Module 1 Flashcards
(37 cards)
Risk factors SUD
younger age
Gender: men greater risk
Living alone
Being unemployed
Very high degree of urbaniyation
(Unrelated to income)
(Unrelated to country of origin)
Individual risk factors
Early aggressive behaviour in childhood
Early drug use ->importance of prevention
Individual Protective factors against SUD
Self-efficacy (belief in self control)
Academic performance
Family risk factors SUD
Lack of parental supervision
Substance abuse by caregivers
Protective family factors SUD
Parental monitoring and support
Peer risk factors SUD
Low refusal skills
Poor social skills
Protective peer factors
Positive relationships
School risk factors SUD
Drug availability at school
Protective school factors SUD
School anti-drug policies
Community risk factors SUD
Community poverty
Community protective factors
Neighbourhood resources
Risk addictive potential drugs
Differs between drug + administration through ingesting or injecting
Moral model of addiction
A sign of moral weakness: drug abuse and drug seeking behaviour is immoral and in conflict with social norms and values
Pharmacological model (mid 19th century)
Addicts are not to blame: highly addictive substance is
->prohibitions: prevent people from getting involved with the substances
Symptomatic model (1930-1950)
Not a condition in itself, but rather a symptom of an underlying neurotic character or personality disorder
-> long-term, insight-oriented psychotherapeutic treatment
Disease model (1940-1960)
There are fundamental biological and psychological differences between addicts and non-addicts, making the former unable to use substances in moderation
-> complete abstinence from substance
Learning theory (1960-1970)
A form of maladaptive learned behaviour that can be unlearned again
->behavioural therapeutic interventions
Bio-psycho-social model (1970-1990)
Both the onset and termination are the result of continuous interaction between biological, psychological and social factors
->multi-modal interventions in which attention is on many factors
Brain disease model (1990 - onwards)
Innate vulnerability forms the basis of repeated use and this repeated use leads to important, difficult to reverse, brain changes leading to impaired cognitive control functions and craving habits
-> pharmacological and behavioural therapeutic interventions
Criticism on the brain disease model
- most people beat addiction by very hard effort. We cannot say this about medical diseases
- people take substances out of choice and also have to make a choice to stop taking them. They are not blameless victims of some illness they have no control over.
Homeostatic account (theory of structural changes)
Structural changes are a CONSEQUENCE of addiction:
->chronic overstimulation of dopamine D2 receptors > brain compensate by downregulating these receptors (explains anhedonia/dysphoria)
Reward deficiency syndrome (RDS) account (theories of structural changes)
Addiction is the RESULT of structural differences
-> individuals with less receptors (chronic dopamine deficiency) have lower reward sensitivity > vulnerable for addiction because they look for stronger stimuli to activate the reward system
2 notable differences in how the dopamine system reacts towards (unexpected) natural vs drug rewards
- the reaction of the dopamine system is much stronger for drug rewards
- the response to the CS is stronger for drug rewards than for natural rewards: the peak is higher. > central to temporal difference account
Temporal difference account
Idea that, because the dopamine response to the CS is stronger for drug rewards, drug-related cues and actions would continue to be reinforced too pathological levels