Motion Flashcards

1
Q

motion

A

change in position/location over time

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2
Q

motion aftereffect (MAE)

A

the illusion of motion in a stationary object after prolonged exposure (adaption) to movement

evidence of interocular transfer - can adapt one eye and still get after image in other

opponent process system

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3
Q

apparent motion

A

brain assuming same object is showing up in a different place after it disappeared from another place

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4
Q

2 ways we perceive motion from what we already know

A

motion detection cells
- particular cells taking info from eye, V1, etc. to digest motion

where pathway

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5
Q

periphery is quite good at _______ ______, meaning this part of the LGN is also good at this

A

detecting motion; Magnocellular layer

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6
Q

where the where path ends

A

parietal cortex

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7
Q

___ cells can code for motion

A

V1

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8
Q

issue with having only V1 cells to detect motion and the solution

A

response would be similar for actual motion vs. two separate images in those receptive fields

solution: add additional system to calculate timing

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9
Q

delay cells

A

as they sound, serve to process a delay from cell 1

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10
Q

multiplication cells

A

only fire when D1 (delay cell of cell 1) and cell 2 simultaneously fire

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11
Q

aperture problem

A

when you (or cells) can only see small window (aperture) of the world, it can become hard to know how objects are moving

through window we see one motion while the whole picture shows us a different motion, true with gratings as well

shows us that we can’t rely on a couple of V1 cells to understand motion alone

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12
Q

what part of the brain has an impact on ability to detect global motion when damaged?

A

hMT (human middle temporal cortex)

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13
Q

another term for hMT (bonus points if you know what cortex this can be categorized as)

A

V5; extrastriate cortex (V+#)

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14
Q

akinetopsia

A

motion blindness
- vision is like a set of still images; for some, kind of like strobe lights or more severe. leaves after images!

caused by damage to hMT

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15
Q

some drugs result in ___ __________ (type of motion blindness)

A

acute akinetopsia

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16
Q

first-order motion

A

changes in luminance over space/time

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17
Q

second-order motion

A

changes in contrast or texture where overall luminance stays the same

18
Q

________ motion seems to be particularly priviledged, why?

A

biological; this is because the motor regions of the brain that is activated when recognizing are the same regions that are involved in the action itself

19
Q

optic flow

A

the changing angular position of points as we move through space

20
Q

focus of expansion (FOE)

A

center point from which motion seems to expand outward from

though to be how pilots start the process of landing a plane by focusing on whether the runway is expanding symmetrically

21
Q

time-to-collision (TTC)

A

helps you decide whether something is coming at you/if you need to move out of the way

optic flow gives much of this information

22
Q

tau

A

size of retinal image relative to expansion rate

basically a heuristic estimate of whether an object is rapidly expanding and taking up a lot of retinal image

23
Q

motion induced blindness

A

not full blindness, but motion can cause static parts of an image to seemingly vanish

static dots will wink out

tied to global position

24
Q

motions of our eyes

A

fixations
- when our eyes are focused on one area

saccades
- when our eyes move sharply between points

micro-saccades
- even when we are fixated, our eyes are making small movement adjustments (preventing fading/adaption)

smooth pursuit
- when our eyes steadily follow motion

reflexive eye movements
- automatic eye movements, e.g. when you move your head without changing fixation

vergence
- rotating eyes inward (converge) or outward (diverge)

25
Q

superior/inferior oblique eye muscles

A

control left to right/rotations

26
Q

superior/inferior rectus eye muscles

A

control up and down

27
Q

lateral/medial rectus

A

control back and forth

28
Q

our eye muscles allow for movement but also …

A

communicate that movement to the brain

29
Q

superior colliculus

A

mid-brain structure critical for controlling eye movements

evidence this brain region gets direct input from retinal ganglion cells, skipping the LGN –> V1 pathway

30
Q

eye movements in the brain get information from the ____ pathway and coordinates with _____ _______ _____ (FEF) to guide eye movements

A

where; frontal eye fields

31
Q

is voluntarily moving your eye vs. jiggling it with your finger the same?

A

no

32
Q

saccadic suppression

A

while you make a saccade, your brain stops taking in new information until fixation, particularly through the magnocellular pathway

which means you can miss small amounts of motion that occur while you move your eyes

doesn’t happen when you jiggle your eye

33
Q

benefit of saccadic suppression

A

world doesn’t “smear” as you move your eyes

34
Q

efference copy

A

when an eye movement is executed, signal is sent to not only the eye, but sensory cortices

35
Q

camparator in the visual system

A

taking change in stimuli and change in efference copy to see what is true stimuli/what happened

36
Q

cells in regions like the parietal cortex and frontal eye fields actually remap their …

A

receptive fields in anticipation of an eye movement

37
Q

receptive field remapping allows us to …

A

do motion perception so quickly, can happen in anticipatory fashion

38
Q

2 places motion happens

A

from the world and from our eyes

39
Q

what 2 parts of our brain allow us to efficently track where and how something is moving in our world

A

mid-brain structures (motor and sensory cortex)
where pathway

40
Q

When a pair of lights flicker on and off in opposite
patterns from each other, this can create _______.

a. Saccadic Suppression
b. Apparent Motion
c. An Efference Copy
d. Second Order Motion

A

b

41
Q

What is the earliest part of the brain that, when
damaged, causes deficits in motion detection?

a. LGN
b. V1
c. hMT/V5
d. Parietal Cortex

A

a