Vestibular Rehab Flashcards

1
Q

1-24 hours (acute) Fullness of ear, hearing loss, tinnitus, vomiting. (+) nystagmus. Pt wakes up in the morning, an have to crawl to the bed (can’t go to work)

A

Ménière’s Disease

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2
Q

What are the key points to examine (to know) in vestibular patients?

A

Is it coming from the brain or from ear?

Is it one side or both?

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3
Q

Oscillopsia

A

decreased gaze stability

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4
Q

as I walk towards a subject, I am not able to stabilize gaze and it becomes blurry (oscillopsia)

A

Bilateral Vestibular Hypofunction

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5
Q

classic vestibular bilateral symptom?

A

oscillopsia

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6
Q

classic unilateral vestibular symptom

A

motion sensitivity

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7
Q

vertigo with strain ex. a pt blow their nose or strain in the toilet, they get vertigo

A

fistula

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8
Q

Vertical NYSTAGMUS

A

CENTRAL Finding until proved otherwise

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9
Q

Direction changing nystagmus; Look to the right, right beating nystagmus – look to the left, left beating:

A

This is a CENTRAL SIGN

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10
Q

Saccades:

A

Look from target to target

  • Significant Overshooting is a Central Sign
  • Multiple movements is a Central Sign
  • Undershoot is considered normal
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12
Q

Ask patient to focus on your nose, slowly move head side to side, observing for visual fixation

What is the name of this test? What does it test?

A

VOR, HEAD THRUST

  • Left - Right discrimination test
  • Decrease in fixation with forced LEFT ROTATION = LEFT DYSFUNCTION, + L Head Thrust test
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13
Q

Left to Right discrimination tests:

A
  1. head thrust
  2. Singleton’s test
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14
Q

Test for BPPV

A
  • HALLPIKE TEST
  • Patient starts in long sit, with head rotated 30 degrees towards the side to test. Ask the patient to keep their eyes open as you quickly bring them into a supine position with their head extended 10 degrees.
  • Classically pt will experience vertigo when placed in the hallpike position (affected ear 30 degrees below horizontal) and should demonstrate a torsional nystagmus. Vertigo is caused by excitation of the posterior semi-circular canal (which has now become gravity sensitive.) BPPV of anterior or horizontal SSC is rare. Patient may report episodes of vertigo over past months or years.
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15
Q
  • Initial episode can be with rolling for the snooze alarm or retrieving object from shelf.
  • complaints of vertigo (room spinning) with static positioning
  • Pt usually knows which positions are involved and avoids them!
  • Symptoms usually abate quickly with movement out of provoking positions.
A

BENIGN PAROXYSMAL POSITIONAL VERTIGO (BPPV)

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17
Q

Otoconia dislodge into the semicircular canals (usually posterior) and become free floating in the endolymph of the canal. When the head is moved into provoking positions endolymph is moved by debris which pulls cupula out of position. In the hallpike expect latency and fatigue nystagmus. Almost all cases of BPPV:

A

CANALITHIASIS

free floating = latency nystagmus

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18
Q

Otoconia become adhered to the cupula (end organ in the ampulla) making it gravity sensitive. Expect immediate nystagmus which may not fatigue.

A

CUPULOLITHIASIS

(could be a stroke)

19
Q

which canal is affected in upgoing, rotary nystagmus to the affected ear

A

Posterior Canal

20
Q

which maneuver would work best for immediate horizontal nystagmus (Cupulolithiasis)?

A

Liberatory maneuver

21
Q

which canal is affected in horizontal nystagmus?

A

horizontal canal

22
Q

which canal is affected in downgoing, rotary nystagmus to the affected ear

A

Anterior Canal

24
Q

which maneuver would work best for latency horizontal nystagmus (Canalithiasis)?

A

BARBEQUE ROLL