WEEK 6 - MOTOR AND COGNITIVE CONTROL Flashcards
(20 cards)
key brain structures involved in movement control
prefrontal cortex, premotor cortex, supplementary motor cortex, primary motor cortex, central suculus, primary somatosensory cortex, posterior parietal cortex
motor cortex
influences OUTCOMES of movement and precision
primary motor cortex
initiates voluntary movement
mirror neurons
activated during prep and observation - develops during learning and experience
cerebellum
involved in balance and coordination and timing of movements
damaged cerebellum
affects rhythm, coordination and speech and impairs activities requiring aim/timing
- does NOT impair continuous motor activity
tests to assess cerebellum function
voluntary eye movements, finger-to-nose test
cellular organisation of cerebellum
Purkinje cells are flat and arranged in sequential planes, and parallel fibres excite these cells which affects cerebellum output
basal ganglia
involved regulating movement and reward responses, appropriate actions, spontaneous and self-initiated behaviours
pathways of basal ganglia in movement
direct = enhances selected movement; indirect = inhibits completing movements and is crucial to learning new actions
damaged basal ganglia
affects movement and response to larger rewards and impairs strength of spontaneous movement
conscious decisions precedes actions
brain activity responsible for movement begins BEFORE conscious awareness which means consciousness may NOT cause actions to be undertaken directly
decision and movement
people are generally accurate in reporting sensory stimuli/movement but LESS so with conscious decisions
Parkinson’s disease
results from loss of dopamine-releasing axons and associated with cognitive deficits and lack of motivation
symptoms of Parkinson’s
rigidity, tremors, slow movements, difficulty initiating voluntary activity
L-DOPA treatment in Parkinson’s
L-DOPA converts to dopamine in brain - increases dopamine release but does not slow neuronal loss
Huntington’s disease
neurological disorder with variable prevalence across regions and associated with damage in cerebral cortex + basal ganglia
symptoms of Huntington’s
arm jerks, facial twitches, tremors, writhing movements
psychological disorders in Huntington’s
Apathy, depression, sleeplessness, memory impairment, anxiety, hallucinations, poor judgement, alcoholism
testing for Huntington’s
autosomal dominant gene - chromosomal exams show likelihood and approximate onset of disease