Respiratory Disfunction of the Newborn Flashcards

(54 cards)

1
Q

what are the 3 critical factors in the development of respiratory distress syndrome

A

preterm unable to produce enough surfactant
muscle coat of pulmonary blood vessels are incompletely developed
ductus arterioles may remain open due to hypoxia

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2
Q

what helps close the PDA

A

hyperoxemia

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3
Q

what could happen if the PDA does not close

A

pulmonary congestion and overload

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4
Q

why are preterm newborns at an increased danger of respiratory obstruction

A
  • the bronchi and trachea are very narrow and mucus obstructs airway
  • positioning
  • weak or absent gag reflex could result in aspiration
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5
Q

if O2 sats are dropping low what should you do

A

position them in a prone position for chest expansion

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6
Q

what are signs of respiratory distress

A
nasal flaring
retractions
crackles
asthma (wheezing)
grunting
cyanosis
tachypnea
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7
Q

what are the two positions you can place the baby in for maintenance of resp function

A

supine with head slightly elevated without hyper flexion and prone position

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8
Q

why do you not want to do a lot of suctioning

A

this could put further stress on the baby

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9
Q

what should be assessed before oral feedings are started

A

infants gag and suck reflex as well as if they are breathing fast or not

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10
Q

why will a baby not go higher than 100% O2 sats on oxygen?

A

because we want to ween the baby off of the oxygen because too much can cause problems to the baby (ROP)

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11
Q

what delivers 400 breaths a minute to the infant and you will see a constant wiggle of their chest

A

high frequency oscillation

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12
Q

what is it when the baby poops in utero and they breath it into their lungs

A

meconium aspiration syndrome

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13
Q

what stops producing due to the meconium build up

A

surfactant

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14
Q

what is the biggest issue with MAS

A

mechanical obstruction because it plugs up the airway, making it sticky

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15
Q

MAS happens to….

A

full term or post term

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16
Q

what are clinical signs of MAS

A

severe respiratory distress shortly after birth and audible rales or rhonchi on auscultation

  • barrel chest
  • not a good apgar
  • PO2 is low
  • Acidosis
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17
Q

what is the management of MAS

A

ventilation and monitoring

surfactant admin

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18
Q

results when the normal vasodilation and relaxation of the pulmonary vascular bed do not occur

A

persistent pulmonary hypertension of the newborn (PPHN)

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19
Q

PPHN happens to…

A

term or near term infants (wimpy white boys don’t do well, african american females do the best)

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20
Q

what are 6 risk factors of PPHN

A
hypoxia
RDS
pneumonia
bacterial sepsis
hypo or hyperthermia
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21
Q

for PPHN babies where do we check blood pressure

A

right arm

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22
Q

why do PPHN babies desat

A

their lung pressure is higher than systemic pressure and this makes blood bypass the lungs (going through PDA)

23
Q

what are assessment finding in a PPHN baby

A
  • term or near term infant
  • low apgar score
  • symp within 12 hrs
  • hypoxia at birth
  • tachypnea
  • retraction/grunting
  • cyanosis
  • hypotension
  • heart murmur
  • met acidosis
24
Q

preductal is the

25
post ductal is the
left arm
26
what helps increase systemic pressure so blood will go to lungs for the PPHN babies
vasopressors
27
why are sedatives and analgesics given to PPHN babies
they are very sensitive and if we touch them they will drop their sats so care is done every hrs
28
delayed clearance of fetal lung fluid and usually resolves by 48-72 hours
transient tachypnea of newborn (TTN) aka wet lungs
29
what is a main cause of TTN
the babies don't get a good squeeze of lungs as they make their way through vaginal canal or a c section is performed so there is no squeezing at all
30
what are clinical manifestations of TTN
respiratory distress and or cyanosis
31
cessation of breathing for 20 sec or longer or for less than 20 sec when associated with cyanosis, pallor, and bradycardia.
apnea
32
what are two types of apnea
central and obstructive apnea
33
caused by preterm infants irregular breathing pattern
central apnea
34
preterm infant when there is a cessation of airflow associated with blockage of the upper airway
obstructive apnea
35
apnea is a dx of _______
exclusion
36
what is often used to treat apnea of prematurity
methylxanthine (caffeine citrate
37
when would you hold the mthylxanthine (caffeine citrate)
when HR is high (>170)
38
surfactant deficiency is the main issue of this and underdeveloped alveoli
respiratory distress syndrome
39
RDS happens to...
usually preterm babies
40
what is a good indicator that RDS babies are getting better
baby is starting to pee more
41
what are risk factors for RDS
low gestational age male predominance maternal diabetes perinatal depression
42
why would maternal diabetes be a risk for RDS
increase sugar and insulin deactivates the surfactant
43
how can you lessen the severity of RDS
giving mom steroids because it helps lung development but it should be given 30-1hr before birth
44
what will the RDS baby look like
gray dusky color nasal flaring grunting
45
how can we manage RDS
``` artificial surfactant given via ET tube resp support and monitoring oxygen supplementation fluid and metabolic management (withhold feeding and give tpn) ```
46
bone or mucus is blocking the nasal cavity
choanal atresia
47
s/s of choanal atresia
cyanosis and retractions at rest noisy respirations difficulty breathing during feeding
48
how do you assess potency of nares
listen for breath sounds while holding mouth closed and alternately compressing each nostril
49
how should you position choanal atresia pt
head elevated
50
what is a sign the baby probably has choanal atresia
put a pacifier in babies mouth and sats drop
51
why is sx for choanal atresia put off
it is too close to the brain so they don't want to do sx on it too soon (track is usually done first)
52
too much oxygen can cause this to happen to the eye
retinopathy of prematurity (ROP)
53
what is the main risk factor for ROP
hyperoxemia
54
how can we prevent ROP
judicial use of supplemental oxygen therapy (for sats usually <93%)