Renal & Urology š Flashcards
(199 cards)
Lactic acidosis following tonic clonic seizureā¦. tx
Since it is usually transient, observe and repeat labs in 2 hours
Managment for patient with posterior urethral injury
Retrograde urethrography first. Then catheterise etc.
Septic shock Tx overview
Focus on finding and treating cause. But do fluid resus, then pressors if that doesnāt work out
Renin and Aldo levels in hypovolemia. And ADH Levels
All elevated. Recall why?? This also means we get low Na in hypovolemia often
Signs that a hematuria is glomerular origin, not lower UT
Proetineuria, brown (rather than pink red), Cr elevation etc.
How to confirm a Dx of renal veing thrombosis
CTA or MRS
How to treat renal vein thrombosis. Given if AKI present or not
AKI present: thrombolysis
No AKI: anticoag
Hyponatremia due to SIADH Tx
Initially do fluid restriction with or without salt tabs. Can give demeclocycline if it doesnāt work. If very severe and have neuro signsā¦. Give hypertonic saline
PUV dx
Do US first, then voiding cystourethrogram, then cystoscope to confirm and ablate the PUV
Rhabdomyolysis can be due to electrical injury? Why? Tx to prevent?
Bone electrical resistance is high, meaning heat is generated internally, killing muscle. Give IV fluid aggressively
When dealing with hypercalcemia in the setting of potential malignancy, order what mainly
PTHrP, CXR, whole body bone scan
Why is pregnancy a risk for pyeloneph
increased progesterone levels cause smooth muscle relaxation and ureteral dilation. Because
of these physiologic changes, pregnant women with untreated ASB are at increased risk for acute pyelonephritis
When txāing UTI in preg, what do we do after
As a test of cure, a repeat urine culture is performed after antibiotic treatment due to the risk of persistent or recurrent bacteriuria.
Membranous nephropathy vs minimal changeā¦
Minimal change is more a acute
1° vs 2° membranous nephropathy⦠which is more adult and which is children
Primary more in adults⦠so if a child has it, assume secondary causes
Cause of edema in nephritic vs nephrotic. MoA, which causes pulmonary edema
Nephrotic is due low albumin, and doesnāt cause pulmonary edema (since fenestrations are large enough to allow albumin equil across the vessels). Where as nephritic will cause pulmonary edema and is due decreased GFR causing huge hydrostatic pressure
When do you reassure in bed wetting in paeds
No other abnormalities, less than 5years old
Extrarenal issues of PCKD
Cerebral aneurysms
⢠Hepatic & pancreatic cysts
⢠Mitral valve prolapse, aortic regurgitation
⢠Colonic diverticulosis
⢠Ventral & inguinal hernias
Risk factors for renal veing thrombosis
Hypercoagulability
⢠Nephrotic syndrome
o Malignancy (particularly renal)
⢠OCP
⢠Volume depletion (infants)
Trauma
Dx of renal vein thrombosis and Tx?
CT or MR angiography
⢠Renal venography
Anticoagulation
⢠Thrombolysis/thrombectomy (if AKI present)
We all know the main CVD risk factors, but what other aspects of chronic kidney disease/dialysis increases risk. Name 3
Anemia of chronic kidney disease
⢠Vascular calcifications (1 phosphorus, 1 calcium)
Oxidative stress related to uremia and dialysis
3 differentials for renal colic not identifying anything on US OR X-ray
- Radiolucent stones (uric acid stones, xanthine stones)
- Small calcium stones (<1-3 mm in diameter)
- Nonstone ureteral obstruction (eg, blood clot, tumor)
Other than UTI, what can cause positive leuk esterase on urinalysisās
Interstitial nephritis
Dx sign of posterior urethral injury, on which modality? Why before catheter
Extravasation of contrast
om the urethra is diagnostic of urethral injury. Urethrography should precede any attempts at urethral (eg, Foley)
atheterization because it can worsen the injury, potentially converting a partial urethral tear into a complete urethral
Iceration