Paeds Flashcards

(171 cards)

1
Q

Give 5 members of paeds haemophilia team

A
GDP, 
paediatric dentist
haematologist
haem specialist nurse
social workers
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2
Q

How is severity of haemophilia assessed

A

% of clotting factors
mild/mod/severe
6-40%/ 2-5%, <1%

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3
Q

How would you treat a patient with moderate haemophilia?

A

enhanced preventative care and rx with GDP/CDS

all tx in hospital setting w/ specialist

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4
Q

Two clinical manifestations of van Willebrands disease

difference between type I and type II vWb disease?

A

mucocutaneous haemotoma
gingival bleeding
post XLA bleeding

type I: quantitative effect on van Willebrand’s factor - loss to 20-50%.
type II: qualitative effect on vW factor
both autosomal dominant
type III: also quantitative effect- autosomal recessive

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5
Q

4 dental procedures that require no augmentation to coagulation factors

A

examination
flouride varnish
small occlusal restorations
supragingival scaling

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6
Q

4 management strategies for patients requiring augmentation of coagulation factors

A

coagulation factor replacement
DDAVP - desmopressin
antifibrinolytics- e.g tranexamic acid
local haemostatic measures

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7
Q

Give 1 possible complication of recombinant clotting factor therapy

A

antibody resistance

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8
Q

3 side effects of desmopressin use

A

hyponatraemia - low serum sodium levels
nausea
diarrhoea
headaches

tachycardia
desmopressin is man made vasopressin (Antidiuretic hormone) used for diabetes, bed wetting, haemophilia A and vWD

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9
Q

Risk of nerve blocks in patients with bleeding disorders

Give one alternative to nerve block

A

muscular haemotoma
- blood could flow into sublingual, submandibular, pterygoid spaces

alternative: articaine infiltration

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10
Q

Max INR for safely treating paediatric patients

A

2.5

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11
Q

When should you prescribe prophylactic antibiotics

A
immunocompromised
hypertrophic cardiomyopathy 
previous infective endocarditis 
valve replacement
cardiac stent
adjunct to tx
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12
Q

6 extra oral features of Down Syndrome

A
rounded skull
small midface
atlantoaxial instability
dysplastic ears
brushfield spots
short, broad neck
dry lips
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13
Q

6 intra oral features of Down Syndrome

A
macroglossia
AOB
microdontia
hypodontia
fissured tongue
delayed eruption
class III occlusion
bifid uvula
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14
Q

4 medical conditions related to Down syndrome

A
cardiovascular defects esp. VSD
cleft lip and palate
deafness
hypothyroidism
mental retardation
acute lymphoblastic leukaemia
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15
Q

Restorative options for pt with Down Syndrome

A

GI restorations- difficult to achieve moisture control
CHX gel or MW
LA if able. GA risk due to atlanto axial instability

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16
Q

What percentage of paediatric patients suffer from asthma?

A

7-19% in the UK

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17
Q

What is asthma?

A
reversible airway obstruction
caused by:
- smooth muscle contraction
- inflammation in respiratory mucosa
- excess mucous secretion
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18
Q

Give 4 signs/symptoms a patient with asthma might display

A

shortness of breath
wheezing
rash
coughing

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19
Q

What medications would you expect a patient with asthma to be taking?

A

corticosteroid inhaler - brown- beclomethasone - preventer

beta 2 agonist inhaler - blue - salbutamol - reliever

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20
Q

How do asthma medications contribute to tooth wear

A
  • xerostomia - .:. sugary/acidic drinks used to aid
  • relaxed lower oesphageal sphincter- gastric reflux
  • inhaler is acidic
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21
Q

What are the dental effects of inhalers and what advice should be given

A
  • candidosis
  • erosion - from intrinsic (GORD) and extrinsic (acidic medication and sugary drinks)
  • xerostomia

advice:
try to rinse with water after every inhaler use,
use spacer is possible

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22
Q

What other considerations should be given to asthma patients?

A
  • –pt will have increased atopy (genetic tendency for allergic reactions) .:. increased chance of allergic reactions - COLOPHONY in fluoride varnish
  • –increased risk of adrenal suppression - from inhaled corticosteroids inhaler
  • –med emergencies risk
  • –SEDATION risk
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23
Q

How is asthma severity established?

A

last hospitalisation
clinical symptoms
measurements- peak expiratory flow, forced resp volume, oxygen saturation

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24
Q

What is cystic fibrosis?

incidence?

A

chromosomal abnormality affected chr7
production of xs thick mucous affecting lungs, pancreas and salivary glands

affects 1 in 2500

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25
Signs and symptoms cystic fibrosis
``` recurrent respiratory infections low weight cough wheeze shortness of breath thickened saliva ```
26
4 intraoral manifestations of cystic fibrosis
thickened saliva ENAMEL DEFECTS delayed eruption increased calculus
27
dental considerations of cystic fibrosis
sedation contraindicated diet advice - high cal, probably sugary diet due to nutritional deficiency ohi - carry toothbrush everywhere antibiotic resistance and prescribing- recurrent infections diabetes and liver disease
28
why is inhalation sedation contraindicated in cystic fibrosis
high oxygen supplementation causes decreased respiratory rate .:. ability to clear secretions reduces in long sedated periods
29
incidence of cancer in u15 y/o
1/600
30
list 5 most commmon childhood cancers
``` leukaemia lymphoma brain tumour wilm's tumour (kidneys) neuroblastoma ```
31
3 tx modalities cancer
chemo radio surgery
32
4 acute intra oral complications cancer tx
infections haemorrhage xerostomia mucositis
33
what is mucositis?
ulceration of all mucosa types in oropharynx/ digestive tract usually as a result of chemotherapy
34
grading of mucositis?
0 none 1 soreness, erythema 2 ulceration, erythema - solid foods tolerated 3 ulceration, erythema - liquid foods only 4 oral alimentation impossible - life threatening
35
mgmt strategies mucositis?
- general- avoid spicey foods, no strong MW - smooth cusps/ sharp edges on restorations - low level laser light therapy - stimulate collagen production - analgesia in form of lignocaine 2% solution - enhanced oh
36
a paediatric patient undergoing chemo for leukaemia presents with bleeding gums. why would this be? what ohi to help? give 3 local and 1 systemic haemostatic options
platelets levels low - <20-30 x10(9)/L avoid toothbrushing. use CHX on swab to clean local: pressure & gauze, ice, topical thrombin systemic: platelet infusion
37
minimum platelet count for injections/xla/scaling
>80 (40-100) x10(9)/L
38
give 3 ways chemotherapy renders patients more susceptible to infection
- inhibits antibody response - abolition of delayed hypersensitivity - neutropenia
39
suppurating pocket assoc w/ 16. how to decide tx?
culture | sensibility testing
40
What is aplastic anaemia?
disease of bone marrow and the hematopoietic cells residing within - causes pancytopenia.
41
4 intra oral signs of aplastic anaemia?
ulceration haemorrhage infection mucosal pallor
42
4 factors to consider before tx of aplastic anaemic?
increased infection risk haemorrhage increased risk of SCCa anaemia
43
what is GVHD? 4 intra oral signs
Graft Versus Host Disease where immune cells from graft tissue recognise host as foreign. a frequent and serious complication following hematopoietic stem cell transplantation (HSCT) lichenoid tissue reaction - can be reticular or erosive xerostomia - due to salivary glands affected .:> increased caries rate limited mouth opening ulceration - can appear as canker sores mucoceles erythema
44
how might a child with renal failure appear at presentation? (3)
underweight - failure to thrive pallid fatigued
45
give 6 intraoral features of a child with renal failure?
GGEEPP ``` gingivitis gingival hyperplasia enamel hypoplasia excess plaque accumulation petechiae pulp obliteration ```
46
when should dental tx be carried out for children with renal failure?
day after dialysis
47
implications of organ transplant for dental tx? (4)
cyclosporin immune suppressant -> gingival hyperplasia corticosteroids -> pulp obliteration bleeding disorders -> platelet dysfunction. care with la- haematoma likely in pterygoid region long term immunosuppression -> poorly controlled infections osseous lesions in jaw
48
5 early clinical features of diabetes
``` polyuria acetone breath irritability weight change thirst fatigue ```
49
5 late clinical features of diabetes
``` dehydration renal dysfunction hypovolaemic shock nausea vomiting ```
50
5 oral manifestations of diabetes
``` xerostomia progressive periodontitis caries candida infection oral neuropathies ```
51
3 features of a hypoglycaemic attack
``` strong, bounding pulse clammy skin confusion hunger nausea ```
52
3 features of hyperglycaemic attack
``` weak pulse dry skin acetone breath frequent urination rapid breathing ```
53
prevalence of epilepsy in children
1/250 | ~0.5%
54
3 intraoral signs of epilepsy
trauma to teeth soft tissue trauma gingival hyperplasia (phenytoin) recurrent oral ulceration
55
what is haemolytic anaemia?
type of anaemia with lysis of erythrocytes (RBCs) | can be intravascular/extravascular
56
how can haemolytic anaemics be diagnosed?
peripheral blood smear
57
3 categories of haemolytic anaemia and eg of each
membrane defects - spherocytosis enzymatic defects - G6PD structural defects - Sickle cell disease, thalassaemia
58
how might a patient with haemolytic anaemia present? (6)
``` pallid fatigued ascites underweight oedema of legs jaundiced shortness of breath FTT failure to thrive ```
59
6 oral related signs of sickle cell disease
``` anaemia ulceration smooth tongue jaw joint pain impaired growth hypercementosis skeletal deformities osteoporosis of jaw ```
60
4 dental implications of sickle cell disease
poor infection control post op antibiotics prevention inhalation risk
61
6 orofacial manifestations of thalassaemia?
``` squirrel like appearance large cheekbones depressed nasal ridge class II malocclusion maxillary protrusion candidosis gingivitis high caries incidence ```
62
6 intraoral manifestations of HIV
``` oral ulceration salivary gland enlargement hairy leukoplakia hepetic stomatitis recurrent candida infections kaposi sarcoma xerostomia ```
63
How is HIV diagnosed
``` ELISA enzyme - linked immuno- sorbent assay ```
64
Current management of HIV
HAART triple therapy ``` highly active anti retroviral therapy ```
65
most common cause of anaemia in children? 4 intraoral signs of this anaemia
iron deficient anaemia ``` glossitis - inflammation causing sore, smooth looking, depapillated tongue candida infections apthous ulceration pale mucosa angular cheilitis ```
66
what type of anaemia does i)b12 ii)fe iii) folate deficiency induce in pts
macrocytic microcytic macrocytic
67
how does the clinical presentation of the tongue differ from patients suffering from iron and b12 deficient anaemias
iron: large, red, smooth, depapillated b12: fiery red, atrophic, sore, beefy
68
gi disease presenting with pigmented lesions in oral cavity and on lips
Peutz Jeghers | brown macules 2-5mm on lips, oral cavity and face
69
``` what is gardner's syndrome intraoral presentation (3) ```
colonic polyposis supernumerary teeth mandibular cysts
70
what is coeliac disease? | 3 intraoral presentation
lifelong autoimmune condition as reaction to gluten apthous ulceration mucosal erythema chronological hypoplasia depapillation of tongue
71
what is crohns disease | 5 intraoral presentation
inflammatory bowel disease (IBD) affecting any part of GI tract ``` full thickness gingivitis cobblestoned mucosa mucosal tags ulceration fissured tongue ```
72
4 strategies to manage intraoral symptoms of crohns
dietary advice steroids- topical/ systemic. betamethasone, hydrocortisone pellets 2.5mg non-cinnamon containing toothpaste correct anaemic deficiencies
73
child brought in by non-parent. what do you need to establish prior to examination?
thorough history inc medical and drug history | consent- if carer has consent, situation, where consenting adult is
74
describe a behavioural management technique name 4 other techniques
tell show do Tell: tell the patient what you’re going to do, Show: show the patient what you’re going to do, Do: get the patient, Plan: long term ``` others: desensitisation modeling distraction parental presence positive reinforcement voice control ```
75
How would you address the issue of non attendance of a child?
- ensure contact details are up to date - explain to parent necessity of attendance of pt and of consenting guardian presence - inform parent of need for child protection involvement in cases of non- compliance - arrange next appt on phone - record everything fully in notes
76
evidence based toothbrushing advice?
2-5 mins / 2 x daily pea sized amount/ 1450ppm toothpaste modified bass technique/ 45 degree angle to gingival margin and brushing away from margin
77
What does a BPE score of 3 mean?
probing depth of 3.5-5.5mm
78
pt attends with fracture of 11. history?
account for missing fragment - swallowed/inhaled/embedded in soft tissue how it happened when it happened
79
List 4 things that determine prognosis of fractured tooth
- type of fracture - mobility - vitality - maturaturity of tooth - open/closed apex
80
Pt presents with yellow/white/brown stains on teeth. Differential diagnosis? How would you differentiate?
amelogenesis imperfecta fluorosis molar incisor hypomineralisation AI - affects all teeth, familial aggregation, on radiograph molars can appear taurodont F- affects all teeth, history of high fluoride in childhood- ingestion/ water. white opacities caries resistant MIH- affects 1s and 6s only. susceptible to caries. well demarcated opacities
81
What questions would you ask parent of child with white/yellow/brown staining?
- childhood systemic illnesses? - measles, rubella, upper respiratory tract infections - pregnancy- did mother suffer from any systemic illnesses prenatally? - premature delivery? - prolonged breast feeding? - traumatic birth? - medications taken? - natural birth?
82
Signs fluorosis? aeitology? how to quantity severity? tx options?
``` varying severity white flecks opaque white lines pitting brown spots ``` quantity of fluoride intake, type of intake, renal function, metabolic function. long term ingestion of high fluoride, fluoridated water of >1ppm Dean's index ``` micro/macroabrasion bleaching composite restorations veneers full crowns ```
83
implications of MIH
- increased sensitivity - increased caries susceptibility - behavioural problems & amp anxiety - difficulty achieving anaesthesia - difficulty restoring lesions - poor aesthetics
84
5 groups of aeitiologies of MIH
- pre/peri and neonatal problems - exposure to environmental contaminants - exposure to fluoride - common childhood illnesses - medically compromised children
85
5 tx options impacted first molars
``` Leave and monitor coronectomy discing of Es orthodontic fixed appliance XLA molar XLA E ```
86
Features of permanent dentition that allow for replacement of primary teeth without crowding?
- Growth of maxilla - Primate space- physiological spacing between primary teeth - proclination of permanent teeth - Extension of dental arch - Leeway space-
87
Leeway space
combined mesiodistal width of primary canine, first and second molar is greater than the combined mesiodistal width of the permanent canine, first and second premolars
88
pt suffering from extrusion. tx
extrusion splint cut and manipulate passive splint (SS) clean and dry area acid etch areas to be splinted fix with composite resin follow up 2 weeks, 4 weeks, 6-8 weeks, 6 months, 1 year, 5 years clinical exam and radiographs, sensibility testing, mobility, TTP, check colour and sinus
89
On a follow up visit post- trauma a radiograph reveals an irregular apical structure. what could be causing this? what else would you expect to see? tx?
external inflammatory root resorption widened pdl loss of lamina dura RCT, CaOH dressing apical surgery
90
avulsion of permanent incisor. pt on phone, what to do.
reassure pt do not handle by root rinse under cold running water for 10s reimplant ASAP (check is definitely not primary tooth) or store in saliva, milk, Hank's saline and visit GDP
91
pt arrives in practice. reimplanted mature tooth closed apex. what to do
leave tooth in place clean area w/ water spray suture gingival lacerations if present verify normal positioning of tooth clinically and rads apply flexible splint- 2 weeks prescribe ABs doxycycline bid/ 7 days - amoxycillin if pt under 12 years old (discolouration risk with tetracyclines) tetanus booster- if uncertain or immunisation/soil contact RCT 7-10 days after reimplant CaOH placed as intracanal medicament for up to a month before obturation. OR antibiotic corticosteroid (odontopaste)
92
patient instructions following reimplant of avulsed tooth
avoid contact sports CHX rinse bid/7days soft food for up to 2 weeks brush w soft toothbrush after each meal
93
what to do if pt presents w/ mature avulsed tooth in storage medium
if saliva, milk or Hank's balanced salt solution and EADT extra oral dry time of <60 minutes can reimplant - clean root surface and apical foramen with stream of saline- remove debris and dead cells - administer LA - irrigate socket with saline - examine socket - if socket wall fracture position this with suitable instrument - reimplant tooth with digital pressure, no force then as before--- suture gingival lacerations if present verify normal positioning of tooth clinically and rads apply flexible splint- 2 weeks prescribe ABs doxycycline bid/ 7 days - amoxycillin if pt under 12 years old (discolouration risk with tetracyclines) tetanus booster- if uncertain or immunisation/soil contact RCT 7-10 days after reimplant CaOH placed as intracanal medicament for up to a month before obturation. OR antibiotic corticosteroid (odontopaste)
94
If pt presents with mature avulsed tooth and EADT >60 minutes what do you do
can do delayed reimplantation - to restore aesthetics, function and maintain alveolar bone contour ankylosis expected + root resorption. soaking in sodium fluoride solution thought to help slow down process -Clean with saline and remove any non-viable soft tissue carefully -RCT can be carried out extraorally or 7-10 days post reimplantation -LA - irrigate socket with saline - examine socket - if socket wall fracture position this with suitable instrument - reimplant tooth with digital pressure, no force -suture gingival lacerations if present =verify normal positioning of tooth clinically and rads -apply flexible splint- 4 weeks -prescribe ABs doxycycline bid/ 7 days - amoxycillin if pt under 12 years old (discolouration risk with tetracyclines) -tetanus booster- if uncertain or immunisation/soil contact
95
If pt presents with avulsed immature tooth reimplanted...
leave tooth in situ clean area w/ water spray suture gingival lacerations if present verify normal positioning of tooth clinically and rads apply flexible splint- 2 weeks prescribe ABs doxycycline bid/ 7 days - amoxycillin if pt under 12 years old (discolouration risk with tetracyclines) tetanus booster- if uncertain or immunisation/soil contact GOAL revascularisation at apex if does not occur need to RCT - if evidence of pulp necrosis follow up - 2 weeks, 4 weeks, 3 months, 6 months, 1 year , 5 year
96
if pt presents with avulsed immature tooth with extraoral dry time <60 minutes...
need to: weigh up risk of revascularisation versus infection related root resorption if saliva, milk or Hank's balanced salt solution and EADT extra oral dry time of <60 minutes can reimplant - clean root surface and apical foramen with stream of saline- remove debris and dead cells - AT THIS POINT- thought to aid revascularisation if application of topical antibiotics (doxycline soak 1mg per 20ml for 5 minutes prior to reimplantation). - administer LA - irrigate socket with saline - examine socket - if socket wall fracture position this with suitable instrument - reimplant tooth with digital pressure, no force then as before--- suture gingival lacerations if present verify normal positioning of tooth clinically and rads apply flexible splint- 2 weeks prescribe ABs doxycycline bid/ 7 days - amoxycillin if pt under 12 years old (discolouration risk with tetracyclines) tetanus booster- if uncertain or immunisation/soil contact AVOID RCT UNLESS EVIDENCE OF PULP NECROSIS
97
if pt presents with avulsed tooth immature and eadt >60 minutes
can do delayed reimplantation - to restore aesthetics, function and maintain alveolar bone contour ankylosis expected + root resorption. soaking in fluoride solution thought to help slow down process -Clean with saline and remove any non-viable soft tissue (necrotic PDL) carefully -RCT can be carried out extraorally or 7-10 days post reimplantation -LA - irrigate socket with saline - examine socket - if socket wall fracture position this with suitable instrument - reimplant tooth with digital pressure, no force -suture gingival lacerations if present =verify normal positioning of tooth clinically and rads -apply flexible splint- 4 weeks -prescribe ABs doxycycline bid/ 7 days - amoxycillin if pt under 12 years old (discolouration risk with tetracyclines) -tetanus booster- if uncertain or immunisation/soil contact
98
eruption dates permanent teeth
``` 6 y/ o - U6s, L6s erupt, L1s begin to 7 y/o - L2s and U1s 8 y/o - U2s 9 y/o - L3s, palpate for Us 10 y/o - U4s, L4s, U5s 11 y/o - L5s, U3s, L7s 12 y/o - U7s 17+ - U+L8s ```
99
Main difference in splinting times and why?
flexible splint for 2 weeks if EADT <60 mins | for 4 weeks if EADT >60mins
100
common outcomes of avulsed teeth (5)
``` discolouration pulp necrosis ankylosis mobility root resorption ```
101
How do osteogenesis imperfecta and dentinogenesis imperfecta relate?
1/2 of patients with osteogenesis imperfecta will suffer from dentinogenesis imperfecta- whilst the other half will have unaffected teeth and just require routine care.
102
What is osteogenesis imperfecta dental implications general signs
brittle bone disease!! lots of different types- range in severity and effects OI always associated with bone fragility. Can affect growth of the jaws. OI does have dental implications though - - class III malocclusion - anterior open bite - impacted teeth - altered dental development ``` general signs: blue sclera of eye multiple bone fractures short height large head easy bruising range of bone deformities- scoliosis ```
103
Mum brings in baby with one erupted tooth gray/blue in colour. What could it be? Any signs to look for if more teeth present?
DI - can occur alone or with OI. enamel is normal but dentine and DEJ is abnormal. Enamel cracks away from dentine, and dentine can grow into pulp chamber .:. - discolouration of tooth, - reduced feeling in tooth - high caries susceptibility - bulbous crowns - slender shorter roots. - pulp chamber obliteration - occult abscess (no obvious signs)
104
Difference between flexible and rigid splint?
flexible - 1 tooth either side | rigid - 2 teeth either side of trauma
105
4 types of healing after root fracture which type would give most poor prognosis?
interposition of the following can occur at the fracture line: - of calcified tissue - of both calcified and connective tissue - of connective tissue - of granulation tissue poor prognosis: granulation tissue would show loss of vitality of tooth and pulpal necrosis/ infection causing an inflammatory response
106
mgmt of root fractures
undisplaced- soft diet and monitor | displaced- LA, reposition, splint
107
root fracture what is it? classification?
fracture of dentine, pulp and cementum horizontal or vertical horizontal - number, location, position, extent. e.g simple apical displaced total horizontal root fracture vertical - separation and position e.g complete intraosseous vertical root fracture
108
History to take after traumatic event SI
``` time and place of event reason for injury any previous dental injuries any pain/ spontaneous any other associated injuries/symptoms- nausea/loss of consciousness/headache systemic health review ``` SI: sensibility testing - can be transient loss of pulp vitality +pulse oximeter radiographs
109
indicators of positive healing in root fractures (5)
``` assymptomatic continued positive pulp testing signs of healing between root fragments absence of apical periodontitis continued root development in immature teeth ```
110
if tooth starts to discolour- yellowy after root fracture what is it
v common finding - 2/3 of root fractures will have partial or complete pulp obliteration coronal pulpal obliteration caused by revascularisation of pulp apical pulp obliteration seen in case of interposition of calcified tissue and those with interposition of calcified and connective tissue.
111
tx for root fracture with pulpal necrosis
long term CaOH therapy followed by conservative RCT within 7-10 days for mature apex for immature apex apexification to be completed first the RCT CaOH has weakening effect on tooth .:. MTA can be used for horizontal root fractures for faster healing
112
contributory factors to pulpal necrosis in root fractures
displacement of coronal fragment use of rigid splints mature apex at time of fracture marginal periodontitis
113
aetiology of vertical root fractures prognosis
extensive restorative tx endodontic tx parafunctional habits can treat e.g guided tissue regeneration or fixation and application of MTA but normally XLA
114
advantages and disadvantages of non vital bleaching
:) easy safe conservative no lab assistance needed (walking bleach technique) ``` :( brittle teeth external cervical resorption relapse may fail over-bleaching ```
115
walking bleach technique
pre-op shade photographs radiographs to check RCT ``` dam gain access remove restoration GP removed to gingival margin ensure good coronal seal 10% carbamide peroxide soaked cotton pledget placed in cavity sealed with GIC pt to return every 2 weeks for up to 10 replacements/ happy with shade then nsCaOH placed to reverse acidity then final restoration ```
116
child ingested toothpaste | 3 questions to ask pt
conc of toothpaste amount swallowed age/weight of child
117
toxic dose fluoride tx if ingested
potentially toxic 5mg/kg a&e immediately oral calcium
118
most common cause of fluorosis in uk
fluoride in water supply
119
family w/3 children aged 1,4,7. live in area with 0.3ppm water supply F-. what supplementation would you suggest
for 1 y/o - fluoride drops 0.25mg for 4 y/o - fluoride chewable tablets 0.5mg for 7 y/o- fluoride MW 225ppm. 1mg needed
120
3 y/o pt presents with blisters on gums. dx? any other features you'd expect tx
primary herpetic gingostomatitis - contagious - herpes simplex virus type 1 - 6 month - 6 y/o - most common viral infection of mouth ``` symptoms: blisters 1-3mm in size painful, erythematous gingiva ulcers on gingiva, lips,mucosa halitosis malaise refusal to eat high fever headache irritability ``` tx: supportive only- reassurance, rest, fluids appropriate analgesia OHI - CHX advise parent of contagious nature- childs eyes refer if concern re: refusal to eat/
121
issues caused by herpes simplex virus reactivation
herpes labialis - cold sores (15-30%) | bell's palsy
122
topical effects of fluoride
promotes remineralisation hydroxyapatite-> fluorapatite bactericidal decrease acid production of plaque
123
effects of primary tooth trauma on a primary tooth
discolouration delayed exfoliation infection loss of vitality
124
effects of primary tooth trauma on a permanent tooth
``` delayed eruption enamel defects arrested development ectopic placement abnormal morphology ```
125
eruption dates primary dentition
Upper: 7, 9, 18, 14, 24 months Lower: 6, 8, 16, 12, 20 months
126
eruption dates permanent dentition
Upper: 7, 8, 11, 10, 10, 6, 12 years Lower: 6, 7, 9, 10, 10, 6, 12 years
127
factors that increase index of suspicion of child welfare in injured child
``` delay seeking tx injuries not matching story inconsistent story contradictory info from child abnormal behaviour/mood child abnormal behaviour parent previous history of injury ```
128
orofacial injuries suspicious of mistreatment
cigarette burns hand/finger marks bites ear/neck injuries
129
how to refer pt on if concerned about welfare
A - Assess H - History E - Examination T - Talk to the child and parent about concerns (if you feel safe) D - Document: everything and in the patients own words M - multiagency (Contact senior colleague and/or e.g. dental protection for advice.) 48 R - refer: Child protection services for information. Social services for referral. Ensure you follow up within 48hours. If possible take photographs with permission. Consider contacting police if you feel child is in immediate danger.
130
indications pulpotomy(6)
``` Avoid XLA, co-operation, space maintenance, caries 2/3 into dentine, exposure >1mm, marginal ridge destroyed, avoid GA, MH precludes XLA, no permanent successor. ```
131
contraindications pulpotomy (6)
``` congenital heart disease immunosuppression suppurating pulp chamber >2/3 root resorption severe pain severe infection severe bone loss space management ```
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Why and how would you carry out pulpotomy?
Indication: exposure of bleeding pulp with no previous symptoms and no clinical/radiological sings of infection. e.g traumatic exposure or large iatrogenic exposure LA and dental dam, Access → remove caries and unroof pulp chamber, Amputate coronal pulp w/ excavator/slow speed, irrigate and dry pulp, control haemorrhage (20s ferric sulphate), assess pulp (→if abnormal bleeding then pulpectomy), Restore→ CaOH/MTA at stumps, GIC luting cement, SSC.
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indication and tx pulpectomy
indication: exposure of non-bleeding or severely hyperaemic pulp, irreversible pulpitis or clinical/radiological signs of periapical periodontitis or acute abscess. LA and dental dam, Access → remove caries and unroof pulp chamber, Amputate coronal pulp w/ excavator/slow speed, remove radicular pulp using barbed broaches. WL from radiograph, file canals 2mm short of apex (important not to go past apex - tooth germs), irrigate w/ CHX, dry canals, obturate with CaOH and iodoform paste (Vitapex), restore with GIC and SSC. vitapex is premixed CaOH and iodoform paste used as temp or permanent root canal filling post pulpectomy
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WHat is AI Give 4 types of amelogenesis imperfecta
disorder of tooth development hypoplastic hypocalcified hypomaturational mixed w/ taurodontism
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Signs and symptoms AI
teeth that are: small discoloured pits/ grooves prone to rapid wear/breakage
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Cause AI
genetic mutation of enamel extracellular matrix molecules e.g amelogenin, enamelin
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Mgmt AI
``` enhanced prevention composite veneers fissure sealants SSC metal onlay ```
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Dental implications AI
``` aesthetics sensitivity caries/acid susceptibility delayed eruption AOB ```
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4 y/o presents. gross caries across anteriors inc smooth surface cause tx
nursing bottle caries cariogenic drinks left in bottle for child to feed on overnight ``` complete/ partial caries removal +/-. temporisation GIC or if severe- XGA Fluoride varnish 4xyear 22600ppm consider supplements advice: ``` ``` avoid taking bottle to bed advise against on demand feeding milk and water only between meals sugar free swaps of foods not soya unless on medical grounds spit dont rinse when toothbrushing, pea sized 1450 w/ parental supervision ```
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3 types of dentinogenesis imperfecta
1. associated with osteogenesis imperfecta 2. autosomal dominant 3. brandywine
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Dental implications dentinogenesis imperfecta
``` aesthetics caries occult abscess poor long term prognosis root fracture ```
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indications SSC
``` >2 surfaces affected/ broken down tx for severe MIH defects post pulpotomy/pulpectomy space maintainer marginal ridge breakfdown ```
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how to place SSC conventionally
- consent - LA, dam - tooth prep - 1mm removal occlusal w/flat fissure bur, clear contacts w/ fine taper bur - crown selection - measure MD, adjust w/ band forming pliers - isolate and dry tooth - mix GIC - seat lingually, snap buccally - look for gingival blanching - remove XS cement w/ probe - check contacts and occlusion
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how does hall technique differ to conventional crown placement
no LA no toothprep no caries removal
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how would you judge a crown has failed
``` secondary caries pulpitis crown lost crown rocking abscess ```
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advantages of planned extraction of FPMs indications of suitability for planned extractions of FPMs
spontaneous space closure reduction in possible ortho need caries free dentition bifurcation of 7s forming class I incisors mild buccal crowding
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2 disadvantages of planned XLA
bad experience GA if goes wrong difficult achieveing anaesthsia
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most common cardiac defect in children? which condition is this highly associated with mgmt of these pt
ventricular septal defect -where connection between two ventricles assoc/w/ down syndrome mgmt: prevention to avoid tx. OHI to minimise endocarditis risk. xla rather than pulptx. consult with cardiologist. avoid sedation refer to specialist care.
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mgmt of external inflammatory resorption
Extirpate the pulp and carry out chemomechanical disinfection. Place non setting CaOH dressing. Reassess to see if resorption has stopped. If yes, complete endo, if not continue w/dressing and plan replacement.
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7 factors making up prevention plan
1. OHI/ tooth brushing instruction 2. Diet Advice 3. Fluoride toothpaste 4. F varnish 5. F supplements 6. Fissure sealants 7. Radiographs 8. Sugar free medication.
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fluoride supplementation in areas of <0.3ppm F-
1mg/day fluoride tablets
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name 3 sources of fluoride found in food and drink
beer tea cucumber bony fish
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clinical trauma review (trauma stamp) (8)
``` colour, displacement (ONLY AT 1st VISIT), mobility, sinus/tender in sulcus, EPT, Ethyl chloride, TTP, Radiographs. ```
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cause of external inflammatory root resorption? clinical signs? mgmt?
infected/necrotic pulp release bacterial toxins, which travel up dentinal tubules and stimulate osteoclasts to resorb the root .:. cementum and bone resorption. mobility, negative sensibility testing, may be TTP moth eaten appearance of PDL and root on RADS RCT, dress with CaOH and review every 3 months. if progressing continue with CaOH. if abating obturate ad monitor
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indications for microabrasion
``` fluorosis ortho decal trauma pre-veneer to mask staining MIH - with predominantly brown lesions- little affect otherwise ``` up to 100ym removed
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benefits and negatives microabrasion
``` :) easy permanent conservative effective easy after care ``` :( caustic acid in surgery only enamel removal unpredictable
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describe process of microabrasion
pre op photos/diagrams/ shade guide/ sensibility/ rads if indicated ppe prepare solutions- pumice, 18% hydrochloric w/ pumice and sodium bicarbonate. labelled well. clean teeth w/ pumice vaseline applied to gingiva dental dam positioned sodium bicarbonate barrier applied to gingiva acid+pumice rubbed onto affected surface 5 seconds with wooden stick washed thoroughly repeated up to 10 times, 100 microns removal if 10 thoroughly clean apply flouride TP soft flex disc used to remove prism-less layer fluoride varnish applied review 4-6 weeks post op rads/photos advice: no coloured foods for 24 hours. anything that would stain a white tshirt.
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3 conditions assoc/ w/ hypoplasia
ectodermal dysplasia down's syndrome cleft lip and palate ED- two or more of ectodermal develop abnormally - skin hair teeth nails sweat glands mucous membranes
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percentage incidence of primary and permanent hypodontia?
primary 0.9% | permanent ~6%
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bleaching product components
10% carbamide peroxide = 3.3% h202 6.6% urea
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Why could a child be anxious before the dentist?
``` 'infection' from parents negative experience previously expectation of pain/uncomfort friends negative/ unusual experiences media uneducated on modern analgesic techniques ```
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How do you measure childhood anxiety?
MCDAS modified child dental anxiety scale pictures of faces for numbers
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8 behavioural management techniques
``` modeling acclimitisation desensitisation tell show do distraction positive reinforcement progressive muscle relaxation hypnotherapy ```
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6 y/o haemophilia A pt attends practice with buccal swelling and grossly carious 85. What is haemophilia A? dx? tx?
genetic disorder caused by deficiency in factor VIII clotting protein periapical abscess assoc/w/85 avoid XLA due to clotting issues atraumatic pulpotomy if poss. antibiotics given if needing to refer XLA -> DDAVP - desmopressin - manufactured factor VIII available for mild-mod haemophilia A atraumatic XLA infiltration LA if possible ensure clot, suture socket oral tranexamic acid given post op, may be required pre-op
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name 3 local haemostatic agents
``` LA w/ vasoconstrictor tranexamic acid thombin, fibrin surgicel ferric sulfate gelfoam ```
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triad of impairment in autism?
social impairment in : - interaction - imagination - communication
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features of autism? (6)
``` hypersensitivity hyposensitivity obsessive learning difficulties tubular sclerosis epilepsy ```
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mgmt of autistic patients dental visits
plan visit: info leaflets, social story, acclimitisation visit timing: first appt of day or first after lunch, child can wait in car environment: quiet surgery, no radio, no interruptions, taste of FV communication: makaton, learning boards, avoid casual chit chat, literal speech extra: oranurse unflavoured toothpaste
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indications for fissure sealants? materials used
``` medically compromised caries in primary dentition high caries risk learning difficulties mental&physical handicap ``` bisGMA resin, GIC
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describe technique for fissure sealant placement
isolate tooth, saliva ejector, cotton wool or dam clean tooth thoroughly - toothbrush/prophy rinse 20 seconds etch 20 seconds 37% phosphoric acid, wash, dry thoroughly, replace cotton wool, check for frosted appearance apply thin fissure sealant in fissures and pits, inc. buccal palatal extension if app. cure 30 secs check with sharp probe check for excess review if GIC do not need as good moisture control and do not need to etch
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4 types of cerebral palsy How could it be further classified?
spastic ataxic athetoid mixed types based on how severely brain damage has affect muscle tone- strength and tension of muscles could be hypo or hypertonia. hypo- low muscle tone, loss of strength and firmness. hypertonia- high muscle tone, causes rigidity and spasmodic movement, monoplegia, - one limb Hemiplegia, - one side of body diplegia, triplegia, quadriplegia, -2,3,4 limbs respectively