Test 2 random tid bits Flashcards

(21 cards)

1
Q

Posner task

A

(attention)
to study top-down processing (have true and false cues, measure time it takes to respond)

  • think paying attention to woman talking in front of you
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2
Q

attentional capture

A

(attention)
to study bottom-up processing (have distractors)

  • think sister tapping you on the shoulder
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3
Q

contingent capture

A

(attention)
to study bottom-up and top-down processing

  • think sister tapping you on the shoulder then responding to exogenous cue with endogenous search task
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4
Q

representational model

A

(attention)
a model of explanation for hemispatial neglect, it says that R temporal lobe is contralateral and ipsilateral and L temporal lobe is contralateral, so when damage to R lobe, have difficulty with left visual field

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5
Q

attentional bias model

A

(attention)
a model of explanation for hemispatial neglect, it says that R and left temporal lobes are both contralateral, but L is more so than R and they compete for resources, so when damage to R lobe, have difficulty in L visual field

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6
Q

guided search model

A

(attention)
a way to search visual field for task, results in activation map

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7
Q

rapid serial visual processing paradigm

A

(attention)
a way to measure our attentional load, when we respond to one cue, our ability to respond to a secondary aspect that captures our attention is diminished, demonstrating the attentional blink

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8
Q

pop-out and conjunction search

A

(attention)
this study is to measure how quickly we can identify our attentional goal in our sensory field. this is made more difficult with salient stimuli (ex. stroop task) and the more attentional resources needed to complete a task, the less likely there are spare resources to attend to anything else

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9
Q

HM

A

Lesion: bilateral medial temporal lobe

Symptoms: anterograde amnesia (couldn’t make new declarative memory), intact WM, can make new nondeclarative memories

Evidence of: MTL separate from WM and non-declarative memory system, episodic memory theory (relies on MTL)

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10
Q

KF

A

Lesion: L temporo-parietal cortex

Symptoms: impaired WM, intact declarative memory

Evidence of: SM and declarative memories as a separate system

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11
Q

MS

A

lesion: R occiptal cortex

Symptoms: impaired perceptual priming, intact explicit recognition (about memory not perception)

Evidence of: involvement of visuo-sensory areas in perceptual priming

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12
Q

KC

A

lesion: MTL damage (kids w hypoxia, damaged HC)

Symptoms: no episodic memories at all, intact semantic memories

Evidence of: relevance of MTL to episodic memory but separation of semantic memories, episodic memory theory

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13
Q

Standard consolidation theory

A

(Memory)

memory consolidation and encoding occurs initially in the HC, then to the associative cortices, so HC is just redundant and unecessary

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14
Q

Multiple trace theory

A

(memory)
episodic memory is all stored in HC, but semantic is elsewhere

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15
Q

Cognitive map theory

A

(memory)
says that the utility of the MTL is that spatial memories are stored in grid cells in the entirhinal cortex, then transferred to the HC

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16
Q

relational memory theory

A

(memory)
takes cognitive map theory further and extends to any relations, not just spatial

17
Q

episodic memory theory

A

(memory)
says that all episodic memories are encoded by the HC, but not semantic (evidence from KC and HM, who have intact semantic but not episodic memory and disrupted HC)

18
Q

declarative memory theory

A

(memory)
all declarative memories are encoded by the MTL (ofc episodic are the first to go bc they’re more repeatedly encoded

19
Q

3 process model

A

(memory)
integrates many theories, says perirhinal ‘what’ stream and the parahippocampal; ‘where’ stream integrates info in the HC

20
Q

top down processing

A

(memory)
for familiarity bc requires more work
dorsal parietal region

21
Q

bottom up processing

A

(memory)
for recollection bc requires less work