Pulmonary Embolism/Obstructive Sleep Apnea - EXAM 3 Flashcards
(32 cards)
Where do PE originate?
- Deep veins of the legs (DVTs)
- Right side of the heart (with atrial fibrillation)
- Upper extremities (rare)
- Pelvic veins ( esp. after child birth or surgery because of increased pressure on vessels
Risk factors of PE
- Obesity
- Sedentary lifestyle
- BC pills
- Pregnancy
- Recent travel
- Recent leg, calf, knee pain
- Hx of heart dysrhythmias
- Family hx of clotting disorder
- Smoker
- Recent surgery
Virchow’s Triad
A patient at risk for the development of a PE often has a predisposition to:
- Venous Stasis
- Damage of the endothelium (inner lining of vein)
- Hypercoaguability
CPAP
Continuous Positive Airway Pressure
- A ventilator mode in which a constant positive pressure is delivered to the airway during inspiration and expiration
- Administered VIA a tight-fitting face or nasal mask or endotracheal/tracheal tube
BiPAP
- A ventilator mode that provides two levels of positive pressure support: higher during inspiration and lower during expiration
- Administered VIA a tight-fitting face or nasal mask
- Better option for patients with COPD
Indications for use of a non-invasive positive pressure ventilation system (NIPPV)
- OSA
- COPD (BiPAP)
- Acute/chronic respiratory failure (hypoventilation)
- Heart Failure
- After extubation to avoid reintubation
- Patient who refuses intubation but desires ventilatory support
- Chest wall or neuromuscular disease (restrictive causes)
Who is a NIPPV not appropriate for?
- Absent ventilation
- Excessive secretions
- Confusion/Decreased LOC
- Facial Trauma
- High O2 requirements
- Hemodynamic instability (shock)
Nursing Considerations of NIPPV
- Patient must be able to breathe spontaneously
- Patient must be able to cooperate with treatment
- Assist with proper fit, care, selection of mask/device
- When used in acute respiratory failure: patient should be NPO to protect from aspiration
Possible Complications of NIPPV
- Poor compliance r/t discomfort of tight fitting mask
- Difficulty communicating when using
- Skin breakdown over face/nose
- Risk of aspiration (when used for resp. distress)
- Nasal congestion/stuffiness
- Excessive dryness (consider humidification)
- Eye irritation
- Gastric distention
What assessments should be made when considering a PE?
- Respiratory Assessment
- O2 Sat
- Rate
- LS
- Assess for cough
- Assess WOB
- Hemoptysis
- Cardiac Assessment
- JVD d/t fluid backup
- cardiac rhythm
- muscle needs O2 to function
- PAIN assessment
- Mental status changes
- Fever
** Anxiety, sudden onset of dyspnea, or tachycardia are common signs of a PE, early recognition of these vague signs can prevent further respiratory complications**
What is a PE?
Blood clot has blocked off blood supply to massive number of alveoli. These alveoli contain O2 that cannot get into blood stream b/c blood flow distal to the embolus is blocked. Depending on amount of lung affected by a PE, patient may experience resp. failure, acute corpulmonale, or sudden circulatory and/or respiratory collapse
What are nursing interventions for PE?
- O2
- Raise HOB
- Bedrest
- Mental Status Assessment (freq. LOC monitoring)
- Freq. SpO2 and VS
- Freq. monitoring of cardiopulmonary status
What respiratory state is a patient in with PE?
Respiratory alkalosis. High pH and PaCO2 are due to hyperventilation, worsened if hyperventilation is prolonged. Low PaO2 is due to occluded pulmonary vessels. Low O2 sat indicates hypoxemia. Mild-moderate hypoxemia with a low PaCO2 is common in a patient with a PE.
V/Q Scan
Intravenous injection of a radioactive isotope in preparation for a scanning device to determine the adequacy of her pulmonary perfusion. VIA face mask patient inhales a radioactive gas as her lungs are again scanned, to determine the distribution of the inhaled gas.
V/Q Mismatch
The V/Q ratio represents the balance between alveolar ventilation and capillary blood flow and determine the adequacy of gas exchange in the lung (normal is 1:1). A mismatch b/w ventilation and oxygenation is the most common cause of hypoxemia. Results of a V/Q scan are expressed as high, med, low probability of a PE.
Spiral (or helical) CT
Noninvasive procedure where patient lies on table while special imaging machine rotates rapidly around the body, taking over 100 pictures in sequence. Provides 3 dimensional information regarding size/location of thrombus.
D-Dimer Test
Blood test. A substance (FDP) produced by the breakdown of thrombus (fibrin degradiaiton). Not specific for PE, recognizes any clot breakdown in the body. A positive D-dimer will increase suspicion of a PE and other tests will be ordered.
Pulmonary Angiography
Invasive procedure involving insertion of a catheter through the antecubital or femoral vein, advanced to the pulmonary artery and injection of contrast medium; allows for visualization/location of emboli.
Nursing Interventions for a PE
- TCDB
- Analgesia
- Prepare for intubation, mechanical ventilation
- Psychosocial support
Treatments of PE
- Anticoagulants/Thrombolytics
- Embolectomy/IVC filter placement
Anticoagulation
Prevention:
Heparin,m lovenox/fragmin, and/or coumadin
Use of PT/INR and APTT in titrating these medications
Teaching on prevention of PE
- How to prevent further dislodgement/jarring of the thrombi by mechanical forces such as:
- Sudden standing
- Sudden position changes
- Changes in rate of blood flow (valsalva manuever)
- Signs/Symptoms of bleeding
What is OSA?
Obstructive sleep apnea.
Partial or complete airway obstruction during sleep. Occurs when tongue/soft palate fall backward and partially/completely obstruct pharynx, causing a period of apnea that can last 15-90 seconds as many as 200-400 times during 6-8 hours of sleep. During apneic episodes, hypoexmia and hypercapnia can result, causing the patient to partially awaken, startle, snort, and gasp, casuing the tongue and soft palate to move forward and airway to open
OSA Risk Factors
- Smoking
- Obesity
- HTN
- COPD