The Child with Respiratory Alterations - Lecture Notes Flashcards

1
Q

One of the most common illnesses in children

A

Respiratory

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2
Q

Nasopharynx

A

smaller, easily occluded during infection

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3
Q

Lymph tissue (tonsils, adenoids)

A

grows rapidly in early childhood; atrophies after age 12

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4
Q

Small oral cavity and large tongue

A

increases risk of obstruction
- especially in unconscious child

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5
Q

Smaller nares

A

easily occluded

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6
Q

Long, floppy epiglottis

A

vulnerable to swelling which results in obstruction

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7
Q

Larynx and glottis

A

higher in neck, increasing risk of aspiration

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8
Q

Immature thyroid, cricoid, and tracheal cartilages

A

may easily collapse when neck is flexed

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9
Q

Fewer muscles are functional in airway

A

less able to compensate for edema, spasm, and trauma

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10
Q

Large amounts of soft tissue and loosely anchored mucous membranes lining airway

A

increase risk of edema and obstruction

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11
Q

smaller lung capacity and underdeveloped intercostal muscles

A

give children less pulmonary reserve

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12
Q

Higher respiratory rates and demands for O2 in young children

A

makes hypoxia easy to occur

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13
Q

where is the airway the smallest?

A

airway is smallest at the cricoid for children younger than 8 years - can be occluded very easily.

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14
Q

what gender is more susceptible to airway obstruction and respiratory distress?

A

male children

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15
Q

Infant and Toddler breathing

A
  • barrel-chested
  • rely heavily on the diaphragm for breathing
  • lack the firm bony structure to ribs/chest makes child more prone to retractions when in respiratory distress
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16
Q

CREBS

A

Cough
Rate/regularity
Effort (work of breathing)
Breath sounds
Saturation

17
Q

Quality of Respirations

A

Rate, regularity, symmetry, effort, accessory muscles, breath sounds, ability to speak (not able to speak is a big sign of respiratory distress)

18
Q

Respiratory Distress Associated Observations

A

Retractions, nasal flaring, head bobbing, snoring, grunting, colour, chest pain, clubbing, cough

19
Q

Respiratory Assessment (QQAPBS)

A

Quality of Respirations
Associated Observations
Quality of pulse
Position of the child
Behaviour change
Signs of dehydration

20
Q

Respiratory Distress

A

what you see initially, showing signs that this child is having difficulty breathing

21
Q

Respiratory Failure

A

when the child can no longer maintain effective gas exchange. there is either functional or structural failure of the mechanisms of respiration

22
Q

Clinical Manifestations of Respiratory Distress (3)

A

Hypoxemia: lack of oxygen in the blood
Hypercapnia: too much carbon dioxide
Alveolar hypoventilation: not enough gas exchange is taking place at the alveoli that are not being ventilated properly

23
Q

3 things that can cause alveolar hypoventilation

A
  1. O2 need exceeds O2 intake. Mismatch between supply and demand
  2. Airway partially occluded: foreign body, edema, swelling, mucus production
  3. Transfer of O2 and CO2 in the alveoli is disrupted: between alveoli and capillary Ventilation perfusion mismatch
24
Q

Progression of Respiratory Failure/Arrest: Cardinal Signs (DTTR)

A
  • Restlessness
  • Tachypnea
  • Tachycardia
  • Diaphoresis
    Subtle, not that particular to any one condition. See there if a child was frightened. Important to notice and take into consideration the entire history of the child
25
Q

Early Decompensation (less obvious): they were managing ok and now they are starting to not manage as well

A
  • mood changes
  • headache
  • increased WOB
  • hypertension
  • exertional dyspnea
  • anorexia
  • increased cardiac output & urinary output
  • CNS symptoms (anxiety, confusion, restlessness, irritability, depressed LOC)
  • nasal flaring
  • retractions
  • expiratory grunting
  • wheezing/prolonged expiration
26
Q

Severe Hypoxia

A
  • hypotension
  • depressed respiration
  • dyspnea
  • bradycardia
  • cyanosis
  • stupor and coma
27
Q

Early Recognition is the Goal

A

important to watch for early signs and intervene early so the child doesn’t go into respiratory arrest. Children’s hearts are usually pretty healthy - most don’t have cardiac arrest, they would have respiratory arrest which if not corrected will eventually cause their heart to fail as well. And if they go into cardiac arrest they have a really poor prognosis

28
Q

Continuum of Respiratory Failure: Initial Signs (DTTRP)

A

Physiology: the child is attempting to compensate for an oxygen deficit and airway blockage. The oxygen supply is inadequate; behaviour and vital signs reflect compensation and beginning hypoxia
- restlessness
- tachycardia
- tachypnea
- diaphoresis
- pale

29
Q

Continuum of Respiratory Failure: Early Decompensation (CNGHHHAMW)

A

Physiology: the child uses accessory muscles to assist oxygen intake; hypoxia persists and efforts now waste more oxygen than is obtained
- Confusion
- nasal flaring
- grunting
- head bobbing
- headache
- hypertension
- anxiety & irritability
- mood changes
- seesaw respirations
- Wheezing

30
Q

Continuum of Respiratory Failure: Imminent Resp Arrestd (CNNDBS)

A

Physiology: the oxygen deficit is overwhelming and beyond spontaneous recovery. Cerebral oxygenation is dramatically affected; central nervous system changes are ominous
- Dyspnea/bradypnea
- bradycardia
- seesaw respiration’s
- cyanosis
- no air movement
- no wheezing