Respiratory emergencies and management Flashcards
(20 cards)
Respiratory Assessment?
FLAPS
Airway?
Check patency - is the airway open and clear?
Look for any obstructions e.g. swelling foreign bodies, secretions.
Noisy breathing - stridor gurgling
Breathing?
RR: 12-20 = normal
Oxygen saturations: normal is 94-98%
Chest expansion check for symmetry and reduced movement on one side.
Use of accessory muscles - intercostal retractions , nasal flaring
Feel?
Palpate the chest wall for abnormalities such as tenderness or rib fractures.
Check for eqaul chest expansion
Look?
Observe rise and fall assess for symmetry, accessory muscle use and any deformities e.g. barrel chest in COPD.
Look at the patients general appearance.
Cyanosis indicating hypoxia.
Nasal flaring intercostal and suprasternal retractions are signs of increased work of breathing
Tripod position
Auscultate?
Wheezing - heard in asthma or COPD
Crackles - fluid in the airways indicating pulomonary oedema or pneumonia.
Reduced or absent breath sounds - possible pneumothorax, pleural effusion.
Percuss?
Tap the chest wall and listen for sounds.
Hyper resonance - pneumothorax
Dullness seen in pleural effuison or pneumonia.
Saturations and speech?
Speaking in short sentences can indicate difficulty in breathing.
Circulation?
HR: Tachycardia - hypoxia or infection.
BP: Monitor for signs of shock or hypotension
CAP refill delayed can result in poor perfusion.
Disability?
Level of consciousness: hypoxia or hypercapnia can cause confusion, drowsiness .
GCS assess patient if they are drowsy or unresponsive.
Acute exacerbation of asthma?
Cannot complete sentences in one breath.
Resps above 25.
Expiratory wheeze.
PMH of asthma
Hyperinflated chest
Fatigue, anxiety
Non productive cough
Management: patient to use their own inhaler, nebulised salbutamol and ipatropium, ECG 12 lead, consider IV hydrocortisone
Life threatening asthma? Management?
Silent chest, cyanosis, bradycardia, unable to talk, decreased LOC.
Oxygen, 1 in 1000 adrenaline ECG 12 lead, consider hydrocortisone, pre-alert to hospital: continuous salbutamol neb and ipratropium single dose.
PEAK flow?
Measures the peak speed of expiration
Done before and after treatment.
COPD?
Hypoxic drive
Chronic Bronchitis?
Dyspnoea above and beyond norm levels
Low PO2 and PCO2 retention
Productive cough
Crackles and wheezes common.
Management: Provide high flow oxygen, monitor the patient carefully consider nebulisation.
Emphysema?
Extreme dyspnoea on exertion
Signs of respiratory distress.
Breathing with pursed lips on expiration
Barrel chest.
Same management
PE?
Blood clot impair the blood supply to the lungs. Caused by a DVT.
Chest pain, breathlessness, abnormal heart trace, coughing, feeling dizzy, altered level of consciousness.
High flow oxygen, IV cannulation, monitor patient carefully.
Pneumonia?
Develops over several days with cough and sputum production, dyspnoea, pleuritic chest pain, weakness, malaise and often myalgia.
Often follows flu viral infection, smoking, alcohol excess, immunosuppression, aspiration.
Productive cough (sputum)
Frothy white/pink sputum - acute LVF
Productive yellow/green/red - infection.
Haemopysis - PE, Chest infection, CA lung.
Abnormal sounds?
Adventitious - fluid in the airway.
Crackles - implies fluid or exudate
Wheeze - implies a decreased airway lumen.
Stridor - implies upper airway obstruction.