Perio Flashcards

1
Q

28 y/o fit and well, FM PAs showing bone loss on multiple teeth
diagnosis?
why? (5)

investigations?

A

generalised aggressive periodontitis

bone loss affecting 3 or more other teeth inc 6s and incisors
age of pt
rapid progression of bone loss
vertical bony defects
pt otherwise fit and well

thorough history (including family)
6ppc
swab of crevicular fluid -

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2
Q

How would you decide prognosis of individual teeth in cases of periodontitis?

A

loss of attachment
mobility
furcation involvement

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3
Q

give reasons for only partial removal of pocket bacteria during scaling? (5)

A
poor operator technique
failure to disrupt biofilm
poor pt OH
pt immunocompromised
difficulty accessing prevents removal
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4
Q

Why do antibiotics fail in aiding pocket bacteria removal?

A

-

  • not in therapeutic range - inadequate concentration
  • lack of vision - antibiotics do not reach site of disease activity
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5
Q

How would you manage a periodontal abscess with systemic involvement?

A
incise and drain
gentle subgingival debridement
hot salty MW
advise optimal analgesia
XLA if tooth poor prognosis
Antibiotics (amoxycillin and metronidazole)
follow up hpt
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6
Q

What would be clinical signs of improved periodontal health?

A

probing depth reduction to <4mm
BOP to <10%
plaque scores of <15%

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7
Q

Space between 13 and 14
Investigations and why?

impediment with implant placement here?

A
SI:
BPE- screening tool
PGI - plaque and bleeding record
6PPC - perio disease chart
PA rads: path detection
study models

implant :
periodontal disease
inadequate bone levels
inadequate space <7mm

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8
Q

What bacteria are involved with ANUG

A

p. intermedia
fusobacterium
spirochetes - treponema

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9
Q

Clinical signs and symptoms of ANUG

A
  • blunted interdental papilla
  • grey slough which when wiped reveals ulcerated tissue
  • halitosis
  • crater like ulcers
  • reverse gingival architecture
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10
Q

5 risk factors for ANUG

A
smoking
young age 
poor OH
immunocompromised
stress
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11
Q

Mgmt for ANUG

A
ultrasonic debridement
oxygenating MW e.g 3% H202 
CHX
OHI
Smoking cessation if needed
Antibiotics if systemic/immunocompromsied - metronidazole 400mg tds/3days
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12
Q

What would you need to say for informed consent for open flap curettage

A
  • Risks- gingival recession + normal oral surgery risks (bleeding, swelling, pain, infection)
  • Other tx options - continue NSPT
  • Risks of no tx- increased mobility, increased pocket depths, increased likelihood of losing teeth
  • Benefits of tx- can effectively debride with DIRECT VISION
  • Outcome- possible reduction in probing depth
  • Recommendation
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13
Q

Pt feels central crushing pain during surgery. What do you think it is?

immediate mgmt?

A

Myocardial Infarction

GTN spray - 2 sublingual actuations
oxygen 15L/min
Aspirin chewed 300mg

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14
Q

In what forms would you deliver post surgical advice? and what would you say- for open flap curettage?

A

written and spoken

avoid smoking for min. 1 week
avoid rinsing for 1 day- after 1 day can rinse with CHX MW(2%, bid) or hot salty
avoid exercise, just rest
do not probe area

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15
Q

Review appt timeframe for open flap curettage?

A

8 weeks

to allow sufficient healing

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16
Q

Bacteria that cause periodontal disease

A

gram negative anaeobic

A. a
P.gingivalis
B.forsythia
T.denticola

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17
Q

What patients might benefit from antibiotics along side hygiene phase therapy?

A
  • pt who is not responding favourably to scaling and debridement therapy/ HPT, with continuing clinical attachment loss
  • pt who test positive for P.ging or A.a in subgingival biofilm - as can invade epithelium of periodontal pockets
  • pt with severe chronic periodontitis and generalised deep pocket depths
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18
Q

Why might antibiotics not aid in reduction in bacteria numbers in perio cases?

A
  • cannot kill bacteria in biofilm .:.
  • biofilm not successfully disrupted during Scale and debridement
  • not therapeutic range - not correct dose
  • poor pt compliance to regime
  • antibiotic resistance
  • antibiotics inactivated by 1st pass metabolism
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19
Q

Why is it that azithromycin and tetracycline are perhaps considered more successful for perio tx than amoxycilin and metronidazole?

A

azi &tet- actively taken up and concentrated by cells

amo&met- enter cells by passive diffusion

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20
Q

What is SIRS

Criteria

A

systemic inflammatory response syndrome

any 2 or more of:
- tachycardia- HR >90bpm
- tachypnoea - RR >20breaths/pm
- leukopenia or bandemia or leukocytosis
<4000 cells/mm3 or >10% or >12000 cells/mm3
- fever or hypothermia - >38 degrees celcius or <36

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21
Q

Pt presents with suppurating abscess - How would you diagnosis

A

-sensibility test - if tooth vital then is perio abscess. if non-vital is endo abscess.
- if TTP laterally is perio lesion, if TTP vertically is endo lesion.
use gutta percha and PA to trace sinus tract to identify origin of lesion

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22
Q

What is a periodontal abscess?

A

localised collection of pus within tissues of periodontium

acute exacerbating of an existing periodontal pocket

caused by: untreated perio/ inadequate RSD/ food packing

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23
Q

Signs and symptoms of perio abscess

A
pain on biting/spontaneous
TTP  (laterally)
swelling
pus 
pocketing at swelling
mobility
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24
Q

How do you manage occlusal trauma in a pt with perio?

A
  • address issue - parafunction/ high restorations
  • bite raising appliance worn at least at night
  • hpt as necessary
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25
Q

What factors can influence localised mobility?

A

occlusal trauma - increasing pdl width

resorption/trauma

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26
Q

When would you advise splinting of mobile teeth?

A

to assist in rsd
when mobility causes discomfort
difficulty eating

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27
Q

Why is there a decrease in mobility after perio tx?

A

growth of junctional epithelium

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28
Q

What can you do to manage pt with widened pdl after rx?

A

Reduce contact in occlusion

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29
Q

What is the role of occlusion in periodontal disease?

A

occlusal trauma itself cannot cause loss of attachment but can exacerbate existing periodontal pockets

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30
Q

What is occlusal trauma?

signs and symptoms?

A

injury to the periodontium
resulting from occlusal forces
that exceed the reparative capacity
of the attachment apparatus

severe attrition
exposed dentine
increased sensitivity
mobility

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31
Q

fremitus?

A

vibration or movement of the tooth in functional occlusion

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32
Q

types of occlusal trauma?

A

primary and secondary

primary- where excessive loads are placed on teeth with normal periodontal tissues
secondary- where normal loads become excessive due to loss of attachment

33
Q

What findings would indicate aggressive periodontitis?

A

severe periodontal destruction inconsistent with OH findings
familial aggregation
early onset
pt otherwise fit and well

34
Q

Differences between localised and generalised aggressive periodontitis?

A

localised:
- localised CAL
- 6s and incisors
- circumpubertal age
- robust antibody response
generalised:
- generalised CAL
- 6s and incisors and at least 3 other teeth
- onset before 30 y/o
- poor serum antibody response
- episodic in nature

35
Q

bacteria indicated in aggressive periodontitis?

A

A.a

P.gingivalis

36
Q

Mgmt of aggressive periodontitis

A

start with NSPT - RSD
2 week CHX MW and SPRAY
Refer to specialist within 6/8 weeks

37
Q

define gingivitis

A

inflammation of the gingiva, characterised by red, swollen tissues which bleed on probing or brushing

38
Q

define chronic periodontitis

A

disease characterised by destruction of junctional epithelium and connective tissue attachment of the tooth, with bone destruction and formation of periodontal pockets

39
Q

What perio features could make a tooth have a poor prognosis? (3)

and if so, what parts of history needed to determine if teeth poor prognosis or not?

A

mobility - less bone support
loss of attachment - less tissue support
furcation involvement- difficulty cleaning
non vitality

smoking, drug history, systemic disease

40
Q

International 1999 Classification of Periodontal disease? (7)

A
gingivitis
chronic periodontitis
aggressive periodontitis
Periodontitis with systemic disease
necrotising ulcerative gingivitis
periodontal abscess
perio-endo lesion
gingival enlargement
developmental or acquired deformities
41
Q

Causes of gingival recession

A
  • chronic periodontal disease
  • inadequate toothbrushing technique
  • occlusal trauma
  • high frenal attachments
  • orthodontics
  • poor fit restorations
  • habits
  • abrasive toothpaste

Generalised gingival recession - always chronic inflammatory periodontal disease

localised-

  • if v shaped localised - occlusal trauma
  • if u shaped localised -
    - inadequate tooth brushing,
    - chronic inflammatory periodontal disease
    - high frenal attachment
42
Q

Disease to cause vertical defects in bone?

how does it cause this?

A

Localised angular periodontitis
Localised aggressive periodontitis/

radius of destruction of plaque is 2mm. .:. depends where plaque are situated. if interproximal bone is >2mm bony defect will be vertical/angular

43
Q

how to classify degree of bone loss?

A

Mild <30%
Mod 30-50%
Severe >50%

44
Q

difference in bacteria between localised and generalised aggressive?

A

localised only has A.a

generalised has both A.a and P. ging

45
Q

Management aggressive periodontitis

A
OHI
Complete Scaling and debridement
Antibiotic therapy- amoxycillin and metronidazole
CHX MW
OHI
46
Q

Clinical signs of periodontal healing

A

Gingival recession
Reduction in BOP
Reduction in probing depth

47
Q

How does each react to traumatic occlusion?

a) healthy periodontium
b) reduced periodontium
c) periodontium with periodontitis

A

a) widening of pdl .:. slight mobility. no LOA, will return to normal if trauma removed
b) widen of pdl but less pdl there. .:. increased mobility
c) loss of attachment when pdl widens, as pdl then breaks

48
Q

What is CHX?

A

bisbiguanide antiseptic

49
Q

Mode of action of CHX

A

dicationic
one cation adheres to pellicle
one cation disrupts bacterial membrane

bacteriocytic and -static

50
Q

Substantivity of CHX

A

12 hours

51
Q

Common doses of CHX

A
  1. 2% 10/20ml bid

0. 15% 15/18ml bid

52
Q

4 side effects of CHX

A
staining
taste disturbance
salivary gland enlargement 
anaphylaxis
sls interaction
53
Q

8 uses of CHX

A
pre op rinse
post intraoral surgery
ANUG
denture stomatitis
tx of dry socket
medically compromised pt
high caries risk pt
pt post jaw fixation for OH
Endo irrigant
Recurrent Oral Ulceration
54
Q

What is included in HPT

A

NSPT non surgical periodontal therapy
Scale and RSD
Removal of plaque retentive factors
OHI

55
Q

Method of proper OHI

A
TIPPS
talk
instruct
practice
plan
support
56
Q

7 parts to record on periodontal chart

A
teeth missing
gingival margin
probing depth
loss of attachment
BOP
mobility
furcation
57
Q

2 disadvantages of perio chart

A
  • doesnt take into account root length - .:. severity misleading
  • probing depths are SUBJECTIVE and can vary greatly
58
Q

How can you measure recession

A

photos w/ probe
pocket chart
study models

59
Q

Mgmt of localised gingival recession

A
atraumatic tooth brushing technique
minimise other risk factors
monitor
treat sensitivity
free soft tissue graft (palatal)
coronal advancement flap
60
Q

Tx for multirooted teeth with furcation involvement (3)

A

guided tissue regeneration
tunnel preparation
hemisection

61
Q

Previously root treated tooth with 9mm probing depth pocket and vertical bony defect. Dx?

initial tx?

A

perio-endo lesion
endo-perio lesion
true combined lesion

re-rct

62
Q

implant factors?

A
pt motivation
smoking
bone quality and quantity
smoking
MH (bisphosphonates)
restorative options
63
Q

How can bony defects be classified?

A

horizontal or vertical

by number of walls 1/2/3

2&3 have better prognosis

64
Q

indications for regenerative periodontal therapy?

A

2&3 walled defects
grade 2 furcation in mandibular molars
grade 2 buccal furcation in maxillary molars

65
Q

Why is diabetes a risk factor in perio disease?

A

WIPA

  • delayed WOUND healing
  • both are pro INFLAMMATORY diseases
  • immunosuppression- impaired PMN neutrophil function
  • ADVANCED glycation end (AGE) products increase tissue destruction
66
Q

2 tests for diabetes diagnosis?

and expected values?

A

RPG random plasma glucose <11.1mmol/L normal, above diabetics.
FPG fasting plasma glucose <7 mmol/L

both need 2 separate OCCASIONS

67
Q

Test for diabetic control

normal value?

A

Hb1ac

<7%

68
Q

4 ways in which smoking affects periodontal tissues

A

CCEB

CHEMOTAXIS and phagocytosis impaired
CYTOKINE production affected
ENZYME catalyse affected
BLOOD flow restricted

69
Q

name the cytokine important in tissue destruction?

what does it do

A

interleukin-1
pro-inflammatory cytokine
stimulates release of enzymes and osteoclasts .:. increasing tissue destruction

70
Q

pt attends with inflamed gingiva extending beyond mucogingival junction. clinical description?

signs?

3 oral conditions that could cause this?
2 risk factors?
2 tx?

A

desquamative gingivitis - clinical description not a condition

erythematous, glazed, friable, haemorrhagic gingiva +/- pain

oral lichen planus OLP
pemphigus vulgaris PV
mucous membrane pemphigoid MMP

risk: smoking, plaque

tx:
- enhanced OHI
- scaling to remove supra and subgingival plaque
- removal of intraoral restorations/ prostheses
- topical or systemic corticosteroids:
- >local lesions: beclamethasone inhaler
- > generalised: betamethasone tablet dissolved as MW

71
Q

Name the 3 meds associated with gingival hyperplasia?

A

phenytoin - antiepileptic
cyclosporin - immunosuppressant
nifedipine - calcium channel blocker

72
Q

How do you manage gingival hyperplasia?

A
plaque still main causation .:.
OHI and RSD
then if not responding-
surgery
liaise with GP re: changing medication
73
Q

Values for BPE

A

0 → black band showing completely, no BOP or plaque retentive factors – No Rx/OHI.
1 → band visible, BOP, no plaque retentive factors – Rx – OHI.
2 → band visible, BOP, supra/sub calc or plaque retentive factors. Rx – OHI, remove PRFs, S&P/RSI.
3 → band partly visible → 3.5-5.5mm – Rx – 6PPC sextant of full mouth if >2 grade 3 measured. OHI, RSI, remove
plaque retentive factors.
4 → band not visible → >5.5mm. Rx – 6PPC full mouth, RSI, remove plaque retentive factors, consider referral. * →
furcation involvement.

74
Q

How is mobility graded?

A

0 - physiological movement (0-0.2mm)
1 - <1mm movement
2 - 1-2mm movement
2 - >2mm movement and vertical movements - rotations and depressions

75
Q

How is furcation graded?

A

0- <3mm horizontal
1 - >3mm horizontal but not through and through
2 - through and through

76
Q

How is gingival recession graded?

A

Miller’s classification

77
Q

How would you decide the prognosis of individual teeth in a pt with aggressive perio? (7)

A
symptoms
bone levels
angular defects
mobility
furcation involvement
is pt maintenance possible?
short tapered roots
78
Q

5 signs and symptoms of periodontal disease

A
signs- 
mobility
furcation involvement
increased probing depth
LOA
BOP
gingival recession
symptoms- 
tooth "loose"
tooth movement
bleeding
food packing
"long in tooth"
79
Q

localised risk factors for periodontal disease

A
overhanging restorations
defective margins
partial dentures
oral appliances
calculus
OH 
tooth malposition