Flashcards in Test 1 Deck (131)
When do anomalies in size/shape occur?
When do anomalies in structure occur?
Histodifferentiation, apposition, calcification (mineralization).
When do anomalies in color occur?
Histodifferentiation, apposition, calcification (mineralization)
What is the most prevalent craniofacial congenital malformation in humans?
Tooth agenesis! Hypodonita!
1-6 teeth absent, not including 3rd molars
More than 6 teeth missing, excluding 3rd molars.
Anodontia and Agenesis
Complete absence of teeth in 1 or both dentitions. Agenesis is in both.
What are the most common missing teeth?
Permanent: MN 2nd premolar, MX lateral incisor, MX 2nd premolar.
If primary tooth doesn't develope, permanent tooth won't develop.
Common occurance. The missing tooth is the most distal of a given type. Can be environmental or genetic.
What are the oral conditions seen in Van der Woude Syndrome?
CL/CP. Lip pits. Adhesions between MX and MN. Taurodontism in 50%.
Variable defects in morphogenesis of ectodermal derivatives includes: hypodontia, anodontia, conical or peg shaped anterior teeth, alveolar ridge deficiency, hypoplastic maxillae, anterior x-bite, decreased vertical dimension.
ED syndrome. Dry hair, severe hypodontia, smooth tongue, nail dysplaisa, hyperhidrosis of palms/soles, hyperkeratosis.
Malformations in eye, umbilical and hypodontia. Affects both dentitions. MN 2nd premolars, then central incisors and MX 2nd premolars. Hyperplastic MX frena, peg shaped or conical inciors, microdontia, enamel hypoplasia, short roots, taurodontism, delayed eruption.
Midline cleavage of the embryonic forebrain. Most severe is cyclopia. Mildest is a single MX central incisor.
Solitary Median MX Control Incisor
Single central incisor. HPE will always have SMMCI but not all with SMMCI have HPE.
Lack of cartilage calcification leads to deficient growth of limbs. Micrognathia, dental crowding, hypodontia, delayed eruption.
Cleft lip and palate
Hypodonita in 80%! Increases with severity of cleft. Agenesis, dysmorphology (microdontia), supernumerary teeth. Most frequently missing: MX lateral incisors followed by MX and MN 2nd premolars.
Pierre Robin Sequence
Hypodontia, especially in the MN. Micrognathia, glossoptosis, CP.
When do disturbances cause hyperdontia?
Initiation and proliferation.
What are the two hyperdontia types?
Supplemental: Normal or typical anatomy.
Rudimentary: Dysmorphic anatomy.
Is hyperdontia more common in permanent or primary?
Where does hyperdontia most often occur?
Where do multiple supernumeraries normally occur?
Accessory fourth molar.
Posteror supernumerary lingual/buccal to molar.
What are the two syndromes with hyperdontia?
Apert and Cleidocranial dysplaisa.
Hyperdontia! Delayed ectopic eruption. Narrow palate!
No clavicles. Multiple supernumerary teeth with delayed eruption and high palate.
What are the syndromes with both hyper and hypodontia?
Downs, crouzon, oral-facial-digital syndrome, type 1.