Skin Problems Flashcards

1
Q

STAND bundle

A

-Score (using the Braden Scale for predicting pressure injury risk)
-Turn (repositioning tubes and decides, turn the pt)
-Apply (bordered foam dressing or barrier cream)
-Nutrition (attention given to nutrition status)
-Discuss (involvement of specialists)

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2
Q

Assessment: Recognize Cues

A

History
-conduct with risk factors in mind
-assess cardiovascular status, cognition/mental status
-identify cause of tissue integrity loss and factors that may impair healing
-Contributing factors
(bedrest, immobility, incontinence, DM/ PDV, malnutrition, decreased sensory perception or cognition problems)

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3
Q

Stage 1 Pressure Injury

A

-intact skin with localized area of non-blanchable erythema (may appear differently in skin with darker pigmentation)
-may be preceded by changes in sensation, temp, or firmness
-color changes are not purple or maroon

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4
Q

Stage 2 Pressure Injury

A

-partial-thickness loss of skin with exposed dermis
-wound bed us viable, pink, or red and moist
-may look like intact or ruptured serum-filled blister

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5
Q

Stage 3 Pressure Injury

A

-full-thickness skin loss with adipose (fat) visible in the ulcer
-granulation tissue and rolled wound edges are often present
-slough/ or eschar may be present
-undermining and tunneling may be present
-subq tissues may be damaged or necrotic

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6
Q

Stage 4 Pressure Injury

A

-full-thickness skin loss with exposed or palpable fascia, muscle, tendon, ligament, cartilage, or bone
-may have slough or eschar
-rolled edges, undermining, tunneling may be present

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7
Q

Arterial Ulcers

A

History:
-pt reports claudication after walking about 1-2 blocks
-rest pain usually present
-pain at ulcer site
-2-3 risk factors prevent
Location:
-end of toes, between toes, deep, ulcer bed pale, with even edges, little granulation tissue
Assessment Findings:
-cool or cold foot
-decreased or absent pulses
-hair loss
-pallor w elevation
-possible gangrene
-when acute, neurologic deficits noted
Treatment:
-treat underlying cause (surgical, revascularization)
-prevent trauma and infection
-pt education, stressing foot care

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8
Q

Venous Ulcers

A

History:
-chronic nonhealing ulcer
-no claudication or rest pain
-moderate ulcer discomfort
-pt reports of ankle or leg swelling
Location:
-ankle area
-brown pigmentation
-ulcer bed pink
-usually superficial, with uneven edges
-granulation tissue present
Assessment Findings:
-ankle discoloration and edema
-full veins when leg slightly dependent
-pulses present
-may have scarring from previous ulcers
Treatment:
-long-term wound care
-elevate extremity
-pt education
-prevent infection

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9
Q

Diabetic Ulcers

A

History:
-diabetes
-peripheral neuropathy
-no reports of claudication
Location:
-plantar area of foot
-metatarsal heads
-pressure points on feet
-deep
-pale, with even edges
-little granulation tissue
Assessment:
-pulses usually present
-cool or warm foot
-painless
Treatment:
-rule our major arterial disease
-control diabetes
-pt education regarding foot care
-prevent infection

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10
Q

Pressure Injuries: Assessment-Recognize Cues

A

Laboratory Assessment
-Clinical indications of infection (cellulitis, exudate changes, increase in injury size or depth)and systemic signs of bacteremia (fever, elevated WBC)
-wound culture is not routinely performed (to see if septic)
-if performed, tissue culture is done (not just wound swab)

Other Diagnostic Assessments:
-arterial blood flow studies is arterial occlusion is suspected
-duplex ultrasound imaging
-blood tests for nutritional deficiencies (prealbumin, albumin, total protein)

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11
Q

Pressure Injuries: Analysis

A

-compromised tissue integrity due to vascular insufficiency and trauma
-potential for infection due to insufficient wound managment

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12
Q

Alginate: Wound Dressing

A

Highly absorbent fabrics or yarns that are derived from natural polysaccharide fiber or seaweed; may be combined with collagen. Alginate forms a gel when it comes in contact with the pressure injury
-Indication: stage 3 and 4 pressure injuries with moderate exudate

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13
Q

Antimicrobial: Wound Dressing

A

Impregnated with antimicrobial agents, such as medical-grade honey, chlorhexidine, or silver ions
-Indication: for pressure injuries with infection

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14
Q

Collagens: Wound Dressing

A

Gels, pads, particles, pastes, powder, sheets, or solutions. Made from bovine, equine, porcine, or avian sources, Used with secondary dressings
Indication: stage 3 or 4 pressure injuries

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15
Q

Foam (including hydropolymers): Wound Dressing

A

Sheets or other shapes of foamed, polymer solutions (usually polyurethane) that have open cells that can hold fluid. May be impregnated or layered
Indication: stage 2 and greater pressure injuries with moderate or heavy exudate

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16
Q

Gauze (moist): Wound Dressing

A

Dry, woven, or nonwoven sponges and wraps made of cotton, polyester, or rayon
Indications: pressure injuries that cannot be dressed with an advanced wound dressing, yet need a moist environment

17
Q

Hydrocolloid (3M Tegaderm hydrocolloid dressing): Wound Dressing

A

Wafers, powders, or pastes (powder and pastes need a secondary dressing). Used in areas that require contouring (heels)
Indications: noninfected stage 2 pressure injuries

18
Q

Hydrogel: Wound Dressing

A

Amorphous, impregnated, or in sheet form. Designed to maintain a moist environment
Indications: Noninfected stage 2 pressure injuries, noninfected stage 3-4 pressure injuries with minimal exudate

19
Q

Superabsorbent(3M Tegaderm superabsorbent dressing)

A

Highly absorptive fiber layers (cellulose, cotton, rayon). Minimizes adherence to the wound while collecting exudate
Indications: heavily exuding pressure injuries

20
Q

Transparent films (3M Tegaderm): Wound Dressing

A

Polymer membranes that are impermeable to liquid, water, and bacteria. Can see through the dressing. (also used to cover IV sites, lacerations, second degree-burns)
Indications: noninfected stage 2 pressure injuries; also for pressure injuries as a secondary dressing when advanced wound dressings are not an option

21
Q

Wet-to-damp saline moistened gauze

A

as with the wet-to-dry technique, necrotic debris is mechanically removed but with less trauma to healing

22
Q

Continuous wet gauze

A

the wound surface is continually bather with a wetting agent of choice, promoting dilution of viscous exudate and softening of dry eschar

23
Q

Topical enzyme preparations

A

proteolytic action on thick, adherent eschar causes breakdown of denatured protein and more rapid separation of necrotic tissue

24
Q

Moisture-retentive dressing

A

spontaneous separation of necrotic tissue is prompted by autolysis

25
Q

Pressure Injuries: Planning and Implementation: Generate Solutions & Take action

A

Nonsurgical Management:
-Physical therapy
-Drug therapy
-Nutrition therapy
-Adjuvant therapies: electrical stim, negative-pressure sound therapy, therapeutic ultrasounds
Surgical Management:
-remove necrotic tissue and skin grafting or use of muscle flaps to close wounds that do not heal by re-epithelialization and contraction

26
Q

Preventing Infection

A

-monitor for S/S on infection (fever, elevated WBC, positive blood cultures)
-report change to Dr.
-sudden deterioration of the wound, with an increase in the size or depth of the lesion
-change in the color or texture of the granulation tissue
-changes in the quantity, color, or odor of exudate
-maintain safe emviromanet
-teach all personnel to use standard precautions and to proper;y dispose of solid dressings and linens

27
Q

Pressure Injuries: Evaluation

A

-experience progress toward wound healing by second intention as evidenced by granulation, epithelialization, contraction, and reduction solution of wound size
-re-establish skin tissue integrity and restore skin barrier function
-remain free from local or systemic infections