Unit 2 Flashcards

(47 cards)

1
Q

one-dimensional model of causality

A
  • attributes causes of behavior to a single cause
  • Is linear in approach (e.g., biological or social or
    psychological)
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2
Q

multi-dimensional model of causality

A
  • attributes causes of behavior to several causes
  • Systemic, interdisciplinary
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3
Q

multidimensional approach: Behavioural influence

A

Phobia as a conditioned response, generalizing to other situations

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4
Q

multidimensional approach: biological influences

A

Overactive vasovagal reaction (vasovagal syncope)

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5
Q

multidimensional approach: emotional influences

A

Behaviour, thoughts, and feelings influences biology

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6
Q

multidimensional approach: social influences

A
  • Influence behaviour and biology
  • Support or rejection can contribute to psychopathology
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7
Q

multidimensional approach: developmental influences

A
  • Developmental critical/sensitive periods - more or less reactive to given situations
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8
Q

gene-environment interactions

A
  • Genes: unit of heredity passed from parent to offspring
  • Behaviour, personality, intelligence, psychological disorders are polygenic (influenced
    by many genes)
  • Environmental factors can also contribute to gene expression (epigenetics)
    About 50% of variability in personality, cognitive, and emotional traits attributed to
    genetic influence
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9
Q

General Conclusions Re:
Gene Involvement in
Psychopathology

A
  1. Genes are involved in the etiology of psychological
    disorders
  2. Contributions of genes to psychological disorders
    come from many genes (polygenic), each having a
    small effect
  3. Genetic contributions cannot be studied in the
    absence of interactions with the environment
    (epigenetics)
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10
Q

gene and environment interaction

A
  • Environment may occasionally turn on certain genes
  • Could lead to changes in biochemical functioning in the brain
  • Brain is plastic – subject to change in the environment
  • Gene-environment interaction models relevant to psychopathology:
    1. Diathesis-Stress Model
    2. Reciprocal Gene-environment Mode
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11
Q

diathesis-stress model

A

psychological disorders result from an interaction between inherent vulnerability and environmental stressors

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12
Q

reciprocal gene-environment model

A
  • Genes may increase the probability that an individual will experience a stressful life event
    -the Hypothesis that people with a genetic predisposition for a disorder may also have a genetic tendency to create environmental risk factors that promote the disorder
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13
Q

types of gene-environment correlations: passive

A

individual has a certain gene, and grows up in an environment that is influenced
by that gene

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14
Q

types od gene-environment correlations: evocative

A

Individual’s genetically influenced characteristics evoke responses from others in the environment

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15
Q

types of gene-environment correlations: active

A

Individuals seek out or create environments that encourage their genetic predispositions

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16
Q

epigenetics

A
  • In a study with rodents that were cross-fostered
  • Maternal behaviour impacted how young rats
    tolerated stress
  • Calm and supportive behaviour by mothers could be
    passed down through generations
  • Epigenetics: Environment can influence DNA by
    changing gene expression
  • Can be passed down to future generations
    (intergenerational transmission
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17
Q

hindbrain

A

coordinates functions that are fundamental to survival, including respiratory rhythm, motor activity, sleep, and wakefulness

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18
Q

midbrain

A

involved in several functions, including motor control, particularly eye movements and processing of vision and hearing

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19
Q

forebrain

A

important for the planning and execution of movements, sensory processing, regulating sleep wake states and behavioral responses to emotions such as stress and fear

20
Q

brain main structure

A

the cerebrum, cerebellum and brainstem. Cerebrum: is the largest part of the brain and is composed of right and left hemispheres. It performs higher functions like interpreting touch, vision and hearing, as well as speech, reasoning, emotions, learning, and fine control of movement

21
Q

peripheral nervous system

A
  • the nervous system outside the brain and spinal cord
    -The sympathetic nervous system prepares the body for “fight or flight,” whereas the parasympathetic nervous system allows the body to “rest and digest.”
22
Q

neurotransmitters

A

Activate or inhibit impulse in the post-synaptic neuron
* Abnormal behaviour can result from;
1. Too much or too little neurotransmitters in the synapse
2. Too few or too many receptors on the dendrites
3. Excess or deficit in amount of transmitter-deactivating substance in the
synapse
4. Reuptake process can be too rapid or too slow
* TOO MUCH EXCITATION OR TOO MUCH INHIBITION → Abnormal
Behaviour

23
Q

central nervous system

A

-made up of the brain and spinal cord
-sending and receiving messages to various parts of the body

24
Q

hypothalamic-pituitary-adrenal (HPA) axis

A
  • Part of the endocrine system
  • Hypothalamus → pituitary gland → adrenal
    gland → CORTISOL
  • Cortisol
  • Increases blood sugar, suppresses immune
    system, alters metabolism
  • Contributes to “fight or flight”
  • Implicated in psychological disorders
25
neurotransmitters: agonists
increase the activity of a NT by mimicking effects
26
neurotransmitters: antagonists
decrease or block of NT
27
neurotransmitters: inverse agonists
produce opposite effect of the NT
28
neurotransmittters: glutamate
* Excitatory effect * Fast acting -exhibitory
29
neurotransmitters: GABA
* Inhibitory effect * Fast acting * Reduces anxiety * Benzodiazepines are a GABA agonist → calming effect -inhibitory
30
neurotransmitters: serotonin (5HT)
* Monoamine category of NT * Regulates behaviour, mood, and thought processes * Low levels of 5HT * Less inhibition, instability, impulsivity * E.g., depression, aggression, suicidality, impulsive overeating, hypersexual behaviours * Selective serotonin reuptake inhibitors directly affect 5HT * Used to treat anxiety, mood, eating disorders -regulates behaviour and mood
31
neurotransmitters: norepinephrine
* Monoamine class of NTs * Part of the endocrine system * Stimulates alpha-adrenergic and beta-adrenergic receptors * Beta-blockers block beta-receptors, keeping blood pressure and heart rate down -increase blood pressure
32
neurotransmitter: dopamine
* Monoamine class of NTs * Implicated in schizophrenia and disorders of addiction * DA activity associated with exploratory, outgoing, and pleasure- seeking behaviours -reward system (pleasure)
33
psychosocial influences on brain structure and function
* Effects of treatment sometimes tells us about the cause * If OCD is caused by a faulty brain circuit
34
placebo effect
* Placebo medications result in behavioural and emotional changes * Mechanism of action: hope, expectation of change * Only seem to work when patients think they are receiving a drug * Suggests psychological factors are another option for changing brain circuits
35
psychosocial influences on brain structure and function: baxter
* Brain imaging before and after treatment with either medication or CBT * Found that the brain circuit had been changed by CBT * Other studies found evidence of changes in the brain following psychological treatment of PTSD, depression, phobias, social anxiety disorder, panic disorder, schizophrenia
36
psychosocial influences on the brain: kennedy
* Patients with major depressive episode treated with CBT and venlafaxine (antidepressant) * Showed similar brain changes as well as some complex differences: * CBT-treated: “Top Down”; changes in cortex affected emotional brain * Venlafaxine-treated: “Bottom-Up”; reaching higher areas of cortex last.
37
psyhcosocial influence on the brain: insel
* Two groups of monkeys * Group 1: free access to toys and treats * Group 2: only had access when the first group did * All monkeys received a dose of GABA inverse agonist (increases anxiety) * Group 1: Responded with anger and aggression * Group 2: Responded with severe anxiety and panic CONCLUSION: Psychosocial factors INTERACT with NTs to impact functioning
38
learned helpessness
* Martin Seligman * Animals receive a shock on their feet * Animals that can do something about it (e.g., escape, press a lever to avoid a shock) function well * Animals that can’t do anything about it become helpless – appear depressed * People react similarly -when human is faced with uncontrollable stress out of there ability to control they become helpless
39
social learning
-Albert bandura * Organisms (individuals) can learn by observing what happens to someone else in a given situation (Modeling or observational learning) * Much of what we learn depends on interactions with other people around us (Social learning theory)
40
prepared learning
* We learn to fear some objects much more easily than others * E.g., snakes and spiders vs. rocks and flowers * One trial learning * Attributed to evolution (survival of ancestors) * Perhaps people who avoided these objects survived in greater numbers and passed down their genes
41
cognitive behavioural therapy
* Developed by Aaron T. Beck * Dealing with faulty attributions and attitudes associated with learned helplessness and depression * Short term therapy – usually 10-20 sessions * Techniques include: * Cognitive Restructuring * Exposure Therapy * Relaxation * Problem Solving
42
emotions: mood and affect
* Subjective feelings * Action tendency – or strong motivation to act * Complex physiological or arousal response * Mood → More persistent period of affect or emotionality * Affect → momentary emotional tone that accompanies what we say or do
43
components of emotion: behaviour, cognitive, physiology
emotion of behaviour: -basic pattern of emotional behaviour (freeze, escape, approach, attack) that differ in fundamental ways -emotional behaviour is a means of communication cognitive aspects of emotion: -appraisals, attributions, and other ways of processing the world around you that are fundamental to emotional experience physiology of emotion: -emotion is a brain function involving (generally) the more primitive brain areas -direct connection between these areas and the eyes may allow emotional processing to bypass the influences of higher cognitive processes
44
emotions and psychopathology
* Dysregulated emotions contribute to almost all psychopathologies * Panic disorder → fear occurring at the wrong time * Bipolar disorder → Cycles of intense joy and happiness and sadness and depression * Depression → uncontrollable and pervasive sadness
45
cultural social and interpersonal factors
* Gender roles * Dramatic gender differences in certain disorders * Insect phobias and mood disorders – likely to be women * Due to cultural expectations of men and women? * May respond differently to treatment * Women maintained gains following treatment for PTSD longer than men * May impact treatment seeking behaviours
46
social effects on health and behaviour
* Higher quality social support and greater number of social relationships * Longer life * Protects against physical disorders such as high blood pressure, catching cold * Protect against depression * Perception of loneliness increases the likelihood of mortality and risk
47
principle of equifinality
* There are various developmental pathways or factors that lead to the same outcome * No single, exclusive cause for a particular psychological disorder * Psychopathology typically results from complex interactions between genetic, biological, behavioural, cognitive, social, cultural, and developmental factors * What may trigger a disorder in one person, may not have the same effect on another -very important for developmental PSYC -there are many ways to achieve a goal