week 1 Flashcards

(110 cards)

1
Q

Pathology

A

study of structural changes in body fluids, cells, tissues and organs

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2
Q

Physiology

A

study of mechanical, physical and biochemical functions of living organisms

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3
Q

Pathophysiology

A

study of abnormalities in physiologic functioning of living beings. To examine disturbances of normal mechanical, physical and biochemical functions either caused by a disease or resulting from a disease or abnormal syndrome or condition

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4
Q

Aetiology

A

causes of disease

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5
Q

Pathogenesis

A

development of disease

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6
Q

Clinical manifestations

A

signs and symptoms

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7
Q

Epidemiology

A

study of disease occurrence, patterns and distribution in human populations

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8
Q

What is Heat Therapy?

A

Heat therapy, or thermotherapy, has been used for centuries to manage pain and aid recovery.

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9
Q

Contact Heating Methods:

A

Hot packs and pads used for localised heating to relieve pain

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10
Q

Paraffin Wax Baths

A

Effective for arthritis and joint stiffness

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11
Q

Hydrotherapy

A

Warm water immersion for muscle relaxation

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12
Q

how does Heat Therapy work

A
  • vasodilation to supply the site with O2 and nutrients
  • rate of local tissue metabolism- increased by which warming promotes healing
  • Pain Modulation: Heat activates Transient Receptor Potential (TRP) channels, which block pain signals.
  • Connective Tissue Effects: Enhances collagen extensibility, making tissues more flexible
  • Muscle Relaxation: Heat lowers nerve sensitivity, reducing spasms & stiffness
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13
Q

Heat Therapy in sports PRIOR to game/ training

A

Prepares muscles for movement by increasing tissue elasticity & blood flow.

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14
Q

Heat Therapy in sports AFTER to game/ training

A

Promotes faster recovery by removing waste products and enhancing glycogen resynthesis.

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15
Q

Heat Therapy in sports in recovery/ injury prevention

A

Regular heat therapy can reduce muscle damage & stiffness.

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16
Q

Heat Therapy in clinical practice/ recommended for conditions including

A

Knee osteoarthritis
Low back pain
Delayed Onset Muscle Soreness

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17
Q

methods of heat transfer

A

convection, conduction, radiation

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18
Q

Possible Physiological Effects of Heat

A

a) Local heating of tissues
b) Vascular effects
c) Metabolic reactions
d) Neuromuscular effects
e) Connective tissue effects
f ) Effects on viscosity

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19
Q

Metabolic Effects of heat

A

rate of chemical reactions increase w temp. this causes an O2 uptake increase and metabolic waste products increase

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20
Q

Neuromuscular Effects of heat therapy

A

Increased nerve conduction velocity with increase tissue temperature
Afferent nerves stimulated by heat may have an analgesic effect by acting on the “pain gate” mechanism
* Reduction of Pain
* Relaxation of muscle spasm

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21
Q

Connective Tissue
Effects from heat therapy

A

Extensibility of collagen tissue has been shown to increase with heat application if simultaneously stretched

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22
Q

Effects on Viscosity from heat therapy

A
  • Raising the temperature of
    liquids lowers their viscosity
  • Influences lymph, blood and joint fluid
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23
Q

Therapeutic Uses of Heat

A
  • Relief of pain
  • Relief of muscle spasm
  • Increase in joint range of
    movement
  • Lengthening of scar tissue
  • Sedation/relaxation
  • Resolution of chronic
    inflammation
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24
Q

Superficial Heating examples

A
  • hot packs
  • wax baths
  • infrared lamps
  • most forms of superficial
    heat application are
    conductive
  • heat is transferred from
    warmer to cooler by
    conduction
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25
Deep Heating examples
* ultrasound * shortwave diathermy (SWD) * microwave diathermy * occurs by conversion of another form of energy to heat * e.g.: energy in ultrasound waves produce heat in deeper tissues
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what are Hot Packs
* Provide a superficial moist heat * Stored in a thermostatically controlled hydrocollator * Wrap in 2-4 layers of towel for insulation before applying to the patient
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what are hot packs advantages
* Ease of preparation and application * Variety of shapes and sizes * Moist, comfortable heat * Relatively inexpensive
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what are hot packs disadvantages
* No method of temperature control once applied to patient * Do not conform to all body parts * Can be awkward to secure * Only retain heat for approx.20 minutes * Passive treatment * May be too heavy * Hot water hazard and spillages
29
what are Paraffin Wax
* Efficient source of superficial heat due to its low melting point * Liquid state allows even distribution of wax * Often used in treatment of rheumatoid arthritis (RA)
30
what are the paraffin wax advantages
* Even distribution * Adds moisture to skin * Can be used as exercise tool after treatment * Comfortable moist heat * Relatively inexpensive
31
what are the paraffin wax disadvantages
* Can only be used for distal extremities (hands and feet) * No method of temperature control once applied to the patient * Only retains heat for approx. 20 minutes
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Guidelines for safe application
a) Contraindications and Precautions b) Skin Tests c) Warnings given d) Monitoring during and after
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contraindications to heat therapy
* Circulatory insufficiency * Risk of dissemination * Exacerbation of existing conditions * To eyes or testes * Broken skin * DO NOT apply over transdermal drug patches or implants as may increase absorption of the drug which can be fatal (Fentanyl) Deep Vein Thrombosis (DVT): Heat application over an area with DVT could dislodge a thrombus, leading to a life-threatening embolism. Local Infection: Heat can exacerbate infection and promote bacterial growth in cases such as abscesses, osteomyelitis and septic arthritis Malignancy: Heat therapy can increase circulation, potentially accelerating cancer growth if applied near a tumour Severely Impaired Circulation: Heating ischaemic tissue could exacerbate tissue breakdown or cause burns due to impaired heat dissipation. Pregnancy: Whole-body heating is dangerous during pregnancy, and local heat (trunk, abdomen, pelvis) should be avoided over the foetal area Recently Radiated Tissue: Heat is contraindicated to an area if it has received radiation. Tissues may respond inadequately due to ‘tissue devitalisation’ for 3–6 months post radiotherapy Acute inflammatory skin disorders such as eczema and dermatitis as heat can exacerbate these conditions Inbuilt stimulators or metal implants: e.g., pacemakers is a CI in first 4 weeks of insertion
34
precautions to heat therapy
* Care using in conjunction with other modalities * Care if lying a patient on heat pack * May need more towels in application Metal Implants: Metal can concentrate heat, raising the risk of burns if heated Epilepsy: Though not directly contraindicated, practitioners should be cautious as thermal interventions may have unknown effects Photosensitivity: Some skin conditions and medications cause increased sensitivity to heat and UV exposure Over Open Wounds: Heat can delay wound healing and cause discomfort
35
Skin Tests: Thermal Sensitivity Test
* Tests the patient’s ability to discriminate hot from cold at the area to be treated * Done for ALL heat applications * Demonstrate on a non-affected area first * Typical items used are test tubes filled with warm and cool water * Care for spillages and dry or change linen immediately * Record the outcome
36
verbal Warning for a Heat Treatment
When having a heat treatment all you should feel is a mild, even, comfortable warmth. If you feel any more than this, or if heat concentrates in any particular spot or it starts to feel uncomfortable, please call me immediately otherwise you may be in danger of being burned. Please do not move or touch any of the equipment during the treatment. Do you understand this warning? Do you have any questions? Are you happy to proceed?
37
Monitoring heat application during
For hot packs, check skin immediately after application and again after 8 minutes and regularly thereafter * Observe skin and ask patient regarding the temperature of the pack * Look for even erythema, * Should be NO concentration of the sensation of heat * Bell/buzzer with patient
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Monitoring heat application after
* Skin should be a nice even erythema after the application * Clean up immediately
39
What is Cryotherapy?
Cryotherapy is the use of cold to treat injuries,
40
Types of Cryotherapy
Ice packs, gel packs Cold Water Immersion
41
cryotherapy in relation to Pain Relief (Hypoalgesia):
Reduces nerve conduction velocity, lowering pain perception. Blocks pain signals via mechanisms like the pain gate theory
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cryotherapy in relation to :Blood Flow Changes (Haemodynamics):
Constricts blood vessels, reducing blood flow and inflammation Effects are greater in superficial tissues than deep muscles
43
cryotherapy in relation to : Inflammation Reduction
Lowers white blood cell activity, reducing inflammation and tissue damage Minimises oxidative stress, preventing unnecessary tissue damage
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cryotherapy in relation to Prevention of Secondary Injury:
Slows metabolism around the injury, limiting cell death and promoting healing
45
Physiological Changes Due to Cooling
* Decreased skin temperature * Decreased muscle temperature * Decreased intra-articular temperature * Changes in blood flow * Decreased tissue metabolic rate * Decrease in nerve conduction velocity rapid locally and gradual deeper
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Changes in Blood Flow (Skin) bc of cryotherapy
* Blood flow decreases immediately during the first minutes of cold application due to vasoconstriction * Autonomic nervous system response triggered by stimulation of thermal receptors in skin * Increased blood viscosity and reduced blood flow * Reduced heat conduction to skin * After 5-10 minutes vasoconstriction gives way to vasodilation * This may last 15 minutes before being replaced by another episode of vasoconstriction
47
Changes to Blood Flow (Muscle) bc of cryotherapy
* Response in muscle to cooling is less dramatic * Decreased blood flow * Vasoconstriction * Increased viscosity of blood
48
Reduced Metabolic Reactions bc of cryotherapy
* Metabolism is a series of chemical reactions * Cooling tissue will reduce the rate of these chemical reactions (Van’t Hoff’s Law) * Opposite to heating tissue * Limits likelihood of ischemia (secondary cell hypoxic damage)
49
Decrease in nerve conduction velocity bc of cryotherapy
* All nerve fibres are not equally affected by cooling * Myelinated nerves and smaller diameter fibres most affected * Smaller type A fibres are most affected * Fast pain, mechanoreceptors, cold receptors * C fibres (unmyelinated) are least affected * Chronic pain * Reduced motor skill * Reduced motor nerve conduction of afferent feedback
50
Proposed Therapeutic Uses of Cold
* Relief of pain * Treatment of acute injury * Secondary cell hypoxia * Effects on motor system * Relief of muscle spasm * Treatment of oedema * Treatment of chronic inflammatory conditions
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Relief of Pain from cryotherapy
* Decreased conduction velocity of pain receptors and neurons * Decreased release of pain- inducing irritants * Release of endorphins and encephalins * Also reduction of muscle spasm
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Treatment of Acute Injury cryotherapy
* Immediate application of cold minimizes the extent of soft tissue damage (secondary cell hypoxia) and bleeding * Decreased metabolism * Decreasing the inflammatory response may limit oedema formation
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cryotherapy Effects on Motor system
* Long applications of ice reduces muscle strength * Short applications increase isometric muscle strength * Reduced motor skills & coordination * Due to slowed nerve conduction
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Reducing muscle spasm cryotherapy
* Reducing pain helps to reduce muscle spasm * Reducing muscle spasm allows increased range of movement
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cryotherapy Treatment of Oedema
No indication that isolated use of ice alone increases function, reduces swelling and pain in acute LAS * Cryotherapy and exercise, greater effect on reducing swelling compared to heat
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cryotherapy Treatment of Chronic Inflammatory Conditions
Cryotherapy may be beneficial in these conditions by relieving pain May also have a role in the management of acute inflammatory flare ups
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Local Immersion
* Involves placing part in mixture of cold water and flaked ice * Temperature can be controlled by varying the amount of ice that is added
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Cold Packs
* Self made using towel and ice flakes * Commercial cold packs * Commercial cold packs may be colder than ordinary ice packs * Potential to cause ice burns * Need to wrap in towel before application * Cold packs are applied for approximately 10-15 minutes (depth of target tissue)
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Ice Towels
* Towel placed in a mix of water and flaked ice and wrung out * Ice flakes will adhere to the cloth of the towel.
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Ice Massage
*Uses blocks of ice for direct massage *Ice is kept moving over the body part
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Cold Compression Units (e.g. Cryocuff)
* Cold water is applied to body parts via a sleeve which introduces compression at the same time as cooling. * Often used after orthopaedic surgery
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Excessive Local Cold on Normal Tissue consequences
* Can result in an ice burn * This is characterised by erythema and skin tenderness a few hours after treatment. * Cryotherapy can also cause injury to superficial peripheral nerves
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Contraindications to cryotherapy
* Circulatory insufficiency * Exacerbation of pre-existing conditions * Cold sensitivity ( e.g. Raynaud’s) * Cold urticaria * Cryoglobulinemia * Regions treated within 3-6 months by radiotherapy * Severe organ states Cold Hypersensitivity (Cold Urticaria): Can cause severe allergic reactions, including anaphylaxis Cryoglobulinemia: Cold exposure can lead to increased blood viscosity, causing blockages in small blood vessels Raynaud’s Disease/Phenomenon pregnancy
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Precautions to cryotherapy
* Unable to communicate * Sensory loss * Note: * Extreme care required when icing near superficial nerve, neuropraxia can be a consequence Local Infection: While cryotherapy is generally safe, care should be taken when applied near infected tissue Recently Radiated Tissue: Radiated tissue may have altered circulation and sensation, increasing the risk of adverse reactions
65
Monitoring During and After cryotherapy
* Cold feeling is noted very quickly * This is followed by burning/aching pain at around 2-7 minutes * Local anaesthesia/analgesia occurs at 5-12 minutes * Check after 5 minutes of application * Even redness during and after application * Shouldn’t be uneven/mottling of skin * Leave bell/buzzer with client
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cryotherapy Warning and Informed Consent
* This is an ice treatment. You will feel gradually increasing cold, which may be followed by a period of discomfort. The area should then go numb. If you feel any extreme discomfort or pain you must call me immediately as there is a danger of an ice burn. Please do not move or touch any of the equipment during treatment. * Do you understand? * Do you have any questions? * Are you happy for me to proceed with treatment?
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Muscle Injuries
 One of the most common injuries in sport  10% - 55% incidence  Types of acute injuries  Contusion  Strain/tear  Chronic/Overuse injuries  Focal thickening/fibrosis  Compartment syndrome  DOMS  Cramps?
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Muscle contusion
A muscle contusion usually occurs when a muscle is subject to a sudden, heavy compressive force, e.g. direct blow to the muscle Overlying skin remains intact
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Muscle contusion - haemotoma
 Muscle compression causes muscle fibre damage and rupture of microvessels  A large area of local hemorrhage (bleeding) is called a haematoma  Triggers an acute inflammatory response to remove the damaged tissue and initiating repair  Pain results from accumulating blood exerting pressure on nerve endings  Pain increases with movement or with pressure to area
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IntrAmuscular hematoma
extravasation of blood within intact muscle fascia results in increased intramuscular pressure, subsequently compresses & limits size of hematoma but can cause acute compartment syndrome.
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IntERmuscular hematoma
develops if fascia surrounding muscle is torn & extravasated blood has access to spread into interstitial & interfascial spaces
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Hematoma Complication: myositis ossificans, what is it
 Occurs when hematoma calcifies within the muscle approx 3 wks post injury  Osteoblasts replace some of the fibroblasts in healing process
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Hematoma Complication: myositis ossificans, what can cause it
rebleed, inappropriate treatment eg heat or massage In case of thigh: knee effusion present, prone knee flexion < 45°
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Hematoma Complication: myositis ossificans, what are the signs
 Don’t improve in expected time frame  Improvements cease with subsequent deterioration  Ongoing signs of inflammation  Increase in morning pain & pain with activity  Night pain  Palpable lump
75
what is a Muscle strain / tear
A muscle is strained or torn when some or all of the fibres fail to cope with the demands placed upon them e.g. excessive tensile force. it can be common in 2 jt muscles and during deceleration.
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mild muscle strain 1st degree
 Minor swelling  Minor discomfort  No or minimal loss of strength  No or minimal restriction of movement  Represents tear of few fibres
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moderate muscle strain 2nd degree
 Significant swelling  Significant discomfort,  Pain on contraction  Loss of strength  Restriction to movement  Represents tear of significant number of muscle fibres
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severe muscle strain 3rd degree
 Complete tear of muscle,  Virtually complete loss of muscle function
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Pathobiology of muscle injury
 Skeletal muscle healing is a repair process (in contrast to bone which regenerates)  Heals with a scar which replaces the original tissue  3 phases of repair  - Inflammation (Destruction phase) - Proliferation (Repair phase) - Maturational (Remodelling phase)
80
Healing of skeletal muscle from initial trauma
 Initial trauma  Ruptured myofibres contract  Hematoma fills gap between myofibre stumps
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Chronic/Overuse Muscle Injury examples
Focal tissue thickening/fibrosis  Compartment syndrome  Delayed onset muscle soreness (DOMS)
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Focal thickening/fibrosis
 Repetitive microtrauma caused by overuse damages muscle fibres  Leads to adhesions between muscle fibres & formation of cross-linkages in fascia  Palpated as firm, taut, thickened bands arranged in the direction of stress  May present as large areas of increased muscle tone & thickening
83
Compartment Syndrome
 Exercise raises intracompartmental pressure & vicious cycle occurs  Muscle hypertrophy may be precipitation factor  Signs - Pain commences during activity, ceases with rest (does not ‘warm up’)  Diagnosis may involve compartment pressure testing
84
Muscle soreness (DOMS)
 Develops 24-48 hours after unaccustomed physical activity  Often more severe after eccentric exercise e.g. downhill running  Etiology unclear, theories proposed include:  Lactic acid  Muscle spasm  Torn tissue  Connective tissue  Enzyme efflux  Tissue fluid
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Muscle cramps
A muscle cramp is a painful, involuntary muscle contraction that occurs suddenly & can be temporarily debilitating. Current theory: related to abnormal neuromuscular control at a spinal level in response to fatigue during or imediately after exercise
86
what is Tendinopathy 3 stages
 3 stages of tendinopathy  Reactive, disrepair, degenerative  Degenerative = Collagen degeneration (disarray & separation)  Increased mucoid ground substance  Increased cells and vascular spaces +/-neovascularization  Absent inflammatory cells
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tendinopathy signs
 Pain sometime after exercise (next day)  Painful at rest, ‘warms up’ with use, then worsens in cool down  Local tenderness & thickening  Swelling or crepitus may be present
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Tendinitis
inflammation of the tendon
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paratenonitis is
inflammation of outer layer of tendon, occurs when tendon rubs of bony prmoinces
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signs and symptoms of paratenonitis
 Acute oedema & hyperaemia  Infiltration of inflammatory cells  Rare occurrence  Signs - Crepitus (due to fibrinous exudate filling tendon sheath)  Difficult to differentiate from tendinosis
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Tendon tear signs
 Sudden onset of pain  Localised tenderness generally occurs at a site of least blood supply
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Tendon Healing- 3 phases of repair
 Inflammation (Destruction phase) - Necrosis predominates - Tenocytes migrate to area  Proliferation (Repair phase) - Synthesis of collagen  Maturational (Remodelling phase) - Repair tissue changes from cellular to fibrous - Alignment of collagen fibres in direction of stress
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Ligament injuries occurs when
abnormal or excessive movement of the joint.
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grade 1 of ligament sprain
stretched fibres but normal ROM on stressing ligament
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grade 2 of a ligament sprain
considerable proportion of fibres torn, stressing of ligament reveals increased laxity, but definite end point
96
grade 3 of a ligament sprain
complete tear of ligament with excessive joint laxity and no end point (be wary using pain as guide)
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Ligament healing
Initially hemostasis activated & fibrin clot formed then inflam response Formation of new tissue Remodelling
98
properties of scar
 Collagen disorganised  Smaller collagen fibres  Immature cross-links
99
Closed (simple) fractures
The skin remains intact over the fracture site
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Open (compound) fractures
The fractured bone disrupts skin integrity, creating a potential pathway for infection
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manifestation of fractures
pain, tenderness, swelling loss of function, nerve function loss, deformity of long bones, blood loss
102
fracture objectives for treatment
reduction- restoration of a fractured bone to its normal postion immobilisation- prevents movement preservation- rehab exercises to restore function
103
fibrocartligous callus formation in bone healing
capillaries continue to develop, tissue develops into granulation tissue, fibroblast produce a fibrocartilgous soft callus bridge (not strong enough to WB)
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Greenstick fractures
Occur when one side of the bone buckles while the opposite side fractures under tensile stress
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Torus (buckle) fractures
Result from axial compressive loading, causing circumferential cortical bulging
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Growth plate (physeal) fractures
Vulnerable sites in children and adolescents, commonly presenting as avulsion fractures when excessive tensile forces detach a portion of bone at the tendon or ligament insertion
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imparied healing bc of bone
malunion (inadequate alignment), delayed union (not in normal time frame), nonunion (failure for bone repair)
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Bone Healing Process
Haematoma Formation: Blood collects at the fracture site Fibrocartilaginous Callus Formation: Soft callus develops within a week Ossification: The soft callus is replaced by hard bone over several months Remodelling: The bone is reshaped to its original form, which may take years
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later complications of fractures
Avascular Necrosis: Loss of blood supply leading to bone death Chronic Pain: Persistent discomfort post-healing Malunion: Abnormal healing alignment leading to secondary complications (e.g., osteoarthritis) Soft tissue injuries – Ligament, muscle, or tendon damage may result in long-term functional impairment.
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Early Complications of fractures
Compartment Syndrome: Increased pressure within muscle compartments Infection: Especially in open fractures Acute compartment syndrome Neurovascular injury Deep vein thrombosis (DVT) / pulmonary embolism (PE)