Motor control Flashcards

1
Q

Is activation of muscle fibres gradual?

A

No; all or none

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2
Q

how is skeletal muscle attached to bone

A

tendon

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3
Q

describe the structure of skeletal muscle

A

potein filaments make up myofibrils that make up muscle fibres that make up muscle fasiculus which make up muscle

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4
Q

what is a motor unit

A

an alpha motor unit (LMN) and all the extrafusal skeletal muscle fibres it innervates

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5
Q

what is the effect of less fibres innervated by a motor neurone

A

greater variation of movement

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6
Q

what happens when an alpha motor neurone depolarises

A

causes contraction of all fibres in that unit

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7
Q

where are alpha MN that control distal muscles located

A

laterally in spinal cord

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8
Q

where are alpha MN that control proximal muscles located

A

medially in spinal cord

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9
Q

what is the motor plate

A

region of muscle fibre plasma membrane that lies directly under terminal portion of axon

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10
Q

what is a neuromuscular junction

A

junction of an axon terminal with motor end plate

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11
Q

what do stretch receptors do

A

monitor muscle length and rate of change of muscle length

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12
Q

describe the structure of stretch receptors

A

peripheral endings of afferent nerve fibres wrapped around modified muscle fibres - whole apparatus = muscle spindle

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13
Q

what do muscle spindles detect

A

change in muscle resistance to stretch (contraction AND relaxation) regardless of current muscle length

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14
Q

describe the structure of intrafusal fibres;

A

middle 1/3; non, contractile, supplied by fast type 1a afferent sensory nerve
end 2/3s; contractile, gamma MN attached, supplied by slow conducting type 2 afferent

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15
Q

how are muscle spindles attached to extrafusal muscle fibres

A

by connective tissue, in parallel

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16
Q

where are extrafusal fibres found, what do they do and what innervates them

A

skeletal muscle, muscle contraction, alpha MN

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17
Q

where are intrafusal fibres found, what do they do and what innervates them

A

muscle spindle, proprioception, gamma MN

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18
Q

what happens if alpha MN alone is activated

A

increased sensitivity of muscle to stretch

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19
Q

what happens if an alpha AND gamma MN activated

A

prevents central 1/3 of spindle going slack during muscle contraction
- so info about muscle length will be continuously available so can adjust accoridngly

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20
Q

what happens when an external force is applied to an intrafusal fibre

A

pulls on intrafusal, receptor ends activated; the more/ faster muscle is stretched the more receptors fire

21
Q

what happens when an AP arrives at a MN

A

extrafusal contraction so muscle shortens so tension removed from spindle so slows rate of firing of stretch receptor so reduction in sensory information

22
Q

what does tension depend on

A

muscle length
load on muscle
degree of muscle fatigue

23
Q

what do GTO do

A

detect changes in muscle tension

24
Q

where are GTO found

A

junction of skeletal muscle/ tendon

25
what are GTO
endings of afferent fibres that wrap around collagen bundles in tendons near their junction with the muscles
26
what are the afferent fibres that lead from GTO to spinal cord
1b fibres that run to anterior horn of spinal cord
27
what happens when muscle is stretched/ attached extrafusal muscle fibre contracts (regarding GTO)
tension exterted on tendon so straightens the collagen bundle so distorts GTO receptor endings so activating them
28
what do 1b fibres do
cause inhibition of a MN of contracting muscle and its synergists
29
why are 1b fibres important
regulate muscle tension at normal range and protect it from overload
30
what is the output of GTO
proportional to muscle tension
31
are GTO afferent fibres faster or slower than muscle spindles
slower
32
what does 1b input do
excites alpha MN
33
what does the afferent from GTO do
inhibits alpha MN
34
what is muscle tone
degree of contraction of muscle/ proportion of Motor units that are active at any time
35
what does high muscle tone look like
feels firm/ rigid, resists passive stretch
36
what does low muscle tone look like
feels soft/ flaccid, offers little resistance to passive stretch
37
what is the contribution of alpha MN activity in a relaxed person
doesn't make significant contribution to resistance to stretch
38
what is the contribution of alpha MN activity when someone is increasingly alert
more activation of alpha MN so muscle tone increases
39
what is hypertonia
high muscle tone
40
what causes hypertonia
UMN disorder; disorders of descending pathways which normally inhibit MN
41
how can you demonstrate hypertonia and what is seen
when joint is moved passively at high speed; increased resistance is due to increased level of alpha MN (LMN) activity which keeps muscle contracted despite attempt to relax it
42
what is spasticity
type of hypertonia muscles don't develop increased tone until they are stretched a bit, then after a brief increase in tone contraction subsides after a short time
43
what is rigidity
type of hypertonia | increased muscle contraction is continual and resistance to passive stretch is constant
44
what is hypotonia
low muscle tone accompanied by weakness, atrophy and decreased/ absent reflex response
45
what causes hypotonia
LMN lesion, neuromuscular junctions, muscles themselves | commonly accompanies disorders of alpha MN
46
what is clasp-knife phenomenon
type of spasticity (hypertonia) period of 'give' occurring after time of resistance - especially when someone bends limb of patient; initially some resistance but after some time resistance falls dramatically
47
what causes clasp-knife phenomenen
1b afferent fibres from GTO inhibiting alpha MN once GTO detect tension - example of inverse stretch (myotatic) reflex
48
when would you expect to see clasp-knife phenomenon
UMN lesion