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Flashcards in Test 1 Deck (131)
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61

When do anomalies in size/shape occur?

Morphodifferentiation

62

When do anomalies in structure occur?

Histodifferentiation, apposition, calcification (mineralization).

63

When do anomalies in color occur?

Histodifferentiation, apposition, calcification (mineralization)

64

What is the most prevalent craniofacial congenital malformation in humans?

Tooth agenesis! Hypodonita!

65

Hypodontia

1-6 teeth absent, not including 3rd molars

66

Oligodontia

More than 6 teeth missing, excluding 3rd molars.

67

Anodontia and Agenesis

Complete absence of teeth in 1 or both dentitions. Agenesis is in both.

68

What are the most common missing teeth?

Permanent: MN 2nd premolar, MX lateral incisor, MX 2nd premolar.
Primary: Incisors
If primary tooth doesn't develope, permanent tooth won't develop.

69

Sporadic Hypodontia

Common occurance. The missing tooth is the most distal of a given type. Can be environmental or genetic.

70

What are the oral conditions seen in Van der Woude Syndrome?

CL/CP. Lip pits. Adhesions between MX and MN. Taurodontism in 50%.

71

Ectodermal Dysplasia

Variable defects in morphogenesis of ectodermal derivatives includes: hypodontia, anodontia, conical or peg shaped anterior teeth, alveolar ridge deficiency, hypoplastic maxillae, anterior x-bite, decreased vertical dimension.

72

Odonto-ochyno-dermal dysplasia

ED syndrome. Dry hair, severe hypodontia, smooth tongue, nail dysplaisa, hyperhidrosis of palms/soles, hyperkeratosis.

73

Rieger Syndrome

Malformations in eye, umbilical and hypodontia. Affects both dentitions. MN 2nd premolars, then central incisors and MX 2nd premolars. Hyperplastic MX frena, peg shaped or conical inciors, microdontia, enamel hypoplasia, short roots, taurodontism, delayed eruption.

74

Holoprosencephaly

Midline cleavage of the embryonic forebrain. Most severe is cyclopia. Mildest is a single MX central incisor.

75

Solitary Median MX Control Incisor

Single central incisor. HPE will always have SMMCI but not all with SMMCI have HPE.

76

Achondroplastic Dwarfism

Lack of cartilage calcification leads to deficient growth of limbs. Micrognathia, dental crowding, hypodontia, delayed eruption.

77

Cleft lip and palate

Hypodonita in 80%! Increases with severity of cleft. Agenesis, dysmorphology (microdontia), supernumerary teeth. Most frequently missing: MX lateral incisors followed by MX and MN 2nd premolars.

78

Pierre Robin Sequence

Hypodontia, especially in the MN. Micrognathia, glossoptosis, CP.

79

When do disturbances cause hyperdontia?

Initiation and proliferation.

80

What are the two hyperdontia types?

Supplemental: Normal or typical anatomy.
Rudimentary: Dysmorphic anatomy.

81

Is hyperdontia more common in permanent or primary?

Permanent!

82

Where does hyperdontia most often occur?

MAXILLARY! Mesodens!

83

Where do multiple supernumeraries normally occur?

MN. Premolars.

84

Distomolar

Accessory fourth molar.

85

Paramolar

Posteror supernumerary lingual/buccal to molar.

86

What are the two syndromes with hyperdontia?

Apert and Cleidocranial dysplaisa.

87

Apert Syndrome

Hyperdontia! Delayed ectopic eruption. Narrow palate!

88

Cleidocranial Dysplasia

No clavicles. Multiple supernumerary teeth with delayed eruption and high palate.

89

What are the syndromes with both hyper and hypodontia?

Downs, crouzon, oral-facial-digital syndrome, type 1.

90

What is the most common variation in tooth size/shape/form?

Permanent MX lateral incisors and 2nd premolars.