Telencephalon- clinical cerebral cortex Flashcards

(47 cards)

1
Q

agranular layer is in which gyrys?

A

post central gyrus

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2
Q

which gyrus is the granular layer?

A

precentral

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3
Q

which cortical projection does interhemispheric connections?

A

commissural

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4
Q

what does the anterior commissure connect? what does the corpus callosum connect?

A

ant commissure connects middle and inferior temporal gyri

CC connects about everything else

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5
Q

which cortical projection connects intracortical areas?

A

association- uncinate, arcuate, superior longitudinal, inf longitudinal, cingulum

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6
Q

which cortical projection descends to subcortical regions?

A

projection

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7
Q

a lesion in the commissural pathways would manifest in a deficit in the…

A

homologous region in the opposite hemisphere

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8
Q

infarct vs penumbra

A

penumbra is potential site for spread of infarction, but not yet irreversibly damaged

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9
Q

deficits associated with left hemisphere damage

A

right sided sensory and motor deficit, aphasia, alexia, gerstmann’s syndrome, tactile agnosia, apraxia (movement deficit), verbal memory impairment, executive reasoning

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10
Q

deficits associated with right hemisphere damage

A

elft sided sensory and motor deficits, arousal, orientation, awareness deficits, neglect of left space, constructional and dressing apraxia, aprosodia

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11
Q

each hemisphere’s spatial awareness capabilities

A

LH- only right spatial awareness

RH- left and right spatial awareness (so loss of RH could still have right spatial awareness)

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12
Q

what is alexia without agraphia? what location would the lesions be in? what artery could be blocked?

A

unable to read written info, even if they write it themselves. damage to visual cortex in the left hemisphere and the splenium of the corpus callosum

-left posterior cerebral artery supplies these

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13
Q

the somatosensory areas of the brain. what are the primary sensations that are processed here?

A

areas 3, 1, 2; touch, proprioception; stimulation produces tingling, numbness

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14
Q

damage to the somatosensory areas causes..

A

contralateral hyperesthesia

astereognosis

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15
Q

the somatosensory association cortices

A

superior parietal areas 5 and 7 (input from 3, 1, 2 and visual area 7)

supramarginal gyrus area 40 (sensory, auditory, and visual input)

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16
Q

damage to superior parietal association results in

A

contralateral loss of tactile discrimination (astereognosis) and inability to recognize forms and body position

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17
Q

damage to supramarginal gyrus results in

A

apraxia, aphasia, spatial neglect

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18
Q

inability to see more than 1 object at a time (usually bilateral damage)

A

simultanagnosia

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19
Q

deficit in reaching under visual guidance that cannot be explained by motor, sensory, visual field defects

20
Q

decreased awareness for the side of the body or objects in space located contralateral to brain injury// what location is commonly damaged and causes this?

A

hemispatial neglect

right parietal lobe

21
Q

what location of lesion is visual agnosia more commonly associated with?

A

inferior temporal lobe and adjacent visual cortex

22
Q

*components of Balint’s syndrome

A

psychic paralysis of gaze with haphazard scanning

optic ataxia

simultanagnosia

23
Q

what causes Balint’s syndrome?

A

bilateral lesions in parieto-occipital cortex due to stroke, trauma, or degenerative disease

24
Q

presentation of hemispatial neglect

A

eyes and head deviate ipsilaterally

eat food on left side of plate, fail to dress left side of body, fail to acknowledge people on the left

25
tests for diagnosing hemispatial neglect
line bisection, line cancellation
26
right posterior parietal cortex lesion results in unilateral visual neglect of what side?
left side (neglect is frequently the result of damage to the right hemisphere!)
27
functions of the inferior areas of the left hemisphere parietal lobe
language, skilled movement, simple math
28
fucntions of the right side inferior parietal lobe
spatial and non-spatial cognition, attention, memory
29
superior areas of parietal lobe on both sides mediates..
reaching, grasping, tactile exploration, oculomotor function, visually guided action and intentions to perform movemnts
30
primary and secondary visual areas
primary- 17 | secondary-18, 19
31
destruction of area 17 results in.... destruction of areas 18 and 19 results in.....
17-visual field defects 18,19- hallucinations, agnosia, alexia
32
describe Anton's syndrome/ what typically causes it
form of cortical blindness in which px denies visual impairment confabulation is common caused by bilateral damage to occipital lobe extending from primary visual cortex to association cortex
33
gyrus for visual association; where do inputs come from?; what does damage result in?
angular gyrus area 39 receives heteromodal input optic radiation damage results in contralateral hemianopia
34
left hemisphere destruction of angular gyrus (visual association) is associated with this syndrome
Gerstmann's syndrome
35
right hemisphere destruction of angular gyrus (visual association) is associated with this deficit
hemi-neglect
36
4 symptoms of gerstmann's syndrome
agnosia (cant ID things), left-right confusion, agraphia, acalculia may or may not have visual field defects
37
a deficit in visual object recognition
visual agnosia
38
rare inability to copy, recognize, or discriminate shapes
apperceptive visual agnosia
39
more common- has shape perception and can draw objects but can't associate the visual object with its meaning; can't name objects or show recognition b y pointing
associate visual agnosia
40
location of lesion that usually causes associative visual agnosia
inferior temporal lobes
41
which plays bigger role in spatial processing and spatially directed movements: occipital cortex or superior partietal lobes?
superior parietal lobes
42
what and where pathways: which is dorsal and goes to posterior parietal lobe? which is ventral and goes to inferior temporal lobe?
where goes to posterior parietal what goes to inferior temporal
43
difference between magno and parvocellular pathways/location?
magno cells have large bodies- operates quickly parvo cells have small bodies- operate slowly with more details LGN
44
auditory areas/what gyrus? input?
41, 42; heschl's gyrus; input from MGN; destruction results in partial deafness
45
association cortex area? location? destruction of LH vs RH?
association cortex is area 22 in posterior superior temporal gyrus LH damage results in Wernicke's aphasia RH damage results in sensory dysprosodia
46
motor areas and their locations
primary- area 4 (precentral gyrus) - hemiparesis premotor- area 6; precentral gyrus - apraxia, dystonia
47
prefrontal cortex damage results in
impaired social behavior, decreased initiation, suck and grasp responses, incontinence, abulia, mutism