Syndromes associated with Ant Uveitis Flashcards
Posner-Schlossman Syndrome (Glaucomatocyclitic Crisis)
What is it?
Acute, recurrent anterior uveitis with markedly elevated IOP!
This can happen multiple times a year for many years. The patient will know they are having an outbreak and come to you
Always the same eye that is effected.
Etiology, demographics, symptoms of Posner-Schlossman Syndrome (Glaucomatocyclitic Crisis)
Etiology- Idiopathic.
Demographics- 20 to 50 years old. Unilateral.
Symptoms- Mild red eye, eye pain, photophobia, halo.
Signs of Posner-Schlossman Syndrome (Glaucomatocyclitic Crisis)
Minimal injection of the conj and/or limbus. White and quiet overall.
Very mild AC reaction. Cells more common than flare.
Few fine KPs on the K endothelium. Non granulomatous.
Increased IOP- 40 to 60
Microcytic K edema due to high IOP
Complications of Posner-Schlossman Syndrome (Glaucomatocyclitic Crisis)
Could cause glaucoma due to inflammatory debris obstructing the TM
How to manage Posner-Schlossman Syndrome (Glaucomatocyclitic Crisis)
It is self limited and resolves spontaneously regardless of treatment.
Attacks usually subside within few hours to few weeks.
Topical steroids during acute attacks. If they do not improve inflammatory response, taper and discontinue.
AND
IOP lowering meds during acute attacks. Avoid prostaglandins- use BB, CAI
When do episodes of Posner-Schlossman Syndrome (Glaucomatocyclitic Crisis) tend to stop?
Almost never seen after the age of 60
What is Fuchs’ Heterochromic Iridocyclitis
Chronic anterior uveitis with non hereditary heterochromia. Lightening of the iris.
Etiology of Fuchs’ Heterochromic Iridocyclitis
Idiopathic
Demographics and laterality of Fuchs’ Heterochromic Iridocyclitis
Typically begins between the ages 20-30
Unilateral
Symptoms of Fuchs’ Heterochromic Iridocyclitis
Typically asymptomatic. If symptoms occur, they are mild redness and pain.
May notice discoloration of the iris and that is why they come in.
Signs of Fuchs’ Heterochromic Iridocyclitis
Minimal to no injection of the conj and/or limbus.
Mild AC reaction. Always cells, no flare.
Diffuse iris stromal atrophy, which causes the lighter colored iris with TIDs.
Diffuse, fine stellate KPs on the K endothelium. Non granulomatous.
Fine vascularization at the angle and iris root.
Complications of Fuchs’ Heterochromic Iridocyclitis
PSC- cataract development. Could be in early 30-40s. Pt may report blurry vision due to the cataract development.
Glaucoma due to inflammatory debris blocking TM
Hyphema due to surgery
Management of Fuchs’ Heterochromic Iridocyclitis
Most cases do not require treatment with steroids– it usually does not respond. Taper if no response.
Elevated IOP does not improve with drops. Treat with glaucoma surgery.
Cataract is seen in over 80% of eyes with
Fuchs’ Heterochromic Iridocyclitis
UGH syndrome
What is it?
Anterior uveitis with elevated IOP/glaucoma and hyphema post cataract surgery.
