Unit 1 - Cardiology (intro to Atrial arrhythmias) Flashcards

(80 cards)

1
Q

What physical exam findings are associated with mild cases of cardiovascular disease?

A

no clinical signs, reduced exercise tolerance

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2
Q

What physical exam findings are associated with moderate cases of cardiovascular disease?

A

tachycardia, tachypnea, dyspnea post exercise, abnormal arterial pulse, jugular distention, edema over ventral abdomen, edema in the distal limbs, and pathologic arrhythmias

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3
Q

What physical exam findings are associated with severe cardiovascular disease?

A

cool extremities, weight loss, reduced capillary refill time, cough, dyspnea at rest, and collapse

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4
Q

What are the reasons for evaluating for cardiovascular disease?

A

obvious signs of heart disease, poor athletic performance, pre-purchase examination, insurance examination

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5
Q

What are the components of a cardiac evaluation?

A

auscultaion, ECG, echo, and exercise testing

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6
Q

What is the normal resting heart rate of the horse?

A

20-40 beats per minute

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7
Q

What is S1?

A

sound 1 - the lub followed by systole

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8
Q

What is S2?

A

sound 2 - the dub followed by diastole

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9
Q

What is S4?

A

the atrial sound

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10
Q

What makes the atrial sound?

A

it arises from ventricular vibrations set up when pressure wave caused by the atrial contraction reaches the ventricle

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11
Q

When arrythmia is an isolated S4 associated with?

A

2nd (and 3rd) degree A-V block

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12
Q

When is S4 absent?

A

in a sinoatrial block

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13
Q

What does S4 immediately preceed?

A

lub

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14
Q

When and where does S1 come from?

A

It arises mostly from the ventricle, after AV valve closure

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15
Q

What does the atrial-S1 interval depend on?

A

the P-Q interval

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16
Q

What is S2 made by?

A

closure of semi-lunar valves and rapid reversal of blood flow

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17
Q

What is S3 made by?

A

early, rapid ventricular filling

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18
Q

Where anatomically is S3 heard? In what percent of horses?

A

left side, 30-50%

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19
Q

What are the semi-lunar valves?

A

aortic and pulmonic valves

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20
Q

Where is S1 the loudest on auscultation?

A

over the mitral valve - in the intercostal space between ribs 5 and 6, right at the caudal edge of the triceos

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21
Q

Where is S2 the loudest on auscultation?

A

over the aortic valve - in the intercostal space between ribs 4 and 5

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22
Q

What makes the P wave on an ECG?

A

right atrial depolarization with a wave spreading from the S-A node across the right atrium, the interatrial septum, and then the left atrium

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23
Q

At rest, how does the P wave appear?

A

it is often bifid

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24
Q

What does the first small ‘hump’ of the p-wave represent?

A

mostly right atrial depolarization

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25
Is the QRS wave due depolarization as a wavefront like the p wave is?
No, the depolarization is NOT as a wavefront across the chambers due to much branching of the conducting system
26
QRS at body surface reflects about ___ of the depolarization of the myocardial mass.
20%
27
What causes the T wave?
repolarization of the ventricles
28
The equine T-wave is very \_\_\_\_.
labile
29
The T-wave changes polarity with \_\_\_\_\_\_.
heart rate
30
What can the T-wave not be used to assess?
heart strain
31
What is ECG only useful for determining?
arrhythmias
32
A.
bifid P wave
33
B.
diastole
34
C.
S4 - ba
35
D.
QRS complex
36
E.
systole
37
F.
S1 - lub
38
G.
T-wave
39
H.
Diastole
40
I.
S2 - dub
41
J.
S3 - ah
42
If a murmur is heard at E, what type of murmur is it?
a systolic murmur
43
If a murmur is heard at B or H, what type of murmur is it?
a diastolic murmur
44
Where will a atrial arrythmia be heard?
at S4
45
Is systole A or B? Diastole?
Systole is A, diastole is B
46
What depth is required for echo in horses? What size transducer?
a depth of 30 centimeters, 3.5 mHz transducer
47
What is echocardiography useful in detecting?
congenital anomalies
48
When should exercise testing take place?
cardiac evaluation should be performed at or immediately after expected level of exercise
49
What are the types of atrial arrhythmias?
wandering pacemaker, premature sinus beats, premature atrial beats, sino-atrial block, sinus bradycardia, post exercise sinus arrhythmia, and atrial fibrillation
50
Is wandering pacemaker a true arrhythmia?
not really, it is an ECG finding
51
How does a wandering pacemaker present on ECG?
the p-wave shape may change from beat to beat, but the rhythm is regular
52
Is wandering pacemaker clinically significant?
no
53
How do premature sinus beat arrythmias present on ECG?
P waves are earlier than normal, but of normal conformation. The QRS wave is normal
54
When do premature sinus beats disappear?
with exercise
55
When do premature sinus beats suggest myocardial disease?
when there are frequent ones - it is not significant if there is one occasionally every 5 minutes
56
What is the recommended treatment for horses that have frequent premature sinus beats?
to rest the horses and reevaluate in 4-6 months
57
How do premature atrial beats appear on ECG?
the same as premature sinus beats, but the P waves are of abnormal conformation
58
What causes the abnormal conformation of P waves in premature atrial beat arrhythmias?
there is an ectopic focus in the atrial muscle
59
When do premature atrial beats suggest myocardial disease? Treatment?
If they are very frequent, it suggests myocardial disease. Rest these horses, and then reevaluate in 4-6 months.
60
How do sino-atrial blocks appear on ECG?
There is no P-wave and a twice normal diastolic pause (2 dubs)
61
What horses are sino-atrial blocks more common in?
ponies
62
What abolishes sino-atrial block?
exercise
63
What mediates sino-atrial block?
it is vagally mediated
64
Is sino-atrial block clinically significant?
not usually
65
What horses typically have sinus bradycardia?
mostly ponies - it is rare
66
How does sinus bradycardia present?
as a very slow irregular atrial rhythm
67
What clinical signs are associated with sinus bradycardia?
reduced exercise or even lethargy
68
What may reslove sinus bradycardia?
rest
69
When are post exercise sinus arrhythmias common?
after light trotting exercise in fit horses
70
How do post-exercise sinus arrhythmias occur?
As the heart rate slows, there are sudden pauses, and then a gradual return to the pre-pause rate
71
Are post-exercise sinus arrhythmias significant? How can they be abolished?
Not usually significant, abolished by more rigorous exercise
72
What clinical signs are associated with atrial fibrilation?
they are variable - they could have none up to heart failure, they may have pulse deficits
73
On auscultation, how does atrial fibrilation sound?
there are no atrial sounds, irregularly spaced group of irregular beats, S1 and S2 vary in intensity due to variable ventricular filling
74
What heart rate is associated with atrial fibrilation?
the heart rate can range from 30-40 up to 80-100
75
What treatment is recommended for atrial fibrillation?
It is best if it is done early in the disease - oral quinidine is a drug of choice
76
How is oral quinidine administered?
via a nasogastric tube
77
What is the protocol for oral quinidine treatment for atrial fibrillation?
Give a test dose of 5 grams on day 1, if no adverse reaction then proceed to day 2 On day 2, give 20 grams as a start up dosage and give an additional 10 grams every 2 hours (make sure to perform an ECG before giving each dose)
78
What dosage should you not exceed for oral quinidine per day?
do not exceed 90 grams/day in a 1000 pound horse
79
What toxic signs are associated with atrial fibrillation treatment of quinidine?
Depression (often), tachycardia, diarrhea (often), colic (often), urticaria, nasal edema, weakness, ataxia, and death
80
Aside from oral quinidine, what can be used to treat atrial fibrillation?
IV quinidine (expensive and risky), Digoxin (if heartrate is greater than 60 bpm), and electrical cardioversion (shocking)