritional needs to stay healthy on a normal diet | Tx: complex carbs (e.g. starch, white/brown rice, bread), high protein, moderate fat
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11. Describe the types, clinical manifestations, and interprofessional and nursing management of anorectal conditions.
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12. Distinguish among the types of viral hepatitis.
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13. Describe the interprofessional and nursing management of the patient with viral hepatitis.
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14. Explain the pathophysiology, clinical manifestations, complications, and interprofessional and nursing management of the patient with cirrhosis.
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15. Describe the clinical manifestations and management of liver cancer.
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16. Distinguish between acute and chronic pancreatitis related to pathophysiology, clinical manifestations, complications, and interprofessional and nursing management.
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17. Explain the clinical manifestations and interprofessional and nursing management of the patient with pancreatic cancer.
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18. Describe the pathophysiology, clinical manifestations, and interprofessional care of gallbladder disorders.
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19. Describe the nursing management of the patient undergoing surgical treatment of cholecystitis and cholelithiasis.
**Cholecystitis:** inflammation of gallbladder r/t gallstones __________ **Cholethiasis:** — BMI must be WNL — Must have small, cholesterol-based stones
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**Know the Assessment of the GI system and ORDER**
**start from RLQ in zig-zag; end at LLQ** Inspection Auscultate Percuss Palpate
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**Preparing patient for colonoscopy. What are the expectations for the nurse?**
-- Check stool to make sure they passed it -- Bowel prep to ensure intestines are cleared out — Consent!
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Steps for placing NG tube What to check for tube feedings
Flush the tube with 30 mL of water - tell patient what you are doing - gather and dispose of all waste in the proper receptacle - lower the head of the bed to no less than 30 degrees -document the feeding, dose, solution, volume, time, and your initials - be sure to document the patients response including any adverse reactions
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Complications of enteral nutrition/feedings
Vomiting Diarrhea Constipation Dehydration — More calorically dense, less water formula contained — Check for high protein content
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Formula for protein intake
**0.8 to 1g/kg of body weight** Example: Patient weighs 70kg x 0.8 = 56g protein DAILY
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Which patients cannot tolerate a high protein diet?
Liver disease patients (cannot process protein well)
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Which tubes will be least the risk for aspiration?
J-tube, because it is at the furthers distance -- Located in the small intestine -- Formula: pre-digested food b/c bypassing stomach (acid contents)
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What must the RN do when preparing a parenteral nutrition (hanging TPN)? SATA
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Slide 19 -- hypovolemic shock
Scenario
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Bland diet
NO SODAS
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Hiatal Hernia Complications
--GERD --Esophagitis --Hemorrhage from erosion (= emergency) --S
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Labs, Dx, RN responsibility in the Dx tests, esophageal varices
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Causes of diarrhea
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What are the causes of consitpation?
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Causes of acute abdominal pain
**Rebound pain (pain after you release pressure)** Abdominal compartment syndrome Acute pancreatitis Appendicitis Bowel obstruction Cholecystitis Diverticulitis Gastroenteritis Pelvic inflammatory disease Perforated gastric or duodenal ulcer Peritonitis Ruptured abdominal aneurysm Internal bleeding/trauma Obstruction
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What is peritonitis? What caused this and what to find in your assessment?
— Infection of peritoneal area — Cause = infection r/t leaky gut; abdomen feels rigid/board-like
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Hernias
— Determined if incarcerated or not? If so —> OR b/c blood flow is not getting to intestinal wall…PROBLEM
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Different types of diet foods
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Why would you have 2 ostomy bags?
Temporary colostomy and resting other part of bowels. Transverse (named b/c uses the part of the colon that goes across) or Double-loop
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Important actions to be done prior receiving a patient for a procedure
— NPO — “When was the last time you flushed the IV?”
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RN Management for *diarrhea*
— Should be considered infectious until proven otherwise
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Diarrhea, fecal incontinence, constipation
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MOA of Bismuth subsalicylate (Pepto-Bismol)
— Decreases secretions and has weak antibacterial activity — Purpose: prevent traveler’s diarrhea
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MOA of Calcium polycarbophil (FiberCon)
— Bulk-forming agent that absorbs excess fluid from diarrhea to form a gel — Purpose: when intestinal mucosa cannot absorb fluid
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MOA of Diphenoxylate with atropine (Lomotil)
— Opioid and anticholinergic — Purpose: decreases peristalsis and intestinal motility
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MOA Ioperamide (Imodium, Pepto Diarrhea Control)
Inhibits peristalsis, delays transit, increases absorption of fluid from stools
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MOA of Octreotide acetate (Sandostatin)
Suppresses serotonin secretion, stimulates fluid absorption from GI tract, decreases intestinal motility
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MOA of Paregoric (Camphorated tincture of opium)
— Opioid — Purpose: decreases peristalsis and intestinal motility
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Fecal incontinence + prevention and treatment
Involuntary passage of stool Tx: bowel training program
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Constipation + prevention and treatment
Decrease in the frequency of BM from what is a patient’s “normal” — Teach patient importance of dietary + activity measures to prevent
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What are the goals of RN management for diarrhea, fecal incontinence, and constipation?
- Cessation and return of normal BM - Normal fluid and electrolyte and acid-base balance - Normalize nutritional status - Prevent skin breakdown
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Problems with diarrhea
— Life-threatening dehydration if severe enough — Electrolyte problems (e.g. HYPOkalemia)
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Infections of the colon and distal small bowels cause which S/Sx before diarrhea? Examples of infections
— Fever (no-to-low grade) — N/V — Frequent bloody diarrhea w/ small volume *Examples: Shigella, Salmonella, C.difficile*
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Collaborative care concerns for diarrhea
— Preventing transmission, replacing F/E, and protecting the skin — Depends on the cause to determine Tx — Acute = self-limiting
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Common causes of acute abdominal pain
— Appendicitis = inflammation of appendix — Peritonitis = hole/infection in the bowel, penetrating the peritoneal — Diverticulitis = cramping/bloating, tenderness — Gastroenteritis = inflammation of stomach + intestines, usually from bacterial toxins/viral infections; N/V/D, cramps, low-grade temp
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Expected output for ostomies
— _Ileostomy:_ >1000mL/day, bile-colored, liquid — _Transverse colostomy:_ small semi-liquid w/ some mucus 2-3 days post surgery (with some blood) — _Sigmoid colostomy:_
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Foods that CAUSE odor for ostomies + treatment
Fish, eggs, asparagus, garlic, beans, dark leafy veggies Tx: buttermilk, cranberry juice, parsley, yogurt DECREASES odor
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Ileostomy Characteristics
— Consistency: liquid-semiliquid — Fluid requirement: increased — Bowel regulation: NOPE — Pouch + skin barriers: YES — Indications for surgery: UC, Crohn’s, or injured colon, familial polyposis, trauma, cancer
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Foods that cause gas in ostomies + treatment
— Dark leafy greens, beer, carbonated beverages, dairy products, and corn — Chewing gum, skipping meals, smoking Tx: yogurt, crackers, toast DECREASE gas
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Colonoscopy Characteristics
— Stool consistency: semi-liquid — Fluid req.: INCREASED — Bowel regulation: NO — Pouch + skin barriers: YES — Indications for surgery: Perforating diverticulum in lower colon, trauma, recto signal fistula, inoperable t muros of colon/rectum/pelvis
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Client education with ostomy involving the small intestine
— Avoid high-fiber foods for the first 2 months postoperative — Chew food well — Increase fluid intake — Evaluate for evidence of blockage when slowing adding high-fiber foods to diet
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Transverse characteristics
— Stool consistency: semi-liquid to semi-formed — Fluid req.: perhaps increased — Bowel regulation: NO — Pouch + skin barriers: YES — Indications for surgery: same for ascending
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Characteristics of stoma appearance
Viable/healthy: — pink/red, beefy-looking, and moist May indicate anemia: — pale Inadequate blood supply to stoma/bowel — blanching, dark red-purple
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Sigmoid (single/double-barreled) characteristics
— Stool consistency: formed — Fluid req: NO ∆ — Bowel regulation: YES, if there is a hx of a regular bowel pattern — Pouch + skin barriers: dependent on regulation — Indications for surgery: cancer of rectum/rectosigmoid area; perforating diverticula, trauma
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Manifestations of stomach ischemia/necrosis
— Expected color: pale pink or blueish purple, ry — Unexpected color = If stoma is BLACK/PURPLE = serious impairment of blood flow and requires immediate attention
102
If stoma is bleeding
Small amount = NORMAL — when small amount is oozing form stoma mucosa when touched b/c of high vascular it’s Moderate/large amount = NOT GOOD — lower GI bleed, coagulation factor deficiency, stomal varices 2nd army to portal HTN
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Peptic Ulcers
— Erosion of GI mucosa resulting from digestive action of HCl acid and pepsin — Develops only in the presence of an acid environment
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Difference between acute vs. chronic peptic ulcers
Acute — superficial erosion — minimal inflammation; resolves quickly when cause is ID’d and removed _________________ Chronic — muscular wall erosion w/ formation of fibrous tissue — continuous for many months/intermittently throughout life; more common
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Post-op care for stoma
— Assess wound, record bleeding, XS drainage, unusual odor — Monitor for edema erythema, drainage around suture line — Fever, HIGH WBC count
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Pathophysiology of Ulcer
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Etiology of peptic ulcer disease (PUD)
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Route of transmission for Hepatitis
Fecal-oral: Hep A, Hep E Blood: Hep B, Hep C, Hep D
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S/Sx of someone experiencing perforation of a peptic ulcer
— board-like abdomen — severe abdominal/back pain that radiates to R shoulder IF HEMORRHAGING: vomiting of blood + shock (^HR/RR, decr BP)
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What is hepatitis + their causes?
*Inflammation of the liver* Causes: — Viral (most common) — Drugs (alcohol) — Chemicals — Autoimmune diseases — Metabolic abnormalities
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Hepatitis A laboratory results
Elevated: ALT, AST, ALP (can also be WNL), total bilirubin, Hepatits A virus antibodies (anti-HAV)
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Clinical manifestations of *Hep A*
— Not chronic & incidence decreased w/ vaccination — RNA virus transmitted via fecal-oral (e.g. contaminated food/water — Vaccinate children @ 1yo *NOTE: the greatest risk of transmission occurs before clinical S/Sx are apparent*
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Hepatitis B laboratory results
Elevated: ALT, AST, ALP (also can be WNL), total bilirubin, HBsAG
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Hep B clinical manifestations
— Transmission: percutaneous, parenteral (concurrent w/ HBV); sexual, shared needles, blood, birth canal — Dx: liver biopsy (intensity and degree of liver damage) — S/Sx: N/V, fever, fatigue, RUQ pain, dark urine/clay stools, joint pain, jaundice — Labs: Elevated AST, ALT, bilirubin, HBV PCR DNA, HBsAg — Tx: Tenofovir, adenovirus dip I odio, interferon alfa-2b, peginterferon alfa-2a, lamivudine, entecavir, & telbivudine (for chronic infxn) — Vaccine?: YES — Prevention: Engerix-b
115
At-risk populations for Hep B
Men who have sex with men Household contact of chronically infected Patients undergoing hemodialysis Health care and public safety workers Transplant recipients HBsAg in the serum for 6 months or longer after infection indicates chronic HBV infection. **NOTE: HBV can live on a dry surface for at least 7 days. HBV is much more infectious than HIV.
116
Normal ranges for Hepatitis laboratory values
Alanine aminotransferase (ALT) = 4-36u/L Aspartate aminotransferase (AST) = 0-35u/L Alkaline phophatase (ALP) = 30-120u/L Total bilirubin level = 0.3-1.0mg/dL Presence of antibodies for Hepatitis infection Enzyme immunoassay (EIA) HCV RNA polymerase chain reaction (PCR)
117
Hepatitis C laboratory results
Elevated = ALT, AST, ALP (or WNL), total bilirubin, anti-HCV, EIA, PCR
118
Clinical manifestations of Hep C (HCV)
Acute: asymptomatic Chronic: liver damage RNA virus transmitted percutaneously IV drug use High-risk sexual behaviors Occupational exposure Dialysis Perinatal exposure Blood transfusions before 1992 NOTE: majority of patients are unaware of their infection and usually develop chronic infection.
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Hepatitis D (Delta) laboratory results
Presence of: identification of intrahepatic delta antigen, anti-HDV, co-infected with HBV
120
Clinical manifestations of Hep D (HDV)
*also called delta virus* Transmission: percutaneous, parenteral (concurrent w/ HBV); sexual, shared needles, blood, birth canal Dx: HBV PCR DNA, HBsAG S/Sx: anorexia, N/V, fever, RUQ pain, dark urine/light stool, joint pain, jaundice (icteric) Lab results: elevated ALT, AST, bilirubin, Tx: NONE Vaccine?: NONE, but vaccination against HBV reduces risk of HDV co-infection Prevention:
121
Hepatitis E laboratory results
Presence of: anti-HEV
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Hep E (HEV) clinical manifestations
*primarily in developing countries (Inda, Asia, Mexico, Africa)* Transmission: fecal-oral, H20-borne Labs: elevated ALT, AST, bilirubin, S/Sx: flu-like (2wk-2mos) Tx: Prevention?: HH, lifestyle, BLEACH
123
Which pain medication should you question after surgery for a patient with Hep B?
Hydrocodone w/ acetaminophen because can acetaminophen can impair liver function.
124
Excessive aspirin vs. acetaminophen can cause damage to which organs?
**Aspirin** to the kidneys and cause elevated aminotransferase levels **Acetaminophen** to liver
125
What happens during an acute viral hepatitis infection?
Acute infection — Liver damage: lysis of infected cells — Cholestasis — Liver cells can regenerate in normal form after resolution of infection Chronic infection — Can cause fibrosis and progress to cirrhosis
126
Active vs. on-going systemic S/Sx of hepatitis viruses
Active: Rash, Angioedema, Arthritis, Fever, Malaise, Cryoglobulinemia, Glomerulonephritis, Vasculitis Intermittent/On-going: Malaise, fatigue, myalgias/arthralgias, hepatomegaly
127
S/Sx during incubation period of hepatitis viruses
Malaise, anorexia, weight loss, fatigue, N/V, abd discomfort, distantes for cigarettes, ↓ Sense of smell, H/A, low-grade fever, arthralgias, skin rashes
128
Serologic Events
— Incubation period — Acute S/Sx: elevated ALT, jaudice (icteric) — Convalescence: begins as jaundice is disappearing, lasts weeks-months — Recovery: homologous immunity to HAV or HBV, but can become reinfected w/ other types and different strains (HCV)
129
What is cirrhosis + different types?
Cirrhosis: extensive scarring of the liver caused by necrotic injury or a chronic Tx to inflammation over a prolonged period of time Types: — Postnecrotic: viral hepatitis/mixed medications or toxins — Laennec’s: chronic EtOH d/o — Biliary: caused by chronic biliary obstruction/Autoimmune disease
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What are the complications of hepatitis?
_Chronic hepatitis:_ — ongoing inflammation of liver — results from HBV, HCV, or HDV — increases risk for liver cancer ___________ _Fulminant hepatitis:_ — XS severe + fatal form of viral hepatitis — Patient goes from viral hep w/in hrs —> severe liver failure — No meds, simply supportive care _______ — Cirrhosis of the liver — Liver cancer/failure — Hepatic encephalopathy: NH3 elevated causing neurological S/Sx (stupor, asterixis [hand-flapping], fetor hepaticus [fruity musty breath], seizures, and coma)
131
Complications for patients w/ cirrhosis?
— Portal HTN: increased portal venous pressure, s pleno metals, large collateral veins, ASCITES, gastric (upper)/esophageal varices (very fragile) — Coagulation defects — Jaundice — Portal-systemic encephalopathy (PSE) w/ hepatic coma (inability to detoxify protein byproducts = elevated NH3
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RF for Cirrhosis when assessing patient
EtOH, chronic HBV, HCV, or HCD, AID (hepatic), Steatohepatitis (causing chronic inflammation), biliary cirrhosis, — — Cardiac cirrhosis —> RHF inducing necrosis/fibrosis d/t lack of blood flow
133
Stages of Hepatic Encephalopathy
(1) — (2) — (3) (4)
134
S/Sx of MEN vs. WOMEN w/ cirrhosis
MEN: gynecomastia (benign growth of the glandular tissue of the male breast), loss of axillary and pubic hair, testicular atrophy, and impotence with loss of libido may occur because of increased estrogen levels. WOMEN: Young = amenorrhea Older = vaginal bleeding. NOTE: liver fails to metabolize aldosterone adequately —> hyperaldosteronism w/ Na+/H2O retention & K+ loss
135
S/Sx of cirrhosis
Fatigue, weight loss, abdominal pain/distention, pruritis, confusion
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Expected laboratory results for patients with cirrhosis
LDH ALT/AST: elevated —> WNL b/c Elevated (initially): LDH, ALT, AST (d/t hepatic inflammation) THEN ALT/AST —> normal (b/c liver unable to create inflammatory response) ALP — elevated b/c of intrahepatic biliary obstruction NH3 =
137
Early vs. Later S/Sx of cirrhosis
EARLY: fatigue (many unaware of liver condition) Vs. LATE: jaundice, peripheral edema, ascites, skin, hematologic , endocrine, and neurologic disorders — Advanced —> liver becomes small and nodular
138
1. Purpose of the liver? 2. What does the liver produce? 3. S/Sx you have coagulation problems?
1. Clotting factors 2. Thrombin 3. Bruising, bleeding gums, PETECHIAE on skin, spider angiomas (telangiectasia/spider nevi), heavy menstrual periods, palmar erythema (red area that blanches w/ pressure) on palms of hands
139
Dx procedures for cirrhosis
— ultrasound — abdominal x-ray/CT scan — MRI — liver biopsy — esophagagogastroduodenoscopy — endoscopic retrograde cholangiopancreatography
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RN care for cirrhosis patients
— RR status — Skin integrity — Fluid balance — VS + pain level — Neurological status: r/t hepatic encephalopathy —> Lactulose — Nutritional status: HIGH carb & protein, moderate fat, and LOW Na+ diet — GI status
141
Therapeutic procedures for Cirrhosis
— Paracentesis — Endoscopic varicela ligation/endoscopic sclerotherapy — Transjugular intrahepatic portosystemic shunt — Surgical bypass shunting procedures = LAST RESORT for portal HTN/esophageal varices — Liver transplantation
142
S/Sx of Hepatorenal Syndrome
— Renal failure w/ azotemia (^Nitrogen levels) — Suddenly decrease in urinary flow (<500mL/24hr) — ^BUN/Cr — Intractable ascites — Spontaneous bacterial peritonitis ››› IV Cefotaxime
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Lab results for Cirrhosis
ELEVATED: AST, ALT, LDH, ALK, GGT, bilirubin, PT/INR DECREASED: platelets, albumin
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Goal + how to manage Cirrhosis
GOAL: slow progress of cirrhosis, prevent, tx any complications — Rest — B-complex vitamins (d/t insufficiency) — AVOID EtOH, ASA, acetaminophen, NSAIDs
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How to manage ASCITES
— Na+ restriction + fluid removal — Albumin — Diuretics (Spironolactone WITH Furosemide (Lasix)) — Tolvaptan (Samsca): correct HYPONa+ by ^ H20 excretion — Paracentesis — Transjugular intrahepatic portosystemic shunt (TIPS): if doesn’t respond well to Diuretics
146
GOAL + how to manage esophageal + gastric varices for cirrhosis patients
GOAL: Prevent bleeding/hemorrhage — Avoid alcohol, aspirin, and irritating foods — Screen for presence with endoscopy — Nonselective β-blocker (Decrease high portal pressure)
147
A Rn is assessing a client who has advanced cirrhosis. The RN should ID which of the following findings as indicators of hepatic encephalopathy? SATA. A. Anorexia B. ∆ in orientation C. Asterixis D. Ascites E. Fetor hepaticus
B, C, E RATIONALE: A & D is present for liver dysƒ(x),
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Which medication classes are metabolized in the liver? SATA. Diuretics Beta-blockers Opioid analgesics Sedatives Acetaminophen
Opioid analgesics, sedatives, & NSAIDs (acetaminophen) are metabolized in the liver and should be AVOIDED for anyone w/ a viral hepatitis
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A RN is caring for a client who has acute pancreatitis. Which of the following serum lab values should the RN anticipate returning to the expected reference range w/in 72hr after tx begins? A. Aldolase B. Lipase C. Amylase D. Lactic dehydrogenase (LDH)
Amylase RATIONALE: Pancreatitis is the most common diagnosis for marked elevations in serum amylase. Serum amylase begins to increase about 3 to 6 hr following the onset of acute pancreatitis. The amylase level peaks in 20 to 30 hr and returns to the expected reference range within 2 to 3 days. — Lipase stays elevated for up to 14days — Aldolase = inflammation of muscles = myositis — LDH = anemia, leukemia, or liver damage
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What is the purpose of an EGD? A. To visualize polyps in the colon B. To detect an ulceration in the stomach C. To ID an obstruction in the biliary tract D. To determine the presence of free air in the abdomen
B. To detect an ulceration in the stomach RATIONALE: EGDs visualize the esophagus, stomach, and duodenum w/ light to ID a tumor, ulcer, or obstruction A. ID polyps = sigmoidoscopy/barium enema in lower GI C. ERCP is performed D. Free air = gas = fluoroscopy/x-ray
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S/Sx of cancerous lesions
-Rectal bleeding is most common -Alternating constipation and diarrhea -Change in stool caliber -Narrow, ribbon-like -Sensation of incomplete evacuation -Obstruction
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Clinical manifestations of colorectal cancer
— Insidious onset — Symptoms often appear in advanced stages — Change in bowel habits — Unexplained weight loss — Vague abdominal pain — Weakness and fatigue — Iron-deficiency anemia and occult bleeding
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Parenteral nutrition uses
ROUTE: IV — Chronic/severe D/V — Complicated surgery/trauma — Gi obstruction — Intractable diarrhea — Severe anorexia nerviosa/malabsorption — Short bowel/gut syndrome — GI tract anomalies + fistulae — No more than 170 calories/L for Dextrose in H2O or dextrose in LRs
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Nutrition/day
CARBS — PROTEIN — 45-65g daily
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RN Mgmt for Parenteral nutrition
Vital signs every 4 to 8 hours Daily weights Blood glucose Check initially every 4 to 6 hours Electrolytes BUN CBC
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Monitor for S/Sx of infection or septicemia
Local: erythema, tenderness, exudate at catheter site Systemic: fever, chills, N/V, malaise
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Enteral delivery options
— Continuous infusion by pump — Intermittent by gravity — Intermittent bolus by syringe — Cyclic feedings by infusion pump
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Common enteral feeding locations
Nasogastric = NG-tube Esophagostomy = E-tube Nasoduodenal/nasojejunal = NJ-tube = nose —> small bowels for children *Gastrostomy = G-tube *Jejunostomy = J-tube Percutaneous endoscopic gastrostomy = PEG-tube *= used extended periods
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NG-tube
*most commonly used for ST feeding problems* — Inserted through the nasal cavity Radiopaque: Allowing visualization from x-ray ↓ likelihood of regurgitation and aspiration when placed in intestine
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G-tube + J-tube
May be used in those needing tube feedings for extended period Patient must have intact, unobstructed GI tract Can be placed surgically, radiologically, or endoscopically *NOTE: Failure to flush the tubing after both drug administration and residual volume determinations can result in tube clogging; small amount of cola can help restore patency*
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BMI ranges + waist circumferences
BMI (kg/m2) = (weight in lbs.) x 703/ (height in inches)^2 Underweight: ≤18.5 WNL: 18.5-24.9kg/m^2 Overweight: 25-29.9 Obesity: 30-34.9 Extremely obesity: ≥40 __________ MEN: ≤40 inches WOMEN: ≤35 inches
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Percutaneous Endoscopic gastrostomy
— Requires esophageal lumen wide enough for endoscope — Can start feedings when bowel sounds are present (~24hr after placement) DO NOT PLACE DRESSINGS BENEATH BUMPER
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Complete proteins vs. incomplete
*Complete:* eggs, fish, meats, milk + milk products, poultry *Incomplete:* legumes, grain, nuts, seeds
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Major minerals vs. Trace elements
*Major minerals:* Ca, Cl, Mg, P, K, Na, S *Trace elements:* Cr, Cu, F, I Fe, Mn, Mo (Molybdenum) Se, Zn
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Malnutrition terms
Cachexia — complicated metabolic syndrome related to underlying illness and characterized by muscle mass loss with or without fat mass loss that is often associated with anorexia; scaphoid appearance Kwashiorkor — seen in children in places w/ limited food resources (e.g. swollen bellies, freq infxns) | Tx: SLOWLY increase calories then protein Marasmus — form of severe malnutrition d/t E deficiency; a child will look emaciated + BW will be ≤60% weight fo r age
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What clinical manifestations of refeeding syndrome may appear when tube feeding is started for a patient in a starvation state? A. Diarrhea B. Vomiting C. Seizures D. Abdominal distention
C. Seizures RATIONALE: s a life-threatening metabolic complication resulting from hypophosphatemia, hypokalemia, hypomagnesemia which manifests with seizures, weakness, acute confusion, shallow respirations, and increased bleeding tendency. Patients receiving total enteral nutrition are generally at risk for diarrhea, vomiting, and abdominal distention due to overfeeding.
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Drug therapies for Obesity
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Extrapyramidal Syndrome S/Sx
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Nutritional Algorithm
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Why would enteral nutrition be ordered?
*aka tube feeding* May be ordered for the patient who has a functioning GI tract but is unable to take any or enough oral nourishment. — Stomach — Duodenum — Jejunum
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Reasons/Indications for enteral nutrition
Anorexia Orofacial fractures Head/neck cancer Burns Nutritional deficiencies Neurologic conditions Psychiatric conditions Chemotherapy Radiation therapy
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Delivery options for enteral nutrition/feeding
Continuous infusion by pump (for critically ill patients) Intermittent by gravity Intermittent bolus by syringe Cyclic feedings by infusion pump
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