motor recovery Flashcards

(32 cards)

1
Q

mm synergies

A

firing of several mm groups simultaneously

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2
Q

normally we move in numerous __ movement patterns

A

variable

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3
Q

after stroke we move in __ ways

A

stereotypical

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4
Q

stereotypical snergies

A

initial: flaccid
followed by: spasticity, hyperreflexia , stereotypical movements
-inflexible, invarient

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5
Q
UE flexor synergy pattern 
scapula
shoulder
elbow
wrist
flexrors thumb
A
elevates/retracts
abd/ER
Flex/sup
flex
flex/add
flex/add
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6
Q

LE extensor synergy

Hip
knee
ankle
great toes

A

adducts/ IR
extends
PF/Inversin
extend

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7
Q

strong components of flexor synergy

A

elbow flexion

hip flexion

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8
Q

strong components of extensor synergy

A

shoulder adducts/ IR

knee extends

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9
Q

Brunnstrom stages of motor recovery

A

1-6

flaccid to spastic to normal

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10
Q

chedoke stroke assesemtn

A

1-7

flaccid to spastic to normal

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11
Q

brunstrom stages with no voluntary movement

A

stage 1 /2

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12
Q

brunstrom spastic movement levels

A

3-6

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13
Q

Motor recovery stage 1

UE

A

flaccid
hyporeflexic
no active reflex / mm

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14
Q

motor recovery 2

UE

A

spasticity
reflex response to facilaition
no voluntary mm

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15
Q

motor recovery stage 3 UE

A

spasticity marked
synergistic movement can be elicited volunitariy
pattern of movement within limited range

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16
Q

motor recovery stage 4 UE

A

spasticity decrease
less resistance to passive
synergy pattern can be reversed
movement possible within greater range

17
Q

motor recovery stage 5

A

spasticity wanes but evident w rapid movement
able to reverse patterns
isolated movement possible
specific movements

18
Q

motor recovery stage 6

A

no resistance to passive movemnt
controlled isolated movement
lacks normal speed

19
Q

chedoke stage 7

A

normal movement, normal speed

no evidence of functional impairment

20
Q

which one has stage 7 chedoke or fugl

21
Q

3 purposes of chedoke

A

stage the recovery
predict outcome
measure clinical important change

22
Q

chedoke 2 inventories

A

physical (shoulder posture arm, hand leg foot)

and activity (gross and walking)

23
Q

associated reactions

A

unintentional involuntary movements caused by lack of inhibition from CNS

can become conditioned

can occur with voluntary action of another limb

24
Q

post stroke shoulder pain from

A
snap malignment
IR humerus
neglect
sensory loss
UE low stage in recover CMSA 123
25
is shoulder sublaxation common? preventable
yes n yes
26
relationship between shoulder subluxation and pain direct or not
not direct
27
can should sublax if increased mm tone
yes
28
how to prevent shoulder sublax
support it education joint approximation maintain ROM (safe below 90)
29
contributing factors to shoulder sublaxation
lack of support in upright / standing pulling on hemiplegic during transfers improper positioning in bed / wc
30
joint approximation
mm stimulation strengthening braces taping
31
complication of spastic UE
``` adhesive capsulitis decrease functional movement loss of scapular mobility loss of humeral alignment contracture/ loss ROM ```
32
best practice for shoulder PROM Arm
do not move shoulder past 90 of shoulder flex / abd UNELSS scapular upwardly rotated and humerus laterally rotated