RSI, reintubation, extubation complications and difficult airway Flashcards
describe aspiration prevention
1) pharmacological aspiration prophylaxis
- goal: reduce amount ( 2.5 pH) of gastric contents
- liquid acid neutralizer (Bicitra)
- prokinetic agent (Reglan)
- acid secretion prevention with proton pump inhibitors (PPIs: Prilosec) and/or H2 antagonists (Pepcid/zantac)
2) preoxygenate/denitrogenate as usual
3) rapid sequence induction (RSI)
- rapid injection of propofol and succs
describe rapid sequence induction (RSI)
objective: prevent pulmonary aspiration
1) decreases time b/w when protective airway reflexes are eliminated and airway protection is secured (seconds matter!) *speeds up intubation
2) attempts to protect airway when protective reflexes are eliminated by occluding gastric pathway to airway (cricoid pressure: occludes gastric pathway to block anything from coming up)
what are RSI indications?
- patients at an increased risk for aspiration of gastric contents
- trauma (includes falls with fractures: once trauma occurs, digestion stops, so even if > 6 hrs consider “full stomach”)
- esophageal obstruction
- NPO guidelines not followed
- acute abdomen and bowel obstruction
- active GI bleeding or upper airway bleeding (nausea)
- pregnancy (consider “full stomach”)
- abdominal mass or ascites increasing risk of gastric content expulsion
- severe GERD (N/V)
describe RSI prep
- check equipment immediately prior to induction
- be sure ETT has stylet
- ensure competent airway personnel present for cricoid pressure
- position for optimal laryngoscopy
describe RSI procedure
- adequate preoxygenation/denitrogenation
- slam induction agent then immediately give paralytic
- perform cricoid pressure maneuver
- if bag mask ventilating while waiting for paralytic effect then do not exceed pressure > 20 cmH2O (introduces air into gastric cavity and gastric insufflation increases risk of aspiration)
- laryngoscopy, intubation, inflate cuff
- *verify ETT position before releasing cricoid pressure
describe cricoid pressure procedure
using Sellick’s maneuver
- thumb and index finger pressing downward on the cricoid ring will compress the esophagus against the cervical vertebrae
- cricoid pressure effectiveness often debated
- RSI with cricoid pressure prevents gastric contents form exiting up through esophagus and insufflation d/t PPV forcing air into stomach
- esophagus to the right of trachea in 75% of people
what are some complications of cricoid pressure?
- may produce poor view during intubation (mask ventilation NOT affected)
- increased risk for esophageal rupture if vomiting
what are contraindications to cricoid pressure?
- airway trauma
- lack of properly trained assistant
- fractured larynx or cricoid
- active vomiting
- esophageal foreign body near cricoid (C6)
- esophageal disease
- cervical fracture near the cricoid (C6)
- no suspected spinal injury
- do RSI w/o cricoid pressure
what are alternatives to RSI?
- regional anesthesia (*always be prepared for a tracheal intubation if regional fails or regional results in airway compromise such as a high spinal)
- awake intubation (maintains protective airway reflexes)
- tracheostomy under local
- delay in surgical procedure (if surgery is non-urgent/emergent)
what are some complications of intubation during laryngoscopy and intubating?
- malpositioning: esophageal intubation, endobronchial intubation, laryngeal cuff supraglottic
- airway trauma: tooth damage, lip/tongue/mucosal laceration, sore throat, dislocated mandible, retropharyngeal dissection
- physiologic reflexes(sympathetic outflow): HTN, tachycardia, intracranial HTN, intraocular HTN, laryngospasm
- tube malfunction: cuff perforation
what are some complications of intubation while the tube is in place?
- malpositioning: unintentional extubation, endobronchial intubation, laryngeal cuff position
- airway trauma: mucosal inflammation and ulceration, excoriation of nose
- tube malfunction: ignition, obstruction
what are some complications of intubation following extubation?
- airway trauma: edema and stenosis (glottis, subglottic, or tracheal), hoarseness (vocal cord granuloma or paralysis), laryngeal malfunction and aspiration
- physiologic reflexes
- laryngospsasm
describe bronchospasms
- occurs during intubation or extubation
- reactive airway disease (RAD) increases risks: asthmatics, bronchitis, URI
- characterize by inability to ventilate
how are bronchospasms treated?
- 100% O2
- beta 2 agonist like albuterol
- lidocaine IV or ETT if intubated
- deepen volatile anesthetic if intubated or masked
- IV atropine, epinephrine, fentanyl
describe steps for emergence
- establish spontaneous breathing first
- increase CO2 level to stimulate breathing (do by decreasing TV and RR)
- stimulate to awaken: verbal and physical (moving arms and calling name); noxious stimulation (ETT movement; surgical field and incision) *blocks are reducing this stimulus
- removal of agents that promote sleep: versed (remains for hours), fentanyl (may or may not remain), volatile agent (remove by ventilation), paralytic (reverse)
describe the steps extubation
- can be done deep or awake depending on:
- surgery (hernia, ENT, sling, carotid endarterectomy want deep since coughing will increase pressure in areas of surgery)
- pt. history (aspiration risk, difficult intubation, OSA want light since pt. needs protective reflexes and want fully awake and breathing)
- desire smooth , atraumatic extubation
- avoidance of coughing: lidocaine, narcotic, pull deeper
what are criteria for extubation?
- adequate NMBA reversal
- 4/4 TOF with sustained 5 second tetanus w/o fade
- TV and RR adequate to eliminate CO2
- strong hand grip, cough, and sustained head lift > 5 sec.
- airway reflexes: swallowing and tongue movement
- maintain oxygenation/ventilation w/o stimulation
what are crucial questions to ask before extubating?
- can I maintain a patent airway post-extubation? (effectively ventilate and oxygenate)
- can I reintubate easily if needed? (don’t take tube if difficult intubation unless positive pt. is awake, following commands, and can maintain ventilation)
- are aspiration risks reduced?
- can pt. maintain oxygentation and ventilation as evidenced by adequate RR rate and TV?
- is paralytic reversed?
- have protective airway reflexes returned? (aspiration risks are reduced)
describe extubation procedure
- suction the oropharyngeal cavity
- administer a positive pressure breath
- remove air from cuff and gently remove ETT while monitoring the airway for exhalation (exhalation during extubation ensures that secretions are projected away from cords)
- do not pull at the end of expiration or secretions will be sucked in on inspiration
- immediately apply supplemental O2 and PPV via facemask while maintaining seal
- listen and feel for air movement (crowing and turbulent sounds indicate air movement but partially obstructed airway; risk laryngospasm)
- PP breathing good with crowing since pushes air in
describe laryngospasms
-glottic closure reflex
-more prevalent in young adults and children
causes:
-light anesthesia, stage 2 extubation (hyperirritable)
-direct glottis, supraglottic stimulation
-irritation from inhaled agents
-foreign bodies on cords (blood/secretions)
-stimulation of periosteum, celiac plexus or dilation of rectum
signs/symptoms:
-high pitched squeaking or crowing indicates some air is moving
-absence of sound indicates complete closure
describe the treatment of laryngospasms
- suction all secretions from cords and mouth
- forward displacement of mandible (jaw thrust) moves tongue off posterior pharynx
- continuous PP with 100% O2
- paralytic agent: Succs 10-20 mg (1/2- 1 cc)
- IV lidocaine
- laryngospasm notch (at soft tissue just behind earlobes, push both sides firmly inward towards skull base while simultaneously pushing anterior similar to jaw thrust; should break within 1-2 breaths)
- work quickly to prevent desaturation and negative pressure pulmonary edema
- reintubate if unable to maintain oxygenation
describe negative pressure pulmonary edema (NPPE)
- results when a strong inspiratory effort against a totally occluded airway generates increased negative intrathoracic cavity pressure (occluded ETT, laryngospasm)
- fluid enters the lungs resulting in pulmonary edema
- more pronounced in young, muscular patients
- light pink or white frothy foam with coughing
- unable to maintain oxygen saturation
- CXR to confirm
how is NPPE treated?
- remain intubated
- Lasix
- 100% O2
- PEEP
- sedation
describe glottic edema
-repeated intubation attempts resulting in trauma to laryngeal structures and glottic opening
-stridor can indicate glottic edema
-results in partial or total airway obstruction
causes:
-ETT too large and tight fitting
-excessive coughing while intubated